ライフサイエンスコーパス: superoxide

1 ses the production of mitochondrial-specific superoxide.

2 onment is caused by AML-induced NOX2-derived superoxide.

3 ation of probes for intra- and extracellular superoxide.

4 iffuse to the nanoplatelet edges and produce superoxide.

5 diffusion-limited reduction and oxidation of superoxide.

6 preassociation of the proton donor with the superoxide adduct and a transition state that requires s

7 A heme superoxide adduct is thought to be the active oxidant in

8 demonstrates the potential of synthetic heme superoxide adducts to mimic the bioinorganic chemistry o

9 hat exclusively express L-OPA1 generate more superoxide and are more sensitive to Ca(2+)-induced mito

10 t phenols/quinols can react with both ferric superoxide and ferric peroxide intermediates formed duri

11 a persistent secondary wave of mitochondrial superoxide and hydrogen peroxide lasting for over 48 h a

12 These interventions restored mitochondrial superoxide and hydrogen peroxide production and inactiva

13 Reactive oxygen species (ROS), such as superoxide and hydrogen peroxide, are mediators of oxida

14 ify the misconceptions prompted by the name "superoxide" and the judgment bias based on the claimed t

15 long with increased free iron, mitochondrial superoxide, and lipid peroxidation, all of which are imp

16 Overproduction of superoxide anion (O(2) (.-) ), the primary cellular reac

17 CFR simultaneously detects superoxide anion (O(2)(*-)) and caspase-3 (casp3) throug

18 Although the superoxide anion (O(2)(-.)) is generated during normal c

19 It catalyses the transformation of the superoxide anion (O2*-) into hydrogen peroxide.

20 significant reduction in hydrogen peroxide, superoxide anion and malondealdehyde contents under stre

21 d that DB infestation significantly increase superoxide anion and malondialdehyde production to two-f

22 t aggregation, adhesion, thrombus formation, superoxide anion generation, and surface activation mark

23 s associated with increased NOX activity and superoxide anion generation.

24 nt of accurate methods for quantification of superoxide anion has attracted tremendous research atten

25 cells was mediated by induction of cellular superoxide anion in cancerous but not normal cells.

26 successfully applied to the determination of superoxide anion in the plant cell samples.

27 rin was proportional to the concentration of superoxide anion in the range from 4 to 40 000 pM with a

28 Superoxide anion production by the phagocyte NADPH oxida

29 pecies, hydrogen peroxide (H(2)O(2)) and the superoxide anion radical (O(2)(.-)), are key redox signa

30 In turn, the ability to generate superoxide anion radical and hydrogen peroxide by ozone-

31 tronic properties, photocatalytic ability in superoxide anion radical-mediated coupling of (arylmethy

32 est scavenging activity was observed for the superoxide anion radical.

33 Inhibition of nanosheet-induced superoxide anion restored the suppression of CSC and EMT

34 While administration of a superoxide anion scavenger during IH did not prevent neu

35 dical scavenging ability, reducing activity, superoxide anion scavenging ability, linoleic acid and p

36 of surface adhesion molecules, generation of superoxide anion, and appearance of reactive oxygen spec

37 ve oxygen species, including singlet oxygen, superoxide anion, hydrogen peroxide, and hydroxyl radica

38 ted lotus slices exhibited reduced browning, superoxide anion, hydrogen peroxide, electrolyte leakage

39 ion of coelenterazine under the oxidation of superoxide anion, the lower the amount aequorin regenera

40 oluminescent probe for in vitro detection of superoxide anion.

41 potential bioluminescent reporter protein of superoxide anion.

42 ata identify leukemia-generated NOX2-derived superoxide as a driver of protumoral p16INK4a-dependent

43 , the continuous generation of hydroperoxyl (superoxide) as a byproduct of aerobic respiration, and t

44 ired O(2) , which reacts with H(2) S to form superoxide, as detected by ESI-MS, a hydroethidine fluor

45 In situ quantification of superoxide associated with corals was enabled by DISCO,

46 similar to SOD5, Cu-only SOD4 can react with superoxide at rates approaching diffusion limits.

47 restrictions of employing the air cathode in superoxide-based batteries.

48 Superoxide-based K-O(2) batteries, invented in 2013, ado

49 lthough using an air cathode is the goal for superoxide-based potassium-oxygen (K-O(2) ) batteries, p

50 A superoxide-bridged dicopper(II) complex, [Cu(II)(2)(XYLO

51 modulation of NADPH oxidase activity, or the superoxide burst.

52 ween the radical cation of the probe and the superoxide, but it significantly increased the lifetime

53 iencies, and that reduction of mitochondrial superoxide by JP4-039 is a promising strategy for adjuva

54 ubiquitous one-electron reduction of O(2) to superoxide by microorganisms outside the cell, remains u

55 ermore, our results show that photogenerated superoxide can induce formation of AgNPs by reducing Ag(

56 akis(2,6-difluorophenyl)porphyrinate), and a superoxide complex, [(F(8))Fe(III)-(O(2)(*-))] (S), are

57 ia HAT reactivity of the partner ferric heme superoxide complex.

58 the lateral root primordia of tt7-2 reduces superoxide concentration and ROS-stimulated lateral root

59 background seawater-normalized extracellular superoxide concentrations near coral surfaces (0-173 nM)

60 ive metabolism enabled by higher periplasmic superoxide defense.

61 We report that indapamide reduced superoxide derived from microglia cultures and that trea

62 contrast, mutations in Copper Chaperone for Superoxide Dismutase (CCSD) resulted in enhanced suscept

63 arkers 8-hydroxy-2'-deoxyguanosine (8-OHdG), superoxide dismutase (Cu-Zn SOD), and thiobarbituric aci

64 ntified the extracellular antioxidant enzyme superoxide dismutase (EC-SOD) as a novel substrate of Ca

65 Manganese superoxide dismutase (MnSOD) functions as a tumor suppre

66 ction of a stable monomeric variant of Cu/Zn superoxide dismutase (mSOD1), an enzyme responsible for

67 phenols, histidine-containing peptides, and superoxide dismutase (SOD) activity have been detected i

68 while 400 ug/ml of extract showed revival in superoxide dismutase (SOD) activity.

69 exogenous delivery of antioxidant enzymes - superoxide dismutase (SOD) and catalase (CAT), encapsula

70 increases of antioxidants (i.e., copper/zinc superoxide dismutase (SOD) and extracellular SOD only in

71 ogenous antioxidants such as catalase (CAT), superoxide dismutase (SOD) and glutathione (GSH).

72 one (GSH), catalase (CAT), peroxidase (POD), superoxide dismutase (SOD) and glutathione reductase (GR

73 ascorbate peroxidase (APX), catalase (CAT), superoxide dismutase (SOD) and peroxidase (POD) in roots

74 The ubiquitous iron/manganese superoxide dismutase (SOD) family exemplifies this defic

75 splayed altered expression of CSDs and other superoxide dismutase (SOD) family members, leading to in

76 Copper/zinc superoxide dismutase (SOD) is a homodimeric metalloenzym

77 Superoxide dismutase (SOD) is a key enzyme that plays a

78 of malondialdehyde (MDA), glutathione (GSH), superoxide dismutase (SOD), catalase (CAT), matrix metal

79 icantly increased activities of catalase and superoxide dismutase (SOD), compared to the OS strains.

80 S. cerevisiae) Cu chaperone for Cu-zinc (Zn) superoxide dismutase (SOD1) activates by directly promot

81 S) homeostasis by repressing a Cu-containing superoxide dismutase (SOD1) and inducing Mn-containing S

82 Here, we examine the trajectory that Cu/Zn superoxide dismutase (SOD1) dimers take over the unfoldi

83 Mutations in Cu/Zn superoxide dismutase (Sod1) have been reported in both f

84 Here, we show that Cu, Zn superoxide dismutase (SOD1) is unique among proteins in

85 on of BMAA into the ALS-linked protein Cu,Zn superoxide dismutase (SOD1) upon translation promotes pr

86 mutations in the metallo-enzyme copper-zinc superoxide dismutase (SOD1).

87 Mitochondrial superoxide dismutase (SOD2) suppresses tumor initiation

88 tional coactivator PGC-1alpha, mitochondrial superoxide dismutase (SOD2), and chemical antioxidants a

89 ess due to hyperacetylation of mitochondrial superoxide dismutase (SOD2), increases HIF1alpha (hypoxi

90 n detached cells by regulating the manganese superoxide dismutase (SOD2).

91 The antioxidant enzyme extracellular superoxide dismutase (SOD3) protects against hypoxia-ind

92 L-6, IL-17, interferon-gamma (IFN-gamma) and superoxide dismutase 1 (SOD) (P < 0.05).

93 II) coordination sphere at the core of human superoxide dismutase 1 (SOD) with 0.7 pm precision.

94 ta, mitochondrial dysfunction, and disturbed superoxide dismutase 1 (SOD1) and Keap1/Nrf2 antioxidant

95 Mutations in the gene encoding superoxide dismutase 1 (SOD1) are the second most common

96 Non-natively folded variants of superoxide dismutase 1 (SOD1) are thought to contribute

97 Mutations in superoxide dismutase 1 (SOD1) cause 15-20% of familial a

98 Mutations in Cu-Zn superoxide dismutase 1 (SOD1) cause familial forms of am

99 and ALS patients harboring mutations in the superoxide dismutase 1 (SOD1) gene.

100 throughout the sequence of the gene encoding superoxide dismutase 1 (SOD1) have been linked to toxic

101 the activity and release of a model enzyme, superoxide dismutase 1 (SOD1) immobilized by polyion cou

102 cid to lysine (E40K) residue substitution in superoxide dismutase 1 (SOD1) is associated with canine

103 Superoxide dismutase 1 (SOD1) is the principal cytoplasm

104 onucleotide that mediates the degradation of superoxide dismutase 1 (SOD1) messenger RNA to reduce SO

105 yotrophic lateral sclerosis (ALS)-associated superoxide dismutase 1 (SOD1) mutant protein induces cha

106 Here, we examined potential inhibitors of superoxide dismutase 1 (SOD1) using ThT-fluorescence inc

107 sis (ALS) and mutations in the gene encoding superoxide dismutase 1 (SOD1) were treated with a single

108 trophic lateral sclerosis (ALS) mouse model, superoxide dismutase 1 (SOD1)(G93A), revealed that these

109 form transiently during aggregation of human superoxide dismutase 1 (SOD1), which is known to form mi

110 lateral sclerosis-associated protein variant superoxide dismutase 1 (SOD1)-A4V, whereas HSPA1L enhanc

111 al-sequence-lacking Acb1 and the antioxidant superoxide dismutase 1 (SOD1).

112 nts for TAR DNA-binding protein (TDP-43) and superoxide dismutase 1 (SOD1).

113 cardial expression of free radical scavenger superoxide dismutase 1 and aldehyde dehydrogenase 2 was

114 ed with mitochondrial dysfunction, disturbed superoxide dismutase 1 and Keap1/Nrf2 antioxidant respon

115 egulation in messenger RNA of shared targets superoxide dismutase 2 (P <= 0.001) and heme oxygenase 1

116 , we observed that inhibitory acetylation of superoxide dismutase 2 (SOD2) at K122 was increased in W

117 drial morphology, elevated protein levels of superoxide dismutase 2 (SOD2), and increased levels of p

118 d proteins and increased antioxidant enzymes superoxide dismutase 2 (SOD2), catalase, glutathione per

119 f specific mitochondrial proteins, including superoxide dismutase 2 (SOD2), depended on 4E-BP1/2.

120 iR-145-5p caused significant upregulation of superoxide dismutase 2 and heme oxygenase 1 protein foll

121 on of oxidative damage markers, and of SOD2 (superoxide dismutase 2), PGC1alpha [peroxisome prolifera

122 elongation in rice (Oryza sativa) (FSD2, Fe-superoxide dismutase 2).

123 In this study, we investigated the effect of superoxide dismutase 3 (SOD3) on LL-37- or KLK-5-induced

124 ABA caused further increases in catalase and superoxide dismutase activities, which led to a signific

125 oxidase, distinct from the established MnSOD superoxide dismutase activity.

126 a higher activity of the antioxidant enzyme superoxide dismutase and a different regulation of the g

127 ated genes, including one of three copies of superoxide dismutase and five novel members of its regul

128 In B. asiatica, high superoxide dismutase and significantly enhanced (p < 0.0

129 polyphenol oxidase and increased activity of superoxide dismutase enzyme.

130 target genes and demonstrated that multiple superoxide dismutase genes contribute to miR398b-regulat

131 ive (SynCav1(+)) mouse with the mutant human superoxide dismutase glycine to alanine point mutation a

132 mutase 1 (SOD1) is the principal cytoplasmic superoxide dismutase in humans and plays a major role in

133 se inactivation are similar, suggesting that superoxide dismutase is calibrated so the oxygen- and su

134 ve stress, because treatment with Tempol, an superoxide dismutase mimetic, rescued kidney injury in k

135 ial fusion and increasing cytoplasmic ROS in superoxide dismutase mutants.

136 d against the proteotoxicity of mutant Cu/Zn superoxide dismutase or C9orf72 dipeptide repeat protein

137 sporter 2, NADP-dependent glyceraldehyde and superoxide dismutase were found significantly upregulate

138 one contents, and activities of catalase and superoxide dismutase were significantly deteriorated in

139 The PEF extracts contained 'superoxide dismutase' (SOD), a known food allergen, osmo

140 BAL superoxide dismutase(SOD), plasma total-antioxidant capa

141 nd its target genes (including mitochondrial superoxide dismutase), (2) enhanced phagocytic activity

142 Scavengers of superoxide radical anion (superoxide dismutase), hydrogen peroxide (catalase), hyd

143 n of defense-related pine genes such as SOD (superoxide dismutase), LOX (lipoxygenase), PAL (phenylal

144 oxidant markers (Glutathione peroxidase, and superoxide dismutase).

145 by well-promoted antioxidant enzymes (i.e., superoxide dismutase, and catalase), strong DPPH-scaveng

146 ies were characterized by higher activity of superoxide dismutase, ascorbate peroxidase and phenylala

147 larly, 10 mmol L(-1) treatment showed higher superoxide dismutase, catalase and ascorbate peroxidase

148 , which depends on the activity of cytosolic superoxide dismutase, SOD-1.

149 responses in catalase, guaiacol peroxidase, superoxide dismutase, soluble protein, lignin, chlorophy

150 Among these was the SodA superoxide dismutase, which is essential for mammalian i

151 O USNPs simultaneously possessing catalase-, superoxide dismutase-, and glutathione peroxidase-mimick

152 (ALS), in which astrocytes expressing mutant superoxide dismutase-1 (mutSOD1) kill wild-type motor ne

153 Superoxide dismutase-1 (SOD1) maturation comprises a str

154 cells in mice expressing the ALS-associated superoxide dismutase-1 (SOD1)(G93A) mutant decreased spi

155 hase II enzymes (heme oxygenase-1, catalase, superoxide dismutase-1) in a time-dependent manner.

156 le such as strongly donating thiolates in Ni superoxide dismutase.

157 orms of either Tar DNA-binding protein 43 or superoxide dismutase.

158 erwise functionally redundant SoxR-regulated superoxide dismutase.

159 The previous studies on superoxide dismutases (Cu, Zn-SODs) showed that the dime

160 ease (CD), contains exclusively Fe-dependent superoxide dismutases (Fe-SODs).

161 Copper (Cu)-only superoxide dismutases (SOD) represent a newly characteri

162 Reactive oxygen species (ROS) like superoxide drive rapid transformations of carbon and met

163 irst K-air (dry) battery based on reversible superoxide electrochemistry is presented.

164 conditions, involving the formation of iron superoxide (FeO(2)Hx with x = 0 to 1), but the puzzling

165 te that this total marine dark extracellular superoxide flux is consistent with concentrations of sup

166 ies could activate aerial oxygen to generate superoxide for completing the vital proton abstraction s

167 vivo administration of pirfenidone decreased superoxide formation, increased healthy mitochondria num

168 ogical or genetic KLF5 inhibition alleviated superoxide formation, prevented ceramide accumulation, a

169 activities due to a high rate of endogenous superoxide formation.

170 It can produce significant amounts of superoxide ([Formula: see text]) and hydrogen peroxide (

171 an enzyme responsible for the conversion of superoxide free radicals into hydrogen peroxide and oxyg

172 neutrophil phagosomes, myeloperoxidase uses superoxide generated by the NADPH oxidase to oxidize chl

173 how that the nucleophilic characteristics of superoxides, generated galvanostatically in an Aluminum/

174 tochondrial membrane potential and increased superoxide generation indicating altered physiology of t

175 diabetes-induced retinal capillary leakage, superoxide generation, leukocyte adherence, and leukotri

176 eightened energetic demand and mitochondrial superoxide generation.

177 Reactive oxygen species - superoxide, hydrogen peroxide and hydroxyl radicals - ha

178 centers, thus propagating the generation of superoxide, hydrogen peroxide, and hydroxyl radicals.

179 and glutelin showed scavenging capacity for superoxide, hydrogen peroxide, nitric oxide and DPPH (1,

180 h ROS modulation, reducing the generation of superoxide/hydrogen peroxide.

181 de flux is consistent with concentrations of superoxide in marine environments.

182 We also examined the importance of superoxide in the mediating pathways and of hematopoieti

183 ps, it becomes clear that the instability of superoxide in the presence of Li ions (Li(+)) and Na ion

184 nd significant upregulation of mitochondrial superoxide, increasing intracellular ROS.

185 Mitochondrial-specific inhibition of superoxide inhibited oxidized LDL-induced NF-kappaB (nuc

186 e process uncover the formation of a Co(III)-superoxide intermediate and its preceding high-valent Co

187 2) electrochemical cell used to generate the superoxide intermediate is also reported to deliver larg

188 the OER as the release of dioxygen from the superoxide intermediate.

189 equilibria among ferric, ferrous, and ferric-superoxide intermediates have been quantified under cata

190 solvents in Li-O(2) batteries that dissolve superoxide intermediates in lithium peroxide (Li(2) O(2)

191 lt in an instability towards highly reactive superoxide intermediates.

192 ntial, reduced ATP production, and increased superoxide ion levels); further, we hypothesized that an

193 oxygen species including singlet oxygen and superoxide ion through both type 1 and type 2 pathways,

194 As a consequence, superoxide is produced at complex III.

195 Peroxide, rather than superoxide, is shown to be the product of O(2) reduction

196 one-electron redox process of O(2)/potassium superoxide (KO(2)).

197 ficant increased aggregate formation, raised superoxide levels (ROS), and altered mitochondrial morph

198 is associated with reduced renal and cardiac superoxide levels and that MTP-131 protects against DKD

199 that mutant myoglobin may result in elevated superoxide levels at the cellular level.

200 High superoxide levels were found in all cells and were decre

201 tional mitochondria, increased mitochondrial superoxide levels, and impaired mitochondrial respiratio

202 ld db/db mice have reduced renal and cardiac superoxide levels, as measured by dihydroethidium oxidat

203 ects against DKD and preserves physiological superoxide levels, possibly by regulating cardiolipin re

204 a chromium(III) complex with an end-on bound superoxide ligand, while the reaction in tetrahydrofuran

205 We confirm that a superoxide-mediated chain reaction, initiated by electro

206 The pH dependence of superoxide-mediated Fe(III) reduction (SMIR) is also con

207 Overall, our results show that superoxide-mediated iron reduction will be important onl

208 The results reveal that superoxide mediates neighbor-neighbor activation of ener

209 Rotenone promoted mitochondrial-generated superoxide (MitoROS), which was exacerbated by ATP13A2 d

210 e cruciality of electrophilicity of the heme superoxide moiety in facilitating the initial indole act

211 cture of 1 may minimize stabilization of the superoxide moiety, resulting in its enhanced reactivity.

212 olized RuBP (near 0.49 V) is compatible with superoxide (O(2) (*-)) production, must be insensitive t

213 In class Ib Mn(2) RNRs superoxide (O(2) (.) ) was postulated to react with the

214 )) on Fe(III) reducibility by photogenerated superoxide (O(2)(*)(-)) and LMCT.

215 OA), the kinetics and reaction mechanisms of superoxide (O(2)(*-)) formation are rarely quantified an

216 al charge transfer (LMCT) and photogenerated superoxide (O(2)(*-)) play an important role in AFO phot

217 etal-dioxygen adduct, whether it exists as a superoxide or a peroxide, which thus merits consideratio

218 that Anf3 consumes oxygen without generating superoxide or hydrogen peroxide.

219 Acute scavenging of superoxide or inhibition of NADPH oxidase improved NO-de

220 f PFOR damage was unaffected by the level of superoxide or peroxide, showing that molecular oxygen it

221 ich binds O(2) reversibly to form the ferric-superoxide porphyrin complex, Fe(III)(TPP)(O(2)(*-)).

222 n impaired mitochondrial function, increased superoxide presence, and detectable protein carbonylatio

223 ccurred via reaction with the oxygen radical superoxide produced by soluble extracellular proteins.

224 tide, SS-31, or Bendavia) in restoring renal superoxide production and ameliorating DKD.

225 c reticulum (ER)-mitochondria communication, superoxide production and apoptosis were evaluated in fi

226 -silenced RAW macrophages depicted increased superoxide production and decreased parasite survival.

227 hosphate (GTP)-bound RAC2 including enhanced superoxide production and increased membrane ruffling.

228 ellular ATP underpins increased myeloid cell superoxide production and phagocytosis associated with i

229 or of site IQ electron leak, an inhibitor of superoxide production by complex I of the mitochondrial

230 Here we show that dark extracellular superoxide production by marine microbes represents a pr

231 the mechanism and function of extracellular superoxide production by the marine diatom Thalassiosira

232 GR was also linked to extracellular superoxide production by whole cells via quenching by th

233 phosphateoxidase) activity and mitochondrial superoxide production coupled with a compromised antioxi

234 Extracellular superoxide production followed a typical photosynthesis-

235 he ecophysiological role(s) of extracellular superoxide production has remained elusive.

236 Here we examined renal superoxide production in a type 2 diabetes animal model,

237 ar ROS and induced significant mitochondrial superoxide production in bronchial epithelial cells (16-

238 ip2(cmo) neutrophils display highly elevated superoxide production in response to a range of stimuli.

239 capacity, Ca(2+) homeostasis, and attenuated superoxide production in response to ischemia and excito

240 lic switch from oxidative phosphorylation to superoxide production in response to its ligand, oxidize

241 Extracellular superoxide production in T. oceanica exudates was couple

242 /db mice and fully preserved levels of renal superoxide production in these mice.

243 er, these results suggest that extracellular superoxide production is a byproduct of a transplasma me

244 We recently reported that reduced superoxide production is associated with mitochondrial d

245 We estimate that extracellular superoxide production represents a gross oxygen sink com

246 increased neutrophil F-actin, and excessive superoxide production seen in patients.

247 Further, elevated mitochondrial superoxide production was noted in tumor cells vs. non-t

248 d in endothelial cells followed by increased superoxide production within 4 hours of blast.

249 stress markers (eg, nitrotyrosine abundance, superoxide production) were also quantified.

250 purposing of the electron transport chain to superoxide production, and NF-kappaB activation.

251 from RAC2[E62K] patients exhibited excessive superoxide production, impaired fMLF-directed chemotaxis

252 eta-glucans (Bbetaglucans) reduced leukocyte superoxide production, inflammatory ADAM17, TNFalpha, nS

253 duration and included vascular permeability, superoxide production, leukotriene generation, leukocyte

254 component p67 (phox) , activates neutrophil superoxide production, whereas interactions with p21-act

255 hydroxylamine while the peroxide, unlike the superoxide, proved capable of deformylating aldehydes.

256 During T. cruzi invasion to macrophages, superoxide radical (O(2) (*-)) is produced at the phagos

257 he detection of the intra- and extracellular superoxide radical anion ([Formula: see text]).

258 Scavengers of superoxide radical anion (superoxide dismutase), hydroge

259 ion of membrane lipids against oxidation and superoxide radical anion scavenging activity.

260 turally dissolved oxygen (approx. 2 mM), the superoxide radical is co-electrogenerated during analyte

261 vamide and these extracts by measuring their superoxide radical scavenging capabilities in a Rotating

262 trochemical system against in-situ generated superoxide radical.

263 vity resulted in significant accumulation of superoxide radicals (WT, 4.54 mumol/mg tissue/min; CatA-

264 SOR protein is a key enzymatic scavenger of superoxide radicals and protects the bacterium from oxid

265 temporal release and dose of the therapeutic superoxide radicals from QDs.

266 g enzymes that can catalyze the reduction of superoxide radicals to hydrogen peroxide and are importa

267 The most potent peptide towards superoxide radicals was the four-amino-acid chain with a

268 d from the reaction between nitric oxide and superoxide radicals, in impairing endothelial AKAP150-TR

269 generated higher levels of nitric oxide and superoxide radicals, resulting in increased local peroxy

270 omotetramer of the 1Fe-SOR class, can reduce superoxide radicals.

271 y correlated with the level of intracellular superoxide radicals.

272 apacity of infusions and beverages, based on superoxide radicals.

273 The superoxide reacts faster with (2,2,6,6-tetramethylpiperi

274 F0389_00796 gene that encodes for a putative superoxide reductase (SOR) enzyme.

275 flammatory functions including phagocytosis, superoxide release and apoptosis.

276 TRPV4 activation also triggered superoxide release.

277 ssembling peptide (Bz-CFFE-NH(2) ) to make a superoxide-responsive, persulfide-donating peptide (SOPD

278 on with an NO synthase inhibitor (L-NAME), a superoxide scavenger (Tempol), and an NADPH oxidase inhi

279 rs was blocked by mitoTEMPO, a mitochondrial superoxide scavenger that reduced oxidative stress and D

280 levels in the presence of Nox2 inhibitor or superoxide scavenger.

281 Superoxide scavenging reduced experimental metastasis.

282 e probes and found reduced fluorescence of a superoxide-selective probe within the primordia of tt7-2

283 e dismutase is calibrated so the oxygen- and superoxide-sensitive enzymes are equally sensitive to ae

284 ed with phenoxazine (or its nitroxide) and a superoxide source were better protected from ferroptosis

285 a the intermediate formation of an iron(III)-superoxide species 3, which could be trapped and spectro

286 e exchange experiments, we discover a cobalt superoxide species as an active intermediate in the OER.

287 (X1)(X2) TMPA)Cu(II) (O(2) (.) )](+) cupric superoxide species was achieved, and they were character

288 electron transfer from the framework to form superoxide species, which are subsequently stabilized by

289 lters the ability of the bacterium to resist superoxide stress when metal starved by the host, reveal

290 echanistically, SIRT3 prevents mitochondrial superoxide surges in detached cells by regulating the ma

291 o-isocaproate or fatty acid oxidation formed superoxides through electron-transfer flavoprotein:Q-oxi

292 enhancement in the reactivity of the cupric superoxide towards phenolic substrates as well as oxidat

293 e ferric iron, which forms the stable ferric-superoxide-TyrB10/GlnE7 complex.

294 of injury from a localized concentration of superoxide was simulated as the spread via passive diffu

295 oxidative stress caused by overproduction of superoxide, we developed a compound that reacts with O(2

296 single electron transfer to form short-lived superoxide, which then recombines to form a peroxide int

297 substantial amounts of molecular oxygen into superoxide, which, after dismutation into peroxide, serv

298 owing SOD1's "electrostatic loop" to attract superoxide with equal affinity (at both redox states of

299 Stabilizing superoxide with large K ions (K(+)) provides a simple bu

300 Free superoxide yields prominent signals that the explosives