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1 ecreted protein 2 was analyzed in the Golden Syrian hamster.
2 ons that mediate mating behavior in the male Syrian hamster.
3  the median and dorsal raphe nuclei), in the Syrian hamster.
4 yloids from three species: human, mouse, and Syrian hamster.
5 pleting hepatotoxin, was administered to the Syrian hamster.
6 esponse in a sensitive animal model, namely, Syrian hamster.
7 arious behaviors and neural functions in the Syrian hamster.
8 as to replication and pathogenesis in golden Syrian hamsters.
9 e brains of young, middle-aged, and old male Syrian hamsters.
10 on reward processes and copulation in female Syrian hamsters.
11 dian pacemaker and peripheral oscillators in Syrian hamsters.
12 ence stimulates offensive aggression in male Syrian hamsters.
13 lity of SARS CoV to infect 5-week-old Golden Syrian hamsters.
14 associated with functional specialization in Syrian hamsters.
15 n of a key hormone, prolactin, in refractory Syrian hamsters.
16 disease in high-dose SARS-CoV-2 challenge in Syrian hamsters.
17 hepatic sterol 27-hydroxylase mRNA levels in Syrian hamsters.
18 ial reward was determined in male and female Syrian hamsters.
19 ical component of conditioned defeat in male Syrian hamsters.
20 e fibers with biotinylated dextran in golden Syrian hamsters.
21 le of such a change is conditioned defeat in Syrian hamsters.
22 hway of bile acid synthesis, in the liver of Syrian hamsters.
23 nd cause a highly lethal HPS-like disease in Syrian hamsters.
24 thed flagella was isolated from the feces of Syrian hamsters.
25 ortens period length of circadian rhythms in Syrian hamsters.
26 flagella isolated from feces of asymptomatic Syrian hamsters.
27 from the inflamed stomachs and ceca of adult Syrian hamsters.
28 mide (GlcCer) in circulating lipoproteins in Syrian hamsters.
29 eases serotonin receptor populations in male Syrian hamsters.
30 s were identified in juvenile and adult male Syrian hamsters.
31 d no effect against other scrapie strains in Syrian hamsters.
32 the expression of conditioned defeat in male Syrian hamsters.
33 evaluated by a toxin neutralization assay in Syrian hamsters.
34  eight different prion strains propagated in Syrian hamsters.
35 g(MH2M) mice] and subsequent transmission to Syrian hamsters.
36 uction of kidney tumors by estradiol in male Syrian hamsters.
37 ffects of endotoxin and cytokines on CETP in Syrian hamsters.
38 luding Omicron BA.4/5 and limited disease in Syrian hamsters.
39 nation inhibition (HAI) titers than RD-Ad in Syrian hamsters.
40 iant (B.1.1.529) sublineage BA.1 variants in Syrian hamsters.
41 nal humoral immune responses in NiV-infected Syrian hamsters.
42 le-9717869, does not cause lethal disease in Syrian hamsters.
43 strains of conventional laboratory mice, and Syrian hamsters.
44  infection and transmission of SARS-CoV-2 in Syrian hamsters.
45 athogenicity of BA.2.75 clinical isolates in Syrian hamsters.
46 a observed following social defeat stress in Syrian hamsters.
47 erolemia and early atherosclerosis in Golden Syrian hamsters.
48  Copenhageni strain Fiocruz L1-130 in Golden Syrian hamsters.
49 antavirus is Andes virus (ANDV) infection of Syrian hamsters.
50 ns and removed periodically for bioassays in Syrian hamsters.
51 3-CTV exhibited no toxicity in the brains of Syrian hamsters.
52 lescence facilitates offensive aggression in Syrian hamsters.
53 involving disruption of adaptive immunity in Syrian hamsters.
54 typhlocolitis in aging (18- to 24-month-old) Syrian hamsters.
55 virus pulmonary syndrome (HPS) in humans and Syrian hamsters.
56 ed three related hypotheses; namely, that in Syrian hamsters: (1) PS potentiation can be elicited bel
57                Single episodes of arousal of Syrian hamsters 2 h before projected activity onset (i.e
58 is peptide has been investigated in the male Syrian hamster, a species in which brain nuclei controll
59 ets of retinal fibers in the early postnatal Syrian hamster, a well-characterized developmental model
60 pregnancy followed by estrogen withdrawal in Syrian hamsters, a first for this species.
61 , most studies have been conducted in golden Syrian hamsters, a species particularly sensitive to acu
62 asured in rapid and slow acetylator congenic Syrian hamsters administered 3,2' -dimethyl-4-aminobiphe
63 dministration of (L)- Met-methyl-d(3) to the Syrian hamster after GSH had been depleted by BB resulte
64       These findings indicate that human and Syrian hamster alpha(v)beta(3) integrins are key recepto
65 he treatment of SARS was evaluated in golden Syrian hamsters, an animal model that supports SARS-CoV
66 ithin the forebrain and midbrain of the male Syrian hamster and addresses the question of whether enk
67 hrough PSI domain residues conserved in both Syrian hamster and human beta(3) integrins.
68 SI domain revealed eight differences between Syrian hamster and human beta(3) integrins.
69 ulated expression of COX-2 in HIT-T15 cells, Syrian hamster and human islets, and other Syrian hamste
70 onditions on reconfiguration dynamics of the Syrian hamster and rabbit prion proteins.
71 protein (PrP) amyloids from human, mouse and Syrian hamster and show that their structural difference
72 omes from 6 HeV-infected tissue samples from Syrian hamsters and 4 tissue samples from a NiV-infected
73 , and single doses of this nAb could protect Syrian hamsters and bank voles challenged with the highl
74 al titers in the lung of SARS-CoV-2-infected Syrian hamsters and block the development of virally ind
75 e-resistant PrP was reported in the urine of Syrian hamsters and humans with prion disease.
76 uated in animal models of dyslipidemia using Syrian hamsters and male Beagle dogs, and all these comp
77 P were sparse within the SCN of Siberian and Syrian hamsters and mouse but were intense in the ventra
78 rticle sequences closest to those present in Syrian hamsters and not mice were also detected in the J
79 /H165A) elicits robust antibody responses in Syrian hamsters and protects against SARS-CoV-2-induced
80 stent immune responses than RD-Ad vectors in Syrian hamsters and rhesus macaques.
81  in protection from acute disease.IMPORTANCE Syrian hamsters are in use as a model of disease caused
82                                              Syrian hamsters are permissive for the replication of sp
83                                              Syrian hamsters are susceptible to infection with certai
84                                Here, we show Syrian hamsters are susceptible to SARS-CoV-2 infection
85                                           In Syrian hamsters, arginine vasopressin (AVP) plays a crit
86 ed in the domestic cat as Orange, and in the Syrian hamster as Sex-linked yellow (Sly), but are curio
87      More broadly, these experiments suggest Syrian hamsters as a novel organism in which to model th
88                      Twenty-four male Golden-Syrian hamsters at 3 months of age were randomly divided
89 atic nucleus (SCN) in urethane-anaesthetized Syrian hamsters at different times of the light-dark cyc
90                                              Syrian hamsters become anemic and exhibit delayed growth
91                               Sequencing the Syrian hamster beta(3) integrin PSI domain revealed eigh
92 I domain polypeptides derived from human and Syrian hamster beta(3) subunits, but not murine or bovin
93  used for mucosal intranasal immunization of Syrian hamsters, both SC-Ad and RC-Ad expressed transgen
94   However, in C57BL/6J mice, 129S2 mice, and Syrian hamsters, BQ.1.1 did not cause increased respirat
95                  Exposure of Sc237 prions in Syrian hamster brain homogenates to 1% SDS and 0.5% AcOH
96 ously distributed across the neuraxis of the Syrian hamster brain.
97 he distribution of 5-HT(3A) receptors in the Syrian hamster brain.
98 c were obtained when CLDs were isolated from Syrian hamster brains.
99 strain could also be transmitted directly to Syrian hamsters, but when derived in this way, its prope
100 n of bile acid metabolism in the male Golden Syrian hamster by measuring the rate of fecal bile acid
101  kinetics of leptospiral infection in Golden Syrian hamsters by a sensitive quantitative real-time PC
102                           Here, we immunized Syrian hamsters by two successive infections with one of
103 ubjective day, circadian activity rhythms in Syrian hamsters can be phase advanced by a variety of st
104 DFTD, and as well as the soft-shell clam and Syrian hamster, can advance studies of tumor biology.
105 und that human adenovirus replicates well in Syrian hamster cell lines and confirmed replication in t
106 lved in structural alterations in neoplastic Syrian hamster cells transformed by chemical carcinogens
107 echanisms activated cph in other MCA-treated Syrian hamster cells.
108  respectively, and was 95% homologous to the Syrian hamster cells.
109 nt with the production of the JEV vaccine in Syrian hamster cells.
110                             Complexes of the Syrian hamster cellular prion protein (PrPC) and synthet
111        This strain was also avirulent in the Syrian hamster challenge model.
112  slow (Bio.82.73/ H-Pat(s) acetylator female Syrian hamsters congenic at the NAT2 locus received a s.
113                   PrP(Sc) from the brains of Syrian hamsters contains the same set of glycans as PrP(
114       To address this question, we gave male Syrian hamsters daily injections of the cell birthdate m
115                      Experimentally infected Syrian hamsters demonstrate the full spectrum of clinica
116             Furthermore, H135-treated golden Syrian hamsters demonstrated significantly lower respira
117 ANDV infection of human endothelial cells or Syrian hamster-derived BHK-21 cells was selectively inhi
118                         Here, we report that Syrian hamster dermal fibroblasts possess substantial le
119 scribed lethal HPS animal model (i.e., adult Syrian hamsters develop HPS and die within 10 to 15 days
120                    Depletion of T cells from Syrian hamsters did not significantly influence early ev
121 ring long "summer-like" photoperiods, female Syrian hamsters display a regular 4-day estrous cycle.
122 ficacious in the clinically validated golden Syrian hamster ear animal model, was designed to be deli
123                                         Male Syrian hamsters either did or did not experience endogen
124  transformation system of carcinogen-treated Syrian hamster embryo (SHE) cell cultures represents mul
125                                           In Syrian hamster embryo (SHE) fibroblasts, epidermal growt
126                                           In Syrian hamster embryo (SHE) fibroblasts, epidermal growt
127 le of c-Fos in apoptosis was examined in two Syrian hamster embryo cell lines (sup+I and sup-II) and
128  and comparisons were made between these two Syrian hamster embryo cell lines.
129 o(a)pyrene [B(a)P]-induced transformation of Syrian hamster embryo cells and benzo(a)pyrene diol-epox
130 anscripts expressed in normal and neoplastic Syrian hamster embryo cells in culture (5.0, 3.5 and 2.0
131                   A preneoplastic variant of Syrian hamster embryo cells, sup(+), exhibits decreased
132             Cph was isolated from neoplastic Syrian hamster embryo fibroblasts initiated by 3-methylc
133                                 Treatment of Syrian hamster embryo fibroblasts with a single dose of
134 sforming mutants of avian pp60(c-src) from a Syrian hamster embryo-derived cell line, 10W, transfecte
135 LTP) was examined in hippocampal slices from Syrian hamsters entrained to a LD 14:10 cycle.
136                                   Adult male Syrian hamsters equipped with a surgically implanted gui
137                                           In Syrian hamsters, even a single social defeat results in
138  that the SCN, IGL, MRN, and DRN of the male Syrian hamster exhibited specific binding of [3H]8-OH-DP
139 ness to estradiol and progesterone in female Syrian hamsters exposed to a short photoperiod is associ
140 eks of the study (HFD-FBX4w group) to Golden Syrian hamsters fed a high-fat diet (HFD) for 8 weeks we
141 (red-bellied tamarins; common marmosets) and Syrian hamsters-following single-dose, atraumatic intran
142   Chronic administration of estrogen to male Syrian hamsters for 7.0 to 9.0 months induces a high fre
143 -HT(3A) receptors across the neuraxis of the Syrian hamster forebrain using immunohistochemistry.
144 ranasal prophylactic dose of decoy protected Syrian hamsters from a subsequent lethal SARS-CoV-2 chal
145 ra virus glycoprotein protected up to 70% of Syrian hamsters from lethal NiV challenge, despite anima
146                         We have now isolated Syrian hamster full-length cDNAs for the cph oncogene an
147                      Unlike rats, studies in Syrian hamsters have failed to detect any alterations in
148                                       Golden Syrian hamster hearts were heterotopically transplanted
149 lly transplanted with brown Norway or Golden Syrian hamster hearts were treated for 50 or 75 days wit
150 ependent phosphodiesterase (PDE) activity in Syrian hamster hearts with hypertrophic cardiomyopathy (
151 dues from the kinase recognition sequence of Syrian hamster HMG-CoA reductase (E.C. 1.1.1.34).
152                  The activity of the class I Syrian hamster HMG-CoA reductase is regulated by phospho
153               The three conserved lysines of Syrian hamster HMG-CoA reductase were mutated to alanine
154   When injected unilaterally into the SCN of Syrian hamsters housed in constant darkness, OFQ/N (1-50
155 icity of BA.4 and BA.5 isolates in wild-type Syrian hamsters, human ACE2 (hACE2) transgenic hamsters
156                                       Golden Syrian hamsters immunized intramuscularly with two injec
157                                              Syrian hamsters immunized with FiloRab1 PBV-processed va
158 h in vitro or on growth and viral fitness in Syrian hamsters in vivo.
159 h in vitro or on growth and viral fitness in Syrian hamsters in vivo.
160 nditioned defeat is a social defeat model in Syrian hamsters in which individuals display increased s
161                           Here, we show that Syrian hamsters, in contrast to mice, are highly permiss
162 tamate agonist NMDA directly into the SCN of Syrian hamsters induced significant phase delays at circ
163 ads and lung pathology scores in male golden Syrian hamsters infected by the SARS-CoV-2 ancestral str
164                                              Syrian hamsters infected with ANDV, but not SNV, develop
165 lymer extraction from brain homogenates from Syrian hamsters infected with Hyper prions and WT mice i
166 ids in purified fractions from the brains of Syrian hamsters infected with Sc237 prions.
167  about PrP 27-30 purified from the brains of Syrian hamsters infected with scrapie.
168 s, as well as primary human neural cells and Syrian hamsters, infected with a clinical variant of SAR
169 y, no signs of prion infection were found in Syrian hamsters inoculated with rPrP fibrils that resemb
170 ing and sequence analysis of portions of the Syrian hamster interleukin 2 (IL-2), IL-4, gamma interfe
171                                      Using a Syrian hamster intraperitoneal model of infection, we de
172                   Thus, the immunosuppressed Syrian hamster is a powerful model to evaluate anti-Ad d
173                   Our findings show that the Syrian hamster is a promising immunocompetent model that
174 p process for estrogen carcinogenesis in the Syrian hamster kidney is proposed.
175 ls: the db/db mouse type 2 diabetes model, a Syrian hamster lipid model, and a dog lipid model.
176                                  In the male Syrian hamster, mating is dependent on chemosensory and
177       For CRM data acquired from midsagittal Syrian hamster ( Mesocricetus auratus ) brain cryosectio
178                                       In the Syrian hamster (Mesocricetus auratus) glutamate activity
179                                          The Syrian hamster (Mesocricetus auratus) has been extensive
180                 Remarkably, infection of the Syrian hamster (Mesocricetus auratus) with L. donovani r
181                                       Golden Syrian hamsters (Mesocricetus auratus) are a well-establ
182 tems modulating this behavior using pubertal Syrian hamsters (Mesocricetus auratus) as an adolescent-
183 al neurons from postnatal day (P)0-P2 golden Syrian hamsters (Mesocricetus auratus) of either sex to
184                                         Male Syrian hamsters (Mesocricetus auratus) treated with low-
185                              Cocaine-treated Syrian hamsters (Mesocricetus auratus) were tested for a
186                                           In Syrian hamsters (Mesocricetus auratus), cells in a subre
187                                           In Syrian hamsters (Mesocricetus auratus), social defeat pr
188 ehavioral effects on intermale aggression in Syrian hamsters (Mesocricetus auratus).
189  of various neurotransmitter systems in male Syrian hamsters (Mesocricetus auratus).
190 or protein-1 (AP-1) activity in both primary Syrian hamster mesothelial cells (SHM) and primary human
191                               Therefore, the Syrian hamster model may become a valuable tool for the
192 tant showed enhanced virulence in the Golden Syrian hamster model of acute C. difficile infection.
193 te showed potent therapeutic efficacy in the Syrian hamster model of COVID-19 and separately, effecti
194                   Here, we describe a lethal Syrian hamster model of EHF using mouse-adapted Ebola vi
195 tahcp6) and tested them for virulence in the Syrian hamster model of infection.
196 er membrane lipoprotein LipL41 in the Golden Syrian hamster model of leptospirosis.
197 associated pathological findings in a golden Syrian hamster model of SARS-CoV infection.
198 t human monoclonal antibodies (hu-mAbs) in a Syrian hamster model of SARS-CoV-2 and in a mouse-adapte
199                            Here, we report a Syrian hamster model that develops progressive lethal pu
200            To this end, we developed a novel Syrian hamster model that is both immunocompetent and re
201               Across four studies, we used a Syrian hamster model to investigate whether acute stress
202                                In the golden Syrian hamster model, p.o. sodium bicarbonate supplement
203 sease; however, by directly manipulating the Syrian hamster model, we continue to eliminate individua
204  which there is a reliable animal model, the Syrian hamster model, which recapitulates important aspe
205 in (ZsG), we visualized virus tropism in the Syrian hamster model.
206 treating SARS-CoV-2 infection using an adult Syrian hamster model.
207 79 limited disease caused by SARS-CoV-2 in a Syrian hamster model.
208 alog, inhibits SARS-CoV-2 replication in the Syrian hamster model.
209 -specific antibodies than RD in a permissive Syrian hamster model.
210 its protective efficacy in the common lethal Syrian hamster model.
211  integrins in hantavirus pathogenesis in the Syrian hamster model.
212 tissue spread of the vector, we employed the Syrian hamster model.
213 nicity of ATCC 23344 in the BALB/c mouse and Syrian hamster models of infection.
214 examine the therapeutic and toxic effects in Syrian hamster models of pancreatic cancer (PaCa).
215 e clock mutation (tau) was discovered in the Syrian hamster more than a decade ago and, using the pow
216 ssion to transgenic mice expressing chimeric Syrian hamster/mouse (MH2M) prion protein (PrP) genes [T
217 of residues within the PSI domains of human, Syrian hamster, murine, and bovine beta(3) integrins ide
218                          As in the analogous Syrian hamster mutant enzyme S871D, P. mevalonii mutant
219 and MS-8209 prolonged the incubation time in Syrian hamsters of the 263K strain of scrapie, but AmB h
220 ve entraining stimulus in fetal and neonatal Syrian hamsters of the same developmental ages used to p
221                                         In a Syrian hamster pancreatic cancer model, tumors similar t
222                   However, expression of the Syrian hamster PCPH and mt-PCPH proteins in haploid yeas
223                                     However, Syrian hamster PKR was still sensitive to K3 inhibition.
224 of peptides based on residues 109-122 of the Syrian hamster prion protein (H1) with a range of substi
225 -ray crystallographic structures of the anti-Syrian hamster prion protein (SHaPrP) monoclonal Fab 3F4
226                            Using recombinant Syrian hamster prion protein [SHaPrP(29-231)], we invest
227 sion cross sections measured for recombinant Syrian hamster prion protein PrP(90-231).
228                           The binding of the Syrian hamster prion protein, SHaPrP(90-231), to model l
229 lts of previous HDX studies on the human and Syrian hamster prion proteins at a higher pH, various se
230  (rPrP) of 142 residues corresponding to the Syrian hamster PrP 27-30 was expressed in Escherichia co
231  recombinant protein (rPrP) corresponding to Syrian hamster PrP 27-30 was expressed in Escherichia co
232 d transgenic mice expressing either mouse or Syrian hamster PrP exclusively in muscle.
233 amination of the dynamics of two recombinant Syrian hamster PrP fragments, PrP(29-231) and PrP(90-231
234 experiments using transgenic mice expressing Syrian hamster PrP in neurons only, MS-8209 extended the
235  this present work, recombinant, full-length Syrian hamster PrP is investigated using EPR methodologi
236 , the secondary structure of the recombinant Syrian hamster PrP of residues 29-231 [PrP(29-231)] is i
237  cellular prion protein (PrPC) and synthetic Syrian hamster PrP peptides were found to mimic many of
238         Studies of an analogous peptide from Syrian hamster PrP verify that sequence alterations in r
239 t similar to those found in the structure of Syrian hamster PrP(90-231).
240     We have investigated the conformation of Syrian hamster PrP(C) on the surface of transfected CHO
241                                              Syrian hamsters readily form dominant-subordinate relati
242  amyloid states were prepared in vitro using Syrian hamster recombinant PrP (rPrP) in the absence of
243                                           In Syrian hamsters, reproductive behaviors are initiated in
244 e were significantly lower, while vaccinated Syrian hamsters revealed protection in a harsh challenge
245                    In vitro amplification of Syrian hamster Sc237 PrP(Sc) displays an optimum pH of a
246 ions of serotonergic and other agents to the Syrian hamster SCN with durations equivalent to the incr
247 l-length prion protein PrP (residues 23-231, Syrian hamster sequence).
248             Synthetic peptides included: (i) Syrian hamster (SHa) hydrophobic core, SHa106-122 (KTNMK
249 NMR has been used to refine the structure of Syrian hamster (SHa) prion protein rPrP(90-231), which i
250 s 120-144 of human (Hu), bank vole (BV), and Syrian hamster (SHa) prion protein, from disordered mono
251 olling susceptibility to foreign prions, two Syrian hamster (SHa) prion strains, Sc237 and DY, were t
252                                              Syrian hamster (SHa) PrP genes of varying length ranging
253   Selection against recombinant (rec) PrP of Syrian hamster (SHa) sequence 90-231 folded into an alph
254 ission of prions through feces, uninoculated Syrian hamsters (SHas) were cohabitated with or exposed
255                  Dermal fibroblasts from the Syrian hamster (SHD cells) are exceptionally resistant t
256                                              Syrian hamsters show reliable territorial aggression, bu
257 nostaining generalized to the SCN and IGL of Syrian hamster, Siberian hamster, and mouse.
258 cating efficiently in normally nonpermissive Syrian hamster smooth muscle (DDT-1), Chinese hamster ov
259             In contrast, in the TG-resistant Syrian hamster smooth muscle cell line DDT/TG 4 microM,
260 ted a series of cell lines from the parental Syrian hamster smooth muscle cell line DDT1-MF2that are
261 t carcinoma cells MCF-7 and MDA-MB-468 and a Syrian hamster smooth muscle cell line DDT1MF2 and were
262  under moderate stringency conditions with a Syrian hamster-specific cph probe.
263 tinguishable from the previously established Syrian hamster strain Sc237, despite having been derived
264 ect virus replication or morbidity in golden Syrian hamsters, suggesting that the gene 7 products are
265  the Mitf gene of the anophthalmic white Wh) Syrian hamster that destabilizes its mRNA and prevents t
266  severe systemic disease in immunosuppressed Syrian hamsters that is similar to that seen in immunoco
267 e changes in blood chemistry in NiV-infected Syrian hamsters that survived or succumbed to disease.
268                                       Golden Syrian hamsters that were intranasally inoculated with S
269                        After transmission to Syrian hamsters, the Me7 strain was indistinguishable fr
270 xpression of the cph proto-oncogene in adult Syrian hamster tissues by northern hybridization using c
271 , Syrian hamster and human islets, and other Syrian hamster tissues.
272         We established an infection model in Syrian hamsters to evaluate the efficacy of small molecu
273 d the brain transcriptome of male and female Syrian hamsters to generate the necessary resources to c
274 se fluoxetine during adolescence predisposes Syrian hamsters to offensive aggression, with demonstrab
275 and decondensation induced by the binding of Syrian hamster transition proteins TP1 and TP2 and prota
276                   In the present study, male Syrian hamsters treated with TRZ (5 mg/kg) at ZT6 signif
277  hK2 was expressed in the adenovirus-induced Syrian hamster tumor cell line AV12-664 (AV12-hK2).
278 lian cells, recombinant PSA was expressed in Syrian hamster tumor cell line AV12-664 (AV12-PSA).
279                             Estrogen-induced Syrian hamster tumors in the kidney represent a useful m
280 n membrane currents in single cells from the Syrian hamster vas deferens cell line DDT1MF-2 were inve
281                                           In Syrian hamsters, vasopressin (AVP) controls a form of sc
282                                           In Syrian hamsters, vasopressin (AVP) in the medial preopti
283 teral inoculation into the sciatic nerves of Syrian hamsters was investigated.
284 lung tissues from SARS-CoV-2-infected golden Syrian hamsters, we show that a specific and selective p
285                            Young male Golden Syrian hamsters, weighing 90 to 120 g.
286                                       Golden Syrian hamsters were immunized at 3 and 6 weeks of age w
287                                       Female Syrian hamsters were immunized via a prime-boost regimen
288                                              Syrian hamsters were inoculated intraperitoneally with b
289  STAT1(-)(/-) mice, Hartley guinea pigs, and Syrian hamsters were inoculated intraperitoneally with R
290                                              Syrian hamsters were split under LDLD with dimly lit nig
291 nogenesis, freshly isolated islets from male Syrian hamsters were transplanted into the right submand
292                                         Male Syrian hamsters were treated with AAS throughout adolesc
293                                              Syrian hamsters were treated, intratracheally, with clod
294 Panama, cause a differential pathogenesis in Syrian hamsters, which could be a useful model for under
295                   Intratracheal infection of Syrian hamsters with Ad14p1 caused a marked, patchy bron
296                                 Infection of Syrian hamsters with ANDV results in a disease that clos
297 nverting enzyme 2 (ACE2) transgenic mice and Syrian hamsters with both viruses resulted in similar vi
298                       Immunization of golden Syrian hamsters with MMS vaccine induced similarly high
299                                 We immunized Syrian hamsters with the best long-term stable FiloRab1
300 inkage map for the euchromatic region of the Syrian hamster X chromosome that places Sly in a region

 
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