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1 f individual fascicles (tibial, peroneal and sural).
2  of myelinated and unmyelinated axons in the sural and medial plantar nerves that immunostain for sub
3 d a higher neuropathy symptom profile, lower sural and peroneal nerve amplitudes, abnormal thermal th
4 hat appeared similar to myelinated fibres in sural and sciatic nerve.
5 gical assessment (Total Neuropathy Score and sural and tibial compound nerve amplitudes), and sensory
6    Moreover, combined interventions improved sural and tibial nerve myelin thickness, hind paw epider
7 served in heterozygous Pmp22 mice as well as sural biopsies from patients with HNPP.
8  by random regeneration in direct suture and sural cable graft groups but not in nerve guide repairs
9              Punches removed from tibial- or sural-innervated planter paw skin were immunolabeled to
10                               Contralesional sural-innervated skin had neither neurite loss nor sprou
11                         Adjacent ipsilateral sural-innervated skin had persistent hyperalgesia withou
12  staining of lateral plantar nerve (LPN) and sural nerve (SN) motor terminals, using the activity-dep
13 nd the F-wave latency (p=0.03) decreased and sural nerve action potential amplitude increased (p=0.04
14  Changes in excitability of the terminals of sural nerve afferents were used to confirm that both loc
15 ith BIPN more frequently demonstrated absent sural nerve amplitudes and diminished distal sensation c
16 ation of myelinated axons in both the distal sural nerve and nerves of the toe.
17 ical stimulation of afferent C fibers in the sural nerve and recorded from single neurons in the vent
18                           Examination of the sural nerve and the auditory nerve adjacent to the brain
19 ibres in the skin or myelinated axons in the sural nerve and toe after vincristine.
20  was evoked by electrical stimulation of the sural nerve and was recorded in the ipsilateral hamstrin
21 vestigated the surgical anatomy of the ovine sural nerve as a potential candidate for facial nerve re
22 long-term outcomes, with the sural or dorsal sural nerve as the most informative.
23               Stimulation of the ipsilateral sural nerve at the malleolus, just before stimulation of
24                             GABA depolarized sural nerve axons and increased the electrical excitabil
25 ibres was a uniform feature in a total of 21 sural nerve biopsies and 'onion bulb' formations and/or
26 ith diabetic neuropathy progression in human sural nerve biopsies and describe their potential utilit
27                                              Sural nerve biopsies from 7 patients with diabetic neuro
28                                              Sural nerve biopsies from six affected individuals and t
29 -4 (CD152) at the protein and mRNA levels in sural nerve biopsies of patients with chronic inflammato
30 eased PMP22 messenger RNA levels in skin and sural nerve biopsies of patients with CMT1A compared wit
31 and nuclear imaging, electroencephalography, sural nerve biopsies, sleep evaluation and neuropsychome
32 y, we analysed PMP22 messenger RNA levels in sural nerve biopsies.
33 nal neuropathy' was proven in 14 (45%) of 31 sural nerve biopsies.
34 3-3 proteins measurement, skin, muscular and sural nerve biopsies.
35                                            A sural nerve biopsy from an affected patient showed marke
36 lectrophysiologic data were evaluated, and a sural nerve biopsy from one affected child was examined
37 tain circumstances replace the more invasive sural nerve biopsy in the morphological and molecular ev
38              Electrodiagnostic studies and a sural nerve biopsy showed features of a dystrophic axona
39                                              Sural nerve biopsy showed mild to moderate selective los
40                       Of the 6 who underwent sural nerve biopsy, 4 had selective loss of small myelin
41 decrease in the size of myelinated fibres on sural nerve biopsy.
42  infected and 2 uninfected animals underwent sural nerve biopsy.
43  area of the antidromic volley evoked in the sural nerve by intraspinal microstimulation in the L4/5
44 unmyelinated fiber function in the hind paw, sural nerve C-fiber morphometry, sciatic nerve neurotrop
45                 The results suggest that the sural nerve can be successfully used for facial nerve re
46        Negative correlations were found with sural nerve conduction velocities (NCVs) (r = -0.65, P <
47                                    The ovine sural nerve descended to the lower leg along the short s
48     Nociceptive nerve function, unmyelinated sural nerve fiber and dorsal root ganglion (DRG) cell mo
49          Nerve gaps were bridged by either a sural nerve graft or a biodegradable collagen nerve guid
50                    ICN was performed using a sural nerve graft.
51                  Enthusiasm has declined for sural nerve grafting because of the associated complexit
52 can be achieved with minimal morbidity using sural nerve grafts, which surgeons commonly use to recon
53 n of a single unit occurred naturally in the sural nerve in some cases.
54 n axonal diameter of myelinated axons in the sural nerve in untreated diabetic rats.
55                                  Because the sural nerve innervates hairy skin, these data suggest th
56             Schwann cells derived from human sural nerve may provide a valuable source of tissue for
57          A loss of > or =500 fibers/mm(2) in sural nerve MFD over 52 weeks was defined as progressing
58                          RESEARCH DESIGN AND Sural nerve myelinated fiber density (MFD), nerve conduc
59                         Neither the proximal sural nerve nor the motor tibial nerve exhibit axon loss
60 M phosphorylation by 2.5-fold (P < 0.001) in sural nerve of BB rats.
61  increases in total levels of p38 and JNK in sural nerve of type I and II diabetic patients.
62                                          The sural nerve of untreated diabetic rats showed a 50% decr
63                   The clinical phenotype and sural nerve pathology in these two families differs in s
64                                     Detailed sural nerve pathology is presented in both cases.
65   Stimulation of the gastrocnemius nerve and sural nerve revealed significant convergence of muscle a
66                               In this study, sural nerve segments from individuals with PMP22 duplica
67 n delivered to tibialis anterior (TA) MNs by sural nerve stimulation.
68 ts were seen with field potentials evoked by sural nerve stimulation.
69                                              Sural nerve transection blocked the postinjury increase
70                            The length of the sural nerve was 14.3 +/- 0.5 cm.
71 sterior edge of the lateral malleolus to the sural nerve was 7.8 +/- 1.8 mm.
72               The number of fascicles in the sural nerve was also significantly lower than in the BB
73                                          The sural nerve was grafted to the BB with end-to-end neuror
74 p) elicited by electrical stimulation of the sural nerve was measured in six normal adults.
75  The mean number of myelinated fibers in the sural nerve was significantly lower than that of the BB
76 were exposed and in situ recordings from the sural nerve were performed to determine compound C-fiber
77                                  Sections of sural nerve were removed from amputated legs of patients
78 omy and the number of fascicles of the ovine sural nerve were similar of those reported in humans.
79                                       In the sural nerve, 20% of the unmyelinated axon profiles immun
80 lation of a 54-kDa isoform of JNK in DRG and sural nerve, and this correlated with elevated c-Jun and
81 ipsilateral foot, and also the contralateral sural nerve, decreased presynaptic inhibition.
82                                       In the sural nerve, where the large majority of myelinated fibe
83 n nearby hairy skin innervated by the spared sural nerve.
84 he peroneal nerve, the tibial nerve, and the sural nerve.
85 egment of the medial cutaneous branch of the sural nerve.
86 0.01) and mean axonal size (P < 0.05) in the sural nerve.
87 ers and profuse regenerative activity in the sural nerve.
88 on was also increased in the DRG, but not in sural nerve.
89 10 sensory potentials were newly observed in sural nerves after treatment.
90 pathies and compared these with normal human sural nerves and those from patients with Guillain-Barre
91 iments were performed on 50 samples of human sural nerves collected during a 52-week clinical trial.
92 tation in mtDNA was more prevalent in distal sural nerves compared to dorsal root ganglia.
93 urofilament distances (NNND) in the axons of sural nerves from patients with anti-MAG paraproteinaemi
94 e sensory latency of both the left and right sural nerves improved on the basis of faster median cond
95 f HIV-SN, freshly isolated mitochondria from sural nerves of macaques infected with a neurovirulent s
96 mtDNA common deletion mutation in postmortem sural nerves of patients with HIV-SN as compared to unin
97 s from four cryopreserved normal adult human sural nerves referenced to the Genome Reference Consorti
98 icantly lower C-fiber conduction velocity in sural nerves than uninfected animals and the magnitude o
99            JAM-C was also expressed in human sural nerves with an expression profile similar to that
100                              In normal human sural nerves, axonal NNND was correlated with axonal dia
101       In the largely cutaneous saphenous and sural nerves, the naked mole-rat had the lowest C:A-fibe
102  (ulnar, deep peroneal) and sensory (median, sural) nerves.
103  and to monitor long-term outcomes, with the sural or dorsal sural nerve as the most informative.
104 Unit isolation was by partial section of the sural or lateral plantar nerves.
105  large myelinated nerve fibers, specifically sural or sciatic nerve conduction velocities, but signif
106 .0001), CNFL (p < 0.0001), IENFD (p = 0.04), sural (p = 0.02) and peroneal motor nerve conduction vel
107 nerve conduction studies (NCS), specifically sural sensory nerve action potential (SNAP) amplitude, w
108 hy, metabolic syndrome associated with lower sural sensory nerve action potential amplitudes.
109 oral dispersion and conduction block, (5) no sural sparing, (6) greater number of fibrillation potent
110  dispersion and conduction block, and absent sural sparing.
111 ced in male mice, but not female mice, under sural-sparing SNI conditions.

 
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