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1 y ) was performed (diagnostic standard [ TAB temporal artery biopsy ]).
2 nts suspected of having GCA should undergo a temporal artery biopsy.
3 Vascular tissue was obtained at the time of temporal artery biopsy.
4 pares the use of invasive procedures such as temporal artery biopsy.
5 erpes zoster antigen was detected on several temporal artery biopsies.
6 l in predicting the likelihood of a positive temporal artery biopsy among patients with a clinical su
7 tion of biochemical markers of inflammation, temporal artery biopsy and positron emission tomography/
8 genesis of the disease but have not replaced temporal artery biopsy as the gold standard for securing
10 od (each p < .001); that of enucleations and temporal artery biopsies decreased significantly 38- and
17 iopsy-positive GCA underwent two consecutive temporal artery biopsies, one prior to therapy and one w
18 cent of the control samples were obtained by temporal artery biopsy performed within 1 year of the bi
19 rd to age, frequency of positive findings on temporal artery biopsy (placebo 87%, MTX 79%), or comorb
22 by angiography and 74 control patients with temporal artery biopsy-proven GCA without large vessel i
25 owed a significant association of VZV DNA to temporal artery biopsy samples positive for GCA compared
32 giant cell arteritis -negative results ( TAB temporal artery biopsy subcohort and total study cohort,
34 ers, with good interobserver agreement ( TAB temporal artery biopsy subcohort, kappa = 0.718; total s
36 comparison with the diagnostic standard TAB temporal artery biopsy ( TAB temporal artery biopsy subc
43 ella-zoster virus antigen) was detectable in temporal artery biopsies taken from individuals with gia
44 ts (98 of 185), temporal artery biopsy ( TAB temporal artery biopsy ) was performed (diagnostic stand
45 ted GCA was examined in peripheral blood and temporal artery biopsies with protein quantification ass
46 ents aged >=50 years with biopsy-proven GCA (temporal artery biopsy within 2 weeks of diagnosis and >