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2 dea of using the Schottky contact to achieve supersensitive and fast response nanowire-based nanosens
3 ng the NR1 subunit of the NMDA receptor show supersensitive behavioral responses to kainic acid and t
4 triatal monoamine levels and no evidence for supersensitive D1, D3, or D4 dopamine receptors in the D
5 both academia and industry, barriers such as supersensitive detection techniques and highly-efficient
6 vation of the nigrostriatal pathway and that supersensitive dopamine D(1) receptors may mediate the e
8 uble-stranded DNA, here we develop a simple, supersensitive, fast and high-throughput platform for th
10 signal transduction mechanisms to produce a supersensitive form of D1- mediated neuronal plasticity.
11 cetylcholine receptor subunits or expressing supersensitive forms of these subunits have begun to tie
14 amine (DA) D1 receptors is the hallmark of a supersensitive molecular response associated with dyskin
16 ine system, direct pathway neurons display a supersensitive response to D1 dopamine receptor agonists
17 nisms underlying the irreversibility of this supersensitive response with long-term dopamine agonist
20 iral therapy and followed intensively with a supersensitive reverse transcriptase PCR assay with a lo
22 tal cortex of PKCepsilon null mice were also supersensitive to allosteric activation by ethanol and f
24 as serine (S)563, phosphorylation at S603 is supersensitive to calcineurin-mediated dephosphorylation
25 negative alleles of cdr1, which render cells supersensitive to Chk1 levels, and suppress the checkpoi
27 ies have suggested that bipolar patients are supersensitive to light suppression of melatonin and tha
30 ggest that the juvenile prefrontal cortex is supersensitive to methylphenidate, and the accepted ther
34 ing protein kinase Cepsilon (PKCepsilon) are supersensitive to positive allosteric modulators of gamm
37 data suggest that neurons in the CPu become supersensitive to the effects of DA agonists after 6-OHD
38 chronic morphine-treated cells which become supersensitive to the excitatory effects of mu-, delta-
39 Here we show that GRK6-deficient mice are supersensitive to the locomotor-stimulating effect of ps
40 n ribonucleotide reductase; rnr4 mutants are supersensitive to the ribonucleotide reductase inhibitor
41 wth of the strain with the pst1-1 allele was supersensitive to the specific histone deacetylase inhib