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1 tode infection via trait-mediated effects on tadpoles'.].
2 motility in the intact albino Xenopus laevis tadpole.
3 sensory vesicle (simple brain) of the Ciona tadpole.
4 the developing visual system of the Xenopus tadpole.
5 -incompetent developmental stages of Xenopus tadpoles.
6 alis heterozygous, but not homozygous mutant tadpoles.
7 e optic tectum in stage 46-49 Xenopus laevis tadpoles.
8 f lung breathing, similar to water-breathing tadpoles.
9 ity profile reflects that of water-breathing tadpoles.
10 ensory inputs in the optic tectum of Xenopus tadpoles.
11 s during tail regeneration in Xenopus laevis tadpoles.
12 ocomotory swimming in post-embryonic Xenopus tadpoles.
13 ulus in the olfactory bulb of Xenopus laevis tadpoles.
14 -brain photoreception in Xenopus laevis frog tadpoles.
15 her studies of network maturation in Xenopus tadpoles.
16 ofacial defects in pre-metamorphic X. laevis tadpoles.
17 m neurons which drive swimming in young frog tadpoles.
18 fects observed on tectal neurons of Stage 45 tadpoles.
19 d to be executed in regeneration-incompetent tadpoles.
20 of cardiac function in translucent wildtype tadpoles.
21 al burdens in a frog kidney cell line and in tadpoles.
22 nile frogs that had been exposed to PBDEs as tadpoles.
23 arms by isolating Bd from dozens of infected tadpoles.
24 y reduced probability of infection in anuran tadpoles.
25 le tectal progenitor cells in Xenopus laevis tadpoles.
26 in triggering the metamorphosis of swimming tadpoles.
27 alamander larvae and their prey, Rana pirica tadpoles.
28 nous D-serine in the brain of Xenopus laevis tadpoles.
29 d visually-guided behavior in Xenopus laevis tadpoles.
30 /CD4(-) iT subset in unmanipulated frogs and tadpoles.
31 potent analog of propofol, in Xenopus laevis tadpoles.
32 eous activity in the optic tectum of Xenopus tadpoles.
33 kin stimuli evoke swimming in hatchling frog tadpoles.
34 s and abnormal tail morphogenesis in Xenopus tadpoles.
35 nate, augment GABA currents, and anesthetize tadpoles.
36 pecifically silence Rx expression in vivo in tadpoles.
37 l regeneration in the regeneration-competent tadpoles.
38 tode infection via trait-mediated effects on tadpoles.
39 ults without their microbiota manipulated as tadpoles.
40 ndance of phylum Fusobacteria in the guts of tadpoles.
41 terior-most spinal nerves of early X. laevis tadpoles.
42 espectively, whereas intermediate-acclimated tadpoles (19-25 degrees C) cleared the greatest proporti
43 as significantly reduced both in oocytes and tadpoles 40 weeks after deprivation, and brain DHA was r
44 olfactory system of mice and Xenopus laevis tadpoles, a discussion arose about the influence of thes
46 evidence that Perkinsea-like protists infect tadpoles across a wide taxonomic range of frogs in tropi
47 rotected the kidney-derived A6 cell line and tadpoles against FV3 infection, decreasing the infectiou
48 ine the growth response of red-eyed treefrog tadpoles (Agalychnis callidryas) to cues from a larval d
50 Consistent with these findings, VPA-treated tadpoles also have increased seizure susceptibility and
52 versus uncoupling of phenotypic evolution of tadpole and adult life-history phases, and for the under
55 examined in RV (frog virus 3 [FV3])-infected tadpoles and adult frogs by quantitative PCR, and a reco
56 Cuban tree frog (Osteopilus septentrionalis) tadpoles and adults and whether any atrazine-induced cha
57 ugh anti-M. marinum immune responses between tadpoles and adults are different, tadpoles are as resis
58 infections by manipulating the microbiota of tadpoles and challenging them with parasitic gut worms a
59 nd retention kinetics were assessed in whole tadpoles and excised tissues using gamma spectroscopy, a
60 n preventing FV3 replication in A6 cells and tadpoles and inferior at promoting tadpole survival.
61 nockdown severely retarded the growth of the tadpoles and led to tadpole lethality prior to metamorph
62 emonstrate overt anesthetic activity in both tadpoles and rats with a potency slightly greater than t
63 s after manipulating the presence/absence of tadpoles and salamanders with offensive (broadened gape
64 portant resources for aquatic consumers like tadpoles and snails, causing bottom-up effects on wetlan
65 (rXlIFN-lambda) conferred less protection to tadpoles and the A6 cell line than rXlIFN, which may be
66 an axis reflecting the developmental rate of tadpoles and the inverse of their size at metamorphosis)
67 cluding annotating the synaptome of swimming tadpoles and tracing the evolutionary origin of cell typ
68 hiopod crustaceans are represented by fairy, tadpole, and clam shrimps (Anostraca, Notostraca, Laevic
69 or decreasing) reduced Bd in culture and on tadpoles, and every concentration tested (0.0106-106 mug
70 ns driving swimming locomotion in young frog tadpoles, and how activity is switched on and off by bri
71 Spontaneous synchrony bouts are rare in most tadpoles, and they instantly emerge from and switch back
72 e median anesthetic concentration (AC50) for tadpole anesthesia, but not when plotted against the ove
74 s between tadpoles and adults are different, tadpoles are as resistant to M. marinum inoculation as a
80 herbivores in Subarctic wetlands, wood frog tadpoles, are capable of increasing their developmental
81 ing limb proprioceptive signals in amphibian tadpoles as a potential sensory substitute after UL migh
86 threat to amphibian populations, with anuran tadpoles being particularly susceptible to these viral i
89 significantly affect bacterial diversity of tadpoles, but significantly increased bacterial diversit
91 ing central nervous system of Xenopus laevis tadpoles by using in vivo time-lapse confocal microscopy
95 In our laboratory experiments, we show that tadpoles can filter feed Bd zoospores and that the degre
97 suggest that a severe infectious disease of tadpoles caused by a protist belonging to the phylum Per
99 Gene expression profiling confirmed poor tadpole CD8(+) T cell response, contrasting with the mar
104 likely occurred because IG predators reduced tadpole densities and anticercarial behaviors, increasin
106 imaging in vivo, we show that pMBP-eGFP-NTR tadpoles display a graded oligodendrocyte ablation in re
108 ti-ring systems, single chain nanoparticles, tadpoles, dumbbells and hairpins, as well as the potenti
109 rdation in the growth and development of the tadpoles during metamorphosis and leads to tadpole letha
110 o the limb margin in amphibians with aquatic tadpoles, Eleutherodactylus coqui, a frog with terrestri
111 ry CD8(+) T cell response in adults, whereas tadpoles elicited only a noninflammatory CD8 negative- a
113 of all the epidermal cell types in the early tadpole epidermis and reinforces the suitability of this
115 rast to our previous findings that X. laevis tadpoles exhibit delayed and modest type I IFN responses
116 We observed induced carbaryl tolerance in tadpoles exposed to 0.5 mg/L carbaryl and also in tadpol
118 o inventory the gut microbial communities of tadpoles exposed to cool (18 degrees C) or warm (28 degr
121 ht representative aquatic species (including tadpoles, fish, water fleas, protozoan, and bacteria) wi
123 that neurons were morphologically simpler in tadpoles from frogs fed the deficient diet compared with
125 malities were significantly greater in 5 dpf tadpoles from the highest exposure group when compared t
126 t of that paper was the production of normal tadpoles from the nuclei of intestinal epithelium cells
127 rial and fungal assemblages were compared in tadpoles from the pond of origin, across metamorphosis,
130 H3K79 methylation play an important role for tadpole growth and development prior to metamorphosis in
133 aled that despite rXlIFN treatment, infected tadpoles had considerable organ damage, including disrup
134 ees C) and cold-acclimated (13-19 degrees C) tadpoles had fewer parasites encyst at warm and cold per
135 tissues are naturally corrected in X. laevis tadpoles has provided valuable insights into the mainten
138 in the United States, in which infected frog tadpoles have an abnormally enlarged yellowish liver fil
140 rogs that had reduced bacterial diversity as tadpoles have three times more worms than adults without
143 ct and indirect effects on key traits of the tadpole host (rates of growth, development and survival)
145 st that these viruses are more pathogenic to tadpole hosts than was previously believed, causing exte
146 g per capita exposure rates of the surviving tadpoles (i.e., via density- and trait-mediated effects)
150 increased the odds of survival by 1.79, and tadpoles in 52-day and 64-day hydroperiod mesocosms were
155 gth of the small intestine in premetamorphic tadpoles, in contrast to X. laevis, where it is present
157 caused large-scale morphological effects in tadpoles, including changes in brain morphology and incr
161 of IBF for pre-metamorphic Rana catesbeiana tadpoles is 41.5 mg/L (95% confidence interval: 32.3-53.
162 of MSI in the optic tectum of Xenopus laevis tadpoles is mediated by the network dynamics of the recr
165 ir hitherto unknown synaptic networks in the tadpole larva of a sibling chordate, the ascidian, Ciona
166 that a particular neuronal cell type in the tadpole larva of the tunicate Ciona intestinalis, the bi
169 al. show that manipulating the microbiota of tadpoles leads to increased susceptibility to parasitic
173 tantly, we show that monomeric Httex1 adopts tadpole-like architectures for polyQ lengths below and a
174 e x-ray experiments reveal that AnBgl1 has a tadpole-like structure, with the N-terminal catalytic do
176 tion and secondary polyphenolic compounds on tadpole (Lithobates sylvatica) exposure and susceptibili
177 climation effects on infection of green frog tadpoles (Lithobates clamitans) by the trematode parasit
178 insea, while only 2.5% histologically normal tadpole livers tested positive (2/81), suggesting that s
180 thesis of chemically multifunctional polymer tadpoles made at high weight fractions of polymer (>10 w
183 ctrophysiological characteristics of Xenopus tadpole midbrain neurons across development and during h
186 mechanisms underlying initiation of a simple tadpole motor response may share similarities with more
187 the responses of adults, here we report that tadpoles mount timely and robust type III IFN gene respo
191 robust FV3-elicited IFN gene expression than tadpoles; nevertheless, they also tolerated substantiall
192 of Bufo terrestris and Hyla cinerea, whereas tadpoles of B. terrestris (an obligate benthos feeder) g
193 lly diluted the risk of chytridiomycosis for tadpoles of Bufo terrestris and Hyla cinerea, whereas ta
196 issue 'fingerprint' in spawn and early-stage tadpoles of the Common frog, Rana temporaria, using atte
198 acterium johnsoniae were administered to 240 tadpoles of the midwife toad, Alytes obstetricans in sem
200 l and metamorph emergence by 58% compared to tadpole-only treatments, and by over 30% compared to non
201 ic regions of tectal neuron dendrites in the tadpole optic tectum requires NMDA receptor activity.
204 dulatory pesticides in the United States, on tadpole (Osteopilus septentrionalis) survival and Bd gro
206 verse of their size at metamorphosis) in our tadpole-parasitic cercarial (trematode) system, would be
207 ermediate hosts, causing opposing effects on tadpole per capita exposure to trematode infection.
209 Thus, in contrast to previous assumptions, tadpoles possess intact antiviral defenses reliant on ty
210 splayed more robust IFN gene expression than tadpoles, possibly explaining why they typically clear F
212 ory metabolic mechanisms) can be observed in tadpoles present in the pond most impacted by pollution
218 phenotype salamanders reduced the number of tadpoles remaining in the pond over time by reducing tad
220 al FV3; however, infection in Xenopus laevis tadpoles revealed that FV3-DeltavIF-2alpha and FV3-Delta
221 that short hairpin RNA knockdown of nudC in tadpole rod photoreceptors, which leads to the inability
222 Members of this clade were present in 38 tadpoles sampled from 14 distinct genera/phylogroups, fr
225 To test these hypotheses, we exposed seven tadpole species to a range of parasite (cercarial) doses
226 silence neurons on just one side of Xenopus tadpole spinal cord and hindbrain, which generate swimmi
227 of the development of neurons in the Xenopus tadpole spinal cord to include interactions between axon
229 bial population signature persisted from the tadpole stage, through metamorphosis, and following prob
230 ring development and persisted into swimming tadpole stages without perturbing the expression of the
231 nal nonlinear topologies including circular, tadpole, star, and H-shaped proteins using genetically e
236 viral burdens, it only transiently extended tadpole survival and did not prevent the eventual mortal
237 Offensive phenotype salamanders reduced tadpole survival and metamorph emergence by 58% compared
239 und that increased litter nitrogen increased tadpole survival, and also increased cercaria production
240 remaining in the pond over time by reducing tadpole survival, not by altering patterns of metamorph
245 o parallel mechanisms have been proposed for tadpole swimming maintenance: postinhibition rebound fir
246 central pattern generator (CPG) for Xenopus tadpole swimming that involves antiphase oscillations of
250 ng non-viral somatic transgenesis in Xenopus tadpole tail muscle, a setting that provides long term e
251 rican species Rana catesbeiana in a cultured tadpole tailfin (C-fin) assay to compare the effluents.
252 ls from adult frog legs or from regenerating tadpole tails do not promote limb regeneration, demonstr
253 scopy is capable of detecting differences in tadpoles that are present in selected ponds with differe
255 s of transcripts between control and exposed tadpoles, there was a significant relationship (r(2) = 0
258 use the optic tectum of awake Xenopus laevis tadpoles to determine how a neuron becomes able to gener
259 We orally exposed Lithobates (Rana) pipiens tadpoles to environmentally realistic levels (0-634 ng/g
260 ngs demonstrate remarkable tolerance of toad tadpoles to extreme metalloid exposure and implicate phy
262 ure model in which we exposed Xenopus laevis tadpoles to pentylenetetrazole (PTZ), a known convulsant
263 he ability of pre-metamorphic Xenopus laevis tadpoles to self-correct malformed craniofacial tissues.
264 The neuronal circuitry that allows frog tadpoles to swim in response to touch is well characteri
265 g the timing of amphibian metamorphosis from tadpoles to tetrapods, through the production and subseq
266 disorders in which we expose Xenopus laevis tadpoles to valproic acid (VPA) during a critical time p
268 aquatic vertebrate, the two-day-old Xenopus tadpole, to investigate how the brainstem and spinal cir
270 By using the intestine of premetamorphic tadpoles treated with or without T3 and for chromatin im
275 expressed in the neural folds, and in early tadpoles undergoing organogenesis gtpbp2 is expressed pr
277 ecies should decrease trematode infection in tadpoles via density- and trait-mediated effects on the
281 n of 1-azidoanthracene to albino stage 40-47 tadpoles was found to immobilize animals upon near-UV ir
282 1-aminoanthracene required to immobilize the tadpoles was significantly increased in the presence of
283 t avoidance assay in freely swimming Xenopus tadpoles, we demonstrate that CB1R activation markedly i
284 g semi-intact preparations of Xenopus laevis tadpoles, we determined the cellular substrate and the s
285 ture and visually evoked Ca(2+) responses in tadpoles, we found that inhibitory neurons cluster into
286 ring regeneration-competent and -incompetent tadpoles, we identified a previously unrecognized cell t
289 ct on embryogenesis because normally feeding tadpoles were formed, consistent with the lack of matern
293 ) effect on Bd growth both in culture and on tadpoles, where low (0.0176-1.76 mug/L) and high (32-176
294 matodes) reduced metacercarial infections in tadpoles, whereas intraguild (IG) predators that consume
295 our developmental strategy to young Xenopus tadpoles, whose brainstem and spinal cord share a core v
296 eover, treatment of regeneration-incompetent tadpoles with immune-suppressing drugs restores myeloid
298 by injecting the tail of Xenopus tropicalis tadpoles with peptide 4.2, a 20-aa sequence derived from