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1 arget an aggressive tumor (grafted F9 murine teratocarcinoma).
2 peutic possibilities of targeting testicular teratocarcinoma.
3 ies glycolipid thought to be unique to human teratocarcinoma.
4 pe although it was derived from a testicular teratocarcinoma.
5 xd3-/- ES cell lines or to generate Foxd3-/- teratocarcinomas.
6  are also the cell of origin of multilineage teratocarcinomas.
7 he undifferentiated areas of the alpha5-null teratocarcinomas.
8 lecules was more disorganized in alpha5-null teratocarcinomas.
9 el formation both in ES cell cultures and in teratocarcinomas.
10 d ectopically into syngeneic mice to develop teratocarcinomas.
11  increased apoptosis observed in alpha5-null teratocarcinomas.
12 luripotent stem cells such as those found in teratocarcinomas.
13 ions that the R-Ras subfamily [R-Ras, R-Ras2/teratocarcinoma 21 (TC21), and M-Ras] of small GTP-bindi
14 at contemporary HK2 virions produced by some teratocarcinoma and breast cancer cell lines, as well as
15  CREB(DIEDML), activates transcription in F9 teratocarcinoma and PC12 cells even in the absence of pr
16 nd motivated re-examination of the cases for teratocarcinomas and liver cancer.
17  self-renewal, relate this to the biology of teratocarcinomas and offer testable hypotheses to expose
18  and in tumor cell lines PA-1 (human ovarian teratocarcinoma) and MCF-7 (human breast adenocarcinoma)
19 uman and murine germ cell tumors (testicular teratocarcinomas) and low in non-germ-cell tumors.
20 ussed evidence for a nonmutational origin of teratocarcinoma, and cited related claims in liver carci
21 ranscriptionally targeted gene expression to teratocarcinoma, and that evaluation in other flt-1-posi
22 plantation embryos, primordial germ cells or teratocarcinomas are currently unique in undergoing prol
23  TF(+/+), TF(+/-), and TF(-/-) teratomas and teratocarcinomas are indistinguishable.
24                                              Teratocarcinomas are tumors that arise from primordial g
25  17 and no other human chromosome on a mouse teratocarcinoma background.
26 ed Wnt-13 could modulate the histogenesis of teratocarcinomas by mediating interactions between sub-p
27 nge the enzyme specificity, we transfected a teratocarcinoma cell line (2102Ep) with vectors encoding
28 euron-like cells (NT2N) derived from a human teratocarcinoma cell line (NTera2/D1) with drugs that de
29 since overexpression of SRF in the embryonal teratocarcinoma cell line F9, which normally expresses l
30 n, in virus-like particles isolated from the teratocarcinoma cell line NCCIT, there is significant ac
31                                    The human teratocarcinoma cell line NTera 2 (NT2) can be induced t
32                   Here we report that in the teratocarcinoma cell line NTera2, p53 is subject to lysi
33                             In PA-1, another teratocarcinoma cell line of human origin that has no or
34 ly induced during the differentiation of the teratocarcinoma cell line P19 along a neuronal lineage a
35      Inhibition of myocardin function in the teratocarcinoma cell line P19CL6 prevented differentiati
36  (hNT) neurons derived from the NTera2 (NT2) teratocarcinoma cell line were shown to remain postmitot
37                  NTera-2/D1 (NT2) is a human teratocarcinoma cell line which can be cultured as a div
38        These results are confirmed in the F9 teratocarcinoma cell line, a model for retinoic acid-ind
39       Furthermore, upon differentiation of a teratocarcinoma cell line, the expression of an active L
40                             In the F9 murine teratocarcinoma cell line, this DR5 RARE is required for
41 tivity of DNMT1, -2, -3a, and -3b in a mouse teratocarcinoma cell line.
42 , and to microcell hybrids made with a mouse teratocarcinoma cell line.
43             We have characterized two murine teratocarcinoma cell lines and find no evidence that end
44 ine zipper motif and has been found in human teratocarcinoma cell lines and some tumor cells.
45 , we describe properties of p40 in the human teratocarcinoma cell lines NTera2D1 and 2102Ep.
46 mplicated in the tumorigenicity of the human teratocarcinoma cell lines PA-1 that contain an activate
47                                           F9 teratocarcinoma cell lines, carrying one or two disrupte
48  detected in six human breast epithelial and teratocarcinoma cell lines.
49 , in association with LINE-1 RNA(s) in human teratocarcinoma cell lines.
50 n, and cytokeratins in a human trophoblastic teratocarcinoma cell, HT-H.
51                                        Human teratocarcinoma cells (GH cells), which produce HERV-K p
52                  F9 embryonic stem cell-like teratocarcinoma cells are widely used to study early emb
53  formation of primitive endoderm in mouse F9 teratocarcinoma cells as does the stimulation of the Fri
54 nhibits cellular proliferation of testicular teratocarcinoma cells by two possible mechanisms, p53-de
55 d (RA) treatment of Jun-deficient, mouse F9, teratocarcinoma cells causes the induction of c-jun expr
56                                    Human NT2 teratocarcinoma cells differentiate into neuron-like NT2
57                                              Teratocarcinoma cells ex vivo in tissue culture are also
58  p53-mediated responses in murine testicular teratocarcinoma cells exposed to the drug.
59                         Because F9 embryonic teratocarcinoma cells form primitive endoderm after stab
60 ssociated A3243G mutant mtDNA into human NT2 teratocarcinoma cells frequently causes mtDNA loss.
61          ras oncogene-transformed PA-1 human teratocarcinoma cells have abundant AP-2 mRNA but, parad
62 or growth, B16F10 melanoma and F9 testicular teratocarcinoma cells have been implanted in C57BL/6J an
63             Previous experiments using human teratocarcinoma cells indicated that p40, the protein en
64 duced differentiation of human Ntera 2 (NT2) teratocarcinoma cells into neuron-like cells (NT2-N cell
65 sis that the p53 protein in undifferentiated teratocarcinoma cells is transcriptionally inactive and
66                                   Testicular teratocarcinoma cells rarely contain mutations in TP53,
67       Overexpression of ZPK in NTera-2 human teratocarcinoma cells results in inhibition of PKA induc
68       Interestingly, Wnt3a stimulation of F9 teratocarcinoma cells results in reduced Dishevelled met
69       Genes activated in mouse totipotent F9 teratocarcinoma cells solely by activation of the Rfz1 c
70 terizing the DNA damage response of isogenic teratocarcinoma cells that differ only in their level of
71       RA induces differentiation of F9 mouse teratocarcinoma cells toward neurons as well as extraemb
72                                        These teratocarcinoma cells undergo p53-mediated apoptosis in
73 ransfer of single human chromosomes to mouse teratocarcinoma cells will allow examination of developm
74                       Cisplatin treatment of teratocarcinoma cells with a wild-type p53 gene resulted
75 93 kidney, SK-N-AS neuroblastoma, and NT2/D1 teratocarcinoma cells with NF2 promoter-luciferase chime
76                     Differentiation of these teratocarcinoma cells with retinoic acid results in a ma
77  retinoic acid-induced differentiation of F9 teratocarcinoma cells, a model of epiblast-to-primitive
78 ariety of cell types, including F9 embryonic teratocarcinoma cells, and can influence axial pattern f
79  enzyme is inducible by retinoic acid in P19 teratocarcinoma cells, and we report the cloning from P1
80 ion microscopy as a GFP-fusion protein in F9 teratocarcinoma cells, Bex3 localized, along with concen
81                                     In mouse teratocarcinoma cells, canonical Wnts that inhibit heart
82 n of MEX3A in OCCC cells, as well as ovarian teratocarcinoma cells, reduced cell survival and prolife
83 of p53 by DNA damage results in apoptosis of teratocarcinoma cells, whereas MDM2, encoded by a p53-re
84               In embryonic stem cell-like F9 teratocarcinoma cells, which are widely used to study re
85 NA, as it is in the p40 RNP complex found in teratocarcinoma cells.
86 ated expression of the murine Hoxa-1 gene in teratocarcinoma cells.
87 gregation-independent myogenic conversion of teratocarcinoma cells.
88 s in p53-wild-type cells but not in p53(-/-) teratocarcinoma cells.
89 lls and depleted NT2 (human neuron-committed teratocarcinoma) cells.
90 n average, an 8-fold decrease in alpha5-null teratocarcinomas compared with control tumors.
91 visualize PE migration in vitro using the F9 teratocarcinoma derived embryoid body outgrowth system a
92                         However, alpha5-null teratocarcinomas displayed less undifferentiated tissue
93 gen-activated protein kinase p38 in mouse F9 teratocarcinoma embryonal cells.
94 yonic carcinoma (hEC) cells are derived from teratocarcinomas, exhibit robust proliferation, have a h
95                           Treatment of mouse teratocarcinoma F9 cells with all-trans-retinoic acid (R
96          Furthermore, treatment of F9 murine teratocarcinoma (F9) cells with 10(-6) M all-trans-retin
97 stem cells to form teratocarcinomas in mice; teratocarcinoma formation was reconstituted by reexpress
98                                              Teratocarcinomas formed by EGFR-deltakinase-expressing E
99 ng endothelium of ES-cell origin occurred in teratocarcinomas from heterozygous Lmo2-lacZ ES cells bu
100 and risk of forming unwanted tissues or even teratocarcinomas have yet to be fully assessed.
101 ) cells or fully differentiated human neuron teratocarcinoma (hNT cells).
102                           In human embryonic teratocarcinoma (hNT2) cells transfected with the human
103  the ability of embryonic stem cells to form teratocarcinomas in mice; teratocarcinoma formation was
104 nger for nonseminomas, and for teratomas and teratocarcinomas in particular (N = 58; CT: OR, 1.63; 95
105  compromised immunodeficient mice to produce teratocarcinomas in which Ry1R expression is absent.
106 ance of this Lmo2 expression was assessed in teratocarcinomas induced in nude mice by subcutaneous im
107 era-2N neurons, but not the parental NTera-2 teratocarcinoma line, decarboxylate [2-(15)N]glutamine t
108 library prepared from the human neuronogenic teratocarcinoma line, NTera2, and cloned a 2373 nucleoti
109 transgene expression levels were detected in teratocarcinoma lines, correlating with levels of flt-1
110 ed in which mice bearing i.p. murine ovarian teratocarcinoma (MOT) were injected i.p. with reporter g
111                                   Testicular teratocarcinomas never contain p53 gene mutations even t
112                               In the case of teratocarcinomas, normal germinal stem cells have the po
113 tinoic acid induced differentiation of human teratocarcinoma NT2 cells in association with attenuatio
114 neuronal differentiation in the NTera2 human teratocarcinoma (NT2) cell line.
115 (Genbank accession number DQ516383) in human teratocarcinoma (NT2) cells that differentiate into hNT
116 G promotes neuronal differentiation of human teratocarcinoma NTERA-2 cells.
117 mor cell lines derived from the embryoblast (teratocarcinoma) or from other lineages failed to do so.
118  also detected in Tera-1, a cell line of the teratocarcinoma origin.
119                                     In mouse teratocarcinoma (P19) cells expressing betaAPP695 or bet
120 ously purified recombinant ATX, based on the teratocarcinoma sequence, retains these same activities.
121  morphogen-induced promotion of F9 embryonic teratocarcinoma stem (F9 stem) cells to primitive endode
122 gs cause the differentiation of F9 embryonic teratocarcinoma stem cells into parietal endoderm, an ep
123               Medium conditioned by mouse F9 teratocarcinoma stem cells stably transfected to express
124 hogen-induced decline in Gialpha triggers F9 teratocarcinoma stem cells to progress to primitive endo
125 p26A1 RAREs in F9 embryonal carcinoma cells (teratocarcinoma stem cells) during RA treatment.
126 h human myeloid leukemia and mouse embryonic teratocarcinoma stem cells, either RA-suppressed CAK pho
127 iption factor expressed at high levels in F9 teratocarcinoma stem cells, embryonic stem cells, and ot
128 immortalized fibroblasts (Balb/c3T3), and F9 teratocarcinoma stem cells.
129 ulate specific subsets of target genes in F9 teratocarcinoma stem cells.
130  RA-resistant myeloid leukemia and embryonic teratocarcinoma stem RARalpha(-/-) cells.
131 onal carcinoma (EC) cell line derived from a teratocarcinoma that differentiate exclusively into post
132 and the growth of TF-deficient teratomas and teratocarcinomas were analyzed.
133                          alpha5-null-derived teratocarcinomas were significantly smaller than the wil
134                 The same mutations result in teratocarcinomas when delivered in early differentiating
135 ted region) of DRD2 in NT2 (neuron-committed teratocarcinoma, which endogenously expresses DRD2) and
136    To search for Wnt genes involved in human teratocarcinomas, with a possible role in human embryoge

 
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