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1 of reactive oxygen species than wild-type E. tarda.
2 a, glomerulonephritis, and porphyria cutanea tarda.
3  commoner sporadic form of porphyria cutanea tarda.
4 sis, we isolated a noncytotoxic strain of E. tarda.
5 bility factor for sporadic porphyria cutanea tarda.
6 gh the skin lesions due to porphyria cutanea tarda.
7 ronic viral hepatitis, and porphyria cutanea tarda.
8  and a putative TonB homolog in Edwardsiella tarda.
9 cohol abuse, hepatitis, or porphyria cutanea tarda.
10 (19 patients) and sporadic porphyria cutanea tarda (65 patients).
11 we discovered that the bacteria Edwardsiella tarda activate EECs through the receptor transient recep
12 eria historically classified as Edwardsiella tarda actually represent three genetically distinct yet
13 re mineralogically and texturally similar to Tarda, also have interconnected nano-scale porosity with
14 ics of inherited disorders porphyria cutanea tarda and hereditary coproporphyria, respectively.
15 ives of tryptophan catabolism produced by E. tarda and other gut microbes activates zebrafish EEC Trp
16 he dermatologic lesions in porphyria cutanea tarda, and diagnose and treat the erythropoietic porphyr
17  15 to 18 m(2)/g: the low values compared to Tarda are likely due to the neoformation of pore-blockin
18 defined as typical motile fish-pathogenic E. tarda are synonymous with Edwardsiella piscicida, while
19 nic E. tarda isolates are consistent with E. tarda as it is currently defined.
20 pping to this region, including OFD1 and SED tarda, as well as Aicardi syndrome and Goltz syndrome.
21 ant to acquired (sporadic) porphyria cutanea tarda, because humans show greater than 60-fold genetic
22              Patients with porphyria cutanea tarda benefit from iron depletion, hydroxychloroquine th
23 een the HLA-A3 antigen and porphyria cutanea tarda but not by others.
24                             Two recent falls Tarda (C2-ung) and Aguas Zarcas (CM2) have specific surf
25 ring and photosensitivity: porphyria cutanea tarda; congenital erythropoietic porphyria; hepatoerythr
26                 A cosmid bearing 40 kb of E. tarda DNA, including wild-type copies of the E. tarda ho
27 e non-acute porphyrias are porphyria cutanea tarda, erythropoietic protoporphyria, X-linked protoporp
28                                           E. tarda escapes from the endocytic vacuole within minutes
29                   Familial porphyria cutanea tarda (f-PCT) results from the half-normal activity of u
30 ah pedigrees with familial porphyria cutanea tarda (F-PCT).
31 In this study, we report that the T3SS of E. tarda facilitates its survival and replication in murine
32 lement, suggesting that they arose in the E. tarda genome by horizontal transfer.
33  effective in vitro repression of the MDR E. tarda growth for approximately four hours, pH resistance
34                            Porphyria cutanea tarda has been described in adult hemodialysis patients.
35   The type III secretion system (T3SS) of E. tarda has been identified as a key virulence factor that
36 ty is due to the previously characterized E. tarda hemolysin activity.
37 da DNA, including wild-type copies of the E. tarda homologs of the transporter-activator protein and
38                  A case of porphyria cutanea tarda in an adult ESRD patient treated with continuous a
39      A proper diagnosis of porphyria cutanea tarda in hemodialysis patients requires fractionation of
40  prevent and treat the pathogen Edwardsiella tarda in striped catfish Pangasianodon hypophthalmus has
41        The pathogenesis of porphyria cutanea tarda in this population is thought to be related to the
42  41 patients with sporadic porphyria cutanea tarda, in whom the frequency of microsatellite alleles t
43  genotypes in each type of porphyria cutanea tarda indicated that C282Y homozygosity is an important
44  present late in life with porphyria cutanea tarda, indicating that not all homozygotes present clini
45         We propose that the host controls E. tarda infection partially by detecting FliC translocated
46 rine bone marrow-derived macrophages, and E. tarda infection triggers pyroptosis of infected macropha
47                            Porphyria cutanea tarda is a disorder of heme biosynthesis resulting from
48                                 Edwardsiella tarda is a Gram-negative enteric pathogen that causes he
49                                 Edwardsiella tarda is a Gram-negative enteric pathogen that causes he
50                            Porphyria cutanea tarda is a liver disease characterized by elevated hepat
51                   Sporadic porphyria cutanea tarda is a skin disease associated with hepatic siderosi
52                            Porphyria cutanea tarda is a skin disease caused by photosensitization by
53                                 Edwardsiella tarda is an enteric pathogen that causes diarrhea, wound
54 wardsiella anguillarum Fish-nonpathogenic E. tarda isolates are consistent with E. tarda as it is cur
55  while atypical nonmotile fish-pathogenic E. tarda isolates are equivalent to Edwardsiella anguillaru
56 st report of novel lytic phages infecting E. tarda isolates in striped catfish Pangasianodon hypophth
57 EtA_WCT72P1, and vB_EtA_DT115P1 infecting E. tarda isolates, with their geographical origins from Mek
58 id emergence of multidrug-resistant (MDR) E. tarda isolates.
59  the frequency in sporadic porphyria cutanea tarda of mutations (Cys282Tyr, His63Asp) in a novel MHC
60 mbers of 530 patients with porphyria cutanea tarda (PCT) and 296 with acute intermittent porphyria (A
61 n is known to enhance both porphyria cutanea tarda (PCT) and experimental uroporphyria.
62 Two major risk factors for porphyria cutanea tarda (PCT) are alcohol consumption and homozygosity for
63 ors for the development of porphyria cutanea tarda (PCT) are alcohol consumption and homozygosity for
64 me, but not all countries, porphyria cutanea tarda (PCT) has been associated with chronic infection w
65 overload that is common in porphyria cutanea tarda (PCT) is uncertain.
66 ted in the pathogenesis of porphyria cutanea tarda (PCT), a disorder characterized by a photosensitiv
67                            Porphyria cutanea tarda (PCT), a liver disease with skin lesions caused by
68                            Porphyria cutanea tarda (PCT), the most common form of porphyria in humans
69 one-third of patients with porphyria cutanea tarda (PCT), the most common porphyria in humans, inheri
70 ittent porphyria (AIP) and porphyria cutanea tarda (PCT).
71 is common in patients with porphyria cutanea tarda (PCT).
72  hepatic manifestations of porphyria cutanea tarda (PCT).
73 rm of porphyria in humans, porphyria cutanea tarda (PCT).
74 orders involving the skin (porphyria cutanea tarda [PCT], vitiligo, and lichen planus); renal (membra
75        However, the specific surface area of Tarda ranges from 33.7 to 81.6 m(2)/g depending on outga
76    Deletion of the flagellin gene fliC of E. tarda results in decreased cytotoxicity for infected mac
77 lanus, Sjogren's syndrome, porphyria cutanea tarda, rheumatoid-like arthritis, or depression.
78                 Spondyloepiphyseal dysplasia tarda (SEDL) is a genetically heterogeneous disorder cha
79 dlin gene cause spondyloepiphyseal dysplasia tarda (SEDT), a rare X-linked chondrodysplasia.
80 high sequence similarity with Orf29/30 of E. tarda strain EIB202, and is renamed EseJ.
81 c bacterial pathogens including Edwardsiella tarda, Streptococcus iniae, Yersinia ruckeri, Flavobacte
82                                          The Tarda surface area is dominated by an interconnected net
83 both familial and sporadic porphyria cutanea tarda, the effect being more marked in familial porphyri
84 the most common porphyria, porphyria cutanea tarda, the identification of an X-linked form of erythro
85 s leukemia and concomitant porphyria cutanea tarda was admitted to the hospital for consolidation che
86                   Familial porphyria cutanea tarda was identified by mutational analysis of the uropo
87 ng more marked in familial porphyria cutanea tarda where anticipation was demonstrated in family stud
88      Efforts to study the interactions of E. tarda with tissue culture cells are complicated by the p
89 le to follow the entry and replication of E. tarda within tissue culture host cells.