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1 pressors reported here, one (JSN2) encodes a tRNAVal, and the other (JSN3) is an antimorphic allele o
3 ts in reduced levels of mature tRNA(Asp) and tRNA(Val) and that altered protein production during dev
4 genes, trnD and trnV, encoding tRNA(Asp) and tRNA(Val), respectively, composing an operon at the attB
9 ding of ethidium bromide to Escherichia coli tRNAVal and an RNA minihelix based on the acceptor stem
10 ed the interactions between Escherichia coli tRNAVal and valyl-tRNA synthetase (ValRS) by enzymatic f
13 healthy growth at 37 degrees C, hypomodified tRNA(Val(AAC)) is at least partially functional and stru
15 , including tRNA(Glu), tRNA(Gly), tRNA(Lys), tRNA(Val), tRNA(His), tRNA(Asp), and tRNA(SeC) to produc
16 3' exonucleases Rat1 and Xrn1 degrade mature tRNA(Val(AAC)) in yeast mutants lacking m(7)G and m(5)C,
17 t both degradation and deacylation of mature tRNA(Val(AAC)) in a trm8-Delta trm4-Delta strain and res
18 rapid tRNA decay (RTD) pathway, since mature tRNA(Val(AAC)) lacking 7-methylguanosine and 5-methylcyt
19 red to human tRNA gene promoters (tRNA(Met), tRNA(Val)), the human small nuclear RNA U6 gene (U6) and
20 -tRNAMetm (CAU anticodon) and mischarged Met-tRNAVal-1 (CAU anticodon) are substrates for the L/F-tra
21 able to hydrolytically deacylate misacylated tRNA(Val) terminating in 3'-pyrimidines but does deacyla
24 A identified a mutation in the mitochondrial tRNA(Val) (mt-tRNA(Val) ) gene, m.1661A>G, present at ne
25 r tRNA(Gly) and tRNA(Leu), the mitochondrial tRNA(Val) and tRNA(Pro)) were strongly associated with t
27 itoribosomes have been shown to integrate mt-tRNA(Val) compared with the porcine use of mt-tRNA(Phe)
28 st and steady-state levels of the mutated mt-tRNA(Val) were greater than in the biopsy material, but
29 estore steady-state levels of the mutated mt-tRNA(Val), consistent with an increased stability of the
30 A showed severe reduction in abundance of mt-tRNA(Val) , and mildly increased mt-tRNA(Phe) , in subje
31 d strikingly, when steady-state levels of mt-tRNA(Val) are reduced, human mitoribosome biogenesis dis
34 the generalized decrease in steady-state mt-tRNA(Val) observed in the homoplasmic 1624C>T-cell lines
37 mutation in the mitochondrial tRNA(Val) (mt-tRNA(Val) ) gene, m.1661A>G, present at nearly 100% hete
41 periments to characterize the interaction of tRNA(Val) with the enzyme provide evidence for two tRNA
42 to the recently refined structural model of tRNA(Val) yields the magnitude, asymmetry, and orientati
43 2'-hydroxyl group, that of the U73 mutant of tRNA(Val) occurred at either the 2' or 3'-hydroxyl group
45 ty determinants to productive recognition of tRNA(Val) at the aminoacylation and editing sites, and b
47 s results have shown that the 3'-terminus of tRNA(Val) is recognized differently at the two sites.
49 e double mutants indicates reduced levels of tRNA(Val(AAC)), consistent with a role of the correspond
51 elix based on the acceptor stem and T-arm of tRNAVal was investigated by 19F and 1H NMR spectroscopy
52 ix, and the 5' side of the anticodon stem of tRNAVal against cleavage by double- and single-strand-sp
53 g the anticodon stem of tRNAPhe with that of tRNAVal, however, converts the tRNA into a good substrat
56 of the acceptor-TpsiC helix of tRNA(Ile) or tRNA(Val) were aminoacylated by cognate synthetases sele
58 emonstrates rapid degradation of preexisting tRNA(Val(AAC)) accompanied by its de-aminoacylation.
59 oacylation and editing sites, and by probing tRNA(Val) for editing determinants that are distinct fro
60 itment of mitochondrial valine transfer RNA (tRNA(Val)) to play an integral structural role, and chan
62 e positions in the 5' flanking region of the tRNA(Val) gene were repaired more efficiently than the g
63 he ends of the anticodon- and T-stems in the tRNAVal.ValRS complex is indicative of enzyme-induced co
64 ase.19F NMR also shows that formation of the tRNAVal-valyl-tRNA synthetase complex does not disrupt t
68 ted mutant tRNAs as well as 3'-end-truncated tRNA(Val) are mixed noncompetitive inhibitors of the ami
70 alyl-tRNA synthetase does not edit wild-type tRNA(Val)(A76) mischarged with isoleucine, presumably be
72 2 by overexpressing a mutant tRNA(AAC)(Val) (tRNA(Val*)) or the RNA component of RNase MRP encoded by
75 aminoacylation of alpha-casein, whereas Val-tRNAVal-1 (UAC), Val-tRNAMetm (UAC), and Arg-tRNAMetm (C