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1 le to the negative sequela of anesthetic and surgical stress.
2 tors within the cecum of mice in response to surgical stress.
3 ogens in the intestinal tract in response to surgical stress.
4 N, SON and MnPO of rats with HF during acute surgical stress.
5 sis due to intestinal P. aeruginosa in which surgical stress (30% hepatectomy) was combined with dire
6 ated behavioural analysis could be detecting surgical stress and/or post-surgical pain.
7 ivated during HF and are sensitive to 'acute surgical stress' and may contribute to the elevated leve
8 ing dysbiosis, perioperative antibiotic use, surgical stress, and immunosuppressive use have each bee
9 documented in Crohn disease, celiac disease, surgical stress, and intestinal obstruction and are asso
10 may provide insights about pathways by which surgical stress contributes to postoperative outcomes.
11 of the Estimation of Physiologic Ability and Surgical Stress (E-PASS) risk model in a Western PHC coh
12 se data provide direct in vivo evidence that surgical stress impairs global NK cell function.
13 patic injury and metabolic dysfunction after surgical stress in aged mice.
14   Dietary restriction promotes resistance to surgical stress in multiple organisms.
15 eurons in the PVN, SON and MnPO due to acute surgical stress in rats with HF, and (2) vagal afferents
16                                 Furthermore, surgical stress in the form of hepatectomy significantly
17 uced withdrawal reflex) and antinociception (surgical stress index, skin conductance algesimeter) wer
18                                              Surgical stress induces age-dependent subclinical hepati
19            Furthermore, we demonstrated that surgical stress or cecal ligation and puncture caused a
20 ke, resulting in protection in two models of surgical stress: renal and hepatic ischemia-reperfusion
21 rioperative pulmonary function, blunting the surgical stress response and improved analgesia.
22                     Protein can modulate the surgical stress response and postoperative catabolism.
23 gainst recurrence during cancer surgery: the surgical stress response, use of volatile anaesthetic, a
24 r improve outcome presumably by blocking the surgical stress response.
25                 Multisystem injury and major surgical stress result in a hypermetabolic state with ac
26                          Clinical markers of surgical stress that were collected included duration of
27 ely attenuated the catecholamine response to surgical stress throughout the postoperative study perio
28 gy of the anomaly and the general effects of surgical stress, tocolytic agents, and anesthesia.
29                       Using a mouse model of surgical stress, we transferred surgically stressed NK c