ライフサイエンスコーパス: temporal lobe epilepsy

1 endent in vivo rat model systems relevant to temporal lobe epilepsy.

2 for further testing as a potential cause of temporal lobe epilepsy.

3 ucture of BC-->GC synapses in a rat model of temporal lobe epilepsy.

4 t seizures on the model of pharmacoresistant temporal lobe epilepsy.

5 ntrol seizures in up to 80% of patients with temporal lobe epilepsy.

6 te to cognitive impairment in drug-resistant temporal lobe epilepsy.

7 ide (LPS) administration in mice with mesial temporal lobe epilepsy.

8 ges 50 and 65 years to treat drug-refractory temporal lobe epilepsy.

9 ocampus from patients with pharmacoresistant temporal lobe epilepsy.

10 ing approach for therapeutic intervention in temporal lobe epilepsy.

11 ion with mossy fibre sprouting, a feature of temporal lobe epilepsy.

12 h seizure susceptibility in mice with mesial temporal lobe epilepsy.

13 about the recurrent excitation hypothesis of temporal lobe epilepsy.

14 may provide a novel avenue for treatment of temporal lobe epilepsy.

15 ter hippocampal seizures in a mouse model of temporal lobe epilepsy.

16 echanism to study cognitive comorbidities of temporal lobe epilepsy.

17 lications for restraining SE-induced chronic temporal lobe epilepsy.

18 namics before spontaneous seizures in medial temporal lobe epilepsy.

19 of hippocampal seizures in a mouse model of temporal lobe epilepsy.

20 uring spontaneous seizures in a rat model of temporal lobe epilepsy.

21 ation processing remain poorly understood in temporal lobe epilepsy.

22 ethylation sensitive non-coding RNA in human temporal lobe epilepsy.

23 on cardiovascular function in a rat model of temporal lobe epilepsy.

24 arges in human patients and animal models of temporal lobe epilepsy.

25 ory function, which is typically impaired in temporal lobe epilepsy.

26 ound in hippocampectomies from patients with temporal lobe epilepsy.

27 ns is similar to that of human patients with temporal lobe epilepsy.

28 4 (TLR4) pathway is known to be involved in temporal lobe epilepsy.

29 ology similar to that of human patients with temporal lobe epilepsy.

30 reported previously for human patients with temporal lobe epilepsy.

31 cial cognitive function that is disrupted in temporal lobe epilepsy.

32 wever, none of these children have developed temporal lobe epilepsy.

33 al lobe resection than verbal memory in left temporal lobe epilepsy.

34 h led to additional damage characteristic of temporal lobe epilepsy.

35 and in vivo optogenetics in a mouse model of temporal lobe epilepsy.

36 gions of the brain and pharmacoresistance in temporal lobe epilepsy.

37 rat model is used frequently to investigate temporal lobe epilepsy.

38 ppocampal network in a mouse model of mesial temporal lobe epilepsy.

39 ycoprotein activity in vivo in patients with temporal lobe epilepsy.

40 seizures in patients with intractable mesial temporal lobe epilepsy.

41 tate gyrus in the mouse pilocarpine model of temporal lobe epilepsy.

42 ncy is important in the amnestic symptoms of temporal lobe epilepsy.

43 h multiple sclerosis, Alzheimer's disease or temporal lobe epilepsy.

44 YP) seizures in the rat pilocarpine model of temporal lobe epilepsy.

45 cuits might contribute to epileptogenesis in temporal lobe epilepsy.

46 n schizophrenia, major depression and mesial temporal lobe epilepsy.

47 were associated with febrile seizures and/or temporal lobe epilepsy.

48 c patients and experimental animal models of temporal lobe epilepsy.

49 mine the clinical utility of imaging TSPO in temporal lobe epilepsy.

50 vitro in surgical samples from patients with temporal lobe epilepsy.

51 l-dependent learning in patients with mesial temporal lobe epilepsy.

52 lzheimer's disease, Parkinson's disease, and temporal lobe epilepsy.

53 in the hippocampus, a region associated with temporal lobe epilepsy.

54 f new treatment strategies for patients with temporal lobe epilepsy.

55 ralization on the mesoscale brain network in temporal lobe epilepsy.

56 R-135a in both experimental and human mesial temporal lobe epilepsy.

57 hippocampi was also effective in a model of temporal lobe epilepsy.

58 entrally affected in Alzheimer's disease and temporal lobe epilepsy.

59 ogenic zone (EZ) in patients with frontal or temporal lobe epilepsy.

60 on dramatically increase in a mouse model of temporal lobe epilepsy.

61 ack inhibitory circuit in a model of chronic temporal lobe epilepsy.

62 suppressing seizures in a male rat model of temporal lobe epilepsy.

63 tent postoperative seizures in patients with temporal lobe epilepsy.

64 roximately one-third of patients with mesial temporal lobe epilepsy.

65 ting of mossy fiber axons, both hallmarks of temporal lobe epilepsy.

66 l circuits in experimental models of chronic temporal lobe epilepsy.

67 thesized to contribute to the development of temporal lobe epilepsy.

68 rain region in the pathophysiology of mesial temporal lobe epilepsy.

69 e epilepsy, the remaining 15 cases for extra-temporal lobe epilepsies.

70 e studied 33 patients with unilateral medial temporal lobe epilepsy (16 left) before, 3 and 12 months

71 e studied 36 patients with unilateral medial temporal lobe epilepsy (19 right) before and 3 and 12 mo

72 e studied 46 patients with unilateral medial temporal lobe epilepsy (25/26 left hippocampal sclerosis

73 erogeneous forms of epilepsy including human temporal lobe epilepsy, a mouse model of acquired tempor

74 mutated in human autosomal dominant lateral temporal lobe epilepsy (ADLTE), regulates postnatal prun

75 ed protein, cause autosomal dominant lateral temporal lobe epilepsy (ADLTE).

76 eported in individuals with substance abuse, temporal lobe epilepsy, amyotrophic lateral sclerosis, m

77 Sixteen patients with unilateral temporal lobe epilepsy and 30 healthy subjects were stud

78 ients were studied, including 30 with mesial temporal lobe epilepsy and 31 with focal neocortical epi

79 blish further parallels between human medial temporal lobe epilepsy and a naturally occurring conditi

80 ctroclinical findings that are suggestive of temporal lobe epilepsy and can be considered as another

81 Brain injury is an etiological factor for temporal lobe epilepsy and can lead to memory and cognit

82 logy is associated with cognitive decline in temporal lobe epilepsy and explored this through clinico

83 We studied 44 patients with unilateral temporal lobe epilepsy and hippocampal sclerosis (24 lef

84 ion in hippocampal tissue from patients with temporal lobe epilepsy and in mice following pilocarpine

85 A levels in the hippocampus of patients with temporal lobe epilepsy and in neural tissues from animal

86 Deja vu can occur as an aura of temporal lobe epilepsy and in some psychiatric condition

87 ved from temporal cortex samples of multiple temporal lobe epilepsy and non-epileptic subjects.

88 Individuals with temporal lobe epilepsy and normal MRI showed a similar p

89 tes of spontaneous seizures in patients with temporal lobe epilepsy and pilocarpine-treated rats that

90 tify the transcriptomic signature of chornic temporal lobe epilepsy and the drugs that reverse it.

91 es in 50-80% of patients with drug-resistant temporal lobe epilepsy and the effect of surgery on work

92 nt clinical observations about resection for temporal lobe epilepsy and the expectations from semanti

93 on of memory encoding networks in refractory temporal lobe epilepsy and the neural correlates of succ

94 t semantic memory is intact in resection for temporal lobe epilepsy and thus casting doubt over the c

95 and UFRs were observed only in patients with temporal lobe epilepsy and were recorded exclusively fro

96 least a year after 3 or more years of active temporal lobe epilepsy, and 17 healthy controls.

97 ral lobe epilepsy, a mouse model of acquired temporal lobe epilepsy, and a mouse model of monogenic D

98 e basolateral amygdala kindling rat model of temporal lobe epilepsy, and it led to prolongation of th

99 has clear clinical utility in patients with temporal lobe epilepsy, and its potential easily transla

100 eralized tonic-clonic seizures in a model of temporal lobe epilepsy, and rescued cognitive impairment

101 n SOV and seizure onset in the TeTX model of temporal lobe epilepsy, as well as the first demonstrati

102 We studied 43 patients with mesial temporal lobe epilepsy associated with hippocampal scler

103 We used an in vivo model of temporal lobe epilepsy based on intrahippocampal kainic

104 In a rat model of temporal lobe epilepsy, basket cell-to-granule cell (BC-

105 nization of memory function in patients with temporal lobe epilepsy before and after left or right an

106 se pathology were significantly activated in temporal lobe epilepsy brain samples, including the c-Ju

107 zures in 50-60% of patients with intractable temporal lobe epilepsy but may impair memory function, t

108 LR) is an effective treatment for refractory temporal lobe epilepsy but may result in a contralateral

109 ffective treatment for medically intractable temporal lobe epilepsy, but can cause memory impairment.

110 ncoding networks within the temporal lobe in temporal lobe epilepsy, but little is known of the extra

111 th transcriptional regulation was evident in temporal lobe epilepsy, but overall few genes previously

112 excitatory neurons is frequently observed in temporal lobe epilepsy, but the extent to which inhibito

113 ibute to the pathogenesis and maintenance of temporal lobe epilepsy, but the underlying cell and mole

114 ties are thought to play a causative role in temporal lobe epilepsy, but their precise contribution h

115 mage have been implicated in the etiology of temporal lobe epilepsy, but whether or not they have a f

116 Seizures in temporal lobe epilepsy can be classified as hypersynchro

117 immunohistochemical analysis of tissue from temporal lobe epilepsy cases revealed increased phosphor

118 In temporal lobe epilepsy cases, hippocampal levels of phos

119 Temporal lobe epilepsy causes severe cognitive deficits,

120 vide new insights for why some patients with temporal lobe epilepsy continue to experience postoperat

121 t the cognitive impairment in drug-resistant temporal lobe epilepsy could be due to perturbations of

122 The studies assessing SPECT use in temporal lobe epilepsy did not reveal a correlation with

123 ties in common epilepsy syndromes, including temporal lobe epilepsy, extratemporal epilepsy, and gene

124 onsiderable neuropsychological literature on temporal lobe epilepsy, few studies have probed semantic

125 clinical trials enrolling 118 patients with temporal lobe epilepsy found greater freedom from seizur

126 proved verbal learning in patients with left temporal lobe epilepsy from preoperatively to 12 months

127 different patient groups (semantic dementia, temporal lobe epilepsy, glioma and stroke) as well as se

128 ed analysis revealed that patients with left temporal lobe epilepsy had greater activation in the lef

129 n that some patients with left resection for temporal lobe epilepsy had measurable anomia.

130 Human temporal lobe epilepsy has been associated with dysfunct

131 for medication-resistant and well-localized temporal lobe epilepsy has good prognosis for seizure fr

132 g genes exhibited altered DNA methylation in temporal lobe epilepsy hippocampus (n = 9) when compared

133 human embryonic stem cells to treat chronic temporal lobe epilepsy in a mouse model.

134 erimental paradigm extensively used to model temporal lobe epilepsy in humans.

135 ting in a status epilepticus model of mesial temporal lobe epilepsy in rats, which is associated with

136 lation-sensitive microRNA were identified in temporal lobe epilepsy including MIR27A, miR-193a-5p (MI

137 ranule cells from control rats and rats with temporal lobe epilepsy induced by pilocarpine hydrochlor

138 Temporal lobe epilepsy is a common and challenging clini

139 Temporal lobe epilepsy is a common and frequently intrac

140 One in 26 people develops epilepsy, and temporal lobe epilepsy is a common form.

141 Temporal lobe epilepsy is a complex neurological disease

142 suggest that visual memory function in right temporal lobe epilepsy is affected differently by right

143 Temporal lobe epilepsy is associated with large-scale, w

144 Temporal lobe epilepsy is associated with significant st

145 Temporal lobe epilepsy is common and can be difficult to

146 Temporal lobe epilepsy is the most common and drug-resis

147 Temporal lobe epilepsy is the most common and often deva

148 Temporal lobe epilepsy is the most common drug-resistant

149 Temporal lobe epilepsy is the most common form of epilep

150 Temporal lobe epilepsy is the most common form of epilep

151 Temporal lobe epilepsy is the most common type of epilep

152 Mesial temporal lobe epilepsy is usually regarded as a polygeni

153 ory formation (e.g., pattern separation) and temporal lobe epilepsy, little is known about activity-d

154 s been suggested that the pathophysiology of temporal lobe epilepsy may relate to abnormalities in va

155 These mice had mesial-temporal lobe epilepsy, microcephaly and corpus callosum

156 pocampi from mice with epilepsy (pilocarpine-temporal lobe epilepsy model) and 100 healthy control hi

157 more, despite induction of MF sprouting in a temporal lobe epilepsy model, KARs were not recruited to

158 recurrent seizures in a pilocarpine-induced temporal lobe epilepsy model.

159 n and 20 women; aged >/=12 years) had mesial temporal lobe epilepsy (MTLE) and disabling seizues for

160 ls (DGCs) are altered in experimental mesial temporal lobe epilepsy (mTLE) and whether their integrat

161 chronically lost in a mouse model of mesial temporal lobe epilepsy (MTLE) as well as in hippocampal

162 that 1 or more particular subtypes of mesial temporal lobe epilepsy (mTLE) exist that are particularl

163 tients with unilateral drug-resistant mesial temporal lobe epilepsy (MTLE) following anterior tempora

164 As mesial temporal lobe epilepsy (mTLE) has been recognized as a n

165 Mesial temporal lobe epilepsy (MTLE) is a chronic neurological

166 Mesial temporal lobe epilepsy (mTLE) is a chronic neurological

167 Mesial temporal lobe epilepsy (MTLE) is a common medically refr

168 Medial temporal lobe epilepsy (MTLE) is associated with limbic

169 The cause of mesial temporal lobe epilepsy (MTLE) is often unknown.

170 Mesial temporal lobe epilepsy (mTLE) is the most common focal e

171 Mesial temporal lobe epilepsy (MTLE) is the most common form of

172 ed neuronal loss and axonal damage in medial temporal lobe epilepsy (MTLE) may lead to the developmen

173 Surgical specimens from patients with mesial temporal lobe epilepsy (MTLE) show abnormalities in tiss

174 Patients with mesial temporal lobe epilepsy (MTLE) without MRI abnormalities

175 c generalized epilepsy (GGE), 21 with mesial temporal lobe epilepsy (mTLE)), we find that people with

176 eatment for patients with intractable mesial temporal lobe epilepsy (mTLE), a third of patients will

177 Mesial temporal lobe epilepsy (mTLE), the most common form of m

178 tment of patients with drug-resistant mesial temporal lobe epilepsy (mTLE).

179 In a mouse model of temporal lobe epilepsy, multicellular calcium imaging re

180 1738 European epilepsy patients with either temporal lobe epilepsy (n = 330) and genetic generalized

181 nt in neuroinflammation, aggravating stroke, temporal lobe epilepsy, neuropathic pain, and various ne

182 Patients with temporal lobe epilepsy often display cognitive comorbidi

183 ale epileptic patients diagnosed with mesial temporal lobe epilepsy or cortical glioma (peritumoral c

184 Temporal lobe epilepsy or limbic epilepsy lacks effectiv

185 Both amoeboid and ramified cells from mesial temporal lobe epilepsy or peritumoral cortex tissue expr

186 or cognitive deficits associated with aging, temporal lobe epilepsy, or transient global amnesia.

187 ectroencephalography for medically resistant temporal lobe epilepsy participated in a visual recognit

188 mory-encoding network in both left and right temporal lobe epilepsy patients across both verbal and v

189 Ictal events occurring in temporal lobe epilepsy patients and in experimental mode

190 Left temporal lobe epilepsy patients showed increased right a

191 Left and right temporal lobe epilepsy patients were impaired on preoper

192 s and temporal lobe cortex of drug-resistant temporal lobe epilepsy patients who underwent temporal l

193 Patients with temporal lobe epilepsy present with a broad spectrum of

194 Humans with medial temporal lobe epilepsy present with white matter patholo

195 Silencing miR-135a in experimental temporal lobe epilepsy reduces seizure activity at the s

196 A sub-group of patients with mesial temporal lobe epilepsy related to hippocampus sclerosis

197 l activity in human patients with unilateral temporal lobe epilepsy relative to age-matched healthy c

198 ular mechanism underlying the development of temporal lobe epilepsy remains largely unknown.

199 Temporal lobe epilepsy represents a major cause of drug-

200 Patients with right temporal lobe epilepsy showed increased left anterior hi

201 In temporal lobe epilepsy, sprouting of hippocampal mossy f

202 in a series of 100 consecutive patients with temporal lobe epilepsy submitted to epilepsy surgery.

203 es and focal epilepsy, which encompasses the temporal lobe epilepsy subtype.

204 ateral to the seizure focus in patients with temporal lobe epilepsy, suggesting increased expression

205 genome-wide DNA methylation changes in human temporal lobe epilepsy that may contribute to the molecu

206 We show in a rat model of temporal lobe epilepsy that spontaneous hippocampal IEDs

207 In the systemic pilocarpine mouse model of temporal lobe epilepsy, the epileptic dentate gyrus exce

208 During temporal lobe epilepsy, the homeostatic regulation of in

209 FOR A SCIENTIFIC COMMENTARY ON THIS ARTICLE: Temporal lobe epilepsy, the most prevalent form of chron

210 Fifty-one patients (77%) had surgery for temporal lobe epilepsy, the remaining 15 cases for extra

211 study, we included subjects with unilateral temporal lobe epilepsy (TLE) before (n = 29) or after (n

212 more than 65,000,000 people worldwide, with temporal lobe epilepsy (TLE) being the most common form.

213 regulated, but reemerges in animal models of temporal lobe epilepsy (TLE) development and patient epi

214 Seizures in temporal lobe epilepsy (TLE) disturb brain networks and

215 on 11 ([11C])-labeled PBR28 in patients with temporal lobe epilepsy (TLE) found increased TSPO ipsila

216 of TLE.SIGNIFICANCE STATEMENT Development of temporal lobe epilepsy (TLE) generally takes years after

217 FSE) produces hippocampal sclerosis (HS) and temporal lobe epilepsy (TLE) has long been debated.

218 Temporal lobe epilepsy (TLE) is a common and commonly de

219 roprotective benefits.SIGNIFICANCE STATEMENT Temporal lobe epilepsy (TLE) is a common and devastating

220 Temporal lobe epilepsy (TLE) is a common epilepsy syndro

221 Temporal lobe epilepsy (TLE) is a devastating disease in

222 Temporal lobe epilepsy (TLE) is a prevalent neurological

223 Temporal lobe epilepsy (TLE) is a prevalent neurological

224 Temporal lobe epilepsy (TLE) is characterized by recurre

225 Mesial temporal lobe epilepsy (TLE) is characterized by stereot

226 e in 26 people develop epilepsy and in these temporal lobe epilepsy (TLE) is common.

227 Temporal lobe epilepsy (TLE) is one of the most common d

228 Temporal lobe epilepsy (TLE) is one of the most common f

229 Medial temporal lobe epilepsy (TLE) is the most common form of

230 Temporal lobe epilepsy (TLE) is the most common type of

231 Temporal lobe epilepsy (TLE) is the most frequent form o

232 dult hippocampal neurogenesis occurs in many temporal lobe epilepsy (TLE) models.

233 Drug-resistant temporal lobe epilepsy (TLE) often requires thorough inv

234 The effects of temporal lobe epilepsy (TLE) on subcortical arousal stru

235 ersely related in the DG of individuals with temporal lobe epilepsy (TLE) or AD and correlate with pe

236 Although most temporal lobe epilepsy (TLE) patients show marked hippoc

237 similar network abnormalities are present in temporal lobe epilepsy (TLE) patients with a history of

238 ation of magnetic resonance imaging-negative temporal lobe epilepsy (TLE) undergoing standard anterio

239 Medically refractory unilateral temporal lobe epilepsy (TLE) with hippocampal sclerosis

240 brain-age for (1) the effect of psychosis on temporal lobe epilepsy (TLE), (2) psychogenic nonepilept

241 A significant proportion of temporal lobe epilepsy (TLE), a common, intractable brai

242 In temporal lobe epilepsy (TLE), although hippocampal atrop

243 model in the field of epilepsy, specifically Temporal Lobe Epilepsy (TLE), and correlate their clinic

244 itivity to localize hippocampal pathology in temporal lobe epilepsy (TLE), but has rarely been evalua

245 ory, and hippocampal sclerosis are common in temporal lobe epilepsy (TLE), but little is known about

246 In animal models of temporal lobe epilepsy (TLE), gene therapy treatments ba

247 In experimental models of temporal lobe epilepsy (TLE), interictal-like activity (

248 y status epilepticus promotes development of temporal lobe epilepsy (TLE), revealing TrkB as a therap

249 Temporal lobe epilepsy (TLE), the most common form of ac

250 tion of pathophysiological pathways in human temporal lobe epilepsy (TLE).

251 ress has been proposed in epileptogenesis of temporal lobe epilepsy (TLE).

252 thesized to contribute to the development of temporal lobe epilepsy (TLE).

253 togenic zone in patients with drug-resistant temporal lobe epilepsy (TLE).

254 icit is a common cognitive disorder in human temporal lobe epilepsy (TLE).

255 t of an epileptic condition resembling human temporal lobe epilepsy (TLE).

256 developing patient-specific cell therapy in temporal lobe epilepsy (TLE).

257 neurogenesis is altered in rodent models of temporal lobe epilepsy (TLE).

258 minimally invasive treatment for intractable temporal lobe epilepsy (TLE).

259 ppocampus and neocortex of rats with chronic temporal lobe epilepsy to demonstrate that subsets of ce

260 This study evaluated a mouse model of temporal lobe epilepsy to test which pathological change

261 and propagation of temporal lobe seizures in temporal lobe epilepsy, using diffusion tensor imaging a

262 Using a rat model of temporal lobe epilepsy, we identified an increase in hip

263 lohippocampectomy and temporal lobectomy for temporal lobe epilepsy were associated with subtle diffe

264 ngs suggest an epilepsy-related tauopathy in temporal lobe epilepsy, which contributes to accelerated

265 selected 16 patients with pharmacoresistant temporal lobe epilepsy who had seizures despite treatmen

266 3 mg/kg; in patients with pharmacoresistant temporal lobe epilepsy, whole-brain K1 increased by only

267 coordinating gene networks in patients with temporal lobe epilepsy will help to identify novel thera

268 in every two patients with pharmacoresistant temporal lobe epilepsy will not be rendered completely s

269 ce of 20 patients with resection for chronic temporal lobe epilepsy with a full battery of semantic a

270 compared miRNA expression patterns in mesial temporal lobe epilepsy with and without hippocampal scle

271 gene are responsible for autosomal dominant temporal lobe epilepsy with auditory features, whereas L

272 alities (Alzheimer's disease (AD) and mesial temporal lobe epilepsy with hippocampal sclerosis (HS))

273 s) in a large consecutive series with mesial temporal lobe epilepsy with hippocampal sclerosis (MTLE-

274 rom 1069 healthy controls and 1249 patients: temporal lobe epilepsy with hippocampal sclerosis (n = 5

275 sample set comprising 959 people with mesial temporal lobe epilepsy with hippocampal sclerosis and 35

276 association study in 1018 people with mesial temporal lobe epilepsy with hippocampal sclerosis and 75

277 reas 20 and 21) from 86 patients with mesial temporal lobe epilepsy with hippocampal sclerosis and 75

278 Seizures in mesial temporal lobe epilepsy with hippocampal sclerosis are ty

279 sis are typically drug-resistant, and mesial temporal lobe epilepsy with hippocampal sclerosis is fre

280 The cause of mesial temporal lobe epilepsy with hippocampal sclerosis is unk

281 Mesial temporal lobe epilepsy with hippocampal sclerosis repres

282 iptional and splicing deregulation in mesial temporal lobe epilepsy with hippocampal sclerosis tissue

283 ebrile seizures, we tested cases with mesial temporal lobe epilepsy with hippocampal sclerosis with (

284 ection to biological understanding of mesial temporal lobe epilepsy with hippocampal sclerosis with f

285 nome-wide significant association for mesial temporal lobe epilepsy with hippocampal sclerosis with f

286 Therefore, using a mouse model of temporal lobe epilepsy with hippocampal sclerosis, a clo

287 dromes, amongst the most common being mesial temporal lobe epilepsy with hippocampal sclerosis.

288 specific to tissue from patients with mesial temporal lobe epilepsy with hippocampal sclerosis.

289 e prolonged febrile seizures group developed temporal lobe epilepsy with mesial temporal sclerosis.

290 ilepsy with hippocampal sclerosis (n = 599), temporal lobe epilepsy with normal MRI (n = 275), geneti

291 Unique methylation profiles were evident in temporal lobe epilepsy with or without hippocampal scler

292 a pilot study of the methylation profiles of temporal lobe epilepsy with or without hippocampal scler

293 dysfunction might be a prime cause of mesial temporal lobe epilepsy with sclerosis and identify novel

294 that the hippocampus of patients with mesial temporal lobe epilepsy with sclerosis is completely devo

295 pping confirmed that in the course of mesial temporal lobe epilepsy with sclerosis, astrocytes acquir

296 changes represent cause or effect of mesial temporal lobe epilepsy with sclerosis, we developed a mo

297 that reproduced key features of human mesial temporal lobe epilepsy with sclerosis.

298 ontrols and kainate-treated rats, a model of temporal lobe epilepsy, with and without tariquidar pret

299 pocampal specimens from patients with mesial temporal lobe epilepsy without (n = 44) and with scleros

300 seizures in patients with intractable mesial temporal lobe epilepsy, without affecting memory.