ライフサイエンスコーパス: synovial

1 ology or highly elevated leukocyte levels in synovial aspiration.

2 chains and Fab-N-linked glycosylation in RA synovial B cell clones reactive to NETs and NET-derived

3 hus, SHM in the IgVH and/or VL regions of RA synovial B cells is necessary for the immunoreactivity t

4 non-avian dinosaurs independently evolved a synovial ball-and-socket joint.

5 ression of cholesterol biosynthesis genes in synovial biopsies of predisposed individuals.

6 enotypic, spatial and functional analyses of synovial biopsy fluorescent activated cell sorted STMs,

7 anscriptomic analysis from ultrasound-guided synovial biopsy of inflamed joints in a well characteriz

8 Fas receptor induces apoptosis of synovial bone and cartilage progenitor populations and p

9 Proportions of synovial bone and cartilage progenitors were assessed by

10 Synovial bone and cartilage progenitors were purified by

11 In the synovial cavity, a state of nonresolving inflammation is

12 element lined with articular cartilage and a synovial cavity, and we demonstrate that this response r

13 Transcriptome analyses find these expanded synovial CD8 T cells to express cycling, activation, tis

14 eceptor CXCR3 is upregulated in the expanded synovial CD8 T cells, while two CXCR3 ligands, CXCL9 and

15 Data from a study with SW982 synovial cell line and primary human synoviocytes showed

16 iocytes and 26,192 chondrocytes: 12 distinct synovial cell types and 7 distinct articular chondrocyte

17 cells, 55% of the cytokines were produced by synovial cells (39% exclusive to synoviocytes and not ex

18 rylated VEGFR2 in articular chondrocytes and synovial cells and reduce levels of phosphorylated VEGFR

19 Of significance, Snail is overexpressed in synovial cells and tissues of CIA rats and RA patients,

20 induces extracellular matrix degradation in synovial cells by repressing PTEN, resulting in increase

21 r matrix-degrading invadosomal structures by synovial cells from collagen-induced arthritis (CIA) rat

22 significantly more pronounced in the primary synovial cells from rheumatoid arthritis patients than t

23 of functional cardiomyocytes from pathogenic synovial cells in RA patients through iPSC reprogramming

24 with catabolic processes in chondrocytes and synovial cells.

25 uired for the prodestructive phenotype of RA synovial cells.

26 odestructive invadosome-forming phenotype of synovial cells.

27 Our results suggest that synovial CG may take part in the degradation of lubricin

28 er expression of PU.1 at B-cell level in the synovial compartment.

29 e explanation for arthritis and (2) negative synovial cultures (if obtained).

30 of meniscal, ligamentous, cartilaginous, and synovial disorders within the knee that are commonly enc

31 In 44% of the patients, synovial effusion was presented.

32 e marrow and soft tissue oedema, presence of synovial effusion, muscular atrophy in the affected extr

33 mice and showed that elevated serum LTB4 and synovial expression of 5-lipoxygenase correlated with in

34 Synovial expression of C5orf30 is inversely correlated w

35 ion in both primary RA SF and the rheumatoid synovial fibroblast cell line, MH7A.

36 flammatory response, mediated by chondrocyte-synovial fibroblast cross-talk, was enhanced by the obes

37 oth transient and longer duration changes in synovial fibroblast membrane potential.

38 nal diversity translates into joint-specific synovial fibroblast phenotypes with distinct adhesive, p

39 of ion channels that are expressed in human synovial fibroblast preparations have begun to provide i

40 sduction was negatively regulated by heme in synovial fibroblast-like synoviocytes from rheumatoid ar

41 itro, exRNA (150-5000 nt) was released by RA synovial fibroblasts (RASF) under hypoxic conditions but

42 itis (RA) is linked to functional changes in synovial fibroblasts (SF) and local infiltration of T ly

43 levels in relation to RANKL expression in RA synovial fibroblasts (SF) and the development of bone er

44 the TNF-alpha signaling pathway in human RA synovial fibroblasts (SFs).

45 howed opposite effects (e.g., osteoarthritis synovial fibroblasts [SF]; GF(-) versus GF(+): 10.7- ver

46 of the melanocortin type 1 receptor (MC(1)), synovial fibroblasts acquire a senescence phenotype char

47 ified by real-time quantitative PCR in human synovial fibroblasts and murine mesenchymal stem cells.

48 he joint-specific origins of mouse and human synovial fibroblasts and synovial tissues.

49 herefore studied the production of ADAMTS by synovial fibroblasts and their contribution to cartilage

50 system, and the availability of adult human synovial fibroblasts are likely to provide new pathophys

51 Our results indicate that synovial fibroblasts exhibit a positional identity that

52 During osteoarthritis, synovial fibroblasts exposed to anomalous mechanical for

53 Here we show transcriptomic differences in synovial fibroblasts from different joint locations and

54 However, synovial fibroblasts from obese OA patients were found t

55 1 and bone morphogenetic protein (BMP)-2, in synovial fibroblasts from RA patients.

56 (hyaluronan and lubricin) and cytokines from synovial fibroblasts have been identified.

57 circulating microparticles, which activated synovial fibroblasts in an IL-1-dependent manner.

58 mediators and induced the repair response of synovial fibroblasts in vitro.

59 Using synovial fibroblasts isolated from RA joints, we found t

60 and BMP-2 decreased IL-34 expression in the synovial fibroblasts or in murine mesenchymal stem cells

61 Therefore, synovial fibroblasts provide the biochemical tools to th

62 nces the cross-talk between chondrocytes and synovial fibroblasts via raised levels of the pro-inflam

63 hanism demonstrated that IL-6 secretion from synovial fibroblasts was induced by chondrocyte-derived

64 Primary human synovial fibroblasts were also examined using flow cytom

65 tro experiments with human keratinocytes and synovial fibroblasts were conducted.

66 ocytes/macrophages, B and T lymphocytes, and synovial fibroblasts, and TLR-induced MIF transcription

67 tein coupled receptor promotes senescence in synovial fibroblasts, enabling amelioration of joint inf

68 highest expression found in macrophages and synovial fibroblasts.

69 al properties of key cells in RA, especially synovial fibroblasts.

70 potent inhibitors of IL-34 expression in RA synovial fibroblasts.

71 these cells shared features of inflammatory synovial fibroblasts.

72 tch target genes are markedly upregulated in synovial fibroblasts.

73 response of BMNC to normal (SF) and inflamed synovial fluid (ISF).

74 The concentration of histamine in synovial fluid (SF) and sera in patients with RA was mea

75 lly bound to C4d were identified from pooled synovial fluid (SF) from four rheumatoid arthritis (RA)

76 ostic utility of the SACOL0688 antigen using synovial fluid (SF) from humans with orthopedic implant

77 The infrapatellar fat pad (FP) and synovial fluid (SF) in the knee serve as reservoirs of m

78 CG) may participate in degrading lubricin in synovial fluid (SF).

79 ound that HA contributes to the formation of synovial fluid aggregates, and HysA can disrupt aggregat

80 endation: ACP recommends that clinicians use synovial fluid analysis when clinical judgment indicates

81 showed gross and analytical improvements in synovial fluid and synovial membrane, with increasing re

82 elevated concentration of PTX3 in plasma and synovial fluid as compared with healthy and osteoarthrit

83 ensated polarized light microscope (CPLM) in synovial fluid aspirated from the patient's joint.

84 eived antibiotics within the month preceding synovial fluid aspiration (48 of whom had PJI), PCR pane

85 vo in monocytes from healthy individuals and synovial fluid cells from RA patients.

86 The cocultures of platelets with synovial fluid cells from rheumatoid arthritis patients

87 ocytes) and their presence in osteoarthritic synovial fluid confirmed.

88 radiographic tests, such as serum urate and synovial fluid crystal analysis and radiographic or ultr

89 piration (48 of whom had PJI), PCR panel and synovial fluid culture sensitivities were 64.5% and 85.4

90 Only 40% had positive synovial fluid culture.

91 Molecular profiling of synovial fluid derived exosomal miRNAs may increase our

92 In conclusion, synovial fluid exosomal miRNA content is altered in pati

93 The synovial fluid exosomes share similar characteristics (s

94 ling, and reports newly identified serum and synovial fluid FAs as predictive biomarkers of OA in obe

95 scent sera from 91 EM patients, in serum and synovial fluid from 141 LA patients, and in serum from 5

96 RNase activity was increased in synovial fluid from RA and OA patients compared with pso

97 ated SOX5 levels were higher in synovium and synovial fluid from RA compared to osteoarthritis patien

98 cellular PLA(2) activity was detected in the synovial fluid from rheumatoid arthritis and gout patien

99 The synovial fluid glycoprotein lubricin (also known as prot

100 Lubricin is a mucinous, synovial fluid glycoprotein that enables near frictionle

101 , with increasing regulatory macrophages and synovial fluid IL-10 concentrations compared with saline

102 OA synovial fluid impacted U3 snoRNA expression by affectin

103 indicating that ADAMTS-5 cleavage occurs in synovial fluid in arthritis.

104 o determine the associations among serum and synovial fluid lipid levels with OA, synovitis, adipokin

105 Both increased and decreased synovial fluid lubricin concentrations have been reporte

106 Synovial fluid lubricin concentrations were correlated w

107 Synovial fluid lubricin concentrations were nearly 16-fo

108 Increased synovial fluid lubricin has been observed following intr

109 RA synovial tissue and synovial fluid macrophages expressed CCR7, which was inc

110 lar traps (NETs) are found abundantly in the synovial fluid of gout patients.

111 SPADE was used to analyze EVs present in the synovial fluid of patients with inflammatory arthritis.

112 Finally, we show that synovial fluid of patients with PJI contains elevated am

113 d whether IgA immune complexes in plasma and synovial fluid of RA patients activate neutrophils.

114 plex with complement component C4d in pooled synovial fluid of rheumatoid arthritis (RA) patients.

115 We detected ITCs in the synovial fluid of the high glucosinolate group, but not

116 d the greater levels of IL-6 detected in the synovial fluid of the obese OA patients.

117 ion, and one with repeated aspiration of the synovial fluid only.

118 as obtained with gels containing only bovine synovial fluid or human serum.

119 Sensitivities of the synovial fluid PCR panel and culture were 55.6% and 76.1

120 We also detect PG(Bb) in 94% of synovial fluid samples (32 of 34) from patients with LA,

121 ic PCR assay panel using 284 prosthetic knee synovial fluid samples collected from patients presentin

122 These same synovial fluid samples contain proinflammatory cytokines

123 Here, clinical records, radiographs, and synovial fluid samples from 30 dogs that sustained RCCL

124 Proteomic analysis of synovial fluid showed significantly distinct profiles be

125 g 379 retrospective, remnant whole-blood and synovial fluid specimens previously submitted to Associa

126 We quantitated NET levels in gout synovial fluid supernatants and detected enzymatically a

127 PG(Bb), which is significantly higher in the synovial fluid than in the serum of the same patient.

128 ated mediators were often >10-fold higher in synovial fluid than serum.

129 Synovial fluid was collected at 24, 96, and 144 h for cy

130 flammatory cells involved in osteoarthritis, synovial fluid was collected early after disease inducti

131 Serum and synovial fluid were collected for lipidomic and adipokin

132 by lubricin (LUB), a component of mammalian synovial fluid with excellent antifouling properties, th

133 e and (iv) exposure to body fluids (blood or synovial fluid) on release kinetics and efficacy of anti

134 les, six clinical specimens (five blood, one synovial fluid) yielded an atypical oppA1 PCR product, b

135 Human Tregs in blood, tonsil, synovial fluid, colon, and lung tissues did not express

136 Of the galectin family members present in synovial fluid, we find that galectin-3 is a specific, h

137 table in fetal bovine serum, human serum and synovial fluid, with varying levels of instability obser

138 Synovial fluid-derived monocytes from patients with spon

139 aracterization of exosomes miRNAs from human synovial fluid.

140 gands, CXCL9 and CXCL10, are elevated in PsA synovial fluid.

141 tics and were pre-coated with human blood or synovial fluid.

142 tic tool had low sensitivity when applied to synovial fluid.

143 gradation and release of components into the synovial fluid.

144 ricant and chondroprotective glycoprotein in synovial fluid.

145 cytes using interleukin-1 and osteoarthritic synovial fluid.

146 OA synovial fluids (SF) stimulated TLR2 and TLR4 receptors

147 CXCL10/CXCR3 axis, with CXCL10 increasing in synovial fluids after injury and Cxcr3(-/-) mice being p

148 d BMP-2 productions were measured in patient synovial fluids by enzyme-linked immunosorbent assay.

149 s with or without IL-1beta or osteoarthritic synovial fluids for 48 h.

150 L-34, TGF-beta1, and BMP-2 were expressed in synovial fluids from RA patients.

151 Succinate is abundant in synovial fluids from rheumatoid arthritis (RA) patients,

152 cesses and mediate leukocyte adhesion in the synovial fluids of arthritis patients and protect agains

153 d the effects of IL-1beta and osteoarthritic synovial fluids on anabolic gene expression and increase

154 lated with the total leukocyte counts in the synovial fluids.

155 Macrophages promote synovial health but become inflammatory when their regul

156 artilage injury, Gdf5-lineage cells underpin synovial hyperplasia through proliferation, are recruite

157 e to total articular cartilage (CC/TAC), and synovial hyperplasia with increased lining cells was fou

158 olymer effectively inhibited joint swelling, synovial hyperplasia, and bone destruction in collagen-i

159 ex non-cartilaginous OA conditions including synovial hyperplasia, osteophyte outgrowth and subchondr

160 tervertebral disc degeneration and extensive synovial hypertrophy and loss of articular cartilage in

161 Cordycepin reduced pain behaviour, synovial inflammation and joint pathology in both OA mod

162 s a wide group of diseases, characterized by synovial inflammation and joint tissue damage.

163 le of LTB4 and its receptor LTB4R1 (BLT1) in synovial inflammation and osteoclast differentiation.

164 Rheumatoid arthritis is characterised by synovial inflammation and proliferation of fibroblast-li

165 Scores for synovial inflammation at DWI and contrast-enhanced MRI a

166 L-1beta-activated signaling pathways causing synovial inflammation in rheumatoid arthritis (RA).

167 replace contrast-enhanced MRI for imaging of synovial inflammation in this patient group.

168 w that LTB4 and its receptor BLT1 exacerbate synovial inflammation in vivo and bone resorption in vit

169 Synovial inflammation is a central feature of osteoarthr

170 Non-resolved persistent macrophage-mediated synovial inflammation is considered as one of the main d

171 s (OA) pain is significantly associated with synovial inflammation.

172 icrotrauma, remodeling of joint tissues, and synovial inflammation.

173 s a chronic autoimmune disorder resulting in synovial inflammation.

174 iated by these cells participate directly in synovial inflammation.

175 contrast-enhanced MRI regarding detection of synovial inflammation.

176 s an autoimmune disease resulting in chronic synovial inflammation.

177 f osteoarthritis includes the involvement of synovial inflammation.

178 t material-enhanced MRI for the detection of synovial inflammation.

179 ward M1-like macrophages that contributed to synovial inflammation.

180 ncreased CGRP(+), TH(+), and GAP-43(+) fiber synovial innervation.

181 munohistochemistry, we found that the lining synovial intima of the stifle (knee) is a target for acu

182 duction of cytokines and chemokines in human synovial intimal resident fibroblast-like synoviocytes (

183 physiology (cell migration in the context of synovial joint autoimmune disease and injury) and tissue

184 erzone formation and fate diversification of synovial joint constituents.

185 Synovial joint development begins with the formation of

186 programs and cell interactions that regulate synovial joint development.

187 tand the aetiology and possible treatment of synovial joint disease.

188 teract in interzone cell differentiation and synovial joint morphogenesis.

189 e, the TMJ has two distinct functions as the synovial joint of the craniofacial complex and also as t

190 The temporomandibular joint (TMJ) is the synovial joint of the craniofacial complex and is subjec

191 rformed single cell RNA-Seq analysis of 7329 synovial joint progenitor cells from the developing muri

192 wound with BMP9 stimulates regeneration of a synovial joint that forms an articulation with the stump

193 t reside in articular cartilage and line the synovial joint.

194 tion enzyme leukocyte esterase (LE) in human synovial (joint) fluid and urine.

195 Connective tissues within the synovial joints are characterized by their dense extrace

196 Synovial joints are the lubricated connections between t

197 Our data support lubricated synovial joints evolving much earlier than currently acc

198 It is assumed that synovial joints first evolved as vertebrates came to lan

199 The stem cells that safeguard synovial joints in adulthood are undefined.

200 Because synovial joints include immune privileged sites, these f

201 erized by the loss of articular cartilage in synovial joints through a process of extracellular matri

202 s (CL) to locally deplete macrophages in the synovial joints to examine the role of macrophages in th

203 ue that enables low friction articulation of synovial joints.

204 orm an internal immunological barrier at the synovial lining and physically seclude the joint.

205 onal ablation in Gdf5-lineage cells prevents synovial lining hyperplasia and decreases contribution o

206 ynovium, exRNA was detectable only in the RA synovial lining layer, whereas extracellular DNA was det

207 -) destructive fibroblasts restricted to the synovial lining layer.

208 of articular cartilage, inflammation of the synovial lining, and changes to the subchondral bone.

209 self-organize three-dimensionally to form a synovial lining-like layer.

210 ether and how neutrophils can regulate their synovial localization in the disease.

211 e osteoarthritis; however, it is unknown how synovial lubricin is affected by knee-destabilizing inju

212 ectin-3 and the galectin-3 interactions with synovial lubricin were found to be decreased in late-sta

213 est that intra-articular BMNC could increase synovial macrophage counts, potentiating the macrophage-

214 unexpected functional diversification among synovial macrophages and have important implications for

215 ly regulates diverse pathologic processes in synovial macrophages including the cell cycle, apoptosis

216 genes was expressed in rheumatoid arthritis synovial macrophages, confirming their expression under

217 (CRIg), found on tissue macrophages such as synovial macrophages, has promising potential to visuali

218 Thus, a cellular cascade of synovial macrophages, NK cells, and neutrophils mediates

219 ighted imaging (DWI) can depict the inflamed synovial membrane in arthritis.

220 rget genes in a dose-dependent manner in the synovial membrane of TMJ.

221 nalytical improvements in synovial fluid and synovial membrane, with increasing regulatory macrophage

222 pha, interleukin (IL)-1beta, and IL-6 in the synovial membrane.

223 Finally, human synovial MSCs transduced with Bmp7 display morphogenetic

224 spontaneously forming NETs from JIA patient synovial neutrophils, and DEK-targeted aptamers reduce N

225 Synovial NK cell production of GM-CSF is IL-18-dependent

226 sing GM-CSF fate reporter mice, we show that synovial NK cells produce GM-CSF in autoantibody-mediate

227 Synovial NK cells promote a neutrophilic inflammatory ce

228 intraobserver reliability for assessment of synovial patterns at MR imaging.

229 c resonance (MR) imaging for differentiating synovial patterns in patients with total knee arthroplas

230 Selected anti-NET RA-rmAbs derived from synovial RA CD19(+) B cells were subjected to overlap-PC

231 Furthermore, synovial samples from human patients with rheumatoid art

232 Herein, a role of polyadenylation in OA synovial samples was investigated, and the potential of

233 Synovial sarcoma (SS) is a rare sarcoma driven by a tran

234 Synovial sarcoma (SS) is an aggressive soft-tissue malig

235 Synovial sarcoma (SS) is an aggressive soft-tissue sarco

236 liposarcoma (HG-MLPS); leiomyosarcoma (LMS), synovial sarcoma (SS), malignant peripheral nerve sheath

237 ique dependency on GBAF (ncBAF) complexes in synovial sarcoma and malignant rhabdoid tumors, both of

238 cell growth and migration across a series of synovial sarcoma cell lines.

239 tified selective cytotoxicity of EA in human synovial sarcoma cells (SW982 cells) and investigated th

240 at EA has a potent cytotoxic effect on human synovial sarcoma cells which is mediated by heteromeric

241 enhanced sarcomagenesis without compromising synovial sarcoma characteristics.

242 Despite the name, synovial sarcoma does not typically arise from a synovio

243 a, dedifferentiated liposarcoma (DDLPS), and synovial sarcoma histological subtype diagnoses, and the

244 investigated EZH2 as a therapeutic target in synovial sarcoma in vitro.

245 Synovial sarcoma is an aggressive soft tissue sarcoma ge

246 Meta-analysis of human synovial sarcoma patient series identified two tumor-gen

247 onfirmed EZH2 expression in the 76% of human synovial sarcoma samples.

248 lial malignancies, these sarcomas (excepting synovial sarcoma) are characterized predominantly by cop

249 X fusion oncoprotein (the hallmark driver of synovial sarcoma) that mediates a direct interaction bet

250 pid signaling associates with progression of synovial sarcoma, a deadly soft tissue malignancy initia

251 on embryonal and alveolar rhabdomyosarcoma, synovial sarcoma, and adult soft tissue sarcomas diagnos

252 a, malignant peripheral nerve sheath tumour, synovial sarcoma, epithelioid sarcoma and Ewing sarcoma

253 und cell tumors (including rhabdomyosarcoma, synovial sarcoma, neuroblastoma, Ewing sarcoma, and oste

254 eting to polycomb-marked genomic regions and synovial sarcoma-specific dependency on PRC1 function.

255 ith different INI1-negative solid tumours or synovial sarcoma.

256 osarcoma, and one (10%) of ten patients with synovial sarcoma.

257 ing therapeutic strategy in the treatment of synovial sarcoma; clinical trials are initiating enrollm

258 Two possible roles for native SSX2 in synovial sarcomagenesis are explored.

259 SSX1 and SS18-SSX2 can each drive comparable synovial sarcomagenesis, independent from other genetic

260 cating a paracrine link between the bone and synovial sarcomagenesis.

261 ath tumors (MPNST), solitary fibrous tumors, synovial sarcomas (SySa), well-differentiated/dedifferen

262 Synovial sarcomas are aggressive soft-tissue malignancie

263 Moreover, in a large panel of human synovial sarcomas, enhanced PI3'-lipid signaling also co

264 Thus, both in the mouse model and in human synovial sarcomas, PI3'-lipid signaling drives CSF1 expr

265 Microscopically, a synovial sheath was present.

266 ore flares and compared these with data from synovial single-cell RNA-seq.

267 The transition of synovial stromal cells into autoaggressive effector cell

268 ) immune effector fibroblasts located in the synovial sub-lining, and FAPalpha(+)THY1(-) destructive

269 steoarthritis progression, cartilage damage, synovial thickening, and osteophyte formation were measu

270 sociated with induction of senescence in the synovial tissue and cartilage protection.

271 so analyzed the expression of neuropilins in synovial tissue and SF, as they may interact with vascul

272 RA synovial tissue and synovial fluid macrophages expressed

273 y expressed in RA B cells from patients with synovial tissue containing ectopic germinal centres comp

274 onocytes, and fibroblasts from 51 samples of synovial tissue from patients with RA or osteoarthritis

275 te receptor beta on activated macrophages in synovial tissue in a preclinical arthritic rat model.

276 moglobin content reflecting the hyperemia in synovial tissue in metacarpophalangeal (MCP) joints of 1

277 In-depth characterization of the synovial tissue in rheumatoid arthritis, as well as psor

278 In vivo, NMT1 loss caused robust synovial tissue inflammation, whereas forced NMT1 overex

279 We hypothesized that synovial tissue macrophages (STM), which persist in remi

280 The reduction of synovial tissue macrophages is a reliable biomarker for

281 Using single-cell RNA sequencing and synovial tissue organoids, we found that NOTCH3 signalli

282 thritis (RA), and macrophages are reduced in synovial tissue shortly after initiation of TNF inhibito

283 In rheumatoid arthritis the synovial tissue undergoes marked hyperplasia, becomes in

284 mononuclear cells that are embedded into the synovial tissue.

285 uced serum amyloid A expression in ileum and synovial tissue.

286 topic germinal centres compared with diffuse synovial tissue.

287 lular DNA was detectable in various areas of synovial tissue.

288 hemokines, and catabolic factors that damage synovial tissues and can activate free nerve endings in

289 derived from CD19(+) B cells within RA human synovial tissues frequently react against NETs.

290 s the expression of CD40-downstream genes in synovial tissues from anti-citrullinated protein Ab-posi

291 ll genes were also significantly enriched in synovial tissues from arthralgia patients.

292 oducing predominantly GM-CSF are expanded in synovial tissues from patients with spondyloarthritis.

293 naive B cells were significantly enriched in synovial tissues from UA, early RA, and established RA p

294 was significantly low in RA serum, SFs, and synovial tissues, as well as in the serum and joints of

295 se to MAC and is detected in human renal and synovial tissues.

296 of mouse and human synovial fibroblasts and synovial tissues.

297 n of PDGFR-alphabeta was also elevated in RA synovial tissues.

298 eumatoid arthritis disease activity and with synovial TNF mRNA expression.

299 gammadelta (sTCR-gammadelta) tetramer from a synovial Vdelta1 gammadelta T cell clone from a Lyme art

300 e spectrum of ligand(s) expression for human synovial Vdelta1 gammadelta T cells as well as the physi