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1 ease 11 L/min and 8 L/min) than those taking terbutaline.
2 RV observed between sexes were attenuated by terbutaline.
3 e randomly assigned formoterol 4.5 microg or terbutaline 0.5 mg as needed by Turbuhaler in daily dose
4 5 microg provided better asthma control than terbutaline 0.5 mg in patients requiring moderate doses
5 horbol 12-myristate 13-acetate (PMA, 10 nM), terbutaline (0.1 mM), or ATP (1 mM), the binding of SP-A
7 voked NO release (isoproterenol; dobutamine; terbutaline; 10(-9) to 10(-5) m) was blocked by the NOS
11 R responses in newborn rats, we administered terbutaline, a beta2AR agonist, on postnatal day 2 and e
13 ) M), the putative beta 2-adrenergic agonist terbutaline also caused preconstricted arterioles and ve
15 rmal R-R intervals (SDNN), were compared pre-terbutaline and post-terbutaline and across phenotype, g
18 h2 cells were exposed to the beta2AR agonist terbutaline before activation by Ag-presenting B cells.
19 usside (endothelium independent agonist) and terbutaline (beta(2)-adrenergic receptor agonist) with a
23 re isometric tension after administration of terbutaline (concentration range, 10(-8) to 10(-4) M), d
25 to determine whether other bronchodilators (terbutaline, diltiazem, and aminophylline) relax bronchi
28 acting beta-agonist formoterol compared with terbutaline, each taken as needed, in patients with mode
29 rnatants, the lower level of IL-2 present in terbutaline-exposed culture supernatants supported the p
33 pre-exposed to Ag and/or the beta 2AR ligand terbutaline for 24 h before being activated by either a
34 The antagonist propranolol competed with terbutaline for beta2AR binding sites and expectedly rig
35 ry response to ACh, sodium nitroprusside and terbutaline in young premenopausal (all P < 0.05) but no
37 acellular cAMP were similar in both subsets, terbutaline induced an increase in cAMP levels in Th1 ce
38 ntagonist atenolol (10(-6) M) did not affect terbutaline-induced dilation in preconstricted arteriole
41 ist, butoxamine, suggests that the effect of terbutaline is mediated by activation of beta2-adrenergi
42 hese structurally related bronchodilators is terbutaline; it is administered as a prodrug, bambuterol
43 > .05) was the most efficacious, followed by terbutaline (maximum relaxation, 72%+/-13% [proximal], 5
44 2)-adrenergic receptor ligands (epinephrine, terbutaline, metaproterenol, salmeterol, propranolol, al
48 ells to either the beta 2AR-selective ligand terbutaline or the sympathetic neurotransmitter norepine
52 ing beta2-agonists albuterol, fenoterol, and terbutaline provide rapid as-needed symptom relief and s
56 of dobutamine (selective beta1-agonist) and terbutaline (selective beta2-agonist) on glycerol releas
60 vivo tests using zebrafish models found that terbutaline sulfate prevents defects in axons and neurom
62 Interestingly, the prediction suggests that terbutaline sulfate, which is widely used for asthma, is
65 ) responses to administration of intravenous terbutaline (TRB) before and after 5 days of low dietary
67 and combined effects of maternal stress and terbutaline (used to arrest preterm labor), autism risk
68 chiral separation of milligram quantities of terbutaline using sulfated cyclodextrin as a chiral addi
71 aline and adrenaline than when salbutamol or terbutaline were present (e.g., log KD propranolol -8.65
72 d of clenbuterol, cimaterol, procaterol, and terbutaline which acted as full agonists for cAMP produc
73 atients may be treated with theophylline and terbutaline, which clinical experience suggests may redu
75 epinephrine > or = formoterol = fenoterol > terbutaline = zinterol = albuterol > salmeterol > dobuta