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1 ve emotional behavior, and a deficit in BNST synaptic potentiation.
2  ongoing learning during waking leads to net synaptic potentiation.
3 , endosome recycling, AMPAR trafficking, and synaptic potentiation.
4 LP-12 secretion and blocked aldicarb-induced synaptic potentiation.
5 panied by a decrease in the initial phase of synaptic potentiation.
6 ic transmission, adjusting the threshold for synaptic potentiation.
7 ion inhibited neuroligin-mediated excitatory synaptic potentiation.
8 minished by nicotine during the induction of synaptic potentiation.
9 in the dentate gyrus during the drug-induced synaptic potentiation.
10 nit composition to occur in conjunction with synaptic potentiation.
11 tion of modular release sites to consolidate synaptic potentiation.
12 ity of CA1 pyramidal cells in the absence of synaptic potentiation.
13 ynapse stabilization after initial phases of synaptic potentiation.
14 lecules are involved in a single pathway for synaptic potentiation.
15 , uncovering the full extent of 5-HT-induced synaptic potentiation.
16 tides but exhibited a reduction of long-term synaptic potentiation.
17 term potentiation, blocked experience-driven synaptic potentiation.
18 f action potentials were necessary to induce synaptic potentiation.
19  occluded both tetanus and forskolin-induced synaptic potentiation.
20 ired for the expression of multiple forms of synaptic potentiation.
21 er ongoing learning during wake leads to net synaptic potentiation.
22 croM) enhanced EPSCs and occluded PS-induced synaptic potentiation.
23 ry and sufficient to mediate the NT3-induced synaptic potentiation.
24 n are implicated in this rapamycin-sensitive synaptic potentiation.
25 eby prevented or diminished the induction of synaptic potentiation.
26 n of BAPTA (10 mM) with adenophostin blocked synaptic potentiation.
27 yanodine receptors, prevents the NT3-induced synaptic potentiation.
28 Thus, NO is necessary for Ca(2+)/CaM-induced synaptic potentiation.
29 E with Ca(2+)/CaM blocked Ca(2+)/CaM-induced synaptic potentiation.
30 D-Fe or carboxy-PTIO blocked tetanus-induced synaptic potentiation.
31 a retrograde messenger in Ca(2+)/CaM-induced synaptic potentiation.
32 ellular) messenger during Ca(2+)/CaM-induced synaptic potentiation.
33 ory behavior, boosting of calcium entry, and synaptic potentiation.
34 mulation of AMPARs in the PSD that underlies synaptic potentiation.
35 plasticity-induced nanoscopic enrichment and synaptic potentiation.
36  AEA signals to astrocytes, inducing lateral synaptic potentiation.
37  and long-term plasticity mechanisms towards synaptic potentiation.
38 -like computations using short-term, Hebbian synaptic potentiation.
39 , both seizures and interictal spikes induce synaptic potentiation.
40 hift to a high-conductance state, leading to synaptic potentiation.
41 rafficking routes to the temporal profile of synaptic potentiation.
42 ties for synaptic tagging and maintenance of synaptic potentiation.
43 sant effect of ketamine results from delayed synaptic potentiation.
44 ished activity-induced spine enlargement and synaptic potentiation.
45 somes into the synaptic membrane, leading to synaptic potentiation.
46 e firing threshold remained unchanged during synaptic potentiation.
47 t, AMPA receptor synaptic incorporation, and synaptic potentiation.
48 s to the plasma membrane, and maintenance of synaptic potentiation.
49 ns, and a lowered threshold for induction of synaptic potentiation.
50  suppressed rCASP6 but not TNF-alpha-induced synaptic potentiation.
51 al conditions and conditions that can induce synaptic potentiation.
52 ating release of the Zn(2+) from vesicles in synaptic potentiation.
53  reduction in neuroligin-mediated excitatory synaptic potentiation.
54 pply of a mobile pool of AMPARs required for synaptic potentiation.
55 ution of the receptors, and favors long-term synaptic potentiation.
56 educing the threshold for activity-dependent synaptic potentiation.
57 amidal neurons show attenuation of long-term synaptic potentiation, a model for neuronal information
58 nucleus of the stria terminalis that undergo synaptic potentiation after attack and traumatic stress
59       These pathways undergo NMDAR-dependent synaptic potentiation after attack.
60 ly widespread changes accompanying long-term synaptic potentiation also reduced the neuron's ability
61 ment (NREM) sleep enhance previously induced synaptic potentiation, although synaptic de-potentiation
62                                    Following synaptic potentiation, AMPARs in intracellular pools con
63 lyculin A, this treatment induced a LTP-like synaptic potentiation and a persistent increase in autop
64 cellular excitability necessary for inducing synaptic potentiation and accelerates the decay of long-
65 slational control by p-eIF2alpha, initiating synaptic potentiation and addiction-related behaviors.
66  medial perforant path, which induced normal synaptic potentiation and Arc in rats with fornix lesion
67 rol were more susceptible to cocaine-induced synaptic potentiation and behavior.
68 lular studies of protein synthesis-dependent synaptic potentiation and behavioural studies of memory
69  LTP induction are necessary for stabilizing synaptic potentiation and by inference may be required f
70 ss modulates neuronal transcription to favor synaptic potentiation and counteract cellular stress, wh
71 sponsible primarily for generating long-term synaptic potentiation and depression, AMPARs are the mai
72 otransmitter release, induction of long-term synaptic potentiation and depression, and activity level
73        NMDA receptors are necessary for both synaptic potentiation and depression, but the precise lo
74  removal of GluR2-containing AMPA-Rs mediate synaptic potentiation and depression, respectively.
75 ve or instructive role on activity-dependent synaptic potentiation and depression, which depends on t
76 behavior, manifested in part by dysregulated synaptic potentiation and extracellular glutamate homeos
77  precede and may initiate this developmental synaptic potentiation and functional tuning.
78                                              Synaptic potentiation and GluR1 delivery were dissociabl
79 that the enduring cocaine-induced changes in synaptic potentiation and glutamate homeostasis are mech
80 es that are known to contribute to long-term synaptic potentiation and hypothesized to subserve learn
81 echanism will be a key target for modulating synaptic potentiation and learning.
82 uA4 in CA1 neurons conferred a PKA-dependent synaptic potentiation and LTP regardless of the developm
83 r LTP and provide a mechanistic link between synaptic potentiation and membrane remodeling during syn
84 hreshold for hippocampal-dependent long-term synaptic potentiation and memory storage in mice.
85 ependent kinase II (CaMKII) has key roles in synaptic potentiation and memory storage in neurons and
86  reward signal, broadens the time window for synaptic potentiation and modulates the outcome of hippo
87 e suppression of both previously established synaptic potentiation and old spatial memory.
88 lower induction threshold for both long-term synaptic potentiation and plasticity-induced spine growt
89 nteractions in mechanisms underlying sensory synaptic potentiation and provide insights into the path
90 ally is sufficient to recapitulate transient synaptic potentiation and reinstate cocaine seeking.
91 nstrate the sequence of events that mediates synaptic potentiation and reinstated cocaine seeking ind
92 es extend to the mechanisms of expression of synaptic potentiation and result in distinct patterns of
93  evidence that waking is associated with net synaptic potentiation and sleep with depression, direct
94  accompanied by impairments in CA1 long-term synaptic potentiation and spatial memory consolidation.
95 ng evidence that BDNF plays a causal role in synaptic potentiation, and exogenous application of BDNF
96 ive-like behaviors in mice, deficits in BNST synaptic potentiation, and increased activity in BNST-CR
97 tion-induced LTP, both spine enlargement and synaptic potentiation are transient.
98 tch recordings suggested that the deficit in synaptic potentiation arose from shunting of dendritic E
99 AEPs after drug elimination, consistent with synaptic potentiation as a mechanism for antidepressant
100                   Interestingly, reversal of synaptic potentiation as well as de novo synaptic depres
101     In particular, whisker experience drives synaptic potentiation as well as the incorporation of CP
102  wakefulness, many cortical circuits undergo synaptic potentiation, as evidenced by the widespread in
103 e concurrent stresses resulting from loss of synaptic potentiation associated with disrupted structur
104 n-derived neurotrophic factor (BDNF) induces synaptic potentiation at both neuromuscular junctions (N
105 rsting activity is necessary for associative synaptic potentiation at CA1 excitatory synapses in adul
106                  These data demonstrate that synaptic potentiation at CA1 synapses is more complex th
107                             Ketamine-induced synaptic potentiation at CA3-CA1 synapses has been propo
108 GluR1 AMPA receptors underlies many forms of synaptic potentiation at glutamatergic synapses througho
109 f beta2-adrenoceptors promoted STD long-term synaptic potentiation at mouse hippocampal excitatory sy
110                                              Synaptic potentiation at PF-PC synapses, induced by the
111 n also significantly reduce the magnitude of synaptic potentiation at SC-CA1 synapses.
112                    We find that aSyn induces synaptic potentiation at the larval neuromuscular juncti
113 dence for an unusual mechanism that mediates synaptic potentiation at the neuromuscular junction (NMJ
114 tes for BDNF-induced Ca2+ elevation and full synaptic potentiation at the NMJ, suggesting a previousl
115 cluding GABA receptors, glutamate signaling, synaptic potentiation, axon guidance, clathrin-mediated
116  learning-induced enhancement limits further synaptic potentiation, but not synaptic depression.
117 fore, the contribution of CLC-3 is to reduce synaptic potentiation by approximately 40%.
118 tic cytosolic Ca2+ ([Ca2+]i) accompanied the synaptic potentiation by BDNF, whereas no change in [Ca2
119  by a cocaine-priming injection, coordinated synaptic potentiation by both NAcore afferents is necess
120 eas no change in [Ca2+]i was observed during synaptic potentiation by CNTF.
121 on, RAP also blocked the enhancing effect of synaptic potentiation by exogenous tPA in hippocampal sl
122 t motor learning induces widespread cortical synaptic potentiation by increasing the net trafficking
123 sGRF2's Rac-GEF activity to be essential for synaptic potentiation by using a molecular replacement s
124 ling models have more recently proposed that synaptic potentiation can occur by the recruitment of ad
125                    Together, while immediate synaptic potentiation capitalizes on available material,
126 nd sufficient to drive spine enlargement and synaptic potentiation concomitantly.
127                                              Synaptic potentiation correlates with an animal's helple
128 Here, we found that ketamine-induced CA3-CA1 synaptic potentiation could be augmented by transiently
129 se M1 slices that DCS induces a long-lasting synaptic potentiation (DCS-LTP), which is polarity speci
130 ne neurons are necessary for cocaine-induced synaptic potentiation, demonstrating that cell type-spec
131 ude kinase and calcium signaling involved in synaptic potentiation, demonstrating that sex is an impo
132                                       There, synaptic potentiation depends on the availability of vis
133 ar signaling that initiates estrogen-induced synaptic potentiation differs between the sexes.
134 on selectively impaired spine plasticity and synaptic potentiation, disrupting neuronal competition f
135 mature death) and showed impaired short-term synaptic potentiation; downregulation of photoreceptor-s
136 ram slow wave activity during sleep reflects synaptic potentiation during wake, and that its homeosta
137                   Another possibility is net synaptic potentiation during wake: stronger coupling amo
138 ARs, CamKII and GSK3beta are consistent with synaptic potentiation during wakefulness and depression
139 that sleep SWA homeostasis may be related to synaptic potentiation during wakefulness.
140                  It has been postulated that synaptic potentiation during waking is offset by a homoe
141  proteasome inhibition on the early phase of synaptic potentiation (E-LTP) induced by theta-burst sti
142 als, but clustered in bursts, induced robust synaptic potentiation (EPSP amplitude 163%; P < 0.01, St
143  molecular mechanism underlying BDNF-induced synaptic potentiation, especially the regulation of Ca2+
144 ted that D1-type receptor activation enabled synaptic potentiation even when postsynaptic activity pr
145 y-PTIO) did not attenuate Ca(2+)/CaM-induced synaptic potentiation, even though MGD-Fe or carboxy-PTI
146 spaced training regimen designed to maximize synaptic potentiation facilitates recognition memory in
147  begins in the axon, but which can influence synaptic potentiation following active backpropagation i
148 ipation of protein kinase Mzeta (PKMzeta) in synaptic potentiation following cocaine exposure.
149 re, the relative effect of a single spike on synaptic potentiation grows as radicalN.
150 aptic plasticity (specifically, long-lasting synaptic potentiation) have been demonstrated to accompa
151 lated by endogenous estradiol, which favored synaptic potentiation in a GluN2B-dependent manner.
152 activity--postsynaptic bursting--accompanies synaptic potentiation in adults.
153  and VAMP7 occludes NMDAR antagonist-induced synaptic potentiation in an intact circuit, confirming t
154 ng the previous waking period, of widespread synaptic potentiation in cortical and subcortical areas.
155 caine-elicited, but not the stress-elicited, synaptic potentiation in DA neurons was blocked by a D1-
156 les leading to spatially structured forms of synaptic potentiation in dendrites, we explored plastici
157 ceptor beta (ERbeta) but not ERalpha rescued synaptic potentiation in diestrus mice by enhancing GluN
158 ue method for inducing spine enlargement and synaptic potentiation in dispersed hippocampal neurons,
159 Inhibition of cpAMPARs eliminated E2-induced synaptic potentiation in females, whereas some synapses
160  FK-506 or an autoinhibitory peptide induced synaptic potentiation in hippocampal slices, which occlu
161 aining integrins are required for persistent synaptic potentiation in hippocampus and regulate hippoc
162 d Ca(2+) channels were involved in mediating synaptic potentiation in oriens, whereas NMDA and adenos
163 Hebbian plasticity to facilitate associative synaptic potentiation in prefrontal excitatory circuits.
164 d activation of NMDA receptors, facilitating synaptic potentiation in response to stimulation at 10-1
165 the balance from synaptic depression towards synaptic potentiation in sleep-promoting neurons underli
166 get of rapamycin kinase activity, suppressed synaptic potentiation in slices from fear-conditioned ra
167 mature spine morphology but can also enhance synaptic potentiation in some cases.
168  of PS-, adenophostin- or Ca(2+)-CaM-induced synaptic potentiation in SP non-pyramidal neurons increa
169  that up-regulated GluN2A, GluN2B, and rapid synaptic potentiation in the accumbens contribute to cue
170 eftriaxone also reversed the cocaine-induced synaptic potentiation in the accumbens core, evidenced b
171 suggest that 11beta-HSD1 deficiency enhances synaptic potentiation in the aged hippocampus and this m
172     CREB activation by fear conditioning and synaptic potentiation in the amygdala and cortical areas
173 rholoenzyme phosphorylation CaMKII maintains synaptic potentiation in the face of CaMKII protein turn
174 tly, the high frequency stimulation-mediated synaptic potentiation in the hippocampal CA1 region was
175                           Moreover, impaired synaptic potentiation in the hippocampal CA1 subregion w
176 alcium sources required for acute E2-induced synaptic potentiation in the hippocampus of each sex and
177 n early, projection-specific, cocaine-evoked synaptic potentiation in the LHb that may represent a pe
178    Consistent with the substance P-dependent synaptic potentiation in the LIPN, the NK1R in the IPN i
179 ne receptor stimulation is necessary for the synaptic potentiation in the NAcore during cocaine-induc
180 as required for contextual fear learning and synaptic potentiation in the vCA1-BA pathway.
181 hat CREB-mediated pathways may contribute to synaptic potentiation in these cells.
182 g changes implicated previously in models of synaptic potentiation in vitro.
183 -dependent long-term potentiation (LTP)-like synaptic potentiation in vivo may be an effective antide
184                              Such persistent synaptic potentiation in VTA DA neurons may represent a
185 rate latent sex differences in mechanisms of synaptic potentiation in which distinct molecular signal
186                        Specifically, network synaptic potentiation increases dramatically with high c
187 a(2+) chelators blocks LTD and drives strong synaptic potentiation, indicating that basal Ca(2+) leve
188 but that PKC activators produce a reversible synaptic potentiation, indicating that PKC activation is
189 , ceftriaxone did not reverse stress-induced synaptic potentiation, indicating that this effect of st
190                                 In parallel, synaptic potentiation induced by a single tetanic stimul
191                    Rapamycin also blocks the synaptic potentiation induced by brain-derived neurotrop
192                                              Synaptic potentiation induced by brain-derived neurotrop
193                                              Synaptic potentiation induced by FK-506 was significantl
194  way that is mechanistically very similar to synaptic potentiation induced by oxytocin, Avpr1b agonis
195       T-type current blockade also prevented synaptic potentiation induced by postsynaptic action pot
196 nant negative TrkB.T1 inhibited two forms of synaptic potentiation induced by the neurotrophin brain-
197 also sharpened the STDP curve, with reliable synaptic potentiation induced only when EPSPs and action
198 as necessary for reinstatement, it inhibited synaptic potentiation initiated by an acute cocaine inje
199  stimulation that normally induces long-term synaptic potentiation instead promoted development of ca
200         Our study suggests that BDNF-induced synaptic potentiation involves coordinated presynaptic a
201 mediated glutamatergic neurotransmission via synaptic potentiation is central to the antidepressant e
202      The results show that CP-AMPAR-mediated synaptic potentiation is common in hippocampal interneur
203 and translation-dependent phase of long-term synaptic potentiation (L-LTP) at the Schaffer collateral
204 PKR increases the late phase of long-lasting synaptic potentiation (L-LTP) in hippocampal slices.
205 tions in synaptic strength such as long-term synaptic potentiation (LTP) and depression (LTD) underli
206 xplain the selective activation of long-term synaptic potentiation (LTP) and long-term depression (LT
207  the UPS to mediate the effects on long-term synaptic potentiation (LTP) has not been investigated.
208      In addition, social interaction induces synaptic potentiation (LTP) in the ventral tegmental are
209                                    Long-term synaptic potentiation (LTP) is induced by synaptic activ
210 es of synaptic contacts) and block long-term synaptic potentiation (LTP), a form of synaptic plastici
211 e, we explored the hypothesis that long-term synaptic potentiation (LTP), potentially able to minimiz
212  selective loss of GluR1-dependent long-term synaptic potentiation (LTP).
213 MPAR) currents are associated with long-term synaptic potentiation (LTP).
214 d genetic manipulations to examine long-term synaptic potentiation (LTP)/long-term synaptic depressio
215           At the cellular level, reversal of synaptic potentiation may be important for neurons to ac
216 in neurons expressing PI3K* elicits a marked synaptic potentiation, mimicking the NT3 effect.
217 ds in hippocampal area CA1 are produced by a synaptic potentiation notably different from Hebbian pla
218                 Because the learning-induced synaptic potentiation occluded HFS-induced LTP, we concl
219 is complex is not critical for mechanisms of synaptic potentiation occurring in immature animals.
220                                          The synaptic potentiation occurs postsynaptically and probab
221     Here, we investigated whether E2-induced synaptic potentiation occurs via presynaptic and/or post
222                              WGs also showed synaptic potentiation of inhibitory inputs both at the m
223 We also found that disruption of PNNs allows synaptic potentiation of normally plasticity-resistant e
224 textual fear memory retrieval, manifested by synaptic potentiation of PV interneuron-mediated inhibit
225 ith AMPAR surface diffusion markedly impairs synaptic potentiation of Schaffer collaterals and commis
226  nicotine directly cause in vivo hippocampal synaptic potentiation of the kind that underlies learnin
227                                              Synaptic potentiation of the MeApv-VmH and MeApv-BNST pa
228 disrupted afferent inputs to CA2 and enabled synaptic potentiation of the normally LTP-resistant syna
229 ered to freely moving mice induces long-term synaptic potentiation of the perforant path connection t
230  of that association is the nicotine-induced synaptic potentiation of the perforant path that was fou
231 adenylyl cyclase activator forskolin induced synaptic potentiation of this pathway that also was asso
232  addition, Abeta oligomers blocked long-term synaptic potentiation only in neurons that expressed Glu
233    Indeed, we observed D1 receptor-dependent synaptic potentiation only when odor-like bursts and opt
234   Sensory experience preferentially produced synaptic potentiation onto nearby dendritic synapses.
235 ers associated with excitatory or inhibitory synaptic potentiation onto principal cells, respectively
236 y an impairment in fear-conditioning-induced synaptic potentiation onto somatostatin-expressing (SOM(
237                    In particular, preventing synaptic potentiation onto somatostatin-positive neurons
238  hippocampal neurons, induction of long-term synaptic potentiation or depression by repetitive synapt
239 ro amygdala slice can induce either enduring synaptic potentiation or depression, depending on whethe
240 ng cleavable or uncleavable pro-BDNF elicits synaptic potentiation or depression, respectively.
241 cling processes in basal conditions and upon synaptic potentiation or depression, spatially and tempo
242 aptic neurotransmitter release, resulting in synaptic potentiation or depression.
243 , and PKC pathways and their contribution to synaptic potentiation or depression.
244  regulated during, other forms of short-term synaptic potentiation or long-lasting synaptic depressio
245 deas about cellular consolidation concerning synaptic potentiation, particularly the relationship bet
246 predictions of dynamic value assignment, the synaptic potentiation persisted after the phasic dopamin
247                                    Clustered synaptic potentiation produced by experience could bind
248  plasticity provides a natural constraint on synaptic potentiation, regulating the inherent instabili
249             Some forms of activity-dependent synaptic potentiation require the activation of postsyna
250                             The induction of synaptic potentiation required excitation of DA neurons
251 eurons in mouse barrel cortex, we found that synaptic potentiation requires postsynaptic, but not pre
252                             This cAMP-driven synaptic potentiation requires the activation of both pr
253 a sustained, alpha7*nAChRs-mediated phase of synaptic potentiation seen in comparable WT/WT circuits
254 berrant appearance of spike-timing-dependent synaptic potentiation (STDP) in PFC slices derived from
255 sion loci of the hippocampal and neocortical synaptic potentiation studies we examined.
256 d of inactivity can reduce the threshold for synaptic potentiation such that subsequent visual experi
257 r with the finding that mature BDNF promotes synaptic potentiation, suggest a bidirectional regulatio
258 s correlates with the induction of transient synaptic potentiation (t-SP) at glutamatergic synapses i
259 s correlated with the induction of transient synaptic potentiation (t-SP) at glutamatergic synapses o
260             Cue-induced transient excitatory synaptic potentiation (t-SP) induced in the nucleus accu
261  cue-induced drug seeking requires transient synaptic potentiation (t-SP) of cortical glutamatergic s
262 ic spikes participate generally in a form of synaptic potentiation that does not require postsynaptic
263  present evidence for localized BDNF-induced synaptic potentiation that is accompanied by spatially r
264 ocretin neurons undergo experience-dependent synaptic potentiation that is distinct from that reporte
265          Mature BDNF facilitates hippocampal synaptic potentiation through TrkB.
266 tion contributed to nicotine-induced in vivo synaptic potentiation, thus, likely contributed to drug-
267 erve terminals prevented the tetanus-induced synaptic potentiation (TISP).
268 estriction of putative neurotrophin-mediated synaptic potentiation to active synapses.
269 tatic mechanism that can prevent the runaway synaptic potentiation to which Hebbian networks are vuln
270 sized that increases in cortical SWA reflect synaptic potentiation triggered by learning.
271 related changes and prevented NMDAR-mediated synaptic potentiation triggered by MOR activation.
272 ring, which was otherwise too weak to induce synaptic potentiation, triggered a long-lasting increase
273 interneurons received passive propagation of synaptic potentiation via the recurrent collaterals of C
274 om spontaneously recycling vesicles triggers synaptic potentiation via the same pathway as NMDAR bloc
275                          This enhancement of synaptic potentiation was absent in mice lacking Egr1.
276                                         This synaptic potentiation was associated with an increase in
277 learning were absent in irradiated mice, and synaptic potentiation was enhanced.
278                               FK-506-induced synaptic potentiation was expressed in adult but not you
279                                      Whereas synaptic potentiation was linked to learned helplessness
280                                              Synaptic potentiation was normal in these associational
281                           A chemical form of synaptic potentiation was produced with a brief bath app
282 lucidate mechanisms underlying CaN-inhibited synaptic potentiation, we co-injected certain agents aff
283 sex difference in the requirement for PKA in synaptic potentiation, we tested how PKA inhibition affe
284           The biologically relevant rules of synaptic potentiation were investigated in hippocampal s
285 y, neural pathways, namely axon-guidance and synaptic potentiation, were also over-represented in MS.
286 etwork firing patterns alter overall network synaptic potentiation when synaptic strengths evolve thr
287 e action potentials was sufficient to induce synaptic potentiation when the presynaptic activity prec
288 akefulness appears to be associated with net synaptic potentiation, whereas sleep may favor global sy
289 to hippocampal CA1 pyramidal neurons induces synaptic potentiation, which can occlude tetanus-induced
290 II 15-30 s after the BTSP protocol inhibited synaptic potentiation, which indicated that DDSC is an e
291  but it has been proposed to sustain Hebbian synaptic potentiation, which is a key component of memor
292  addition, Ca(2+)-CaM (40:10 microM) induced synaptic potentiation, which occluded PS-induced potenti
293 ted AMPA receptor (AMPAR) trafficking during synaptic potentiation, which was distinct from actin's s
294 ly reduced the magnitude of timing-dependent synaptic potentiation while leaving the magnitude of tim
295 c Ca2+ elevation and restricted BDNF-induced synaptic potentiation, while knockdown of the TrkB recep
296 cy (0.2 Hz), which does not by itself induce synaptic potentiation, will produce long-lasting synapti
297                                    Thus, the synaptic potentiation within the BNST in response to ILC
298 ocking postsynaptic spiking diminished basal synaptic potentiation without affecting apical plasticit
299 f carboxy-PTIO alone blocked tetanus-induced synaptic potentiation without affecting basal synaptic t
300 on in cellular excitability in parallel with synaptic potentiation would be a negative feedback mecha

 
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