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1 phased small interfering RNA in maize anther tapetal and archesporial cells.
2 merly AtMYB103) regulate genes essential for tapetal and pollen development.
3 ed from male cats were transplanted onto the tapetal area of female cats after native RPE was debride
4   Our work suggested that the pathway of two tapetal-bHLH subfamilies is conserved in all land plants
5 itical role in the BR-mediated regulation of tapetal cell degeneration and pollen development in Sola
6 n and seed development by triggering PCD and tapetal cell degradation.
7 mal and 24-nt meiotic phasiRNAs dependent on tapetal cell differentiation.
8          These lipids are released following tapetal cell disintegration and are relocated to form th
9 tion and inhibits periclinal division once a tapetal cell is specified.
10 equence of the premature degeneration of the tapetal cell layer during microspore development.
11 e 6 anthers and are more concentrated in the tapetal cell layer later.
12 ouble mutant anthers lack development of the tapetal cell layer, which accounts for the microspore ab
13 likely cross-link extensins to contribute to tapetal cell wall integrity during anther development.
14 microspores/ pollen grains (gametophyte) and tapetal cells (sporophyte) of B. napus.
15 tenation in multivesicular endosomes in both tapetal cells and developing pollen grains as well as mo
16 me genes may be expressed in the sporophytic tapetal cells and in gametophytic tissues, they are regu
17 velopment by regulating protein transport in tapetal cells and microspores.
18 mutant anthers display swollen, hypertrophic tapetal cells and pollen grains, suggesting disrupted ce
19  Transgenic plants exhibited GUS activity in tapetal cells and pollen of the developing anthers indic
20                                              Tapetal cells and their mitochondria changed in the volu
21 ds (e.g. flavonols) produced by pollen grain tapetal cells are deposited in the pollen wall.
22                        Unusually for plants, tapetal cells are specified very early in development, a
23 plasma membrane and secreted proteins in the tapetal cells at the free microspore stage, contributing
24 ly from the mitochondria into the cytosol of tapetal cells before the gross morphological changes ass
25  indicated that the protein is secreted from tapetal cells during the early microspore stage.
26  is targeted to the endoplasmic reticulum in tapetal cells in vivo.
27 m at different stages, using isolated single tapetal cells in which the in vivo morphology and volume
28                              The ablation of tapetal cells interferes with pollen production and resu
29 to suppress trans-differentiation of somatic tapetal cells into meiocytes, we find that mac1 anthers
30  not RF2B, accumulates to high levels in the tapetal cells of anthers.
31 ese vacuolar inclusions were not observed in tapetal cells of double mutants of abcg26 and genes enco
32 are localized in microspore mother cells and tapetal cells of meiotic and post-meiotic stage anthers.
33 accumulate to high levels in rapidly growing tapetal cells of pea anthers.
34 morphology beginning at anther stage 4, with tapetal cells that have excess and/or enlarged vacuoles
35 ail to elongate, and there are fewer, larger tapetal cells that retain, rather than secrete, their co
36 ere ABCG26-exported polyketides traffic from tapetal cells to form the sporopollenin backbone, in coo
37 nsport and assembly of exine components from tapetal cells to microspores in the intact anthers of Ar
38 he ago1d mutant predominantly show excessive tapetal cells with little starch accumulation during pol
39  that produces excess microsporocytes, lacks tapetal cells, and abnormally maintains middle layer cel
40 siRNAs, develop short anthers with defective tapetal cells, and exhibit temperature-sensitive male fe
41 g laser capture microdissection, we analyzed tapetal cells, meiocytes and other somatic cells at seve
42 CPPR1 led to abnormal plastid development in tapetal cells, prolonged tapetal programmed cell death (
43 roduction and programmed cell death (PCD) in tapetal cells, resulting in delayed or premature tapetal
44 e differentiation of the microsporocytes and tapetal cells, suggesting that EMS1 mediates signals tha
45 asm in sunflower causes premature PCD of the tapetal cells, which then extends to other anther tissue
46 ned by biphasic protein expression in anther tapetal cells, with an initial peak around pollen meiosi
47 n, accumulated large fluorescent vacuoles in tapetal cells, with corresponding loss of fluorescence o
48 arietal cells then predominantly to daughter tapetal cells.
49 ) are contributed by surrounding sporophytic tapetal cells.
50 localized to endoplasmic reticulum of anther tapetal cells.
51 ollen coat and premature degeneration of the tapetal cells.
52 lationship between the development of ml and tapetal cells.
53  densely stained cytoplasm typical of normal tapetal cells.
54 within the large cytoplasmic lipid bodies of tapetal cells.
55 inated secretory activity of the surrounding tapetal cells.
56 re specifically localised in the interior of tapetal cytoplasmic lipid bodies where they were associa
57 tal cells, resulting in delayed or premature tapetal degeneration, respectively.
58 sgenic for the construct exhibited premature tapetal degradation and subsequent pollen abortion.
59 petum of B. napus anthers and that following tapetal degradation, these proteins, possibly in modifie
60                                              TAPETAL DEVELOPMENT and FUNCTION1 (TDF1) is an essential
61 EMALE FUNCTION) and promoting male function (TAPETAL DEVELOPMENT AND FUNCTION1 [aspTDF1]).
62 and wheat, abiotic stresses lead to abnormal tapetal development resulting in delayed PCD.
63 arnase or the antisense RTS genes interrupts tapetal development, resulting in deformed non-viable po
64  either homo- or heterodimers to choreograph tapetal development.
65 regulated and is required later than MS23 in tapetal differentiation.
66 lix (bHLH) transcription factor required for tapetal differentiation; transcripts localize initially
67  both genes accumulate to high levels in the tapetal (endothelium) layer surrounding the embryo sac.
68 ains, studies on promoter gene regulation of tapetal expressed genes are very few and there are no re
69  identified in the TAPNAC promoter and other tapetal expressed promoters.
70 edicted amino acid sequences of three of the tapetal-expressed oleosin-like genes.
71 llen wall and coat is mainly associated with tapetal function, we used 3D imaging to quantify geometr
72 sport (ESCRT) components, ISTL1 and LIP5, in tapetal function.
73 P1 (ins/ins) eyes show discolouration of the tapetal fundus with varying onset and disease progressio
74 ecific cis regulatory sequences that control tapetal gene expression.
75 igand result in male-sterile plants in which tapetal initials fail to proliferate.
76 uction of late expression of EMS1 in the few tapetal initials in ems1 plants results in their prolife
77 ad normal fundus examination (30%), 10 had a tapetal-like reflex (TLR; 37%), 5 had scattered peripher
78              This unexpected autonomy of the tapetal 'lineage' is discussed in the context of tissue
79 m buds, coinciding with the maximal rates of tapetal lipid accumulation.
80 floating lipid body fraction obtained from a tapetal/locular fluid extract from maturing anthers and
81 s are surrounded by concentric monolayers of tapetal, middle layer and endothecial cells.
82  experiments also show that integrity of the tapetal monolayer is crucial for the maintenance of the
83 ROCYTES 1 (EMS1) in forming the monolayer of tapetal nurse cells in Arabidopsis.
84 ve periclinal divisions, in the ms32 mutant, tapetal precursor cells fail to differentiate, and, inst
85 2-ref mac1-1 double mutant is unable to form tapetal precursors and also exhibits excessive somatic p
86 stid development in tapetal cells, prolonged tapetal programmed cell death (PCD) and tapetum degradat
87 protease (UNDEAD), may control the timing of tapetal programmed cell death (PCD).
88 entified bHLH142 as having a pivotal role in tapetal programmed cell death and pollen development.
89 42 exhibited retarded meiosis and defects in tapetal programmed cell death.
90 he expression of AP37 and AP25, which induce tapetal programmed cell death.
91                      Our results reveal that tapetal siRNAs are sufficient to reconstitute germline m
92                                              Tapetal siRNAs govern germline methylation throughout th
93 rosporangia expand at similar rates and the 'tapetal' space at the periphery of mutant locules become
94 dentify cis regulatory sequences that confer tapetal specific expression.
95 a hairpin RNA construct under the control of tapetal-specific A9 promoter, which was used to generate
96 s specific to SAMDC homologues in anther and tapetal-specific activity of A9 promoter as shown with G
97 nstrated by expressing the pehA gene using a tapetal-specific promoter and treating the mature plants
98  upstream of bHLH51 and bHLH122, controlling tapetal specification and maturation.
99 st twofold more rapidly than normal prior to tapetal specification, suggesting that MAC1 regulates ce
100 wed that 24-nt siRNAs produced in the anther tapetal tissue can methylate male meiocyte genes in tran
101 polyamine biosynthesis, has been targeted in tapetal tissue of tomato using RNAi to examine its effec
102 for the function of CLE19 in maintaining the tapetal transcriptional regulation of pollen exine genes

 
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