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1 tients with STAT1 GOF mutations had impaired terminal maturation.
2 ncluding GATA-1 to block differentiation and terminal maturation.
3 vitro, we show that IL-33 directly inhibited terminal maturation.
4  be down-regulated to allow cells to undergo terminal maturation.
5 cell cycle in chondrocytes and to defects in terminal maturation.
6 onsistent with a role of T-bet in regulating terminal maturation.
7 peptidergic neurons and promote motor neuron terminal maturation.
8 1 transitional cell state (PATS) en route to terminal maturation.
9 ine megakaryocytes, which also have impaired terminal maturation.
10 ATA-1 proliferation arrest and Epo-dependent terminal maturation.
11 iently than C/EBPalpha-ER and did not direct terminal maturation.
12 CML), Bcr-Abl(+) myeloblasts fail to undergo terminal maturation.
13 ferentiation as the cells eventually undergo terminal maturation.
14 ortant role in megakaryocyte endomitosis and terminal maturation.
15  to maintain erythroid cell viability during terminal maturation.
16 d in differentiating oligodendrocytes before terminal maturation.
17  modes of migration that are associated with terminal maturation.
18 id cells undergoing erythropoietin-dependent terminal maturation, although transcripts derived from t
19 onstrated that MCM10 is required for NK cell terminal maturation and acquisition of immunological sys
20      Both cell types fail to complete normal terminal maturation and are present in decreased numbers
21  revealed that although AAMphi had undergone terminal maturation and differentiation, they entered a
22  and Mst2 act as molecular rheostats for the terminal maturation and effector differentiation program
23 at GATA3 is a critical regulator for NK cell terminal maturation and egress out of the BM and that im
24                     This attachment promotes terminal maturation and enucleation of erythroblasts.
25                   Our data indicate that the terminal maturation and enucleation of human primitive R
26  organs, Ag-driven B cell activation induces terminal maturation and Ig isotype class switch (class s
27 esults identify T-bet as a key factor in the terminal maturation and peripheral homeostasis of NK and
28 d, expanded in vitro, and induced to undergo terminal maturation and platelet release.
29 n erythrocyte precursors, facilitating their terminal maturation and protecting against oxidant stres
30 c cytokines because it is necessary both for terminal maturation and regulation of lineage-specific m
31 o the sinusoidal vascular niche, the site of terminal maturation and thrombopoiesis.
32 enic lineage probably influences chondrocyte terminal maturation and turnover of the cartilage matrix
33 o the erythroid lineage, vary further during terminal maturation, and are strongly associated with ch
34 ecreased efficiency, caused a delay in their terminal maturation, and did not invoke V(alpha)14iNKT c
35  a key role in CD4(+) T-cell priming, B-cell terminal maturation, and immunoglobulin (Ig) class-switc
36  developing NK cells promotes functionality, terminal maturation, and long-term survival.
37  that share many morphologic features during terminal maturation but have marked differences in cell
38  proplatelet formation and other features of terminal maturation, but continued to proliferate aberra
39 NKT) cells in the periphery due to a halt in terminal maturation, but despite this deficiency, T-bet(
40          Most chloroplast proteins undergo N-terminal maturation, but this is poorly understood due t
41 hat is, direct inhibition of T cell-mediated terminal maturation by DC.
42 hermore, mice lacking T-bet exhibited both a terminal maturation defect of iNKT cells and a predomina
43 n recovery and similar to beta(Delta/Delta), terminal maturation defects in both bone marrow and sple
44  perturb either axonal arborization or nerve terminal maturation, depending on the stage of deletion.
45 es Itk and Rlk provide important signals for terminal maturation, efficient cytokine production, and
46 ocytes arise from a common progenitor, their terminal maturation follows very different paths; erythr
47 ast cell line that undergoes GATA1-dependent terminal maturation, identifying 2616 GATA1-responsive g
48 on of cell migration as a feature of NK cell terminal maturation in this system.
49 E dysregulation in BM erythroid cells during terminal maturation in vivo.
50 demonstrate that 1) human DCs do not undergo terminal 'maturation' in response to TNF-alpha, 2) DC ph
51 r completely each of the changes that typify terminal maturation, including (a) secretion of relative
52 n from mesenchymal progenitor cells to their terminal maturation into hypertrophic chondrocytes.
53 c interaction with T cells stimulates their "terminal maturation" into fully professional DC.
54                       It is unclear how iNKT terminal maturation is regulated and what factors mediat
55 irement for NE openings during erythroblasts terminal maturation led us to examine a potential role f
56                        Spdef is required for terminal maturation of antral mucous gland cells to prot
57                The absence of Spdef impaired terminal maturation of antral mucous gland cells, as ref
58  repressor, has long been known to drive the terminal maturation of B cells into plasma cells.
59 caspase-3-uncleavable GATA-1 mutant restores terminal maturation of beta-TM erythroblasts, which may
60 The Sox6 transcription factor is crucial for terminal maturation of definitive red blood cells.
61 hage interactions play a central role in the terminal maturation of erythroblasts, including enucleat
62 tween erythroblasts and macrophages promotes terminal maturation of erythroid cells by suppressing ap
63                Our studies revealed that the terminal maturation of human erythroblasts is associated
64                                          The terminal maturation of human erythroblasts requires sign
65 e the epigenetic changes associated with the terminal maturation of human erythroblasts, we performed
66                                              Terminal maturation of invariant NKT (iNKT) cells from s
67  both normal regulation of proliferation and terminal maturation of megakaryocytes, and further, that
68 ar to those found in progenitor cells delays terminal maturation of MEL and G1ER cells, two cell mode
69                        Platelets also affect terminal maturation of monocytes in vitro.
70  data suggest that Sall3 is required for the terminal maturation of neurons destined for the glomerul
71                 Commitment, development, and terminal maturation of NK cells were largely unaffected
72 esults show that in addition to the impaired terminal maturation of NK cells, human STAT5b mutation l
73  are profound effects on the development and terminal maturation of NK cells.
74 e subset-specific Ly49 receptors and thereby terminal maturation of NK cells.
75 regulated cyclin E activity causes defective terminal maturation of nucleated erythroblasts in vivo H
76         These observations indicate that the terminal maturation of OL progenitors derived from multi
77 role in globin switching; and (3) during the terminal maturation of red cells, where it helps control
78 ietic stem cell survival and quiescence, and terminal maturation of select blood lineages.
79  into lung and airway have been established, terminal maturation of the cells remains a vexing proble
80 rs orchestrate myelin gene expression during terminal maturation of the mammalian CNS.
81 ulted in the successful Ly49 acquisition and terminal maturation of the NK cells; however, it could o
82 rrier is established through the coordinated terminal maturation of the retinal pigment epithelium (R
83 membrane or in the cytoplasm allowed for the terminal maturation of transduced progenitor cells.
84 sentation, have been termed T cell-mediated "terminal maturation" of DC, Here we report that XS52 cel
85 hly expressed in the Mo lineage up to 80% at terminal maturation, present on 20% to 30% of maturing M
86 es and what the signals are that drive their terminal maturation remain unclear.
87 stration of polycistronic RNA processing, 3'-terminal maturation, RNA editing, turnover, and translat
88 ha and alpha, is important for completion of terminal maturation steps.
89 shion, with the CD56(bright) stage preceding terminal maturation to the CD56(dim) stage, considered t
90 w that Nurr1 and Pitx3 cooperatively promote terminal maturation to the midbrain DA neuron phenotype
91                    XS52 cells also underwent terminal maturation upon exposure to lipopolysaccharide
92  CD56bright NK cells, and this impairment in terminal maturation was also observed in Irf8-/-, but no
93 lls are karyotypically normal and capable of terminal maturation with approximately 50% enucleation.