ライフサイエンスコーパス: synaptic plasticity

1 stigation of more powerful directed types of synaptic plasticity.

2 ng of synaptic input patterns with long-term synaptic plasticity.

3 detecting circuit-specific learning-induced synaptic plasticity.

4 uit to ameliorate motor symptoms and recover synaptic plasticity.

5 c transmission and mediate multiple forms of synaptic plasticity.

6 functions in engram physiology beyond aiding synaptic plasticity.

7 impairments in hippocampal neurogenesis and synaptic plasticity.

8 elty suppressed feeding tests, and increased synaptic plasticity.

9 ynamic SUMOylation processes associated with synaptic plasticity.

10 ve rapid, data efficient learning with local synaptic plasticity.

11 amine and prevented scopolamine induction of synaptic plasticity.

12 n the brain and modulates different forms of synaptic plasticity.

13 ession (NMDAR-LTD) is a long-lasting form of synaptic plasticity.

14 e the cellular correlate for mGluR-dependent synaptic plasticity.

15 re the influence of extracellular calcium on synaptic plasticity.

16 fects in mice of either sex on cognition and synaptic plasticity.

17 e a lasting impact on hippocampal memory and synaptic plasticity.

18 n HD mice and evoked a sustained increase of synaptic plasticity.

19 ic connections through a phenomenon known as synaptic plasticity.

20 , brain regions associated with learning and synaptic plasticity.

21 fferences in the effects of junk-food on NAc synaptic plasticity.

22 e in neuronal survival, differentiation, and synaptic plasticity.

23 itive to PCBD are pathways with key roles in synaptic plasticity.

24 with regions known to exhibit high levels of synaptic plasticity.

25 termittent hypoxia (dAIH) that evokes robust synaptic plasticity.

26 lter the experimental outcome with regard to synaptic plasticity.

27 n extensively characterized as a mediator of synaptic plasticity.

28 t during severe hypoxia except for long-term synaptic plasticity.

29 -serine and therefore affect NMDAR-dependent synaptic plasticity.

30 cascade, which has been previously linked to synaptic plasticity.

31 he NMDAR to dendritic spine shrinkage during synaptic plasticity.

32 at modulate the ability to elicit subsequent synaptic plasticity.

33 uscle contraction, apoptosis, secretion, and synaptic plasticity.

34 y contribute to local protein changes during synaptic plasticity.

35 receptors rescues both NMDAR activation and synaptic plasticity.

36 e performance by altering activity-dependent synaptic plasticity.

37 nges in synaptic transmission that influence synaptic plasticity.

38 the hippocampus, a key area for learning and synaptic plasticity.

39 factor (BDNF) is a potent modulator of brain synaptic plasticity.

40 synaptic efficacies endowed with short-term synaptic plasticity.

41 cal dynamics of small molecules that control synaptic plasticity.

42 els can account for the observed homeostatic synaptic plasticity.

43 bunit composition and altered NMDA-dependent synaptic plasticity.

44 ssion, neurotrophic factors, and measures of synaptic plasticity.

45 receptor trafficking, which is essential for synaptic plasticity.

46 akening and dendritic spine shrinkage during synaptic plasticity.

47 can have profoundly negative consequences on synaptic plasticity.

48 e, innate immunity, synapse development, and synaptic plasticity.

49 rare mutations in genes that are critical to synaptic plasticity.

50 tions for various MAPs in activity-dependent synaptic plasticity.

51 ssociated with depression-like behaviors and synaptic plasticity.

52 parallel fiber (PF) synapses, triggering PF synaptic plasticity.

53 ner and defines a neuronal subset primed for synaptic plasticity.

54 naptically located receptors that can impair synaptic plasticity.

55 d be due to an underlying diversity in their synaptic plasticity.

56 g synaptogenesis, synaptic transmission, and synaptic plasticity.

57 fluences on neurotransmission and short-term synaptic plasticity.

58 for this process, by serving as triggers for synaptic plasticity.

59 to synapses as well as neurotransmission and synaptic plasticity.

60 contributor in heroin-induced cell-specific synaptic plasticity.

61 n characterized as a critical event for this synaptic plasticity.

62 th previously been implicated in hippocampal synaptic plasticity.

63 al neurons in the strata, and a magnitude of synaptic plasticity adequate for each neuronal stratum.

64 quencies and show that neurogranin regulates synaptic plasticity along three modalities.

65 In addition, altered short- and long-term synaptic plasticity, along with an increased spine densi

66 ers are characterized by impaired functional synaptic plasticity and abnormal dendritic spine morphol

67 ocesses have distinct effects on homeostatic synaptic plasticity and animal behavior.

68 s are key regulators of neurodevelopment and synaptic plasticity and are unique in their requirement

69 We describe differences in synaptic plasticity and behavior when optogenetically dr

70 PA receptor (AMPAR) expression is central to synaptic plasticity and brain function, but how these ch

71 he present study examines sex differences in synaptic plasticity and cellular activation occurring in

72 d morphology are altered as a consequence of synaptic plasticity and circuit refinement during adoles

73 as the underlying mechanism for deficits in synaptic plasticity and cognition with obesity and insul

74 ses, suggesting a role for RNA regulation in synaptic plasticity and cognition.

75 port that caspase-2 plays a critical role in synaptic plasticity and cognitive flexibility.

76 lements of four broadly categorized forms of synaptic plasticity and discuss their functional capabil

77 highlighted that dysregulated miRNAs target synaptic plasticity and dopaminergic signaling pathways,

78 nderlying motor deficit by assessing in vivo synaptic plasticity and E/I balance in the primary motor

79 mental role of the endocannabinoid system in synaptic plasticity and emotional memory processing.

80 ncreased general mRNA translation, bolstered synaptic plasticity and enhanced long-term memory.

81 gesting RSK is required for learning-related synaptic plasticity and enhancement in neuronal excitabi

82 aired with tactile rehabilitation to enhance synaptic plasticity and facilitate recovery of sensory f

83 Cortical inflammation, synaptic plasticity and gamma-aminobutyric acid (GABA) s

84 ar-derived synaptic organizer that regulates synaptic plasticity and hippocampal-dependent memory.

85 -4 significantly reduces basal transmission, synaptic plasticity and impairs postsynaptic receptor tr

86 lar pathway is triggered by the induction of synaptic plasticity and in response to object location l

87 tle is known about stress-induced inhibitory synaptic plasticity and its relevance for neuropsychiatr

88 GRIP1 in regulating AMPAR trafficking during synaptic plasticity and learning and memory.

89 m channel, SK2, contributes to impairment of synaptic plasticity and learning in AS mice.

90 lcNAcylation leads to defects in hippocampal synaptic plasticity and learning.

91 bitory synaptic transmission and rescued the synaptic plasticity and long-term memory deficits in DS

92 tive conditioning by molecular processes for synaptic plasticity and long-term memory.

93 ating evidence supports the role of sleep in synaptic plasticity and memory consolidation.

94 r, partial blockade of Rac1 activity rescues synaptic plasticity and memory deficits in Cc2d1a cKO mi

95 protein kinase II (CAMK2) is a key player in synaptic plasticity and memory formation.

96 tion of activity-dependent genes, as well as synaptic plasticity and memory formation.

97 roprotective actions of physical exercise on synaptic plasticity and memory in AD mice.

98 ct against Abeta-induced damage of long-term synaptic plasticity and memory, or from amyloid depositi

99 igomeric tau-induced impairment of long-term synaptic plasticity and memory.

100 s that are vital for regulation of long-term synaptic plasticity and memory.

101 ockout did not affect life span but impaired synaptic plasticity and memory.

102 pression of proteins essential for long-term synaptic plasticity and memory.

103 ng a multitude of brain functions, including synaptic plasticity and motor planning.

104 in astrocytic Glu uptake can play a role in synaptic plasticity and neurodegeneration.

105 pheric asymmetry, we investigated changes in synaptic plasticity and neuronal excitability of BLA neu

106 role of endogenous calcineurin in regulating synaptic plasticity and nociceptive transmission and sug

107 rmacological activation of betaARs modulates synaptic plasticity and opens therapeutic opportunities

108 However, diverse forms of synaptic plasticity and pathophysiological conditions ar

109 tive mRNA transcripts from genes involved in synaptic plasticity and psychiatric disease.

110 ings suggest that LB differentially programs synaptic plasticity and PV/perineuronal net development

111 gated (HCN) channels are major regulators of synaptic plasticity and rhythmic activity in the heart a

112 Synaptic plasticity and seeking behavior induced by drug

113 resultant insulin resistance (IR) modulates synaptic plasticity and the corresponding behavioral fun

114 hippocampus, and regulates the induction of synaptic plasticity and the hippocampus-dependent mnemon

115 They are critically involved in synaptic plasticity and their availability has been repo

116 h biologically realistic dynamics, including synaptic plasticity and time-varying inputs.

117 the ability to orchestrate multiple forms of synaptic plasticity and to adapt to sensory patterns in

118 ng changes in neurons' learning flexibility (synaptic plasticity) and epigenetic misregulation in ani

119 stnatal week, can enable hippocampal memory, synaptic plasticity, and alter hippocampal excitability

120 e in the formation of presynaptic terminals, synaptic plasticity, and axonal growth and regeneration.

121 le of autophagy in the pre- and postsynapse, synaptic plasticity, and behavior.

122 d may negatively affect neuronal morphology, synaptic plasticity, and cognitive function.

123 ts of Abetaos on glutamatergic transmission, synaptic plasticity, and dendritic spine structure.

124 protein kinase associated with excitability, synaptic plasticity, and excitability disorders, with th

125 for hippocampal basal neurotransmission and synaptic plasticity, and further supports the notion tha

126 gene, is essential for learning and memory, synaptic plasticity, and maturation of neural networks.

127 for the APP family in neuronal excitability, synaptic plasticity, and memory in adulthood, despite th

128 f biologic processes, including development, synaptic plasticity, and regeneration after injury, as w

129 MARCKS supports development, synaptic plasticity, and regeneration after injury.

130 scular inflammation, recovery of hippocampal synaptic plasticity, and restoration of hippocampus-depe

131 the molecular mechanisms of synaptogenesis, synaptic plasticity, and synaptic toxicity.

132 likely a crucial parameter in determining PF synaptic plasticity, and the occurrence of hyperpolariza

133 ch excess glutamate can negatively influence synaptic plasticity, and we discuss the relevance of the

134 oscillations, synaptic transmission, and/or synaptic plasticity are impaired following kindled seizu

135 tion, numbers, and change at synapses during synaptic plasticity are tightly regulated by neuronal ac

136 fibres and to detect short-lived changes in synaptic plasticity as measured by the application of cu

137 GNIFICANCE STATEMENT Long-lasting changes in synaptic plasticity associated with memory formation are

138 nscribed from enhancer regions that regulate synaptic plasticity-associated gene expression, includin

139 ntire organism, including a robust change in synaptic plasticity at glutamate synapses onto corticotr

140 by endocannabinoids that endow bidirectional synaptic plasticity at identified BLA-NAc synapses.

141 ring the second postnatal week, but inhibits synaptic plasticity at later developmental stages.

142 lative to L5, requiring 48 h of SAT to drive synaptic plasticity at thalamic and intracortical inputs

143 ncreased synaptic transmission and long-term synaptic plasticity at the Cornu Ammonis (CA) 3-CA1 syna

144 tation/inhibition (E/I) balance and aberrant synaptic plasticity at the cortical level.

145 ually evoked network dynamics and short-term synaptic plasticity at the SAC-DSGC synapse.

146 dependent transcription factor important for synaptic plasticity, at single-cell resolution.

147 Motor learning depends on synaptic plasticity between corticostriatal projections

148 ed sessions, which we attribute to long-term synaptic plasticity between interneurons and pyramidal c

149 ronal immediate early gene with key roles in synaptic plasticity, brain development, and behavior.

150 mice exhibited normal cognitive function and synaptic plasticity but had increased dendritic spine de

151 are involved in neuronal differentiation and synaptic plasticity but the molecular mechanisms behind

152 n kinase II (CaMKII) regulates many forms of synaptic plasticity, but little is known about its funct

153 nist of NMDA receptors (NMDARs) required for synaptic plasticity, but mechanisms that terminate D-ser

154 for myelin plasticity that would complement synaptic plasticity by adjusting conduction velocity for

155 Mathematical analysis shows how short-term synaptic plasticity can coordinately change amplitude an

156 These data establish that UPF2 regulates synaptic plasticity, cognition, and local protein synthe

157 y, a recently discovered hippocampal form of synaptic plasticity combines the above elements, while l

158 ial activation, and GABA signaling and lower synaptic plasticity compared with Ctrl-Hum mice.

159 rebrain cholinergic neurons (BFCNs) modulate synaptic plasticity, cortical processing, brain states a

160 n might serve as a presynaptic substrate for synaptic plasticity coupling distinct forms of release.S

161 2 markedly ablates tau pathology and rescues synaptic plasticity defects in tau P301S transgenic mice

162 elineate a working conceptual model in which synaptic plasticity deficits described in animal models

163 e function, brain insulin receptor function, synaptic plasticity, dendritic spine density, microglial

164 tformin equally improved cognitive function, synaptic plasticity, dendritic spine density, microglial

165 )-HNK, and that ketamine-induced hippocampal synaptic plasticity depends on 4E-BP2 and, to a lesser e

166 built a biochemically detailed model of post-synaptic plasticity describing CaMKII, PKA, and PKC path

167 TATEMENT In Purkinje neurons, parallel fiber synaptic plasticity, determined by coincident activation

168 ling growth is mechanistically distinct from synaptic plasticity driven by neuronal activity and requ

169 mits NMDA receptor-dependent corticostriatal synaptic plasticity during an early critical period of p

170 Blocking RARalpha-dependent homeostatic synaptic plasticity during an EE experience by ablating

171 increased receptor membrane trafficking and synaptic plasticity during memory reconsolidation.

172 l the unrecognized functions of autophagy in synaptic plasticity, endocytic recycling, and memory.

173 fundamental to the roles of the receptor in synaptic plasticity, even when expressed alongside wild-

174 We propose that the direction of ChC synaptic plasticity follows homeostatic rules that depen

175 persistent activity, excitatory feedback, or synaptic plasticity for storage.

176 Abnormal synaptic plasticity has been implicated in several neuro

177 lthough standard, correlation-based, Hebbian synaptic plasticity has been the primary focus of neuros

178 nce of isoform-specific functions of GSK3 in synaptic plasticity has not been fully explored.

179 Signalling pathways leading to post-synaptic plasticity have been examined in many types of

180 udies on the effects of sleep deprivation on synaptic plasticity have yielded discrepant results.

181 le-cell system to study aspects of defective synaptic plasticity in Coffin-Lowry Syndrome (CLS), a co

182 functional signaling protein that suppresses synaptic plasticity in dendritic spines of hippocampal n

183 MS medium spiny neurons, suggesting that MOR synaptic plasticity in DMS is less synapse-specific than

184 the importance of intrinsic excitability and synaptic plasticity in engrams, and the lifetime of an e

185 roduces AD-like impairments in cognition and synaptic plasticity in experimental systems.

186 nriched environment (EE) engaged homeostatic synaptic plasticity in hippocampal circuits, thereby red

187 ts after traumatic brain injury and enhanced synaptic plasticity in hippocampal slices.

188 ntenance is likely supported either by local synaptic plasticity in hippocampus or by activity patter

189 ntly reverses cocaine-induced behavioral and synaptic plasticity in male and female rodents.SIGNIFICA

190 that MAP2 participates in activity-dependent synaptic plasticity in mature hippocampal networks.

191 n and inter-neuronal crosstalk, and modulate synaptic plasticity in neural networks; extracellular gl

192 synthesis of RA is essential for regulating synaptic plasticity in regions of the brain involved in

193 brate and vertebrate nervous systems display synaptic plasticity in response to behavioral experience

194 This gating of synaptic plasticity in stress by astrocytic metabolic ne

195 amate presynaptic activity and alteration of synaptic plasticity in the basolateral amygdala (BLA), i

196 amate receptors (NMDARs) plays a key role in synaptic plasticity in the central nervous system (CNS).

197 0 d of IH (IH(30)) on adult neurogenesis and synaptic plasticity in the dentate gyrus.

198 of neuron-astrocyte signaling contributes to synaptic plasticity in the DLS of male and female mice.

199 tatus epilepticus, and postulates a role for synaptic plasticity in the emergence of epileptic foci.

200 The discovery of impaired hippocampal synaptic plasticity in the heterozygous mouse model shed

201 tic transmission and Amh-regulated long-term synaptic plasticity in the hippocampus.

202 ion of several mRNAs important for long-term synaptic plasticity in the hippocampus.

203 te that microglial roles in surveillance and synaptic plasticity in the mouse brain are modulated by

204 Seeking addictive drugs is regulated by synaptic plasticity in the nucleus accumbens core and in

205 ing evidence has established a firm role for synaptic plasticity in the pathogenesis of neuropathic p

206 ted toward a higher excitation, and impaired synaptic plasticity in the PFC such as those observed in

207 ases levels of synaptic proteins crucial for synaptic plasticity in the prefrontal cortex.

208 effects of oxytocin-MCH are associated with synaptic plasticity in the reward and fear circuits reve

209 We report that LB conditions enhanced synaptic plasticity in the right, but not the left BLA o

210 H) mice) display motor deficits and impaired synaptic plasticity in the striatum.

211 evealed that 3 mo of OLT1177 diet can rescue synaptic plasticity in this mouse model of AD (P = 0.007

212 We propose a novel model of synaptic plasticity in which aberrant neural networks ar

213 Both types of synaptic plasticity involve the control of postsynaptic

214 nucleus accumbens, opioid-induced excitatory synaptic plasticity involves presynaptic and postsynapti

215 Homeostatic synaptic plasticity is a stabilizing mechanism engaged b

216 We hypothesized that the cell type-specific synaptic plasticity is associated with parallel cell-spe

217 Dysregulation of proteins involved in synaptic plasticity is associated with pathologies in th

218 As synaptic plasticity is crucial for learning, we examine

219 conformation of calmodulin and its impact on synaptic plasticity is less clear.

220 ission, experimental evidence indicates that synaptic plasticity is metabolically demanding as well.

221 lace fields, dendritic inhibition along with synaptic plasticity is necessary for place field stabili

222 Altered synaptic plasticity is often associated with major depre

223 How Msp300 expression is regulated during synaptic plasticity is poorly understood.

224 Synaptic plasticity is well accepted to represent the ce

225 attenuates learning, memory, spine density, synaptic plasticity (L-LTP), and potentiates perseverati

226 However, hippocampal synaptic plasticity, learning, and memory are impaired i

227 an altered expression of markers involved in synaptic plasticity, learning, and memory formation such

228 MPARs) is a fundamental mechanism underlying synaptic plasticity, learning, and memory.

229 Reduced synaptic plasticity, manifested by impaired long-term po

230 (PKC)epsilon, (also of PKCalpha) on impaired synaptic plasticity/maturation and spatial learning and

231 that a deficit or alteration in hippocampal synaptic plasticity may contribute to the intellectual d

232 t two decades, extensive work on homeostatic synaptic plasticity mechanisms have shown that they dive

233 have shown that they diverge from classical synaptic plasticity mechanisms that process and store in

234 r, it is unclear how MAP2 is associated with synaptic plasticity mechanisms.

235 to the mutual influence between network and synaptic plasticity mechanisms.

236 cerebellar granule cells and participates in synaptic plasticity, motor control and learning that are

237 ealed involvement of these altered miRNAs in synaptic plasticity, nervous system development, and neu

238 ivation and enhances long-term potentiation, synaptic plasticity, neurogenesis and hippocampal-depend

239 released from neural cells are implicated in synaptic plasticity, neuron-glia interface, neuroprotect

240 terpretation of the multiple forms of neural synaptic plasticity observed experimentally, including s

241 Hence, inhibitory synaptic plasticity occurs in parallel with excitatory s

242 inoids that regulate NMDA receptor-dependent synaptic plasticity of glutamatergic synapses in the pre

243 e mechanisms can trigger spatially organized synaptic plasticity on various spatial and temporal scal

244 AG and RMTg inhibitory synapses but prevents synaptic plasticity only at PAG synapses.

245 brate and vertebrate neurons, coinduced with synaptic plasticity or in isolation.

246 Arc, a neuronal gene that is critical for synaptic plasticity, originated through the domesticatio

247 image sensor array features photon-triggered synaptic plasticity owing to its quasi-linear time-depen

248 ebellar-based learning is thought to rely on synaptic plasticity, particularly at synaptic inputs to

249 3 inflammasome improves behavioral tests and synaptic plasticity phenotypes in a murine model of the

250 tes inside the cytosol, inducing the loss of synaptic plasticity potential.

251 This form of synaptic plasticity primarily involves the regulation of

252 repressor Wilm's Tumor 1 (WT1) as a critical synaptic plasticity regulator that decreases memory stre

253 way mediates effects on downstream ARC-based synaptic plasticity related to these competing memory sy

254 Synaptic plasticity requires a tight control of mRNA lev

255 We show how various synaptic plasticity rules allow for the emergence of div

256 We find that when synaptic plasticity rules are naively implemented, train

257 still unclear whether biologically plausible synaptic plasticity rules can organize neuronal activity

258 dent plasticity, it is still unclear whether synaptic plasticity rules inferred from in vitro experim

259 urnover of D-serine and its effects on NMDAR synaptic plasticity.SIGNIFICANCE STATEMENT Despite the p

260 e and elimination of dendritic spines during synaptic plasticity.SIGNIFICANCE STATEMENT Signaling thr

261 ocannabinoids contribute to pathway-specific synaptic plasticity.SIGNIFICANCE STATEMENT We examined t

262 These receptors contribute to synaptic plasticity, spine maturation and circuit develo

263 Spike-timing-dependent synaptic plasticity (STDP) is a leading cellular model f

264 ike transmission by modeling both short-term synaptic plasticity (STP) and nonsynaptic effects.

265 al networks is influenced by both short-term synaptic plasticity (STP) as well as nonsynaptic factors

266 s that incorporated the signature short-term synaptic plasticity (STP) profiles of the inhibitory par

267 neurons, based on known forms of short-term synaptic plasticity (STP).

268 can be 'silently' maintained via short-term synaptic plasticity (STSP) without the need for persiste

269 +) signalling yielded pronounced homeostatic synaptic plasticity, suggesting a critical role for this

270 Because of its involvement in synaptic plasticity, SynGAP has emerged as a critical pr

271 ave identified specific forms of homeostatic synaptic plasticity that are elicited by these drugs use

272 essenger cAMP is an important determinant of synaptic plasticity that is associated with enhanced neu

273 Here, we reveal a hidden form of inhibitory synaptic plasticity that prevents accumulation of excita

274 ketamine elicits a unique form of functional synaptic plasticity that shares several attributes and m

275 eceptor (NMDAR) is vitally important for the synaptic plasticity that underlies learning.

276 ent inhibition of GABA interneurons promotes synaptic plasticity that underlies rapid antidepressant

277 ICANCE STATEMENT Repetitive drug use induces synaptic plasticity that underlies the formation of long

278 Synaptic plasticity, the activity-dependent change in ne

279 ic inhibition is a main factor for decreased synaptic plasticity, the cellular phenomenon underlying

280 ion of specific mRNAs has been implicated in synaptic plasticity, the tightly controlled mechanisms t

281 Mitochondria influence synaptic plasticity through calcium buffering and are cr

282 transitions between awake and SWS sleep, and synaptic plasticity to allow the change of synaptic conn

283 ecific ion channels may work in concert with synaptic plasticity to promote vocal learning.

284 dependent gene expression is integral to the synaptic plasticity underlying learning and memory; howe

285 ting diverse aspects of neurodevelopment and synaptic plasticity varies according to cellular redox s

286 Neurotrophins promote neuronal survival and synaptic plasticity via activating the tropomyosin recep

287 Translational control of long-term synaptic plasticity via Mechanistic Target Of Rapamycin

288 Potential homeostatic synaptic plasticity was either absent or masked by large

289 dress the necessity of GluA2 phospho-Y876 in synaptic plasticity, we generated phospho-deficient GluA

290 or genes involved in presynaptic homeostatic synaptic plasticity, we identified an essential role for

291 o Abeta-induced impairments in cognition and synaptic plasticity, whereas LCMT-1 gene-trap mice showe

292 uronal properties but specifically inhibited synaptic plasticity, which is regulated by NFIA in astro

293 ic regulatory roles played by neurogranin on synaptic plasticity, which provide mechanistic explanati

294 lactate supply alone rescued stress-impaired synaptic plasticity, which was blocked by inhibiting neu

295 astrocytic Ca(2+) elevation and facilitates synaptic plasticity, while activation of beta-adrenergic

296 oided by precisely balancing labile forms of synaptic plasticity with more stable forms.

297 lasticity occurs in parallel with excitatory synaptic plasticity, with the ensuing interruption of th

298 sal anxiety, and correlated with a marker of synaptic plasticity within the basolateral amygdala.

299 nce of dendritic spine remodeling in driving synaptic plasticity within the CNS.

300 t the outcome of activity-dependent forms of synaptic plasticity, yet activity-independent processes