ライフサイエンスコーパス: temporal lobe

1 052 +/- 0.0020 mm/year, P = 0.0006) anterior temporal lobe.

2 both visual association areas and the medial temporal lobe.

3 prominent in the fusiform gyrus and lateral temporal lobe.

4 lateral and localized beyond the ipsilateral temporal lobe.

5 pplied a virtual lesion to the left anterior temporal lobe.

6 statement of neural activity in the anterior temporal lobe.

7 sociated with faster rates of atrophy in the temporal lobe.

8 e visual cortex, fusiform gyrus, and lateral temporal lobe.

9 reas first and then propagates to the medial temporal lobe.

10 own of the BBB in the hippocampus and medial temporal lobe.

11 ay was associated with ripples in the medial temporal lobe.

12 by progressive atrophy of the frontal and/or temporal lobes.

13 y matter tissue density changes in bilateral temporal lobes.

14 rostructure in tracts connecting frontal and temporal lobes.

15 progressive degeneration in the frontal and temporal lobes.

16 Sylvian fissure of the frontal, parietal and temporal lobes.

17 ed architecture that spans the occipital and temporal lobes [1-14], where visual categorizations unfo

18 with the strongest modulation in the medial temporal lobe (33 of 416) and in particular the right hi

19 Although single neurons in the medial temporal lobe activate to represent locations in the env

20 with focal neurodegeneration in the anterior temporal lobe, affecting primarily the ventral, occipito

21 rease in gamma spectral power over bilateral temporal lobes after stimulation.

22 d functional significance of less accessible temporal-lobe alpha activity we analyzed responses to se

23 e as debilitating as the more commonly known temporal lobe amnesia, yet the precise contribution of d

24 principally responsible for diencephalic and temporal lobe amnesia.

25 s of structural abnormalities in frontal and temporal lobes, amygdala, and insula are less consistent

26 relationships (i.e. episodic memory: medial temporal lobe and angular gyrus; semantic memory: left a

27 ar implant, the patient underwent MRI of the temporal lobe and brain ( Figs 1 - 4 ) and thin-section

28 ar implant, the patient underwent MRI of the temporal lobe and brain and thin-section CT of the tempo

29 lving the orbitofrontal cortex, inferomedial temporal lobe and cerebellum.

30 etween rather than within the visual, medial temporal lobe and default mode networks, whereas during

31 actions underlie the common core symptoms of temporal lobe and diencephalic anterograde amnesia.SIGNI

32 We examine intracranial EEG in the human temporal lobe and find robust alpha oscillations that ar

33 apidly peaking activity in the left anterior temporal lobe and later engagement of the medial prefron

34 The representational formats of medial temporal lobe and neocortex are sufficient to determine

35 types that have been described in the medial temporal lobe and posterior parietal cortex, discuss the

36 In OA, FTP binding increased in bilateral temporal lobe and retrosplenial cortex, accompanied by a

37 y in women is not just limited to the medial temporal lobe and significantly contributed to greater r

38 l regions; language: left posterior superior temporal lobe and supramarginal gyrus; executive functio

39 s (the 'peak clusters') were in the anterior temporal lobe and the precentral gyrus.

40 mpared to TD participants in the frontal and temporal lobes and several sub-lobar regions previously

41 ach PET ligand: one weighted to the anterior temporal lobe, and another weighted to posterior temporo

42 mposition by the cerebral hemispheres, short temporal lobe, and long sylvian fissure.

43 [0.11]), less severe atrophy of the lateral temporal lobe, and lower mean (SD) cerebrospinal fluid l

44 ial reward prediction signals in the insula, temporal lobe, and prefrontal cortex, while DA depletion

45 NCE STATEMENT Subregions in the human medial temporal lobe are critically involved in episodic memory

46 Thus, two temporal lobe areas extend the core face-processing netw

47 se formats are not only determined by medial temporal lobe areas, but essentially also by the neocort

48 examine intracranial EEG (iEEG) in the human temporal lobe as participants perform a verbal paired as

49 ed extensive regions in the bilateral medial temporal lobes as well as the bilateral posterior cingul

50 fluctuations in neural firing in the medial temporal lobe, as well as in the mid-to-anterior cingula

51 The anterior temporal lobes (ATL) have become a key brain region of i

52 ted tomography, and 0.91 and 0.89 for medial temporal lobe atrophy on magnetic resonance imaging (MRI

53 led a significant hypometabolism in the left temporal lobe (BAs 20, 36 and 38), in the right frontal

54 7 regions of interest, especially the medial temporal lobe (beta=0.66-0.76, t=3.90-5.58, FDR-correcte

55 levels of miR-298 varied in postmortem human temporal lobe between AD patients and age-matched non-AD

56 ealed significant hypometabolism in the left temporal lobe (Brodmann areas [BAs] 20, 36, and 38), in

57 rior and middle temporal areas, and anterior temporal lobe, but also parts of the control network as

58 e medial frontal cortex (MFC) and the medial temporal lobe, but it remains unknown how these structur

59 s of tau aggregates and neuronal loss in the temporal lobe, but primary age-related tauopathy lacks t

60 Tau PET binding was elevated in the temporal lobe, but the most prominent loci of pathology

61 ate that stimulation targeted outside of the temporal lobe can modulate hippocampal activity and impa

62 either the medial diencephalon or the medial temporal lobes can result in profound anterograde amnesi

63 ive covariance with metabolism in the medial temporal lobe, cerebellum, brainstem, basal ganglia, tha

64 y within specific elements of frontal-medial temporal lobe circuits, with a central role for the dors

65 future experience within neocortical-medial temporal lobe circuits.

66 anterior and posterior cingulate, insula and temporal lobes (Cohen's d effect sizes: -0.10 to -0.14).

67 degeneration in the prefrontal and anterior temporal lobes compared to patients with mixed apraxia o

68 to a loss of structural stability in medial temporal lobe connectivity in a way that matched propose

69 nal evolutionary modifications affecting the temporal lobe connectivity pattern.

70 cortex connectivity and stronger hippocampal-temporal lobe connectivity.

71 cortex connectivity and stronger hippocampal-temporal lobe connectivity.

72 CE STATEMENT Frontal neural networks and the temporal lobes contribute to reward-guided learning in m

73 focal hippocampal atrophy within the medial temporal lobes, correlative atrophy in the mediodorsal t

74 a and tau pathologies in the hippocampus and temporal lobe cortex of drug-resistant temporal lobe epi

75 supported by similar interaction effects on temporal lobe cortical thickness (whole-brain voxelwise

76 Medial temporal lobe dependent cognitive functions are highly v

77 ing a mechanism by which IEDs disrupt medial temporal lobe-dependent declarative memory retrieval pro

78 nknown, however, how the organization of the temporal lobe differs across several anthropoid primates

79 recordings in the human neocortex and mesial temporal lobe during rhythmic onset seizures.

80 CD was most prevalent in the anterior medial temporal lobe (entorhinal and perirhinal cortices, anter

81 tients with unilateral drug-resistant mesial temporal lobe epilepsy (MTLE) following anterior tempora

82 Mesial temporal lobe epilepsy (MTLE) is a chronic neurological

83 Mesial temporal lobe epilepsy (mTLE) is a chronic neurological

84 The cause of mesial temporal lobe epilepsy (MTLE) is often unknown.

85 Mesial temporal lobe epilepsy (MTLE) is the most common form of

86 Surgical specimens from patients with mesial temporal lobe epilepsy (MTLE) show abnormalities in tiss

87 c generalized epilepsy (GGE), 21 with mesial temporal lobe epilepsy (mTLE)), we find that people with

88 Mesial temporal lobe epilepsy (mTLE), the most common form of m

89 study, we included subjects with unilateral temporal lobe epilepsy (TLE) before (n = 29) or after (n

90 regulated, but reemerges in animal models of temporal lobe epilepsy (TLE) development and patient epi

91 Seizures in temporal lobe epilepsy (TLE) disturb brain networks and

92 of TLE.SIGNIFICANCE STATEMENT Development of temporal lobe epilepsy (TLE) generally takes years after

93 Temporal lobe epilepsy (TLE) is a common and commonly de

94 roprotective benefits.SIGNIFICANCE STATEMENT Temporal lobe epilepsy (TLE) is a common and devastating

95 Temporal lobe epilepsy (TLE) is a devastating disease in

96 Temporal lobe epilepsy (TLE) is a prevalent neurological

97 Temporal lobe epilepsy (TLE) is characterized by recurre

98 e in 26 people develop epilepsy and in these temporal lobe epilepsy (TLE) is common.

99 Medial temporal lobe epilepsy (TLE) is the most common form of

100 Temporal lobe epilepsy (TLE) is the most common type of

101 dult hippocampal neurogenesis occurs in many temporal lobe epilepsy (TLE) models.

102 The effects of temporal lobe epilepsy (TLE) on subcortical arousal stru

103 brain-age for (1) the effect of psychosis on temporal lobe epilepsy (TLE), (2) psychogenic nonepilept

104 model in the field of epilepsy, specifically Temporal Lobe Epilepsy (TLE), and correlate their clinic

105 ory, and hippocampal sclerosis are common in temporal lobe epilepsy (TLE), but little is known about

106 t of an epileptic condition resembling human temporal lobe epilepsy (TLE).

107 developing patient-specific cell therapy in temporal lobe epilepsy (TLE).

108 neurogenesis is altered in rodent models of temporal lobe epilepsy (TLE).

109 minimally invasive treatment for intractable temporal lobe epilepsy (TLE).

110 blish further parallels between human medial temporal lobe epilepsy and a naturally occurring conditi

111 ved from temporal cortex samples of multiple temporal lobe epilepsy and non-epileptic subjects.

112 Individuals with temporal lobe epilepsy and normal MRI showed a similar p

113 We used an in vivo model of temporal lobe epilepsy based on intrahippocampal kainic

114 se pathology were significantly activated in temporal lobe epilepsy brain samples, including the c-Ju

115 immunohistochemical analysis of tissue from temporal lobe epilepsy cases revealed increased phosphor

116 In temporal lobe epilepsy cases, hippocampal levels of phos

117 Temporal lobe epilepsy causes severe cognitive deficits,

118 t the cognitive impairment in drug-resistant temporal lobe epilepsy could be due to perturbations of

119 Temporal lobe epilepsy is a complex neurological disease

120 Temporal lobe epilepsy is associated with significant st

121 Temporal lobe epilepsy is common and can be difficult to

122 Temporal lobe epilepsy is the most common drug-resistant

123 Temporal lobe epilepsy is the most common form of epilep

124 Mesial temporal lobe epilepsy is usually regarded as a polygeni

125 ale epileptic patients diagnosed with mesial temporal lobe epilepsy or cortical glioma (peritumoral c

126 Both amoeboid and ramified cells from mesial temporal lobe epilepsy or peritumoral cortex tissue expr

127 s and temporal lobe cortex of drug-resistant temporal lobe epilepsy patients who underwent temporal l

128 Silencing miR-135a in experimental temporal lobe epilepsy reduces seizure activity at the s

129 A sub-group of patients with mesial temporal lobe epilepsy related to hippocampus sclerosis

130 Temporal lobe epilepsy represents a major cause of drug-

131 ppocampus and neocortex of rats with chronic temporal lobe epilepsy to demonstrate that subsets of ce

132 in every two patients with pharmacoresistant temporal lobe epilepsy will not be rendered completely s

133 alities (Alzheimer's disease (AD) and mesial temporal lobe epilepsy with hippocampal sclerosis (HS))

134 rom 1069 healthy controls and 1249 patients: temporal lobe epilepsy with hippocampal sclerosis (n = 5

135 reas 20 and 21) from 86 patients with mesial temporal lobe epilepsy with hippocampal sclerosis and 75

136 Mesial temporal lobe epilepsy with hippocampal sclerosis repres

137 iptional and splicing deregulation in mesial temporal lobe epilepsy with hippocampal sclerosis tissue

138 specific to tissue from patients with mesial temporal lobe epilepsy with hippocampal sclerosis.

139 e prolonged febrile seizures group developed temporal lobe epilepsy with mesial temporal sclerosis.

140 ilepsy with hippocampal sclerosis (n = 599), temporal lobe epilepsy with normal MRI (n = 275), geneti

141 eported in individuals with substance abuse, temporal lobe epilepsy, amyotrophic lateral sclerosis, m

142 eralized tonic-clonic seizures in a model of temporal lobe epilepsy, and rescued cognitive impairment

143 ties in common epilepsy syndromes, including temporal lobe epilepsy, extratemporal epilepsy, and gene

144 or cognitive deficits associated with aging, temporal lobe epilepsy, or transient global amnesia.

145 In temporal lobe epilepsy, sprouting of hippocampal mossy f

146 In the systemic pilocarpine mouse model of temporal lobe epilepsy, the epileptic dentate gyrus exce

147 and propagation of temporal lobe seizures in temporal lobe epilepsy, using diffusion tensor imaging a

148 in the hippocampus, a region associated with temporal lobe epilepsy.

149 c patients and experimental animal models of temporal lobe epilepsy.

150 f new treatment strategies for patients with temporal lobe epilepsy.

151 ralization on the mesoscale brain network in temporal lobe epilepsy.

152 R-135a in both experimental and human mesial temporal lobe epilepsy.

153 hippocampi was also effective in a model of temporal lobe epilepsy.

154 entrally affected in Alzheimer's disease and temporal lobe epilepsy.

155 ogenic zone (EZ) in patients with frontal or temporal lobe epilepsy.

156 on dramatically increase in a mouse model of temporal lobe epilepsy.

157 ack inhibitory circuit in a model of chronic temporal lobe epilepsy.

158 suppressing seizures in a male rat model of temporal lobe epilepsy.

159 tent postoperative seizures in patients with temporal lobe epilepsy.

160 roximately one-third of patients with mesial temporal lobe epilepsy.

161 ting of mossy fiber axons, both hallmarks of temporal lobe epilepsy.

162 l circuits in experimental models of chronic temporal lobe epilepsy.

163 te to cognitive impairment in drug-resistant temporal lobe epilepsy.

164 ing approach for therapeutic intervention in temporal lobe epilepsy.

165 ter hippocampal seizures in a mouse model of temporal lobe epilepsy.

166 arges in human patients and animal models of temporal lobe epilepsy.

167 h led to additional damage characteristic of temporal lobe epilepsy.

168 fist, and pincer are strongly predictive of temporal lobe EZ.

169 tural connectivity between these regions and temporal lobe face patches.

170 showed thicker cortical regions in the right temporal lobe (FDRq = 0.05).

171 all categories of patients except for extra-temporal lobe focal epilepsy showed a significant increa

172 ated with TDP-43, starting within the medial temporal lobe, followed by early involvement of the temp

173 regions for posterior cortical atrophy, left temporal lobe for logopenic progressive aphasia and medi

174 c progressive aphasia and medial and lateral temporal lobe for typical Alzheimer's disease.

175 gions of tau accumulation in the frontal and temporal lobes for all phenotypes and key regions of atr

176 with LATE-NC also had atrophy in the medial temporal lobes, frontal cortex, and other brain regions.

177 n "process" and "representational" models of temporal lobe function is challenged.

178 nce in cognitive tests assessing frontal and temporal lobe functioning.

179 rols in frontal lobe (g = -0.47; p < 0.001), temporal lobe (g = -0.84; p < 0.001), parietal lobe (g =

180 nd Abeta were predominantly localized in the temporal lobe, however, sex differences in extra-tempora

181 e signal abnormalities located in the medial temporal lobe in 16 of 37 patients (43%; 95% confidence

182 on new insights into the role of the medial temporal lobe in auditory cognition.

183 ated with brain tau deposition in the medial temporal lobe in MCI participants (r = 0.43 for early MC

184 eft inferior frontal gyrus and left anterior temporal lobe in the process of flexible feature modulat

185 ACS and functional connectivity of bilateral temporal lobes, in line with the bilateral increase in g

186 ted regional tau deposition in the bilateral temporal lobes including the entorhinal cortex.

187 Recordings from the medial temporal lobe, including the hippocampus, reveal the exi

188 suggests a functional explanation for medial temporal lobe involvement in visual memory for relationa

189 Coma, temporal lobe involvement, hematoma volume, and electrog

190 tio, 4.88; CI, 1.36-17.57; p = 0.015); coma, temporal lobe involvement, intraparenchymal hemorrhage v

191 The medial temporal lobe is critical for both spatial navigation an

192 The medial temporal lobe is one of the most well-studied brain regi

193 uency (3-12 Hz) power distributed across the temporal lobe is significantly related to memory perform

194 PND (non-displaceable binding potential)) in temporal lobes, lateralising according to their clinical

195 m (P < 0.001) involving the occipital lobes, temporal lobes, limbic system, cerebellum, and frontopar

196 ysical interaction preference, which, in the temporal lobe, mapped onto a fine-grain pattern of objec

197 SYN + AD performed worse than SYN-AD on a temporal lobe-mediated naming task (t(27) = 2.1, p = 0.0

198 these face patches and regions of the medial temporal lobe memory system (including the hippocampus),

199 e a communication dynamic between the medial temporal lobe memory system and the neocortex.

200 o provide reward prediction error signals to temporal lobe memory systems, but the role of these sign

201 The IT projects directly to the medial temporal lobe (MTL) [19], where neurons respond selectiv

202 ng evidence points to the role of the Medial Temporal Lobe (MTL) and Medial Prefrontal Cortex (mPFC)

203 dynamically coupled between the human medial temporal lobe (MTL) and temporal association cortex.

204 ll-that theta power increases in left medial temporal lobe (mTL) are impaired in schizophrenia, as is

205 The medial temporal lobe (MTL) contains "concept cells" that respon

206 ions of memory-impaired patients with medial temporal lobe (MTL) damage who took a 25-min guided walk

207 both sexes with unspecific unilateral medial temporal lobe (MTL) damage, one male with selective bila

208 ral activity pattern reinstatement in medial temporal lobe (MTL) during the replay phase of the secon

209 ncertain, but genotype differences in medial temporal lobe (MTL) functional activity and structure at

210 pocampal formation located within the medial temporal lobe (MTL) in primates.

211 The medial temporal lobe (MTL) is a locus of episodic memory in the

212 The medial temporal lobe (MTL) is known as the locus of spatial cod

213 The medial temporal lobe (MTL) is known to support episodic memory

214 Using medial temporal lobe (MTL) recordings from 96 neurosurgical pat

215 ted functional integration across the medial temporal lobe (MTL) subsystem of the default network.

216 organized using the circuitry in the medial temporal lobe (MTL) that supports spatial processing and

217 STATEMENT By recording from the human medial temporal lobe (MTL) while subjects recall items experien

218 medial parietal cortex (MPC) and the medial temporal lobe (MTL), structures known to be engaged duri

219 to memory loss through disruption of medial temporal lobe (MTL)-dependent memory systems.

220 e medial prefrontal cortex (mPFC) and medial temporal lobe (MTL).

221 nt of cortical representations by the medial temporal lobes (MTL).

222 The hippocampus and surrounding medial-temporal-lobe (MTL) structures are critical for both mem

223 ry, relies on subregions in the human medial temporal lobes (MTLs).

224 partial]: frontal lobe, n = 243 vs n = 117; temporal lobe, n = 244 vs n = 137; parietal lobe n = 240

225 ynaptic boutons (SBs) in layer 4 (L4) of the temporal lobe neocortex (TLN) were quantitatively invest

226 In the medial temporal lobes, non-amnestic patients had less atrophy a

227 The marked atrophy of the medial temporal lobe observed in AD patients may explain the in

228 Deep inside the temporal lobe of the brain, the hippocampus has a centra

229 erconnected structures located in the medial temporal lobe of the mammalian brain.

230 cterized by neuronal loss in the frontal and temporal lobes of the brain.

231 rospectively analyzed ictal semiology of 489 temporal lobe or frontal lobe seizures recorded over a 6

232 and ventral tegmental area (SN/VTA), medial temporal lobe, or subsequent memory performance.

233 y the relative overgrowth of the frontal and temporal lobes over the insula, corresponding to domains

234 gnificant decrease in the ipsilateral mesial temporal lobe (p = 0.02), parahippocampal area (p = 0.03

235 ) than with SUVRCB (Pearson r: from 0.51 for temporal lobe [P = 0.002] to 0.82 for precuneus [P < 0.0

236 of neurodegeneration affecting the anterior temporal lobe, partial compensation appears to be possib

237 sistently active neurons in the human medial temporal lobe phase lock to ongoing slow-frequency (1-7

238 cted to the same brain regions in the mesial temporal lobe, precuneus cortex, and angular gyrus.

239 increases in connectivity within the medial temporal lobe predicted improved performance of a separa

240 tal cortex and a key component of the medial temporal lobe-prefrontal cortex circuit.

241 found that the resulting imbalance of medial temporal lobe-prefrontal cortex connectivity partially m

242 Encoding activity in the medial temporal lobe, presumably evoked by the presentation of

243 mans, the invasion of dorsal tracts into the temporal lobe provides a further specialization.

244 The right Anterior Temporal Lobe (rATL), putatively involved in semantic in

245 requency theta power are seen across lateral temporal lobe recording sites and persist throughout the

246 y attenuated inhibitory self-coupling within temporal lobe regions and excitatory projections between

247 robust against various thresholds and medial temporal lobe regions defining elevated tau.

248 In subjects with lower global SUVr, temporal lobe regions showed the greatest flortaucipir r

249 N) centered on posterior parietal and medial temporal lobe regions, but the temporal dynamics of thes

250 emporal lobe epilepsy patients who underwent temporal lobe resection (n = 19), in comparison with age

251 ) before (n = 29) or after (n = 56) anterior temporal lobe resection and healthy volunteers (n = 124)

252 e acquired before and after elective partial temporal lobe resection in 25 patients for intractable e

253 ced in those who remained seizure-free after temporal lobe resection, normalizing the rate of atrophy

254 ell project to the cortical motor system and temporal lobe, respectively.

255 a (40 Hz) activity in the right parietal and temporal lobes, respectively.

256 s to the hippocampus, we control the risk of temporal lobe seizures during a specific time period.

257 portant in the generation and propagation of temporal lobe seizures in temporal lobe epilepsy, using

258 ents with frontal and 54.5% of patients with temporal lobe seizures.

259 are associated with memory impairment after temporal lobe seizures.

260 appeared as well in the brain region (in the temporal lobe) spatially separate from but most connecte

261 y auditory areas and moving laterally on the temporal lobe: spectral features are found in the core o

262 LATE-NC appears to affect the medial temporal lobe structures preferentially, but other areas

263 wing that clear asymmetries exist within the temporal lobe structures subserving the core system and

264 ted regions of the limbic system, the medial temporal lobe structures-the hippocampus and amygdala as

265 dine reliably induces seizure-like events in temporal lobe structures.

266 and whether this occurs in structures in the temporal lobe, supplying cortical inputs to the hippocam

267 whereas a region in the left dorsal anterior temporal lobe supports object-color knowledge in both th

268 Children were more often drug-free; temporal lobe surgeries had the best seizure outcomes; a

269 ot be rendered completely seizure-free after temporal lobe surgery.

270 The medial temporal-lobe system is essential for the formation and

271 of how this region may interact with medial temporal lobe systems involved in configural object proc

272 elation between behavioral response to right temporal lobe tACS and functional connectivity of bilate

273 ep spindle coupling predicted greater medial temporal lobe tau burden.

274 A temporal lobe tau PET meta-region of interest was used.

275 nchored neural representations in the medial temporal lobe that are modulated by one's own as well as

276 describes the memory system, centered on the temporal lobe that builds specific memory traces.

277 nal cortex, a region located in the anterior-temporal lobe that is critical for object memory.

278 ctivity between dorsolateral PFC (DLPFC) and temporal lobe that mediated the effect of pain intensity

279 eft inferior frontal gyrus and left anterior temporal lobe that related to our measures of feature mo

280 hology was extensive and involved the medial temporal lobe, the diencephalon, cerebral cortex, basal

281 e area and volume but greater right inferior temporal lobe thickness, surface area, and volume than t

282 n pathways for synchronous discharges in the temporal lobe, though critical microcircuit-level detail

283 SM-OCT of ex vivo human temporal lobe tissue reveals fine structures including c

284 oral lobe epilepsy (MTLE) following anterior temporal lobe (TL) resection.

285 eizure generalization from the epileptogenic temporal lobe to broader brain networks in these patient

286 The frontal cortex and temporal lobes together regulate complex learning and me

287 Change in iron levels over time in the temporal lobe was associated with cognitive decline in i

288 The temporal lobe was involved in 71.9% of operations.

289 Temporal lobe was the most common NHS location (16 of 51

290 During gamma-tACS over the right temporal lobe, we observed an increase in accuracy on a

291 R2* values in the temporal lobe were associated with longitudinal changes

292 and multivariate responses in left anterior temporal lobe were predicted by degree of conceptual bri

293 al thickness in the left middle and inferior temporal lobe; whereas PTSD negatively related to cortic

294 the right ventral frontal area and the left temporal lobe, which represented a close mirror image of

295 iors has been linked to major differences in temporal lobe white matter in humans compared with monke

296 cally compared the organization of the major temporal lobe white matter tracts in the human, gorilla,

297 ional tissue characteristics of preoperative temporal lobe white matter tracts known to be important

298 in the anatomic association of the superior temporal lobe with other regions of whole-brain networks

299 upling of network hubs in both antero-mesial temporal lobes, with development of an abnormal excitato

300 st that sensory cues proceed into the medial temporal lobe within the first 500 ms.