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1 , which replaces the flagellum of the motile swarmer cell.
2 thesis of the pili filaments in the daughter swarmer cell.
3 re only present at the flagellar pole of the swarmer cell.
4 on, resulting in a stalked cell and a motile swarmer cell.
5 vides asymmetrically, producing a mother and swarmer cell.
6 change that culminates in the formation of a swarmer cell.
7 d accumulates preferentially in the daughter swarmer cell.
8 aL mutant was unable to differentiate into a swarmer cell.
9 hter cells, a stalked cell and a flagellated swarmer cell.
10 mer cell to an elongated, highly flagellated swarmer cell.
11 lation migration across surfaces, called the swarmer cell.
12 to and silences the origin of replication in swarmer cells.
13 he CtrA protein and the size distribution of swarmer cells.
14 he incipient stalked pole in differentiating swarmer cells.
15  form that persists at the flagellar pole of swarmer cells.
16 tion of the early cell division gene ftsZ in swarmer cells.
17 tative swimmer cells to fully differentiated swarmer cells.
18 pA is fully expressed only in differentiated swarmer cells.
19 lts in transcription of the ftsZ promoter in swarmer cells.
20 acteria into successively thicker cohorts of swarmer cells.
21 ssion of flagellin that is characteristic of swarmer cells.
22 ibited a threefold increase in expression in swarmer cells.
23 longated (10- to 80-mum), highly flagellated swarmer cells.
24 ersed in the newly divided progeny stalk and swarmer cells.
25 ng sessile-stalked and piliated, flagellated swarmer cells.
26 ganisms is faster growing and may detach as "swarmer" cells.
27                                           In swarmer cells, a short form of PodJ is localized at the
28 e stalked cell, unphosphorylated DivK in the swarmer cell activates an intricate transcriptional casc
29 ntus divides asymmetrically to a flagellated swarmer cell and a cell with a stalk.
30  asymmetrically to produce a non-replicating swarmer cell and a replicating stalked cell.
31 ium Caulobacter crescentus produces a motile swarmer cell and a sessile stalked cell at each cell div
32 s rise to two different cell types: a motile swarmer cell and a sessile stalked cell.
33 lls divide asymmetrically, yielding a motile swarmer cell and a sessile stalked cell.
34 s asymmetric cell division to yield a motile swarmer cell and a stalked cell in the gram-negative bac
35 ter crescentus divides asymmetrically into a swarmer cell and a stalked cell, a process that is gover
36 t cell types at each cell division, a motile swarmer cell and an adhesive stalked cell.
37 rsed throughout the cytoplasm of the progeny swarmer cell and is localized to the pole of the stalked
38 alA-flaB locus from wild-type swimmer cells, swarmer cells and cells obtained after urinary tract inf
39        Transcription of ftsZ is repressed in swarmer cells and is activated concurrently with the ini
40    The initial stage of attachment occurs in swarmer cells and is facilitated by flagellar motility a
41 he fliL transcriptome with that of wild-type swarmer cells and showed that nearly all genes associate
42 cious swarming mutants are also constitutive swarmer cells and swarm on minimal agar medium.
43 origin is located at the flagellated pole of swarmer cells and, immediately after the initiation of D
44 that SpoT is required for this phenomenon in swarmer cells, and in the absence of SpoT, carbon-starve
45 ional flagellar pole, remain at this pole in swarmer cells, and localize at the stalk tip after the s
46                                              Swarmer cells are generally more flagellated and longer
47                                 H. neptunium swarmer cells are highly motile via a single polar flage
48                                              Swarmer cells are specially adapted to rapidly transloca
49 vision and contain FtsZ, whereas the progeny swarmer cells are unable to initiate DNA replication and
50 ntration, FtsZ was artificially expressed in swarmer cells at a level equivalent to that found in pre
51 h slowdown, but the effect is more severe in swarmer cells because they have a longer G1 phase.
52 ypothesize that this novel mechanism acts on swarmer cells born in a biofilm, where eDNA can accumula
53 e processes in Caulobacter and is present in swarmer cells but absent from stalked cells.
54 Instead, doubling of the average length of a swarmer cell by suppression of cell division effectively
55 e inappropriate production of differentiated swarmer cells, called pseudoswarmer cells, under nonindu
56 es a large gene system and differentiates to swarmer cells capable of movement over and colonization
57                                   In progeny swarmer cells, CcrM is completely degraded by Lon before
58                                           In swarmer cells, CpdR is in the phosphorylated state, thus
59 rives progression of the coupled stalked and swarmer cell cycles of the bacterium Caulobacter crescen
60                        Expression of FtsZ in swarmer cells did not alter the timing of cell constrict
61 nsistent with this observation, H. neptunium swarmer cells did not respond to any chemotactic stimuli
62                                           As swarmer cells differentiate into stalked cells (G1/S tra
63      FtsZ is synthesized slightly before the swarmer cells differentiate into stalked cells and the i
64 period under typical culture conditions, the swarmer cell differentiates into a replicative stalked c
65 diated by the synthesis of a holdfast as the swarmer cell differentiates into a stalked cell.
66 ntiate showing the essential role of flaA in swarmer cell differentiation and behaviour.
67 se results suggest that FliL plays a role in swarmer cell differentiation and implicate FliL as criti
68 ditions inhibiting flagellar rotation induce swarmer cell differentiation and implicating a rotating
69 ression of fliL+ in wild-type cells prevents swarmer cell differentiation and motility, a result also
70 the addition of polyvinylpyrrolidone induced swarmer cell differentiation and resulted in a fourfold
71 utrescine restored both the normal timing of swarmer cell differentiation and the ability to migrate
72 tical to transduction of the signal inducing swarmer cell differentiation and virulence gene expressi
73 ession of wosA also resulted in constitutive swarmer cell differentiation in liquid medium, a normall
74 al through the chemotaxis pathway and induce swarmer cell differentiation in response to signals othe
75 tudy also suggests that despite constitutive swarmer cell differentiation in wosA-overexpressing stra
76               A search for genes controlling swarmer cell differentiation of Vibrio parahaemolyticus
77            ZapA activity is not required for swarmer cell differentiation or swarming behaviour, as Z
78                                              Swarmer cell differentiation parallels an increased expr
79 l transduction from solid surfaces to induce swarmer cell differentiation, possibly via alterations i
80 ile the latter two classes were defective in swarmer cell differentiation, representative LPS mutants
81 f growth that are formed as cyclic events of swarmer cell differentiation, swarming migration, and ce
82 zation of urinary tract surfaces is aided by swarmer cell differentiation, which is initiated by inhi
83 ogen, iron limitation is a signal modulating swarmer cell differentiation.
84 e two-component systems cooperate to promote swarmer cell differentiation.
85  other proteins and virulence factors during swarmer cell differentiation.
86 lator FlhD(4)C(2), which ultimately controls swarmer cell differentiation.
87 g growth in liquid and on agar plates during swarmer cell differentiation.
88 16-fold higher in the disA background during swarmer cell differentiation.
89             DisA overexpression also blocked swarmer cell differentiation.
90 k and holdfast occur at the same pole during swarmer cell differentiation.
91  by flagella is thought to trigger bacterial swarmer-cell differentiation, an important step in patho
92 A fliL mutation that results in constitutive swarmer cell elongation also increased wosA transcriptio
93 regulator that controls the establishment of swarmer cell fate.
94     Expression of umoA (a known regulator of swarmer cells), flgF, and flgI increased significantly i
95                        This event primes the swarmer cell for the impending transition into a stalked
96 ivated in liquid broth and hyper-flagellated swarmer cells from solid medium.
97          These mutants were constitutive for swarmer cell gene expression, inappropriately expressing
98  its flagellar motors, which in turn control swarmer cell gene expression.
99  inappropriately expressing high levels of a swarmer cell gene fusion product when grown in liquid.
100           When the operon was overexpressed, swarmer cell gene transcription was induced in liquid cu
101 cells were extremely long and thus resembled swarmer cells harvested from a surface.
102 iately after cell division and (ii) a motile swarmer cell in which DNA replication is blocked.
103 e rapidly away from a colony, analogously to swarmer cells in bacteria with flagella.
104  media, thereby increasing the proportion of swarmer cells in mixed culture.
105 , result in the inappropriate development of swarmer cells in noninducing liquid media or hyperelonga
106 ue to an increase in the number of elongated swarmer cells in the population.
107 , and in the absence of SpoT, carbon-starved swarmer cells inappropriately initiated DNA replication.
108 entus differentiates from a motile, foraging swarmer cell into a sessile, replication-competent stalk
109                  Upon differentiation of the swarmer cell into a stalked cell, full length PodJ is sy
110 ation initiator, upon differentiation of the swarmer cell into a stalked cell.
111 is synthesized during the differentiation of swarmer cells into replicating stalked cells.
112 genesis of the single polar flagellum of the swarmer cell is the best-studied aspect of this developm
113          PodJ(S), sequestered to the progeny swarmer cell, is subsequently released from the polar me
114                   Because CtrA is present in swarmer cells, is degraded at the same time as ftsZ tran
115          Upon depletion of available carbon, swarmer cells lacking the ability to synthesize ppGpp or
116 trogen limitation significantly extended the swarmer cell life span.
117 thesize that nitrogen limitation extends the swarmer cell lifetime by delaying the onset of a sequenc
118 d a technique for observing isolated E. coli swarmer cells moving on an agar substrate and confined i
119                                              Swarmer cells of Caulobacter crescentus are devoid of th
120                                       Motile swarmer cells of Hyphomicrobium strain W1-1B displayed p
121                                  We adsorbed swarmer cells of Serratia marcescens to polydimethylsilo
122 ion of external environmental cues, sets the swarmer cell on a path to differentiate into a stalked c
123 n noninducing liquid media or hyperelongated swarmer cells on agar media.
124 rium can differentiate into hyperflagellated swarmer cells on agar of an appropriate consistency (0.5
125       The terminus moves from the end of the swarmer cell opposite the origin to midcell.
126 ntrolling the decision to be a highly mobile swarmer cell or a more adhesive, biofilm-proficient cell
127 ls, we were unable to detect the holdfast in swarmer cells or at the flagellated poles of predivision
128 umerous unsheathed lateral flagella move the swarmer cell over surfaces.
129  discovered a binding partner of PopA at the swarmer cell pole that together with PopA regulates the
130 ntegrated into the membrane at the incipient swarmer cell pole, where it initiates flagellar assembly
131                         Here, we show that a swarmer-cell-pole scaffold, PodJ, forms biomolecular con
132                    Furthermore, we show that swarmer cells produce more ppGpp than stalked cells upon
133                                       Unlike swarmer cells, pseudoswarmer cells are not hyperflagella
134                                       Unlike swarmer cells, pseudoswarmers displayed increased activi
135 motility is a random dispersal mechanism for swarmer cells rather than a stimulus-controlled navigati
136 ost-division degradation of FtsA and FtsQ in swarmer cells reduces their concentration to 7% and 10%
137 t extract (PYE) medium contain >70% and >80% swarmer cells, respectively.
138 luorescence microscopy showed that, in these swarmer cells, simply increasing FtsZ concentration was
139 ve from these results that the C. crescentus swarmer cells swim more efficiently than both E. coli an
140               Forward swimming C. crescentus swarmer cells tend to get physically trapped at the surf
141  and produces cells that look like wild-type swarmer cells, termed "pseudoswarmer cells," that are el
142 similar daughter cells: a stalked cell and a swarmer cell that assembles several pili at the flagella
143 ed cell competent for DNA replication, and a swarmer cell that is unable to initiate DNA replication
144 m divides asymmetrically to produce a motile swarmer cell that represses DNA replication and a sessil
145                The morphological change from swarmer cell to stalked cell is a result of changes of f
146 comitantly with the transition of the motile swarmer cell to the sessile stalked cell.
147 e half-life of FtsA increases from 13 min in swarmer cells to 55 min in stalked cell types, confirmin
148 d, increasing ca. twofold from low levels in swarmer cells to a maximum immediately prior to cell div
149 lyzed the adaptive response of C. crescentus swarmer cells to carbon starvation and found that there
150 ci contour lengths in Caulobacter crescentus swarmer cells to determine the in vivo configuration of
151 ances initial attachment and enables progeny swarmer cells to escape from the monolayer biofilm.
152                             Both swimmer and swarmer cells transcribe flaA, but not flaB.
153 ngated, hyperflagellated, and multinucleated swarmer cell type when it is grown on a surface.
154 egained motility yet produced differentiated swarmer cells under noninducing conditions transcribed f
155                                        Thus, swarmer cells utilize at least two independent signaling
156        Differentiation of the speB mutant to swarmer cells was delayed by two hours relative to wild-
157                                    Elongated swarmer cells were only rarely observed.
158 a typical Gram-negative rod) to an elongated swarmer cell when grown on a solid surface.
159 ble of differentiating into hyperflagellated swarmer cells when plated on a solid agar surface.
160 ed cells but degraded in the non-replicative swarmer cell where ClpAP alone degrades FtsA and both Cl
161 e, are evenly distributed between mother and swarmer cells, whereas hpnN is required for the C(35) ho
162 intracellular location of SMC showed that in swarmer cells, which do not replicate DNA, the protein f
163 obacter crescentus is asymmetric, yielding a swarmer cell with several polar pili and a non-piliated

 
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