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1 and was 30-fold above control levels at the terminal stage.
2 p53 or INK4a mutations upon progression to a terminal stage.
3 r myelin mutants, were not obvious until the terminal stage.
4 wn about SMG cell differentiation during the terminal stages.
5 ive, involving the entire cerebral mantle in terminal stages.
7 stic technology, roughly 80% of cases are in terminal stages at the time of diagnosis, and a large pr
9 oplasts contain both OspA and OspB and are a terminal stage in the bactericidal process induced by Fa
11 ur studies suggest that TIG3 facilitates the terminal stages in keratinocyte differentiation via acti
12 10,000-fold difference between SY and FU at terminal stages, indicating that PrP-res content does no
14 of congenic BALB/c.lambdaLIZ N5 mice in the terminal stage of a plasmacytoma induction experiment, 2
19 also facilitates the membrane fusion at the terminal stage of cytokinesis by controlling the localiz
22 entrosome-related abscission site during the terminal stage of cytokinesis, implicating RalA as a cri
24 t direct activation of genes involved in the terminal stage of differentiation may cause neuronal dif
27 ns in approximately 130 days and reached the terminal stage of disease in approximately 150 days.
35 nge alcohol accentuates loss of body mass at terminal stage of simian immunodeficiency virus (SIV) in
36 icles (PSVs) with the plasma membrane at the terminal stage of spore differentiation in Dictyostelium
37 mation of the membrane attack complex at the terminal stage of the complement activation cascades via
38 levels of human CD59, a CRP that acts at the terminal stage of the complement cascade, we present evi
41 a transcriptional repressor that acts at the terminal stage of the neuronal differentiation pathway a
43 eed conditions that mark the initial and the terminal stages of a given differentiation pathway and c
49 are a set of enzymes that participate in the terminal stages of bacterial peptidoglycan assembly.
51 were used to characterize progression of the terminal stages of Caulobacter crescentus cell division.
54 necessary and independent roles in both the terminal stages of CI assembly and in coenzyme Q metabol
55 (CTBs) revealed that HCMV infection impedes terminal stages of differentiation and invasion by vario
56 trations decreased to undetectable levels at terminal stages of disease in animals that succumbed to
58 rain tissues, edema was detected only at the terminal stages of disease, likely caused by the leakage
62 erythropoiesis is important in promoting the terminal stages of erythroid development and may further
63 rized and is believed to be critical for the terminal stages of erythroid differentiation, but the ad
64 proliferation to differentiation during the terminal stages of erythropoiesis is a tightly controlle
66 hosphatase inhibitor okadaic acid during the terminal stages of expression to create a hyperphosphory
68 orter to hydrolyze Glc-6-P to glucose in the terminal stages of glycogenolysis and gluconeogenesis.
69 bits a selective role being required for the terminal stages of granulocyte development but is dispen
73 uent reason for sedation was delirium in the terminal stages of illness (median, 57.1%; range, 13.8%
74 ted rhesus macaques, at the asymptomatic and terminal stages of infection, were cultured in ethanol 2
75 nce of spontaneous diabetes, 5) arrested the terminal stages of islet cell destruction, and 6) disrup
78 provide an understanding of tumor growth at terminal stages of malignancy, they do not address tumor
79 ptors may have a specific role in regulating terminal stages of myelination, such as myelin membrane
80 iferation leading to astrogliosis during the terminal stages of neurodegeneration in Sandhoff disease
82 hat receptor expression is restricted to the terminal stages of olfactory neuron differentiation, and
86 e megakaryocytes resulted in a defect in the terminal stages of platelet production and had a mild ef
94 expression patterns between the initial and terminal stages of the differentiation pathway, coupled
95 The paradox is that with the exception of terminal stages of the disease or chemotherapy treatment
97 ed metal enrichment events coincide with the terminal stages of Tiwanaku culture (1000 to 1200 A.D.)
101 ficult to identify by analyzing cells within terminal stage tumors, whose identity could be concealed
103 nd retinal pigment epithelium in all but the terminal stages, when subtle changes were noted within t
104 of non-GABAergic to GABAergic neurons in the terminal stages, which implies that non-GABAergic neuron
105 y relevant stages such as advanced stage and terminal stage with in vivo 2-deoxy-2-[(18)F]fluoro-D-gl