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1         The primary endpoint was the rate of surgical wound infection.
2  using a clinically relevant murine model of surgical wound infection.
3 ptible S. aureus which frequently cause deep surgical wound infections.
4   Empyema was most common (42%), followed by surgical wound infections (29%), mediastinitis (16%), st
5 may be a significant component in some mixed surgical wound infections and that surgical management a
6 hypothermia both increases susceptibility to surgical-wound infection and lengthens hospitalization.
7  extraintestinal infections (pneumonia, deep surgical wound infection, and vertebral osteomyelitis wi
8 ide updated guidelines for the prevention of surgical wound infections based upon review and interpre
9 fection in patients at risk of bacteremia or surgical wound infection but failed to reach their clini
10  is common during major surgery, may promote surgical-wound infection by triggering thermoregulatory
11 onchial wash, blood, sinus drainage, and two surgical wound infections from separate patients in Texa
12             The development and treatment of surgical wound infections has always been a limiting fac
13                  We developed a minipig deep surgical wound infection model offering 3 independent ef
14 o elicit strong inflammatory responses; in a surgical wound infection model with Pseudomonas aerugino
15 reus strain in both the s.c. abscess and the surgical wound infection models in WT mice.
16 r years, bloodstream, respiratory tract, and surgical wound infections predominated.
17                                              Surgical wound infections remain a significant source of
18 c. abscess formation, hindpaw infection, and surgical wound infection, S. aureus multiplied in the ti
19 nistration of such drug in a murine model of surgical wound infection significantly reduced the bacte
20        CD4(+) alphabeta T cells homed to the surgical wound infection site of WT animals.
21 of mixed wound infection, from a pool of 400 surgical wound infections that we have studied, in which
22                             During S. aureus surgical wound infection, the presence of IFN-gamma at t
23 omodulator in the treatment of MSSA and MRSA surgical wound infection through enhancement of the loca
24 rees C in the normothermia group (P < 0.001) Surgical-wound infections were found in 18 of 96 patient