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1 stasis, whereas excess RA is well known as a teratogen.
2 transition metal cadmium is an environmental teratogen.
3 require treatment with isotretinoin, a known teratogen.
4    Marginal biotin deficiency may be a human teratogen.
5 n the early 1960s after it was found to be a teratogen.
6 result from exposure to this important human teratogen.
7  belief that cocaine is a uniquely dangerous teratogen.
8 ly in Mosmo-/- embryos are refractory to the teratogen.
9 Moreover, astrogliosis may be common to both teratogens.
10 odium valproate and carbamazepine, are human teratogens.
11 oxification of environmental carcinogens and teratogens.
12  "proximate zone" of most likely exposure to teratogens.
13 caused by in utero exposure to environmental teratogens.
14 tiologies include the local formation of the teratogen acetaldehyde or oxygen radicals by fetal ethan
15 s well as the causal involvement of specific teratogens acting at the earliest stages of neurulation.
16  retaining sensitivity to another known limb teratogen, all-trans retinoic acid (atRA).
17 ilbestrol (DES), a known human developmental teratogen and carcinogen.
18 lysis indicated that the interaction between teratogen and genotype was highly significant (P < or =
19 Although hyperthermia is a recognized animal teratogen and maternal fever has been associated with bi
20 d suggest apoptosis as a potential target of teratogens and genetic defects that are associated with
21           Prevalence of prenatal exposure to teratogens and prenatal care initiation by gestational w
22 ren (5000+) worldwide exposed to this potent teratogen, and the many informative cases in which the e
23  high, as DHCR7-inhibitors can be considered teratogens, and are commonly used by pregnant women.
24                             New uses for old teratogens as well as novel chemical modulators of devel
25 rly exposure to valproic acid (VPA), a known teratogen associated with ND in humans.
26 ecular marker of biomass burning and a known teratogen, being the most abundant (>70%).
27          Thalidomide has a dark history as a teratogen, but in recent years, its derivates have been
28 sy and bipolar disorder and is also a potent teratogen, but its mechanisms of action in any of these
29                          Ethanol (EtOH) is a teratogen, but its teratogenic mechanisms are not fully
30  in the abundance of the protein target of a teratogen can change birth defect outcomes by quantitati
31 een studied for more than 30 years as potent teratogens capable of inducing cyclopia and other birth
32                 Here we identify a potential teratogen causing CHD in mice: maternal iron deficiency
33            IMPORTANCE Zika virus (ZIKV) is a teratogen causing devastating sequelae to the newborns w
34                          RA is also a potent teratogen, causing multi-organ birth defects in humans.
35 Antiseizure medications (ASMs) are potential teratogens commonly prescribed for multiple indications.
36                                              Teratogens could disrupt the two cellular processes to a
37          Here we show that the plant-derived teratogen cyclopamine, which inhibits the Hh response, i
38 ugh a mechanism similar to that of the known teratogen cyclopamine.
39 xamine craniofacial defects by a known human teratogen, cyclopamine.
40 served when cocultures were treated with the teratogen cyclophosphamide.
41         THC therefore acts as a 'conditional teratogen', dependent on a complementary but insufficien
42                                        These teratogens did not prevent the sterol modification of Sh
43 assessments to identify sequelae of prenatal teratogens early in life may improve long-term outcomes
44             Although alcohol is a recognized teratogen, evidence is limited on alcohol intake and ora
45 ant exposure were compared with those of non-teratogen-exposed controls.
46 urred for 48.9% (95% CI, 48.4%-49.5%) of all teratogen-exposed pregnancies (2.5% [2.4-2.5] of all pre
47               It has also been proposed that teratogen exposure may potentiate the effects of genetic
48                          Results showed that teratogen exposure was followed by decreased levels of R
49 into the approximate timing of the causative teratogen exposure.
50  screening platform to mitigate the risks of teratogen exposures in human.
51 apeutic interventions to mitigate effects of teratogen exposures.
52      We propose that increased deposition of teratogens/fetotoxicants to the embryonic compartment is
53 ward screening of 298 FDA-approved drugs and teratogens for growth defects using over 2,400 cortical
54  Maternal diabetes, genetic factors and some teratogens have been shown to be associated with its pat
55                            Ethanol acts as a teratogen in developing fetuses causing abnormalities of
56 n current human doses, ivermectin is a known teratogen in mammals.
57 mentally ubiquitous contaminant, is a potent teratogen in mice.
58  only TCAA appeared to be a specific cardiac teratogen in the fetus when imbibed by the maternal rat.
59 zopanib, vandetanib, and everolimus are also teratogens in these models.
60 The fetal brain is sensitive to a variety of teratogens, including ethanol.
61 f teratogens to achieve this; testing single teratogens independently and not examining combinatorial
62 ome-wide search for susceptibility genes for teratogen-induced clefting in the AXB and BXA set of rec
63 an harm neurons subjected to excitotoxic and teratogen-induced injury.
64 omal regions for both forms of clefting when teratogen-induced.
65                             We conclude that teratogen induction of p53-dependent apoptosis in the de
66 h both multiple genetic loci and exposure to teratogens influencing susceptibility.
67                   We find that mutations and teratogens interact in predictable ways to cause birth d
68 gesting that the mechanism of action of this teratogen involves a pathway(s) requiring Paraxis activi
69                        Isotretinoin, a known teratogen, is strictly regulated through the iPLEDGE pro
70                We investigated mechanisms of teratogen-mediated blockade of maternofetal transport by
71 xification of and protection from cadmium, a teratogen, mutagen and potentially lethal heavy metal.
72 posed rat embryos to valproic acid, a second teratogen newly linked to autism.
73  basis underlying HPE is not known, although teratogens, non-random chromosomal anomalies and familia
74 e analysed the effects of several classes of teratogens on cartilage formation using 200 independent
75                 Retinoic acid (RA), a potent teratogen, produces a characteristic set of embryonic ca
76  life.IMPORTANCEZika virus (ZIKV) is a known teratogen responsible for microcephaly in neonates born
77                                 In addition, teratogens such as jervine, which inhibit the response o
78 genital human syndromes and to the action of teratogens such as thalidomide.
79               Despite its history as a human teratogen, thalidomide is emerging as a treatment for ca
80 myeloma and inflammatory diseases, is also a teratogen that causes birth defects, such as limb trunca
81                       In utero exposure to a teratogen that directly inhibits SMO reduces the penetra
82                                 Alcohol is a teratogen that induces a variety of abnormalities includ
83 , an effective anti-acne therapy, is a known teratogen that is strictly regulated through the iPLEDGE
84 um could be a target of genetic mutations or teratogens that cause human conotruncal heart defects.
85 ryonic exposure to ethanol or retinoic acid, teratogens that cause increased cell death.
86 nd epilepsy but it requires a combination of teratogens to achieve this; testing single teratogens in
87 s as epigenetic intermediaries, which permit teratogens to shape complex, divergent developmental pro
88 gate the mechanism of action of a well-known teratogen, valproic acid (VPA).
89 A during pregnancy also use alcohol, a known teratogen, we examined whether local brain volumes diffe
90  embryos treated with valproic acid, a known teratogen which affects somite segmentation, showed pert
91  genetic or genomic variation to exposure to teratogens, which elicit diverse cell and molecular resp
92 DM (classes A1 and A2) is not an established teratogen yet.