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1 eliorated by treatment with the ER chaperone tauroursodeoxycholic acid.
2 oxybutyrate, or the anti-apoptotic bile acid tauroursodeoxycholic acid.
3 t fiber, which was inversely associated with tauroursodeoxycholic acid.
4 by PLIN2OE or using the ER stress inhibitor, tauroursodeoxycholic acid.
5                               Treatment with tauroursodeoxycholic acid, a chemical chaperone drug tha
6              Finally, oral administration of tauroursodeoxycholic acid, a chemical chaperone that inh
7 of protein folding by the chemical chaperone tauroursodeoxycholic acid, a hydrophilic bile salt, prev
8 ion with increasing amounts of the bile salt tauroursodeoxycholic acid, Abcg5 became fully rate-limit
9 pregnant females with the chemical chaperone tauroursodeoxycholic acid alleviated ER stress, restored
10 cal chaperones, such as 4-phenylbutyrate and tauroursodeoxycholic acid, also attenuated ER Ca(2+) dep
11 d patients with IPF, and ER stress inhibitor tauroursodeoxycholic acid ameliorates ER stress and fibr
12                                              Tauroursodeoxycholic acid and taurochenodeoxycholic acid
13 ER) stress was inhibited by pharmacological (tauroursodeoxycholic acid) and genetic (overexpression o
14 ical chaperones, sodium 4-phenylbutyrate and tauroursodeoxycholic acid, both with chaperone propertie
15 ved by combining d-beta-hydroxybutyrate with tauroursodeoxycholic acid but not with probucol.
16 n contrast to taurochenodeoxycholic acid and tauroursodeoxycholic acid, GCDCA induced apoptosis in a
17                                      PBA and tauroursodeoxycholic acid improved ER stress indexes in
18  treatment with the ER stress-relieving drug tauroursodeoxycholic acid improved metabolic and neurode
19 ugh induction of microRNA (miR)-34a, whereas tauroursodeoxycholic acid induced SIRT1 expression witho
20 mpanied by reduced glycodeoxycholic acid and tauroursodeoxycholic acid levels.
21  especially in glomeruli, and suggested that tauroursodeoxycholic acid might be useful for the early
22 ereas application of the chemical chaperones tauroursodeoxycholic acid or 4-phenylbutyric acid allevi
23 nt of PCOS mice with an ER stress inhibitor, tauroursodeoxycholic acid or BGP-15, decreased interstit
24 ingly, cells exposed to ER stress inhibitor, tauroursodeoxycholic acid partially mitigated all these
25  acid, serotonin, short chain fatty acids or tauroursodeoxycholic acid shows a similar effect to IF i
26 posed to glycochenodeoxycholic acid (GCDCA), tauroursodeoxycholic acid, taurochenodeoxycholic acid, a
27 nhibition of ER stress, by administration of tauroursodeoxycholic acid to control mice, prevented upr
28    Administration of the chemical chaperone, tauroursodeoxycholic acid, to Eif2s1(+/)(tm1RjK) heteroz
29                     Small chemical chaperone tauroursodeoxycholic acid treatment of Pak2-CKO mice sub
30                                              Tauroursodeoxycholic acid treatment significantly improv
31                                     Finally, tauroursodeoxycholic acid treatments were used to demons
32 n vivo is converted to its taurine conjugate tauroursodeoxycholic acid (TUDC), is a mainstay for the
33 olic acid (TCA), glycocholic acid (GCA), and tauroursodeoxycholic acid (TUDCA) all activated ERK1/2 i
34                          Here, we pinpointed tauroursodeoxycholic acid (TUDCA) as an efficient therap
35 ob/ob mice with the ER stress-relieving drug tauroursodeoxycholic acid (TUDCA) causes long-term ameli
36      Intriguingly, attenuation of the UPR by tauroursodeoxycholic acid (TUDCA) diminishes Mst1/2 muta
37                      Recently, the bile acid tauroursodeoxycholic acid (TUDCA) has been shown to have
38 pecies glycoursodeoxycholic acid (GUDCA) and tauroursodeoxycholic acid (TUDCA) have long been known t
39                      We assessed the role of tauroursodeoxycholic acid (TUDCA) in inhibition of caspa
40 ed by co-infusion of the ER stress inhibitor tauroursodeoxycholic acid (TUDCA) into the lateral cereb
41   UDCA is conjugated to taurine in vivo, and tauroursodeoxycholic acid (TUDCA) is a potent hepatocell
42                                              Tauroursodeoxycholic acid (TUDCA) is considered an artif
43  expression in jck mice by administration of tauroursodeoxycholic acid (TUDCA) or tolvaptan impeded t
44 hat acute inhibition of brain ER stress with tauroursodeoxycholic acid (TUDCA) partially reversed obe
45 virus, we found that the molecular chaperone tauroursodeoxycholic acid (TUDCA) significantly inhibits
46           Moreover, the choleretic effect of tauroursodeoxycholic acid (TUDCA) was impaired in InsP(3
47 es to insulin resistance, and treatment with tauroursodeoxycholic acid (TUDCA), a bile acid derivativ
48 is, we investigated the protective effect of tauroursodeoxycholic acid (TUDCA), a bile acid, on ER st
49                                              Tauroursodeoxycholic acid (TUDCA), a chemical chaperone
50 have previously reported that treatment with tauroursodeoxycholic acid (TUDCA), a hydrophilic bile ac
51 odel of progressive DN and treated mice with tauroursodeoxycholic acid (TUDCA), a specific inhibitor
52 wo well-established TGR5 agonists, RG239 and tauroursodeoxycholic acid (TUDCA), acutely promoted stim
53                                              Tauroursodeoxycholic acid (TUDCA), an endogenous bile ac
54           TCA, taurodeoxycholic acid (TDCA), tauroursodeoxycholic acid (TUDCA), glycocholic acid (GCA
55 ne the efficacy of an ER chemical chaperone, tauroursodeoxycholic acid (TUDCA), in preserving cones i
56 in vitro effects of an endogenous bile acid, tauroursodeoxycholic acid (TUDCA), on astrocyte and micr
57                            Administration of tauroursodeoxycholic acid (TUDCA), which reportedly inhi
58 mical chaperones sodium 4-phenylbutyrate and tauroursodeoxycholic acid were found to reduce tunicamyc
59 ss signaling, like 4-phenylbutyrate (PBA) or tauroursodeoxycholic acid, will inhibit the disruption o