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1 e is a plausible major driver of thalidomide teratogenicity.
2 Fi use during pregnancy carries low risk for teratogenicity.
3 change; skin disorders; hair disorders; and teratogenicity.
4 use is limited by systemic side effects and teratogenicity.
5 further raises concern about potential human teratogenicity.
6 In animals it may cause some teratogenicity.
7 here are substantial data regarding risk for teratogenicity.
8 neffective or not tolerated, because of high teratogenicity.
9 women of child-bearing potential because of teratogenicity.
10 the biophysical properties of viral RNA and teratogenicity.
11 ted with medication use during pregnancy: 1) teratogenicity, 2) perinatal syndromes (neonatal toxicit
12 n ligase, is a direct target for thalidomide teratogenicity and antitumor activity of IMiDs (now know
13 , is a direct protein target for thalidomide teratogenicity and antitumor activity of immunomodulator
14 teratogenic in the zebrafish embryo model of teratogenicity and has a low predicted human dose, indic
15 ential because of antiepileptic drug-related teratogenicity and hormonal interactions; although studi
19 AP4K kinases, which were associated with the teratogenicity and testicular toxicity observed in rats
20 acodynamics (PD, anticonvulsant activity and teratogenicity) and pharmacokinetic (PK) analysis of fou
21 malformations, growth impairment, behavioral teratogenicity, and neonatal toxicity) is reviewed for t
23 re human data regarding potential behavioral teratogenicity are needed in order to understand both th
25 erozygous Shh null mutations exacerbated PBO teratogenicity at all doses tested, including 33 mg/kg.
26 We found no evidence of embryotoxicity or teratogenicity based on the risk of miscarriage, stillbi
27 risk of chronic toxic effects and issues of teratogenicity for women that may affect the choice of d
28 icity: intrauterine fetal death, morphologic teratogenicity, growth impairment, behavioral teratogeni
30 pread clinical use by 1962 because of severe teratogenicity--has antiangiogenic and immunomodulatory
31 l uncertain risk of short term and long term teratogenicity, however, must be weighed against the ris
36 These results indicate that cadmium-induced teratogenicity may be due to the ability of the metal to
37 e nanoparticles can significantly reduce the teratogenicity of bisphenol A, triclosan and 17alpha-eth
39 id, and to the toxicity, carcinogenicity, or teratogenicity of foreign compounds, including drugs.
42 JNK1/2 signaling, 2) JNK1 contributes to the teratogenicity of hyperglycemia, and 3) both JNK1 and JN
50 illamine therapy to protect their fetus from teratogenicity, only to undergo serious deterioration an
53 ion using a validated rat model of valproate teratogenicity that mimics the human scenario of chronic
54 ganogenesis, developmental defects, and drug teratogenicity, with promising applications in tissue an