ライフサイエンスコーパス: terminal repeat

1 nt downstream transcription from the 3' long terminal repeat.

2 er sequences in the U3 region of the 3' long terminal repeat.

3 d eight, respectively, are duplicated in the terminal repeat.

4 ing of Tat at the TAR region of the HIV long terminal repeat.

5 differential DNA methylation at its 5'-long terminal repeat.

6 stranded RNA40 (ssRNA40) from the HIV-1 long terminal repeat.

7 obin locus within the self-inactivating long-terminal repeat.

8 F-kappaB and its recruitment to the HIV-long terminal repeat.

9 form the capsid, and the right-hand inverted terminal repeat.

10 y differ by the presence of 3(3R) or 4(4R) C-terminal repeats.

11 x that is associated with repressed HIV long terminal repeats.

12 ked by adeno-associated virus (AAV) inverted terminal repeats.

13 showed binding of RPA1 and RPA2 to the KSHV terminal repeats.

14 tures, followed by the nucleation of three N-terminal repeats.

15 anded DNA (dsDNA) genome with large inverted terminal repeats.

16 ipts is promoted by endogenous intronic long terminal repeats.

17 tor genomes that consisted of linear, 1-long-terminal-repeat (1-LTR), and 2-LTR circular DNAs.

18 transcripts, a moderate reduction of 2-long terminal repeat (2-LTR) circles, and a relatively large

19 HIV-1 DNA, including 2-long terminal repeat (2-LTR) circles, and multiply spliced (m

20 Two-long terminal repeat (2-LTR) circles, which are formed in the

21 al HIV DNA, integrated HIV DNA, and two long terminal repeat (2-LTR) circles.

22 efined by an increase in the level of 2-long terminal repeat (2-LTR) circles.

23 DNA to circularization in the form of 2-long terminal repeat (2-LTR) circles.

24 nal plasma CMV (P = 0.04), detectable 2-long terminal repeat (2-LTR), and lower nadir CD4(+) (P < 0.0

25 An increase in 2-long-terminal-repeat (2-LTR) circles in the depleted FACT com

26 egrated viral DNA, particularly the two-long-terminal-repeat (2-LTR) circles.

27 omes containing two copies of the viral long terminal repeat (2LTR circles) were analyzed in using dr

28 extension at the viral U5 end of the 3' long terminal repeat (3'-LTR), which is a poor substrate for

29 tissue from axilla was positive for the long-terminal repeat (33 copies per 10(6) cells) and env (26.

30 litates repression by LANA bound to the KSHV terminal repeats, a function that requires the MeCP2 C t

31 ugs and tested these compounds on HIV-1 long terminal repeat-activated transcription.

32 Two degenerate amino-terminal repeats also interact with the DNA.

33 subunits (named CasC1-6), with its 5' and 3' terminal repeats anchored by CasD and CasE, respectively

34 semble that is highly polarized toward the C-terminal repeat and cap.

35 Both P-TEFb recruitment to the HIV long terminal repeat and enhanced HIV processivity were block

36 d cryptic splice sites in the HIV 5iota long terminal repeat and gag gene as well as in the beta-glob

37 rictive chromatin structures at the HIV long terminal repeat and limiting P-TEFb levels contribute to

38 ons in the regulatory elements in their long terminal repeats and differed in a helical segment of en

39 The DNA of the element is marked by long terminal repeats and encodes a single large protein with

40 NMT3a/3b in suppressing retrotransposon long terminal repeats and long interspersed elements, respect

41 essing of extraneous CRISPR RNAs from Cas12a terminal repeats and sequence- and context-dependent los

42 ted replication fork protection at the viral terminal repeats and that Tim-dependent recombination-li

43 subgenomic DNA fragments, spanning the long terminal repeats and the Gag gene, are excised in vivo,

44 tational analysis of the HERV-K (HML-2) long terminal repeat, and treatments with agents that inhibit

45 ranulatus genome, especially Gypsy-like long terminal repeats, and there has also been an expansion i

46 ut any nucleation step by using the intact N-terminal repeats as a template.

47 c gene family expansions, the number of long-terminal-repeat-based retrotransposons, and mechanisms o

48 romatin readers of the bromodomain and extra-terminal repeat (BET) family, which includes BRD4.

49 unction analysis indicates that several long-terminal-repeat bursts that occurred from 5.7 million ye

50 load was associated with undetectable 2-long terminal repeat circles (P < .001) and HIV-negative or i

51 a smaller increase in the number of two-long terminal repeat circles than for virus specifically bloc

52 duction (ie, HIV-specific antibodies, 2-long terminal repeat circles) and markers of immune activatio

53 ompetent reservoirs, proviral DNA, or 2-long-terminal repeat circles, although APOBEC3G, TRIM5alpha,

54 RNA transcription or more detectable 2-long terminal repeat circles.

55 013), and frequency of detectable HIV 2-long terminal repeat circular DNA (P=.013) were significantly

56 normally in MX2-expressing cells, but 2-long terminal repeat circular forms of HIV-1 DNA are less abu

57 We demonstrate that the foamy virus long terminal repeats contain an insulator element that binds

58 )-RNA strand genome of retroviruses and long terminal repeat-containing retrotransposons reflects a m

59 un-described polarity effect, in which the N-terminal repeats contribute more to affinity than C-term

60 erally true that a T/SxxH motif close to the terminal repeats contributes little or even negatively t

61 We further found that the C-terminal repeat (CTR) domain of Spt5, which is dispensab

62 PP1 dephosphorylates an Spt5 carboxy-terminal repeat (CTR), but not Spt5-Ser666, a site betwe

63 motes Paf1C recruitment by phosphorylating C-terminal repeats (CTRs) in Spt5, and we show that Kin28

64 many 2C transcripts are initiated from long terminal repeats derived from endogenous retroviruses, s

65 nd recognize an unexpected abundance of long terminal repeat-derived and LINE1-mobilized transposed e

66 considerably reduced mLANA binding to viral terminal repeat DNA as assessed by electrophoretic mobil

67 However, during HIV-1 infection, two-long terminal repeat DNA circles (2-LTRcs) are also generated

68 ependent on the RNA polymerase II (RNAPII) C-terminal repeat domain (CTD) and the phosphorylation of

69 we found that Bur1 phosphorylates the Spt5 C-terminal repeat domain (CTD) both in vivo and in isolate

70 ytic subunit of the major elongation-phase C-terminal repeat domain (CTD) kinase in Saccharomyces cer

71 teins bind the hyper-phosphorylated RNAPII C-terminal repeat domain (CTD) phosphorylated on both Ser2

72 alter this Rtf1 domain or delete the Spt5 C-terminal repeat domain (CTR) disrupt the interaction bet

73 formins, Sca2 is monomeric, but has N- and C-terminal repeat domains (NRD and CRD) that interact with

74 and chimeric transcripts initiated from long terminal repeats during zygotic genome activation.

75 We show that a long terminal repeat element inserted into intron 35 exposes

76 e as well as the transposon cargo flanked by terminal repeat element sequences.

77 demonstrate that DNA hypomethylation at long terminal repeat elements representing the most recent ge

78 lf-inactivating vectors devoid of viral long-terminal-repeat enhancers have proven safe; however, tra

79 n dominance, temporally associated with long-terminal-repeat expansion in the A subgenome that also r

80 HDAC3 contribute to repression of HIV-1 long terminal repeat expression in the HeLa P4/R5 cell line m

81 ion rate included the density of exons, long terminal repeats, GC content of the gene, and DNA methyl

82 ntiviral vectors with self-inactivating long terminal repeats, have been shown to have improved safet

83 (iii) However, the carboxy-terminal repeat in the VEEV HVD is indispensable for VEE

84 shock factor binding HREs within their long terminal repeats in seven Brassicaceae species.

85 Insertions of long terminal repeats in the past 5 million years are respons

86 BRCT repeats (breast cancer gene 1 [BRCA1] C terminal repeats) in the Dpb11 protein [5, 6].

87 of unique antibodies identified bound the C-terminal repeat-in-toxin (RTX) domain.

88 c2(jeb)) due to a murine leukemia virus long terminal repeat insertion in Lamc2 (laminin gamma2 gene)

89 ep 78/68 polypeptide in conjunction with AAV terminal repeat integrating elements.

90 n immunodeficiency virus type 1 (HIV-1) long terminal repeat is present on both ends of the integrate

91 ow that this retrotransposon with LTRs (Long Terminal Repeats) is widely distributed among the Rubiac

92 e D sequence, from both ends of the inverted terminal repeats (ITRs) in the adeno-associated virus se

93 Using the analysis of two long terminal repeat junctions in HIV-infected cells, we show

94 tant inhibition of NF-kappaB and HTLV-1 long terminal repeat (LTR) activation.

95 s have been reported to stimulate HIV-1 long terminal repeat (LTR) activity.

96 : MGEScan-long terminal repeat (LTR) and MGEScan-non-LTR are successful

97 t, the two orthologs similarly restrict long terminal repeat (LTR) and non-LTR retrotransposons (MusD

98 NF-kappaB sites in the U3 region of the long terminal repeat (LTR) are critical for Cav-1-mediated in

99 e RNA polymerase II (RNAP II) on the 5' long terminal repeat (LTR) but not on the 3' LTR.

100 tone proteins at the HIV type 1 (HIV-1) long terminal repeat (LTR) by histone deactylases (HDACs) can

101 were assayed for total HIV-1 DNA and 2-long terminal repeat (LTR) circles by quantitative polymerase

102 mestic cat cell susceptibility and FeLV long terminal repeat (LTR) copy number, similar to observatio

103 Domestic cat enFeLV env and long terminal repeat (LTR) copy numbers were determined for e

104 found that repressive histone marks and long terminal repeat (LTR) DNA methylation could be detected

105 to the Repbase collection of known ERV/long terminal repeat (LTR) elements to annotate the retrovira

106 annotated transcripts overlapping with long-terminal repeat (LTR) elements, several thousand of whic

107 or octamer complex with the viral cDNA long terminal repeat (LTR) ends termed an intasome.

108 e transcriptional activity of the viral long terminal repeat (LTR) from Vpr-deficient proviruses was

109 ut 11.7% with intact genomes and normal long terminal repeat (LTR) function.

110 c reprogramming effect of ZL0580 on HIV long terminal repeat (LTR) in microglia.

111 we show that methylation of the HIV 5' long terminal repeat (LTR) in the latent viral reservoir of H

112 , but rather transcription of the HIV-1 long terminal repeat (LTR) is increased in IL-4-producing CD4

113 formation at several targets within the long terminal repeat (LTR) of HIV.

114 entified a transcript that contains the long terminal repeat (LTR) of lambda-olt 2-1 and shows a simi

115 demonstrate that HspBP1 inhibits HIV-1 long terminal repeat (LTR) promoter activity.

116 dition, we detected Tip110 at the HIV-1 long terminal repeat (LTR) promoter and found that Tip110 exp

117 ng of viral transcription driven by the long terminal repeat (LTR) promoter of HIV-1.

118 he G-quadruplex structures in the HIV-1 long terminal repeat (LTR) promoter suppresses viral transcri

119 sulted in decreased Tat-dependent HIV-1 long-terminal repeat (LTR) promoter transactivation as well a

120 of NucDHS in the proximal region of the long terminal repeat (LTR) promoter was associated with the a

121 by the mouse mammary tumor virus (MMTV) long terminal repeat (LTR) promoter were morphologically indi

122 this cell line was specific for the MLV long terminal repeat (LTR) promoter, as normal levels of repo

123 coding the viral proteins is flanked by long terminal repeat (LTR) regions from the retrovirus.

124 le for structural discovery of complete long terminal repeat (LTR) retroelements, which are widesprea

125 long interspersed elements (LINEs), and long terminal repeat (LTR) retroelements, which include endog

126 Interestingly, we find that a long terminal repeat (LTR) retrotransposon insertion upstream

127 cing of all 13 intact copies of the Tf2 long terminal repeat (LTR) retrotransposon.

128 s known repeats including a majority of long terminal repeat (LTR) retrotransposons (13.67%).

129 recombination with specific classes of long terminal repeat (LTR) retrotransposons and organize into

130 from these isoforms appeared to target long terminal repeat (LTR) retrotransposons and other unrelat

131 Long terminal repeat (LTR) retrotransposons are an abundant c

132 Long terminal repeat (LTR) retrotransposons are closely relat

133 Endogenous retroviruses and long terminal repeat (LTR) retrotransposons are mobile geneti

134 Retroviruses evolved from long terminal repeat (LTR) retrotransposons by acquisition of

135 B homologues have been shown to silence long terminal repeat (LTR) retrotransposons by recruiting his

136 d mouse intracisternal A particle (IAP) long terminal repeat (LTR) retrotransposons in cultivated cel

137 om either insertions of low-copy number long terminal repeat (LTR) retrotransposons or deletions, the

138 rly twofold increase in the deletion of long terminal repeat (LTR) retrotransposons via solo LTR form

139 BEL/Pao-like long-terminal repeat (LTR) retrotransposons were annotated fr

140 d-repeat transposable elements (MITEs), long terminal repeat (LTR) retrotransposons, and non-LTR retr

141 genous retroviruses (ERVs), also called long terminal repeat (LTR) retrotransposons, begins with tran

142 e the performance of methods annotating long terminal repeat (LTR) retrotransposons, terminal inverte

143 Long terminal repeat (LTR) retrotransposons, the most abundan

144 obilization of specific low-copy number long-terminal repeat (LTR) retrotransposons, which differ amo

145 me surveillance role by controlling Tf2 long terminal repeat (LTR) retrotransposons.

146 transposable elements, 77% of which are long terminal repeat (LTR) retrotransposons.

147 dant elements in eukaryotes are the non long terminal repeat (LTR) retrotransposons.

148 ithin the Ty3/gypsy-like superfamily of long terminal repeat (LTR) retrotransposons.

149 tail here the contribution of different long terminal repeat (LTR) sequences for the establishment of

150 ter phenotypically resembles endogenous long terminal repeat (LTR) sequences, pointing to a select ro

151 bp insulator located in the foamy virus long terminal repeat (LTR) that has high-affinity binding to

152 activates transcription from the viral long terminal repeat (LTR) through recruitment of cellular CR

153 In these species the CMTs silence long terminal repeat (LTR) transposons in the distal chromati

154 ped to the NF-kappaB sites in the HIV-1 long terminal repeat (LTR) U3 and could be transferred to MLV

155 he transcriptional activity of the XMRV long terminal repeat (LTR) was found to be higher than the Mo

156 BRG1 associates with Tax at the HTLV-1 long terminal repeat (LTR), and coexpression of BRG1 and Tax

157 1 RNA by recruiting P-TEFb to the HIV-1 long terminal repeat (LTR), and we show that inhibition of HS

158 owing the reduction in H3K27 at the HIV long terminal repeat (LTR), subsequent exposure to the HDACi

159 trinsic toggling of HIV's promoter, the long terminal repeat (LTR), to generate bimodal ON-OFF expres

160 lity group protein A1 (HMGA1) and viral long terminal repeat (LTR), which led to higher levels of HIV

161 ntrinsic NF-kappaB activator, increased long terminal repeat (LTR)-dependent XMRV transcription.

162 We show that long terminal repeat (LTR)-derived transcripts contribute ext

163 We found two instances of long terminal repeat (LTR)-driven provirus transcription but

164 ncreases HIV infection by enhancing HIV long terminal repeat (LTR)-driven transcription via the NF-ka

165 mas transiently transfected with an HIV long terminal repeat (LTR)-luciferase reporter that contained

166 elongation complex essential for HIV-1 long terminal repeat (LTR)-mediated and general cellular tran

167 In the mouse, long terminal repeat (LTR)-retrotransposons, or endogenous re

168 he CD28-responsive element of the HIV-1 long terminal repeat (LTR).

169 tivating the HIV-1 promoter, termed the long terminal repeat (LTR).

170 sequences in the U3 region of the viral long terminal repeat (LTR).

171 E, in the surface glycoprotein (SU) and long terminal repeat (LTR).

172 ranscription initiation site on the HIV long terminal repeat (LTR).

173 lation of histones located at the viral long terminal repeat (LTR).

174 me2) that colocalizes with a retroviral long terminal repeat (LTR).

175 ment (HRE) localized in the proviral 5' long terminal repeat (LTR).

176 s well as the coactivator CBP, with the long terminal repeat (LTR).

177 IM1 complex for activation of the viral long terminal repeat (LTR).

178 s (TE), including endogenous retroviral long terminal repeats (LTR), short and long interspersed nucl

179 sed nuclear elements (LINE), but not in long terminal repeats (LTR).

180 'Copy-and-paste' long-terminal-repeat (LTR) retrotransposons have been particu

181 ects on nuclear histone acetylation and long-terminal-repeat (LTR) transcription.

182 rom their well-characterized effects of long-terminal-repeat (LTR)-driven gene expression.

183 HIV-1 test v2.0 (targeting gag and the long terminal repeat [LTR]).

184 tion of, the integrated viral promoter (long terminal repeat [LTR]).

185 We tag hPSCs by GFP, expressed by the long terminal repeat (LTR7) of HERVH endogenous retrovirus.

186 ammaRV/LV) with self-inactivating (SIN) long terminal repeats (LTRs) and internal moderate cellular p

187 ogenous retroviruses and their solitary long terminal repeats (LTRs) compose >40% of the human genome

188 contains identical 5' and 3' peripheral long terminal repeats (LTRs) containing bidirectional promote

189 intrachromosomal recombination between long terminal repeats (LTRs) flanking GAP1.

190 Retrotransposons often carry long terminal repeats (LTRs) for retrovirus-like reverse tran

191 Retrotransposons containing long terminal repeats (LTRs) form a substantial fraction of e

192 yza genomes, and based on the dating of long terminal repeats (LTRs) of FRetro3 it was amplified in t

193 of nucleotide substitution between two long terminal repeats (LTRs) of individual orthologous LTR-re

194 trong promoter/enhancer elements in the long terminal repeats (LTRs) of murine leukemia virus (MLV)-b

195 cyte harboring active versus restricted long terminal repeats (LTRs) revealed that the gene expressio

196 nabled the accurate prediction of 20.5% long terminal repeats (LTRs) that doubled the previous estima

197 ichment in transposable elements (TEs): long terminal repeats (LTRs) were randomly located across the

198 tering of telomeres and retrotransposon long terminal repeats (LTRs)) were observed throughout the ce

199 s retroviral elements (ERVs) containing long terminal repeats (LTRs), are silenced through trimethyla

200 tains identical DNA sequences, known as long terminal repeats (LTRs), at its 5' and 3' ends.

201 lized to endogenous retrovirus-K (ERVK) long terminal repeats (LTRs), which act as imprinted promoter

202 his is flanked by two loxP sites in its long terminal repeats (LTRs).

203 transposable elements (TEs), including Long-Terminal-Repeats (LTRs) and SINE-VNTR-Alus (SVAs), that

204 ith transgenic NF-kappaB reporters (HIV-long terminal repeat/luciferase [HLL]), we found exaggerated

205 of the mouse mammary tumor virus (MMTV) long-terminal repeat (MMT mice).

206 oter, and the mouse mammary tumor virus long terminal repeat (MMTV-LTR).

207 The non-long terminal repeat (non-LTR) retrotransposon R2 is inserted

208 Non-long terminal repeat (non-LTR) retrotransposons are highly ab

209 Many non-long terminal repeat (non-LTR) retrotransposons lack internal

210 ave the hallmarks that characterize non-long terminal repeat (non-LTR) retrotransposons; they have al

211 trotransposition activity of the L1 non-long-terminal-repeat (non-LTR) retrotransposon in both HeLa a

212 Additionally, mutation of the inverted terminal repeat of the rAAV genome, which has been propo

213 The long terminal repeats of lymphomagenic P-MLVs are differentia

214 found TINATs to be encoded in solitary long terminal repeats of the ERV9/LTR12 family, which are epi

215 owed that a minimal region in the ARC2 and N-terminal repeats of the LRR domain coordinate the activa

216 ith FXR family members are mediated by the C-terminal repeating peptide of HVD.

217 ontrol of the mouse mammary tumor virus long terminal repeat promoter, develop multifocal hyperplasia

218 rotein enhances Tat-mediated HIV-1 LTR (long terminal repeat) promoter activity.

219 ransferase, the cysteine protease, and the C-terminal repeat region can be aligned within three domai

220 Zn(2+) coordinates to the flexible, N-terminal repeat region of PrP(C) and drives a tertiary c

221 The C-terminal repeat region of Spt5, which has been implicate

222 e-long, noncoding RNA segment in the 5' long terminal repeat region of viral transcripts.

223 a panel of ATIp mutants indicated that the C-terminal repeat region was required for inclusion format

224 associating with proviral DNA at the 5' long terminal repeat region, recruiting KAP1 and HP1, and imp

225 coverage was obtained in the highly GC-rich terminal repeat regions.

226 , generating DNA products with heterogeneous terminal repeat registers.

227 lies of long interspersed element 1 and long terminal repeat retroelements, which are disparately met

228 be explained by the presence of similar long terminal repeat retroelements, which were enriched at th

229 -silencing pathways, particularly copia long terminal repeat retrotransposon in Drosophila melanogast

230 Tf1, a long-terminal repeat retrotransposon in Schizosaccharomyces p

231 Previous studies of Tf1, a long terminal repeat retrotransposon in Schizosaccharomyces p

232 Ty1, a long terminal repeat retrotransposon of Saccharomyces, is str

233 This reduced capacity for long terminal repeat retrotransposon silencing and removal in

234 The Saccharomyces cerevisiae long terminal repeat retrotransposon Ty3 integrates within on

235 he Saccharomyces cerevisiae genome, the long terminal repeat retrotransposon Ty3 is found at RNA poly

236 walnut short interspersed element (SINE) and terminal-repeat retrotransposon in miniature (TRIM) elem

237 Ms2 allele in the ms2 mutant has acquired a terminal-repeat retrotransposon in miniature (TRIM) elem

238 findings echo an earlier study with the long-terminal-repeat retrotransposon of Saccharomyces cerevis

239 We initially investigated 510 long terminal repeat-retrotransposon (LTR-RT) families compri

240 Analyzing the long terminal repeat-retrotransposon (LTR-RT) type of TE, we

241 Long terminal repeat retrotransposons (LTR-RTs) are prevalent

242 ansposable elements (TEs), particularly long terminal repeat retrotransposons (LTR-RTs), in recombina

243 marily by the periodic amplification of long terminal repeat retrotransposons (LTR-RTs).

244 ous end joining, mediates clustering of long terminal repeat retrotransposons at centromeres in fissi

245 Terminal repeat retrotransposons in miniature (TRIMs) ar

246 Terminal repeat retrotransposons in miniature (TRIMs) ar

247 and evolutionary and genomic studies of long terminal repeat retrotransposons in other genomes.

248 ure (TRIMs) are a unique group of small long terminal repeat retrotransposons that are difficult to i

249 genome due to a rapid amplification of long terminal repeat retrotransposons that occurred 38 millio

250 Unlike other long terminal repeat retrotransposons, TRIMs are enriched in

251 activity predominately driven by Gypsy long terminal repeat retrotransposons, which extended the low

252 erived from internal deletions of large long terminal repeat retrotransposons.

253 y of the replication strategies used by long terminal repeat retrotransposons.

254 Structural analysis of EBV terminal repeats revealed a banding pattern consistent w

255 Finally, analysis of KERV long terminal repeat sequences using massively parallel seque

256 le and mismatches/insertions or deletions in terminal repeats, showing their alignment or base-pairin

257 genous retroviruses [ERVs] and 488 solo long terminal repeats [sLTRs]) within the C57BL/6J mouse geno

258 Short terminal repeats (STRs) were performed simultaneously on

259 ee sites are located in a region of the KSHV terminal repeat subunit previously recognized as a minim

260 ial unfolding, when allowed to refold, the C-terminal repeats successively regain structures without

261 roviruses and proviruses devoid of a 5' long terminal repeat, suggesting that the expression of HERV-

262 Here we tested a set of long terminal repeat TE families for roles as enhancers in mo

263 all single-stranded DNA genome with inverted terminal repeats that form hairpin structures.

264 st five nucleosomes, is found at the 5' long terminal repeat, the location and modification state of

265 the 3' end of the coding region and the long terminal repeat, this retrotransposon family contains a

266 at repressed NEERV by binding the NEERV long terminal repeat to recruit the transcriptional regulator

267 was further located to a position within the terminal repeat (TR) and its deletion resulted in lower

268 LANA binds to KSHV terminal repeat (TR) DNA and simultaneously associates w

269 LANA binds to KSHV terminal repeat (TR) DNA and tethers the viral episomes

270 1,162-amino-acid protein that acts on viral terminal repeat (TR) DNA to mediate KSHV episome persist

271 is an 1,162-amino-acid protein that tethers terminal repeat (TR) DNA to mitotic chromosomes to media

272 sociated herpesvirus (KSHV) initiates at the terminal repeat (TR) element and requires trans-acting e

273 The prevalence of piggyBac(PB)-like terminal repeat (TR) elements in the human genome raises

274 LANA acts on the KSHV terminal repeat (TR) elements to mediate KSHV episome ma

275 LANA binds cooperatively to the terminal repeat (TR) region of the viral episome via adj

276 d LANA-mediated recruitment of NAP1L1 at the terminal repeat (TR) region of the viral genome.

277 d nuclear antigen (LANA), which binds in the terminal repeat (TR) region of the viral genome.

278 with the BAC vector cassette inserted in the terminal repeat (TR) region.

279 o the viral origin of replication within the terminal repeat (TR) region.

280 bound with high occupancy to the KSHV genome terminal repeats (TR) and to a few minor binding sites i

281 s (KSHV) genomes is mediated in cis by their terminal repeats (TR).

282 f the KSHV-encoded LANA protein to the viral terminal repeats (TR).

283 LANA self-associates to bind KSHV terminal-repeat (TR) DNA and to mediate its replication.

284 LANA binds the KSHV terminal-repeat (TR) sequence through its carboxy-termin

285 LANA binds the KSHV terminal-repeat (TR) sequence to mediate DNA replication

286 btelomere features such as the start of each terminal repeat tract, SRE identity and organization, an

287 and cohesin bind within 3 kb of the start of terminal repeat tracts at many, but not all, subtelomere

288 tallography have deciphered several inverted terminal repeat-transposase complexes that are intermedi

289 on by CHART revealed EBER2's presence at the terminal repeats (TRs) of the latent EBV genome, overlap

290 y enriched within repeat regions such as the terminal repeats (TRs).

291 esulted in reduced binding of LANA with KSHV terminal repeats (TRs).

292 (nt) pri-miRNA, encoded within the BFV long terminal repeat U3 region, that is subsequently cleaved

293 red to the cell surface is mediated by the C-terminal repeating units.

294 nctional annotations (e.g. centromeres, long terminal repeats) using 3D genome reconstructions from H

295 pulations whose genomes had identical direct terminal repeats versus those with circularly permuted r

296 all eight open reading frames (ORFs) in the terminal repeat was investigated in IgM(+) WBC from koi

297 A pair of inverted terminal repeats was detected in YSLPV1, suggesting that

298 ells and the replication-competent ASLV long terminal repeat with a splice acceptor/tv-a glioma mouse

299 d specific DNA sequences containing inverted terminal repeats with similarity to piggyBac transposons

300 iven the shift toward self-inactivating long terminal repeats with weaker promoters to control the ri