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1  and CCCTC-binding factor (CTCF) in anaphase/telophase.
2 c spot that persists until the completion of telophase.
3 d transiently released in early anaphase and telophase.
4 pindle during anaphase and to the midzone at telophase.
5 ed at the midline during late anaphase/early telophase.
6 ome earlier and accumulated gradually during telophase.
7 d at the spindle midzone during anaphase and telophase.
8 ganization of SC35 is restored subsequent to telophase.
9 d in the zone of microtubule overlap late in telophase.
10 the proper spacing of daughter nuclei during telophase.
11  anaphase, leading to segregation defects at telophase.
12 hase transition and closed 90 s later during telophase.
13 concentrates near the cleavage furrow during telophase.
14 ther, robust silencing is not observed until telophase.
15 coincident with nuclear envelope assembly in telophase.
16 osophila embryos, actin caps assemble during telophase.
17 d to kinetochores from late prophase to late telophase.
18 ntry of the proteins into the nucleus during telophase.
19 eentry into the newly formed nucleus in late telophase.
20  the cleavage furrow during prophase through telophase.
21 ase, and localizes to the spindle midbody at telophase.
22 ly the phragmoplast during late anaphase and telophase.
23 es with midzone microtubules in anaphase and telophase.
24 nd to the cleavage furrow and midbody during telophase.
25 t the onset of M-phase and reassemble during telophase.
26 e structures then appear to fuse together by telophase.
27 ng prometaphase and back to the chromatin in telophase.
28 re most likely carried through anaphase into telophase.
29 with the cleavage furrow during anaphase and telophase.
30 hores in M phase cells from late prophase to telophase.
31 es gradually diminishes, and is gone by late telophase.
32  were significantly longer than wild type at telophase.
33 d foci (NDF) between early anaphase and late telophase.
34  and immediately resumes full binding in ana/telophase.
35  midzone and the midbody during anaphase and telophase.
36 y region of the spindle in late anaphase and telophase.
37 as cells progress through anaphase and begin telophase.
38 nd remains centromere associated until after telophase.
39 usters in the period from metaphase to early telophase.
40 at the midzone of these same spindles during telophase.
41 itosis, is relocalized to the midbody during telophase.
42 and clustered near the poles in anaphase and telophase.
43 he volume of the equator during anaphase and telophase.
44 ormalities and micronucleus formation during telophase.
45 metaphase and translocated to the midbody at telophase.
46 and myosin-IIB form a complex in vivo during telophase.
47  at the reassembling nuclear envelope during telophase.
48 es perpendicular to the division site during telophase.
49 ssion during metaphase and separation during telophase.
50 es and localized to the division site during telophase.
51 c phases, including metaphase, anaphase, and telophase.
52 e, peaks at metaphase, and decreases through telophase.
53 nding nucleoplasm before leaving in anaphase/telophase.
54 vated during nuclear reformation in anaphase/telophase.
55 iled abscission of the cleavage furrow after telophase.
56 th by transiently elongating during anaphase/telophase.
57 rometaphase, but not metaphase, anaphase and telophase.
58  cortical dynein, followed by a reduction in telophase.
59 r entry into prophase and that it resumes in telophase.
60 metaphase, and then quickly disappears after telophase.
61 ion zone that forms between sister-asters in telophase.
62 process of chromosome decondensation at late telophase.
63 recedes formation of nuclear speckles during telophase.
64 re-associates with chromatin during anaphase/telophase.
65 se and remains excluded from DNA until early telophase.
66 res in early mitosis and near the spindle in telophase.
67 ) breaks down at prophase and reassembles at telophase.
68 broad, mixed cell-type transcriptomes before telophase.
69 zone during anaphase and the mid-body during telophase.
70 fects at metaphase, but these are rescued by telophase.
71 aberrant microtubule bridges during anaphase/telophase.
72 s its kinase activity from metaphase through telophase.
73 rects erroneous anaphase orientations during telophase.
74 with DNA replication, and dissociates by the telophase.
75 es in anaphase, and chromatin bridges during telophase.
76 ondensation was prematurely lost in anaphase/telophase.
77  in the cancer cells during meta-, ana-, and telophases.
78                                At the end of telophase, a portion of Cdc15p is located at the mother-
79                                       During telophase, a significant amount of Plk3 was also detecte
80                                           At telophase, active MAP kinase localized at the midbody.
81           We have found that Scc1 returns in telophase after DSBs and that it partially reconstitutes
82 rosomes and spindle poles from interphase to telophase and at the midbody during cytokinesis.
83  We find that CID assembly initiates at late telophase and continues during G1 phase in somatic tissu
84 toplasm and at the spindle poles, and during telophase and cytokinesis stimulated PSKs are present in
85                                       During telophase and cytokinesis, many Golgi stacks redistribut
86 ndle microtubules, where it remained through telophase and cytokinesis.
87 bidopsis and rice cells undergoing anaphase, telophase and cytokinesis.
88 s detected weakly on midbody microtubules at telophase and cytokinesis.
89  at the contractile ring and mid-body during telophase and cytokinesis.
90  prophase, reassociating again at the end of telophase and cytokinesis.
91 in cell cycle stages other than anaphase and telophase and Dbf2 kinase was prematurely active during
92  a lesser degree, Mcm2 onto chromatin during telophase and early G1 when Mcm2-7 are normally recruite
93 igh DII levels were observed in cells during telophase and early G1, suggesting that low auxin signal
94 xpansion during chromosome decondensation in telophase and early G1.
95 it fusome material to spindle midbodies near telophase and for normal fusome organization.
96                                           In telophase and G1 unbudded cells, no localization was obs
97 ar bodies (PNBs) appeared in nuclei in early telophase and gradually disappeared as nucleoli formed,
98 shed staining of anaphase and no staining of telophase and interphase centrosomes.
99 ained high near the equatorial plane through telophase and into cytokinesis, whereas the phosphorylat
100 rtment conformation forms transiently during telophase and is subsequently modulated by a second fold
101 re, we show that FIP3 binds to Cyk-4 at late telophase and that centralspindlin may be required for F
102 pindle midzone/cleavage furrow (anaphase and telophase), and midbody (cytokinesis) during cell divisi
103 re both localized on midbody microtubules at telophase, and also interacted with each other during mi
104 cleavage furrow and midbody during anaphase, telophase, and cytokinesis, implicating a role in the co
105 ase-anaphase transition, impede anaphase and telophase, and impair a cell's ability to arrest in G1 o
106 tubules in interphase and the midbody during telophase, and its protein levels decrease as cells exit
107 s and spindle poles during metaphase through telophase, and partially co-localized with chromatin dur
108 ation of Runx foci is completely restored in telophase, and Runx proteins are equally partitioned int
109 ated Exocyst and ESCRT machinery during late telophase, and therefore that these two distinct facets
110 cm proteins with chromatin took place during telophase, approximately 30 min after the destruction of
111  is believed to be the result of a prolonged telophase arrest that has been recently identified in RN
112 llowing the change in centromere position in telophase-arrested cells upon depolymerization and subse
113 hase chromosome state is formed and identify telophase as a critical transition between condensin- an
114 ndle during mitosis, in perichromatin during telophase, as well as in the midbody during cytokinesis.
115                     cdc5-1 mutants arrest at telophase at the nonpermissive temperature due to the fa
116 ific displacement of H2A.Z from chromatin in telophase-blocked cells, regardless of the silencing sta
117                                       During telophase both isoforms colocalized to the contractile r
118 rest in interphase, prophase, metaphase, and telophase but not anaphase.
119 phase chromosomes and bind to chromosomes in telophase but not in metaphase.
120                             Cells blocked in telophase (but not at metaphase) are also able to establ
121 ated suppression of LATS1 or MOB1A prolonged telophase, but had no effect on the length of the earlie
122 s and appeared in prenucleolar bodies during telophase, but it did not colocalize with p80-coilin in
123 e regulated, peaking in metaphase-anaphase B/telophase, but its function remains unknown.
124  CHMP7 license its assembly at the NE during telophase, but restrict its assembly on the ER at this t
125 Transcription is known to restart in bulk by telophase, but whether de novo transcription at the mito
126 interactions are not established until after telophase, by which time the nuclear envelope has reasse
127 ent in unbudded G1 cells, as well as in late telophase cells after spindle disassembly.
128 nt of ESCRT-I/II complexes to the midbody of telophase cells initiates ESCRT-III assembly into two ri
129 s of prolonged mitosis, we isolated anaphase-telophase cells that were just finishing a mitosis of no
130  centromere protein (INCENP) in anaphase and telophase cells.
131  explaining how cohesin can be reloaded onto telophase chromatin in the absence of securin and cyclin
132 gous chromosomes in paired late-anaphase and telophase chromosomal masses were highly correlated.
133                                         This telophase chromosome movement suggests that the surface
134 ked chromosome decondensation, and inhibited telophase chromosome movement.
135 d a small pool of cohesin complexes bound to telophase chromosomes in wild-type cells and show that t
136 t pBAF is associated with the core region of telophase chromosomes.
137  cytoplasm, partition stochastically, and in telophase coalesce to generate a functionally and struct
138                                           In telophase, colocalization of growing microtubules ends f
139 us and colocalize again with the DNA in late telophase, concomitantly with the appearance of the nucl
140 ays and immunofluorescence, we show that, by telophase, condensin-mediated loops are lost and a trans
141 erine 19 phosphorylation during anaphase and telophase, consistent with an activating phosphorylation
142                              The fidelity of telophase correction also relies on the tension-sensitiv
143  division orientation plasticity provided by telophase correction may enable progenitors to adapt to
144 nding protein 1 (53BP1) are absent until the telophase/cytokinesis stage.
145  suppressed in mitotic mammalian cells until telophase/cytokinesis.
146 ates the inclusion of lagging acentrics into telophase daughter nuclei.
147 e-segregating acentric chromosomes enter the telophase daughter nucleus.
148                                   Second, in telophase, decondensing chromosomes often moved rapidly
149 bility, but imp1delta mutant cells exhibit a telophase delay and mild temperature-sensitive lethality
150 tribution of midzone microtubule bundles and Telophase Disc 60 protein (TD60) rather than the positio
151  activation in vitro requires two cofactors, telophase disc-60kD (TD-60) and microtubules.
152 STB association lasts from G1/S through late telophase during the cell cycle.
153 aughter nuclei and the nucleolus during late telophase/early G1.
154 vels in cortices prepared from late anaphase/telophase embryos.
155 r RENT (regulator of nucleolar silencing and telophase exit) complex at the rDNA region.
156 e RENT (regulator of nucleolar silencing and telophase exit) silencing complex, and Fob1, which recru
157 d RENT (regulator of nucleolar silencing and telophase exit).
158 d RENT (regulator of nucleolar silencing and telophase exit).
159 d RENT (regulator of nucleolar silencing and telophase exit).
160  kinase (DDK) accumulates at kinetochores in telophase, facilitated by the Ctf19 kinetochore complex.
161  of microtubule dynamics, and no anaphase or telophase figures were observed.
162 at successfully proceed through anaphase and telophase, forming two daughter nuclei separated by a mi
163 ansition from early to late anaphase, and by telophase FP-PP1gamma also accumulates at the cleavage f
164  in "anaphase," and bundling into arrays in "telophase." Furrow induction usually occurs at multisite
165 n kinase that plays an important role in the telophase/G1 transition.
166 the role of DBF2 and MOB1 in controlling the telophase/G1 transition.
167  to abnormally high AIR-2 levels at the late telophase/G1 transition.
168     These factors localize to centromeres in telophase/G1, when new CENP-A chromatin is assembled.
169                                           In telophase, GM130 is dephosphorylated as the Golgi fragme
170                                       During telophase, Golgi cisternae are regenerated and stacked f
171                       Cells in metaphase and telophase have no detectable focus.
172                                       Around telophase, Hof1p is phosphorylated and the double rings
173                                           In telophase, hRif1 localized to chromosomes, and in interp
174  perpendicular, to the ooplasmic membrane at telophase I, and some oocytes extruded the entire spindl
175 servable defect occurs in microsporocytes at telophase I, where some chromosomes are scattered throug
176 he formation of radial microtubule arrays at telophase II and consequently leads to defects in postme
177 vated in vivo oocytes were enucleated at the telophase II stage, electrofused with donor somatic cell
178 disrupts the radial microtubule system after telophase II, and affects the proper establishment of nu
179 le as it transits into anaphase II and later telophase II, becoming associated with the midzone micro
180  was undetectable from late prophase I until telophase II.
181 progressed to pronuclear, MIII, and anaphase/telophase III stages.
182 are rapidly imported into daughter nuclei in telophase, immediately bind chromosomes as individual co
183 se, whereas nucleation remained high through telophase, implying the presence of additional regulator
184 slow cleavage-furrow ingression during early telophase in dividing spermatocytes.
185 ision, chromosomes from prometaphase through telophase in human RPE1 cells.
186  while it was concentrated at the midbody in telophase in meiotic oocytes.
187 the beginning of mitosis and reforms at late telophase in the daughter nuclei.
188  from the division site during metaphase and telophase in the tan1 air9 mutant.
189 ng early mitosis and defective reassembly at telophase, increased formation of multiple spindle poles
190  specifically in the cleavage furrow late in telophase independent of contractile ring constriction.
191  rapidly and irreversibly disassemble during telophase is less clear.
192         This increase of H3K27ac in anaphase/telophase is required for posttranscriptional activation
193 lation persists until spindle disassembly in telophase, is increased in PPP6C knockout cells, and is
194 ules show strong KRIT1 staining and, in late telophase, KRIT1 stains the midbody remnant most strongl
195 ith the nascent nuclear envelope (NE) during telophase, lamin C remains in the interior, surrounding
196 specific uncoiling of mitotic chromosomes in telophase, large-scale reorganization of interphase chro
197 he role of residual cohesin in DSB repair in telophase (late mitosis).
198 se stable binding of Mis18 to centromeres in telophase licenses them for CENP-A deposition.
199 y, An-Mad1 and An-Mlp1 redistribute from the telophase matrix and associate with segregated kinetocho
200 ssociation of ECT2 from the mid-body at late telophase may be required for the recruitment of FIP3 an
201                             We show that, in telophase, MCM2 and MCM4 maintain transient interactions
202 mammalian pre-RC assembly takes place during telophase, mediated by post-translational modifications
203 ragmoplast reaches the cell cortex, cortical-telophase microtubules are incorporated into the phragmo
204 last reaches the cell cortex, these cortical-telophase microtubules transiently interact with the div
205 dles and form distinct bands associated with telophase midbodies.
206 , cytokinesis was oriented transverse to the telophase mitotic array and was less well aligned with t
207                                           At telophase, MPP10 was found in cellular structures that r
208                              At anaphase and telophase, myosin II moves to the cleavage furrow and ap
209 n regulating anaphase spindle elongation and telophase nuclear positioning via inhibition of Klp2, a
210                                              Telophase nuclei also played a predominant role in posit
211                        Microtubules from the telophase nuclei interdigitated throughout the plane of
212     The reentry of processing complexes into telophase nuclei is suggested by the presence of pre-rRN
213                                              Telophase nuclei were competent for transcription and pr
214              In zygotes developing normally, telophase nuclei were positioned parallel to the polar g
215 patial correlations between the growth axis, telophase nuclei, and the division plane were analyzed i
216 d structures, similar to micronuclei, during telophase of a DNA elimination mitosis.
217 mbiguously showing that from prometaphase to telophase of mammalian cells, most of the ER is organize
218 kinase signaling in one daughter cell during telophase of mitosis.
219 rrested eggs and because CENP-E reappears in telophase of mouse oocytes activated in the absence of p
220 otic cells must form a single nucleus during telophase or exclude part of their genome as damage-pron
221 RE contacts that are normally resolved after telophase persist deeply into G1-phase in CTCF-depleted
222 r invagination channels at late prophase and telophase, potentially suggesting roles for such channel
223 olgin-positive acceptor compartment in early telophase preceded the accumulation of a Golgi glycosylt
224          Notably, gene domains emerge in ana/telophase prior to completion of the first round of tran
225                                       During telophase, pY ERK associates with newly formed Golgi ves
226 e and continued to rise through anaphase and telophase, reaching a maximum of 7 times interphase rate
227           Loss of Clp1 from the cytoplasm in telophase renders cells sensitive to perturbation of the
228 embly and Cse4 exit during anaphase and late telophase, respectively.
229 its the degradation of mitotic cyclins after telophase, resulting eventually in cell death.
230 aphase bridges were observed to persist into telophase, resulting in chromosomal exclusion from the r
231 normal anaphase but then develop an abnormal telophase spindle and fail to undertake cytokinesis.
232 ced--relies on asymmetric positioning of the telophase spindle midzone, which specifies the cleavage
233 uired both to establish the structure of the telophase spindle to provide a framework for the assembl
234 addition, loss of ASE1 function destabilized telophase spindles, and expression of a nondegradable As
235 nvelope (e.g. metaphase, anaphase, and early telophase stages), these ARPs were excluded from the con
236                                  At anaphase/telophase, staining shifted to the midbody and the inter
237  associates with decondensing chromosomes in telophase, suggesting a role for YY1 in early marking of
238 levels that are elevated during anaphase and telophase, temporally correlating with H3-K9 acetylation
239 tacts between cis-regulatory elements in ana/telophase that are dissolved upon G1 entry, co-incident
240 61F gains fusome-dependent interactions near telophase that mediate its incorporation into these stru
241 NT (for regulator of nucleolar silencing and telophase), that also contains Cdc14 and the silencing r
242                                       During telophase the NDF disappeared with a concomitant appeara
243      Brd4 binding to M/G1 genes increased at telophase, the end phase of mitosis, coinciding with inc
244                                      In late telophase, the Golgi ribbon began to be reformed by a dy
245                                       During telophase, the nuclear envelope (NE) reforms around daug
246                                           In telophase, the staining shifted from the centrosomes to
247  the division plane were not observed before telophase; the earliest division marker detected was a p
248 ly- and late-replicating chromatin from late telophase throughout G1-phase.
249 increased rapidly during the transition from telophase to cytokinesis, whereas cell volume increased
250 released from the nucleolus at late anaphase/telophase to dephosphorylate important regulators of Cdc
251 s required for cell cycle progression at the telophase to G1 cell cycle transition.
252 uired to maintain nuclear spacing during the telophase to interphase transition.
253 NGLED1 organize cortical microtubules during telophase to mediate phragmoplast positioning at the fin
254                    Midbodies function during telophase to regulate the abscission step of cytokinesis
255 ning, we show that LGN also functions during telophase to reorient oblique divisions toward perpendic
256 neighboring nonsister centrosomes during the telophase-to-interphase transition of the cortical divis
257 e, histone acetylation, and CTCF in anaphase/telophase, transcription in cytokinesis, and long-range
258 hat accumulate at the cell cortex during the telophase transition in maize (Zea mays) leaf epidermal
259 ciated from chromatin during the anaphase-to-telophase transition, coincident with the dissociation o
260                          During the anaphase-telophase transition, lamin B1 begins to become concentr
261 from poles to midzone during the anaphase-to-telophase transition.
262 y the formation of four colinear clusters at telophase, two per daughter cell.
263 ould segregate their lytic components during telophase, using imaging flow cytometry, confocal micros
264                         During metaphase and telophase, we suggest that its activity is downregulated
265 ic Golgi fragments, seen in prometaphase and telophase, were found to localize adjacent to endoplasmi
266 ession until some point during late anaphase/telophase when it is rapidly dephosphorylated.
267 , a putative methyl transferase, only during telophase when rDNA gene transcription and pre-rRNA meth
268 similarly on, and around, kinetochores until telophase when they transiently localize near the spindl
269 ome, are recruited to the cell plate at late telophase, when primary PD are formed, and remain associ
270 en rDNA transcription ceases, and reforms in telophase, when rDNA transcription resumes.
271 nexin 11 is recruited to the midbody in late telophase, where it forms part of the detergent-resistan
272 alizes on chromosomes from metaphase through telophase, whereas Ser-988-phosphorylated BRCA1 resides
273        OGT colocalized to the midbody during telophase with Aurora B.
274 ic entanglements are removed during anaphase/telophase, with remaining ones removed during early G1,
275 ccumulated in nuclei in late anaphase and in telophase, with the exception of a pool of cIAP1 that as

 
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