ライフサイエンスコーパス: susceptible cell

1 rus to receptors present on the surface of a susceptible cell.

2 istant subpopulation in a main population of susceptible cells.

3 receptors is essential for HSV infection of susceptible cells.

4 seminate viral particles to target organs or susceptible cells.

5 rains of primate immunodeficiency viruses to susceptible cells.

6 ate RNA degradation in neurons than in other susceptible cells.

7 ogenic potential of the Env protein in other susceptible cells.

8 between highly susceptible cells and poorly susceptible cells.

9 TLV-1) on both highly susceptible and poorly susceptible cells.

10 s that were infectious when transfected into susceptible cells.

11 ng viral destabilization after attachment to susceptible cells.

12 onstrate that the drug inhibits infection of susceptible cells.

13 the ligands TNF-alpha and TRAIL to kill the susceptible cells.

14 tic cytopathic effect by forming syncytia in susceptible cells.

15 minate the inserted sequences upon spread in susceptible cells.

16 n the formation of cation-selective pores in susceptible cells.

17 coreceptors for entry of HIV-1 strains into susceptible cells.

18 ae that inhibits protein synthesis and kills susceptible cells.

19 through stimulation of apoptotic programs in susceptible cells.

20 le Tva proteins to inhibit ALV(A) entry into susceptible cells.

21 ixed populations of antibiotic-resistant and susceptible cells.

22 te for P. falciparum) within a population of susceptible cells.

23 inding to RI and RII, and of PKA activity in susceptible cells.

24 gion of p6(Gag), Y36S-L41P, could not infect susceptible cells.

25 -Fas antibodies rapidly induces apoptosis in susceptible cells.

26 ral genome from the endosomal compartment of susceptible cells.

27 ation may be restricted by the quiescence of susceptible cells.

28 infection by preventing virus attachment to susceptible cells.

29 bserved between phenotypically resistant and susceptible cells.

30 variants pseudoviruses increased entry into susceptible cells.

31 sing both phenotypically phage-resistant and susceptible cells.

32 e-4 (DPP4) and is necessary for infection of susceptible cells.

33 eria within a clonal population of otherwise susceptible cells.

34 a relatively low rate of virion infection of susceptible cells.

35 iron from cefiderocol, preventing uptake by susceptible cells.

36 the same outer-membrane transporter to enter susceptible cells.

37 cysteines are necessary for McpM to inhibit susceptible cells.

38 anisms associated with the entry of EAV into susceptible cells.

39 ubsequently confer cancer stemness traits to susceptible cells.

40 e required for facilitating virus entry into susceptible cells.

41 te degradation in the malignant hyperthermia-susceptible cells.

42 s essential for EV-D68 to bind to and infect susceptible cells.

43 ultimately leads to premature senescence of susceptible cells.

44 rus type 1 (HTLV-1) and can spread HTLV-1 to susceptible cells.

45 us was recovered after RNA transfection into susceptible cells.

46 virus particles which can be transmitted to susceptible cells.

47 tibodies that neutralize viral attachment to susceptible cells.

48 d viral integration and replication in HIV-1-susceptible cells.

49 ch are expressed to various degrees in HIV-1 susceptible cells.

50 ing transfection and subsequent infection of susceptible cells.

51 infect intraepithelial and subepithelial HIV-susceptible cells.

52 establishes sustained latent persistence in susceptible cells.

53 subunits that bind globotriaosylceramide on susceptible cells.

54 p53] host mRNA export following infection of susceptible cells.

55 inheritance due to loss of heterozygosity in susceptible cells.

56 icrodomains for entry into and egress out of susceptible cells.

57 ure of Etx pore assembled on the membrane of susceptible cells.

58 xhibited similar one-step growth kinetics in susceptible cells.

59 tors and induces primarily necrotic death in susceptible cells.

60 s B virus (HBV) in attachment and entry into susceptible cells.

61 not needed for HBV attachment and entry into susceptible cells.

62 hrough binding the proteoglycan receptors of susceptible cells.

63 had infected the same small subpopulation of susceptible cells.

64 mplex intracellular pathway in order to kill susceptible cells.

65 verting enzyme 2, the functional receptor on susceptible cells.

66 terminal repeat of p65 and p50 proteins from susceptible cells after exposure to HRF, and the binding

67 ansfer human immunodeficiency virus (HIV) to susceptible cells, although the underlying mechanism is

68 to (gp120/gp41)(3)] attaches the virion to a susceptible cell and induces fusion of viral and cell me

69 s B3 (CVB3) utilizes polyamines to attach to susceptible cells and initiate infection.

70 n E9 protein toxin to enter the cytoplasm of susceptible cells and kill them by hydrolysing their DNA

71 titers of HTLV-1 pseudotypes between highly susceptible cells and poorly susceptible cells.

72 viruses as they trigger cytotoxicity against susceptible cells and secrete proinflammatory cytokines

73 clear how these three classes of agents kill susceptible cells and whether they utilize the same cyto

74 ixed populations of antibiotic resistant and susceptible cells, and also track pole age and growth ra

75 s-susceptible cells, binds preferentially to susceptible cells, and competes with mature virus for bi

76 sing, differences between O2-tolerant and O2-susceptible cells, and differences between acute and chr

77 for attachment and binding to receptor(s) on susceptible cells, and GP2, which mediates viral and cel

78 the leptin receptor conferred resistance in susceptible cells, and leptin stimulation enhanced prote

79 ereby decreasing the viral burden, access to susceptible cells, and the chronic activation of the imm

80 vo consequences of both the accessibility of susceptible cells, and their heterogeneous susceptibilit

81 ciencies of infection of resistant cells and susceptible cells are similar, but resistant cells (C3H/

82 in AcH in aqueous solution and vesicles from susceptible cells, as well as the ability of trifluoroet

83 particle) upon interaction with receptors on susceptible cells at 37 degrees C.

84 onment, leading to an increase in flavivirus-susceptible cells at the infected bite site.

85 , as well as the state of differentiation of susceptible cells at the time of infection, affects the

86 nt variable in this context is the number of susceptible cells available for virus replication.

87 ut switch to the lysogenic cycle later (when susceptible cells become scarce) is favoured over a bet-

88 binds with saturable character to poliovirus-susceptible cells, binds preferentially to susceptible c

89 of the Fas/FasL system induces apoptosis of susceptible cells, but may also lead to nuclear factor k

90 ptosis-inducing ligand) induces apoptosis in susceptible cells by binding to death receptors 4 (DR4)

91 Infection of susceptible cells by herpes simplex virus (HSV) can lead

92 Upon infection of susceptible cells by HIV-1, the conical capsid formed by

93 Hepatitis C virus (HCV) gains entry into susceptible cells by interacting with cell surface recep

94 utralizing antibodies block viral entry into susceptible cells by targeting the HIV-1 envelope glycop

95 HIV-1 can disseminate between susceptible cells by two mechanisms: cell-free infection

96 /RV) produce less genomic RNA than congenic, susceptible cells (C3H/He).

97 Expressing Abs on the surface of HIV-1-susceptible cells can be a new way to fight HIV-1.

98 opic expression of the HLA-DR complex in non-susceptible cells conferred susceptibility.

99 te outbreaks of phage infections in pools of susceptible cells continue to occur.

100 We find that inhibitor treatment of susceptible cells derepresses the Polycomb repressive co

101 The block to expression in less susceptible cells does not appear to result from the abi

102 dic platform was capable of delineating drug susceptible cells, drug tolerant, and drug resistant cel

103 Upon coculture with CD4(+)-susceptible cells, epithelial cells can effectively tran

104 qCXCL16 acts as an EAV entry receptor in EAV-susceptible cells, equine monocytes.

105 eumococci in mouse lungs were outcompeted by susceptible cells even during Cm treatment.

106 iate the lipid-mediated apoptotic cascade in susceptible cells, failed to either induce PT or release

107 In less susceptible cells, Fas transduces apoptosis-independent

108 to ensure the maintenance of a local pool of susceptible cells for additional rounds of virus replica

109 in the infant mucosa provide a large pool of susceptible cells for ingested HIV-1 at birth and during

110 WNV antibodies do not always protect susceptible cells from WNV infection in vitro.

111 The sequence data revealed that the EHV-1-susceptible cells had acquired an E. caballus MHC-I cDNA

112 s in many transformed cells due, in part, to susceptible cells harboring defects in the IFN system.

113 human immunodeficiency virus (HIV) spread to susceptible cells has been documented, there is comparat

114 We showed that DNA in antibiotic-susceptible cells has increased accessibility upon expos

115 nfection strategy when half of the available susceptible cells have been infected.

116 st in vitro, to promote virus replication in susceptible cells.IMPORTANCE AKAV and SBV are the etiolo

117 riptomic profiling of susceptible versus non-susceptible cells in combination with genome-wide CRISPR

118 Understanding why and how susceptible cells in motor and nonmotor regions of the b

119 The mechanisms by which viruses kill susceptible cells in target organs and ultimately produc

120 ns may contribute to tumor initiation within susceptible cells in the CNS by limiting DNA damage repa

121 of the dose required for 50% infectivity of susceptible cells in tissue culture (TCID(50)).

122 HIVs and simian immunodeficiency viruses to susceptible cells in trans.

123 ssays in measuring recent HIV-1 infection of susceptible cells in vivo needs to be reevaluated.

124 Skp2 becomes an essential survival gene when susceptible cells incur Rb1 deficiency.

125 curli fibres provide a barrier that protects susceptible cells independent of genes required for biof

126 How does the number of susceptible cells influence the growth potential of the

127 nveloped virus.IMPORTANCE Virus entry into a susceptible cell is the first step of infection and a si

128 r understanding of how HIV is transmitted to susceptible cells is critical to devise effective strate

129 d by bacteria binds to membrane receptors of susceptible cells, is cleaved proteolytically, attaches

130 brane protein (p38.5) from a prototypical NK-susceptible cell line (K562) that preferentially bound t

131 Equivalent fractions from a second scrapie-susceptible cell line also stimulate conversion.

132 ells from infected tissues were mixed with a susceptible cell line, the subtype C isolates were outco

133 ed from gradient centrifugation of a scrapie-susceptible cell line.

134 xpressed on the surface of MARC-145, a PRRSV-susceptible cell line.

135 erlotinib decreased c-fos mRNA levels in the susceptible cell lines A431, CAL27, and HN11; however, b

136 ed 28S rRNA cleavage was detected in all MHV-susceptible cell lines and all MHV strains tested.

137 structure and assembly of PaV, we identified susceptible cell lines and developed a reverse genetic s

138 We also used susceptible cell lines and our modified cell blot proced

139 tudied in an attempt to identify potentially susceptible cell lines for virus propagation in vitro an

140 g that use of a combination of different HRV-susceptible cell lines is the best approach for the reco

141 Using genomic approaches, we show that the susceptible cell lines overexpress cytidine deaminase (C

142 SteviX4 induces ferroptotic cell death in susceptible cell lines, and target identification experi

143 KCP and/or K8.1 remained infectious in KSHV-susceptible cell lines, including epithelial, endothelia

144 In vitro, even in the most susceptible cell lines, more than 50% of cells latently

145 -FADD association and caspase-8 cleavage) in susceptible cell lines.

146 sible for attenuation of MLB astroviruses in susceptible cell lines.

147 ar stomatitis virus, and influenza virus, in susceptible cell lines.

148 esistant mitochondria via elimination of Fas-susceptible cells may suggest the existence of a shared

149 infectivity, or it could block receptors on susceptible cells near the sites of transmission.

150 are small polypeptides (7-14 kDa) which kill susceptible cells of closely related fungal species.

151 In susceptible cells only, sigma antagonists evoke a rapid

152 ent modalities such as gene therapy of HIV-1-susceptible cell populations, must be capable of engende

153 ny antineoplastic agents induce apoptosis in susceptible cells raises the possibility that factors af

154 However, susceptible cells remain extant in the wild, implying th

155 Transfection of these cosmid clones into MDV-susceptible cells resulted in the generation of a recomb

156 n-resistant revertants, isolated from highly susceptible cells, revealed a gene expression signature

157 To further understand how rotaviruses enter susceptible cells, six different polarized epithelial ce

158 R) as the receptor through which they infect susceptible cells, some CVB strains are known for their

159 y intermediate because it accumulates inside susceptible cells soon after infection and drugs which i

160 The ferroptosis-susceptible cell state can either pre-exist in cells tha

161 olon tumors in mice and led to a ferroptosis-susceptible cell state.

162 the activation or differentiation state of a susceptible cell such as a macrophage.

163 ation and secretion of H1B from target HIV-1 susceptible cells, suggesting that ubiquitinated H1B is

164 s with an EGFR complementary DNA renders non-susceptible cells susceptible to HCMV.

165 its increased binding to and infectivity for susceptible cell targets.

166 ant cancers contained a higher percentage of susceptible cells than cancers from noncastrated mice.

167 l persisters, a subpopulation of genetically susceptible cells that are normally dormant and tolerant

168 ompetition assays in vitro were performed in susceptible cells that were either interferon type I/III

169 to elute unproductively from the surface of susceptible cells, their precise role remains unclear.

170 ute induction of EphA2 may purge genetically susceptible cells, thereby uncovering a more aggressive

171 Upon entering susceptible cells, these ABPs enzymatically degrade esse

172 Syringacin M kills susceptible cells through a highly specific phosphatase

173 Viruses are thought to spread across susceptible cells through an iterative process of infect

174 1b were serially diluted and cocultured with susceptible cells to amplify virus.

175 We suggest that delivery of sfRNA to new susceptible cells to inhibit type I IFN induction before

176 ghlights kidney proximal tubule cells as key susceptible cells to injury.

177 We determined whether exposure of susceptible cells to Tat protein of HIV could result in

178 he minimum inhibitory concentration (MIC) of susceptible cells to the MIC of the least susceptible, s

179 ld promote cell proliferation and predispose susceptible cells to tumorigenesis.

180 cell activation can be abrogated by exposing susceptible cells (tumor and mycobacteria-infected cells

181 ers in fetal brain development, are the most susceptible cell type for HCMV infection in the fetal br

182 cDNAs from this EHV-1-susceptible cell type were inserted into EHV-1-resistant

183 This platform identified susceptible cell types and demonstrated dynamic traffick

184 We show that FIPC-1 induces ferroptosis in susceptible cell types and labels cellular proteins in a

185 of regional and developmental variations in susceptible cell types and supportive microenvironments

186 tively high cAMP signaling pathway output in susceptible cell types can elicit DNA replication stress

187 The susceptible cell types in the cerebral cortex and the mo

188 le KSHV establishes latency in virtually all susceptible cell types, LECs support spontaneous express

189 To identify susceptible cell types, Multi-Marker Analysis of Genomic

190 Abnormal growth and maturation of susceptible cell types, particularly neurons and oligode

191 pitopes can lead to increased infectivity of susceptible cells via antibody-dependent enhancement (AD

192 NFalpha family that can trigger apoptosis in susceptible cells via respective death receptors (DRs).

193 BDV G-mediated attachment to BDV-susceptible cells was cholesterol independent, but G loc

194 L activity levels in congenic resistant and susceptible cells were compared.

195 At the initiation of treatment, susceptible cells were either small and at early stages

196 heptapeptide facilitates McC transport into susceptible cells, where it is processed releasing a non

197 can be transmitted in cocultures to adherent susceptible cells, which become infected, express viral

198 olyamine depletion limits CVB3 attachment to susceptible cells, which is rescued by incubating virus

199 MDV concurrently loses its ability to infect susceptible cells while gaining the capacity to infect i

200 Furthermore, pretreatment of susceptible cells with anti-beta(3) antibodies such as c

201 studies of IFN production after infection of susceptible cells with measles virus (MeV) have often re