コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ecreted protein 2 was analyzed in the Golden Syrian hamster.
2 ons that mediate mating behavior in the male Syrian hamster.
3 the median and dorsal raphe nuclei), in the Syrian hamster.
4 yloids from three species: human, mouse, and Syrian hamster.
5 pleting hepatotoxin, was administered to the Syrian hamster.
6 esponse in a sensitive animal model, namely, Syrian hamster.
7 arious behaviors and neural functions in the Syrian hamster.
8 as to replication and pathogenesis in golden Syrian hamsters.
9 e brains of young, middle-aged, and old male Syrian hamsters.
10 on reward processes and copulation in female Syrian hamsters.
11 dian pacemaker and peripheral oscillators in Syrian hamsters.
12 ence stimulates offensive aggression in male Syrian hamsters.
13 lity of SARS CoV to infect 5-week-old Golden Syrian hamsters.
14 associated with functional specialization in Syrian hamsters.
15 n of a key hormone, prolactin, in refractory Syrian hamsters.
16 disease in high-dose SARS-CoV-2 challenge in Syrian hamsters.
17 hepatic sterol 27-hydroxylase mRNA levels in Syrian hamsters.
18 ial reward was determined in male and female Syrian hamsters.
19 ical component of conditioned defeat in male Syrian hamsters.
20 e fibers with biotinylated dextran in golden Syrian hamsters.
21 le of such a change is conditioned defeat in Syrian hamsters.
22 hway of bile acid synthesis, in the liver of Syrian hamsters.
23 nd cause a highly lethal HPS-like disease in Syrian hamsters.
24 thed flagella was isolated from the feces of Syrian hamsters.
25 ortens period length of circadian rhythms in Syrian hamsters.
26 flagella isolated from feces of asymptomatic Syrian hamsters.
27 from the inflamed stomachs and ceca of adult Syrian hamsters.
28 mide (GlcCer) in circulating lipoproteins in Syrian hamsters.
29 eases serotonin receptor populations in male Syrian hamsters.
30 s were identified in juvenile and adult male Syrian hamsters.
31 d no effect against other scrapie strains in Syrian hamsters.
32 the expression of conditioned defeat in male Syrian hamsters.
33 evaluated by a toxin neutralization assay in Syrian hamsters.
34 eight different prion strains propagated in Syrian hamsters.
35 g(MH2M) mice] and subsequent transmission to Syrian hamsters.
36 uction of kidney tumors by estradiol in male Syrian hamsters.
37 ffects of endotoxin and cytokines on CETP in Syrian hamsters.
38 luding Omicron BA.4/5 and limited disease in Syrian hamsters.
39 nation inhibition (HAI) titers than RD-Ad in Syrian hamsters.
40 iant (B.1.1.529) sublineage BA.1 variants in Syrian hamsters.
41 nal humoral immune responses in NiV-infected Syrian hamsters.
42 le-9717869, does not cause lethal disease in Syrian hamsters.
43 strains of conventional laboratory mice, and Syrian hamsters.
44 infection and transmission of SARS-CoV-2 in Syrian hamsters.
45 athogenicity of BA.2.75 clinical isolates in Syrian hamsters.
46 a observed following social defeat stress in Syrian hamsters.
47 erolemia and early atherosclerosis in Golden Syrian hamsters.
48 Copenhageni strain Fiocruz L1-130 in Golden Syrian hamsters.
49 antavirus is Andes virus (ANDV) infection of Syrian hamsters.
50 ns and removed periodically for bioassays in Syrian hamsters.
51 3-CTV exhibited no toxicity in the brains of Syrian hamsters.
52 lescence facilitates offensive aggression in Syrian hamsters.
53 involving disruption of adaptive immunity in Syrian hamsters.
54 typhlocolitis in aging (18- to 24-month-old) Syrian hamsters.
55 virus pulmonary syndrome (HPS) in humans and Syrian hamsters.
56 ed three related hypotheses; namely, that in Syrian hamsters: (1) PS potentiation can be elicited bel
58 is peptide has been investigated in the male Syrian hamster, a species in which brain nuclei controll
59 ets of retinal fibers in the early postnatal Syrian hamster, a well-characterized developmental model
61 , most studies have been conducted in golden Syrian hamsters, a species particularly sensitive to acu
62 asured in rapid and slow acetylator congenic Syrian hamsters administered 3,2' -dimethyl-4-aminobiphe
63 dministration of (L)- Met-methyl-d(3) to the Syrian hamster after GSH had been depleted by BB resulte
65 he treatment of SARS was evaluated in golden Syrian hamsters, an animal model that supports SARS-CoV
66 ithin the forebrain and midbrain of the male Syrian hamster and addresses the question of whether enk
69 ulated expression of COX-2 in HIT-T15 cells, Syrian hamster and human islets, and other Syrian hamste
71 protein (PrP) amyloids from human, mouse and Syrian hamster and show that their structural difference
72 omes from 6 HeV-infected tissue samples from Syrian hamsters and 4 tissue samples from a NiV-infected
73 , and single doses of this nAb could protect Syrian hamsters and bank voles challenged with the highl
74 al titers in the lung of SARS-CoV-2-infected Syrian hamsters and block the development of virally ind
76 uated in animal models of dyslipidemia using Syrian hamsters and male Beagle dogs, and all these comp
77 P were sparse within the SCN of Siberian and Syrian hamsters and mouse but were intense in the ventra
78 rticle sequences closest to those present in Syrian hamsters and not mice were also detected in the J
79 /H165A) elicits robust antibody responses in Syrian hamsters and protects against SARS-CoV-2-induced
81 in protection from acute disease.IMPORTANCE Syrian hamsters are in use as a model of disease caused
86 ed in the domestic cat as Orange, and in the Syrian hamster as Sex-linked yellow (Sly), but are curio
89 atic nucleus (SCN) in urethane-anaesthetized Syrian hamsters at different times of the light-dark cyc
92 I domain polypeptides derived from human and Syrian hamster beta(3) subunits, but not murine or bovin
93 used for mucosal intranasal immunization of Syrian hamsters, both SC-Ad and RC-Ad expressed transgen
94 However, in C57BL/6J mice, 129S2 mice, and Syrian hamsters, BQ.1.1 did not cause increased respirat
99 strain could also be transmitted directly to Syrian hamsters, but when derived in this way, its prope
100 n of bile acid metabolism in the male Golden Syrian hamster by measuring the rate of fecal bile acid
101 kinetics of leptospiral infection in Golden Syrian hamsters by a sensitive quantitative real-time PC
103 ubjective day, circadian activity rhythms in Syrian hamsters can be phase advanced by a variety of st
104 DFTD, and as well as the soft-shell clam and Syrian hamster, can advance studies of tumor biology.
105 und that human adenovirus replicates well in Syrian hamster cell lines and confirmed replication in t
106 lved in structural alterations in neoplastic Syrian hamster cells transformed by chemical carcinogens
112 slow (Bio.82.73/ H-Pat(s) acetylator female Syrian hamsters congenic at the NAT2 locus received a s.
117 ANDV infection of human endothelial cells or Syrian hamster-derived BHK-21 cells was selectively inhi
119 scribed lethal HPS animal model (i.e., adult Syrian hamsters develop HPS and die within 10 to 15 days
121 ring long "summer-like" photoperiods, female Syrian hamsters display a regular 4-day estrous cycle.
122 ficacious in the clinically validated golden Syrian hamster ear animal model, was designed to be deli
124 transformation system of carcinogen-treated Syrian hamster embryo (SHE) cell cultures represents mul
127 le of c-Fos in apoptosis was examined in two Syrian hamster embryo cell lines (sup+I and sup-II) and
129 o(a)pyrene [B(a)P]-induced transformation of Syrian hamster embryo cells and benzo(a)pyrene diol-epox
130 anscripts expressed in normal and neoplastic Syrian hamster embryo cells in culture (5.0, 3.5 and 2.0
134 sforming mutants of avian pp60(c-src) from a Syrian hamster embryo-derived cell line, 10W, transfecte
138 that the SCN, IGL, MRN, and DRN of the male Syrian hamster exhibited specific binding of [3H]8-OH-DP
139 ness to estradiol and progesterone in female Syrian hamsters exposed to a short photoperiod is associ
140 eks of the study (HFD-FBX4w group) to Golden Syrian hamsters fed a high-fat diet (HFD) for 8 weeks we
141 (red-bellied tamarins; common marmosets) and Syrian hamsters-following single-dose, atraumatic intran
142 Chronic administration of estrogen to male Syrian hamsters for 7.0 to 9.0 months induces a high fre
143 -HT(3A) receptors across the neuraxis of the Syrian hamster forebrain using immunohistochemistry.
144 ranasal prophylactic dose of decoy protected Syrian hamsters from a subsequent lethal SARS-CoV-2 chal
145 ra virus glycoprotein protected up to 70% of Syrian hamsters from lethal NiV challenge, despite anima
149 lly transplanted with brown Norway or Golden Syrian hamster hearts were treated for 50 or 75 days wit
150 ependent phosphodiesterase (PDE) activity in Syrian hamster hearts with hypertrophic cardiomyopathy (
154 When injected unilaterally into the SCN of Syrian hamsters housed in constant darkness, OFQ/N (1-50
155 icity of BA.4 and BA.5 isolates in wild-type Syrian hamsters, human ACE2 (hACE2) transgenic hamsters
160 nditioned defeat is a social defeat model in Syrian hamsters in which individuals display increased s
162 tamate agonist NMDA directly into the SCN of Syrian hamsters induced significant phase delays at circ
163 ads and lung pathology scores in male golden Syrian hamsters infected by the SARS-CoV-2 ancestral str
165 lymer extraction from brain homogenates from Syrian hamsters infected with Hyper prions and WT mice i
168 s, as well as primary human neural cells and Syrian hamsters, infected with a clinical variant of SAR
169 y, no signs of prion infection were found in Syrian hamsters inoculated with rPrP fibrils that resemb
170 ing and sequence analysis of portions of the Syrian hamster interleukin 2 (IL-2), IL-4, gamma interfe
182 tems modulating this behavior using pubertal Syrian hamsters (Mesocricetus auratus) as an adolescent-
183 al neurons from postnatal day (P)0-P2 golden Syrian hamsters (Mesocricetus auratus) of either sex to
190 or protein-1 (AP-1) activity in both primary Syrian hamster mesothelial cells (SHM) and primary human
192 tant showed enhanced virulence in the Golden Syrian hamster model of acute C. difficile infection.
193 te showed potent therapeutic efficacy in the Syrian hamster model of COVID-19 and separately, effecti
198 t human monoclonal antibodies (hu-mAbs) in a Syrian hamster model of SARS-CoV-2 and in a mouse-adapte
203 sease; however, by directly manipulating the Syrian hamster model, we continue to eliminate individua
204 which there is a reliable animal model, the Syrian hamster model, which recapitulates important aspe
215 e clock mutation (tau) was discovered in the Syrian hamster more than a decade ago and, using the pow
216 ssion to transgenic mice expressing chimeric Syrian hamster/mouse (MH2M) prion protein (PrP) genes [T
217 of residues within the PSI domains of human, Syrian hamster, murine, and bovine beta(3) integrins ide
219 and MS-8209 prolonged the incubation time in Syrian hamsters of the 263K strain of scrapie, but AmB h
220 ve entraining stimulus in fetal and neonatal Syrian hamsters of the same developmental ages used to p
224 of peptides based on residues 109-122 of the Syrian hamster prion protein (H1) with a range of substi
225 -ray crystallographic structures of the anti-Syrian hamster prion protein (SHaPrP) monoclonal Fab 3F4
229 lts of previous HDX studies on the human and Syrian hamster prion proteins at a higher pH, various se
230 (rPrP) of 142 residues corresponding to the Syrian hamster PrP 27-30 was expressed in Escherichia co
231 recombinant protein (rPrP) corresponding to Syrian hamster PrP 27-30 was expressed in Escherichia co
233 amination of the dynamics of two recombinant Syrian hamster PrP fragments, PrP(29-231) and PrP(90-231
234 experiments using transgenic mice expressing Syrian hamster PrP in neurons only, MS-8209 extended the
235 this present work, recombinant, full-length Syrian hamster PrP is investigated using EPR methodologi
236 , the secondary structure of the recombinant Syrian hamster PrP of residues 29-231 [PrP(29-231)] is i
237 cellular prion protein (PrPC) and synthetic Syrian hamster PrP peptides were found to mimic many of
240 We have investigated the conformation of Syrian hamster PrP(C) on the surface of transfected CHO
242 amyloid states were prepared in vitro using Syrian hamster recombinant PrP (rPrP) in the absence of
244 e were significantly lower, while vaccinated Syrian hamsters revealed protection in a harsh challenge
246 ions of serotonergic and other agents to the Syrian hamster SCN with durations equivalent to the incr
249 NMR has been used to refine the structure of Syrian hamster (SHa) prion protein rPrP(90-231), which i
250 s 120-144 of human (Hu), bank vole (BV), and Syrian hamster (SHa) prion protein, from disordered mono
251 olling susceptibility to foreign prions, two Syrian hamster (SHa) prion strains, Sc237 and DY, were t
253 Selection against recombinant (rec) PrP of Syrian hamster (SHa) sequence 90-231 folded into an alph
254 ission of prions through feces, uninoculated Syrian hamsters (SHas) were cohabitated with or exposed
258 cating efficiently in normally nonpermissive Syrian hamster smooth muscle (DDT-1), Chinese hamster ov
260 ted a series of cell lines from the parental Syrian hamster smooth muscle cell line DDT1-MF2that are
261 t carcinoma cells MCF-7 and MDA-MB-468 and a Syrian hamster smooth muscle cell line DDT1MF2 and were
263 tinguishable from the previously established Syrian hamster strain Sc237, despite having been derived
264 ect virus replication or morbidity in golden Syrian hamsters, suggesting that the gene 7 products are
265 the Mitf gene of the anophthalmic white Wh) Syrian hamster that destabilizes its mRNA and prevents t
266 severe systemic disease in immunosuppressed Syrian hamsters that is similar to that seen in immunoco
267 e changes in blood chemistry in NiV-infected Syrian hamsters that survived or succumbed to disease.
270 xpression of the cph proto-oncogene in adult Syrian hamster tissues by northern hybridization using c
273 d the brain transcriptome of male and female Syrian hamsters to generate the necessary resources to c
274 se fluoxetine during adolescence predisposes Syrian hamsters to offensive aggression, with demonstrab
275 and decondensation induced by the binding of Syrian hamster transition proteins TP1 and TP2 and prota
278 lian cells, recombinant PSA was expressed in Syrian hamster tumor cell line AV12-664 (AV12-PSA).
280 n membrane currents in single cells from the Syrian hamster vas deferens cell line DDT1MF-2 were inve
284 lung tissues from SARS-CoV-2-infected golden Syrian hamsters, we show that a specific and selective p
289 STAT1(-)(/-) mice, Hartley guinea pigs, and Syrian hamsters were inoculated intraperitoneally with R
291 nogenesis, freshly isolated islets from male Syrian hamsters were transplanted into the right submand
294 Panama, cause a differential pathogenesis in Syrian hamsters, which could be a useful model for under
297 nverting enzyme 2 (ACE2) transgenic mice and Syrian hamsters with both viruses resulted in similar vi
300 inkage map for the euchromatic region of the Syrian hamster X chromosome that places Sly in a region