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1 itivity of peripheral nerves to light touch (tactile allodynia).
2 s tactile stimulation elicits pain behavior (tactile allodynia).
3 rn, has been implicated in the expression of tactile allodynia.
4 ontribute to peripheral inflammation-induced tactile allodynia.
5  proprioceptive deficits and fail to develop tactile allodynia.
6 5-LOX (Zileuton) dose-dependently attenuated tactile allodynia.
7 carrageenan-induced thermal hyperalgesia and tactile allodynia.
8 ateral spinal dorsal horn of rats displaying tactile allodynia.
9 ulation do not display a learning deficit or tactile allodynia.
10 these afferents contributes to the resulting tactile allodynia.
11 h dimethylfumarate (DMF) and BHB reduced the tactile allodynia.
12  and intraepidermal nerve fiber loss but not tactile allodynia.
13 2delta-1 subunit and reversed injury-induced tactile allodynia.
14 hermore, to the behavioural manifestation of tactile allodynia.
15 2delta-1 subunit upregulation and diminished tactile allodynia.
16 rog/day) for 5 days significantly attenuated tactile allodynia.
17 onset and diminished in rats recovering from tactile allodynia.
18 th necessary and sufficient for inflammatory tactile allodynia.
19  nerve-ligated rats and its correlation with tactile allodynia, a neuropathic pain state defined as r
20 ain, where both male and female rats display tactile allodynia, a pathological coupling between KCC2-
21           SP16 also prevented development of tactile allodynia after partial nerve ligation and this
22 normal chow after 16 weeks on HFD alleviated tactile allodynia and essentially corrected thermal hypo
23 ion of activin A protein alone caused robust tactile allodynia and increased CGRP in the DRG.
24                                              Tactile allodynia and mechanical hyperalgesia were asses
25 ation of dynorphin A(1-17) antiserum blocked tactile allodynia and reversed thermal hyperalgesia to a
26 ed rats, i.th. morphine was inactive against tactile allodynia and showed diminished in potency again
27 s delivering morphine displayed time-related tactile allodynia and thermal hyperalgesia (i.e., opioid
28 k-out mice developed significantly increased tactile allodynia and thermal hyperalgesia in both the e
29 sed with DAMGO, but not saline, demonstrated tactile allodynia and thermal hyperalgesia of the hindpa
30          Correlating with the development of tactile allodynia and thermal hyperalgesia, spinal CaMKI
31       SCI animals also exhibited symptoms of tactile allodynia and thermal hyperalgesia.
32 terestingly, peripheral nerve injury induces tactile allodynia and upregulates Ca(V)3.2 channels and
33 locity (SNCV) deficits, thermal hypoalgesia, tactile allodynia, and a remarkable ( approximately 78%)
34 motor and sensory nerve conduction deficits, tactile allodynia, and thermal hypoalgesia in the absenc
35 .th.) morphine is ineffective in suppressing tactile allodynia at fully antinociceptive doses in thes
36 d and dorsal root ganglia (DRG) of rats with tactile allodynia because of either tight ligation of th
37 XA(3), or HXB(3) evoked profound, persistent tactile allodynia, but 12(S)-HpETE and HXA(3) produced r
38         Selective reversal of injury-induced tactile allodynia by NPY receptor antagonists would have
39 siRNA normalized mechanical hyperalgesia and tactile allodynia caused by SNL but had no significant e
40        People living with FM also experience tactile allodynia, cold-evoked pain, paraesthesia and dy
41                                     Profound tactile allodynia developed in all the resiniferotoxin-t
42                               Also, profound tactile allodynia developed in all the RTX-treated rats
43 hic pain model in which gabapentin-sensitive tactile allodynia develops after tight ligation of the l
44                                    Moreover, tactile allodynia did not develop as an unwanted side ef
45   Systemic A-134974 dose-dependently reduced tactile allodynia (ED(50)=5 micromol/kg, i.p.) for up to
46 es were lateralized and in proportion to the tactile allodynia exhibited by SNI animals.
47 stration of the TAT-4BB reversed M3G-induced tactile allodynia in a dose-dependent manner but did not
48 t tool molecule 20 (AM-1488), which reversed tactile allodynia in a mouse spared-nerve injury (SNI) m
49 n a hyperalgesia at the site of injury and a tactile allodynia in areas adjacent to the injury site.
50 togenic diet treatment significantly reduced tactile allodynia in both rats and mice, though with a s
51 in this circuit underlies the development of tactile allodynia in chronic injury.
52      Finally, we analyzed the development of tactile allodynia in diabetic mice lacking expression of
53                  Importantly, the absence of tactile allodynia in diabetic NF-kappaB p50(-/-) mice su
54 lazine completely blocked the development of tactile allodynia in diabetic rats, whereas relatively m
55                   The complete inhibition of tactile allodynia in experimental diabetes by sulfasalaz
56 entration able to activate the HCAR2-reduced tactile allodynia in female WT mice, but not in the HCAR
57 , leading to inflammaraft formation, induced tactile allodynia in naive mice.
58 algesic tolerance, thermal hyperalgesia, and tactile allodynia in response to chronic intrathecal mor
59 hese compounds, 23 dose dependently reversed tactile allodynia in the Chung model of neuropathic pain
60 algesia in the inflammatory state, decreased tactile allodynia in the neuropathic state, and no chang
61 f formalin-induced flinching, and attenuated tactile allodynia in the spinal nerve ligation model of
62 mically and spinally can effectively relieve tactile allodynia in this animal model of postherpetic n
63 .8 muM, respectively) and carrageenan-evoked tactile allodynia in vivo.
64 ct of systemic and intrathecal gabapentin on tactile allodynia induced by resiniferotoxin in rats.
65                                  The delayed tactile allodynia induced by RTX is likely attributable
66  nerve crush resulted in a patchy but marked tactile allodynia manifesting first at 3 weeks and persi
67 viated diabetes-induced thermal hypoalgesia, tactile allodynia, motor and sensory nerve conduction ve
68 cation of icilin (0.1nM to 1microM) affected tactile allodynia or thermal hyperalgesia after SNL, but
69  syndrome) or by mechanical hyperalgesia and tactile allodynia (pain in response to nonpainful stimul
70                       Behavioral testing for tactile allodynia, performed one week prior to STZ injec
71                                              Tactile allodynia produced by placing a plastic cuff aro
72 ly affected by inflammation, suggesting that tactile allodynia results from the continuing firing of
73  such as the loss of tactile sensitivity and tactile allodynia seen in patients who have diabetes, in
74 n after SNL contributes nerve injury-induced tactile allodynia.SIGNIFICANCE STATEMENT Neuropathic pai
75 as more potent (ED(50)=10 nmol) in relieving tactile allodynia than delivering the compound by intrac
76 ited time-dependent and reversible cutaneous tactile allodynia that was maintained throughout and tra
77 lish pain phenotypes, including long-lasting tactile allodynia, that scale with the extent of stimula
78 novel AK inhibitor A-134974 potently reduces tactile allodynia through interactions with spinal sites
79 ats with streptozotocin-induced diabetes and tactile allodynia, using in situ hybridization and immun
80 elopment of diabetes-induced retinopathy and tactile allodynia was investigated.
81                                              Tactile allodynia was present in both injured and adjace
82                     Moreover, injury-induced tactile allodynia was reversed by inhibiting and exacerb
83 ssess the role of NO in nerve injury-induced tactile allodynia, we examined neuronal NO synthase (nNO
84 orn neurons may result in the development of tactile allodynia, where non-painful stimuli gain the ca
85 males and females display a severe sustained tactile allodynia which is reduced by gabapentin but not