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1 nt to the chronic injury of aging-associated testicular atrophy.
2 viable, but sterile flies, exhibiting severe testicular atrophy.
3  counts, abnormal sperm morphology, and mild testicular atrophy.
4 tin mice aged 19 to 22 months showed greater testicular atrophy and decreased average seminiferous tu
5 of partial androgen insensitivity, including testicular atrophy and decreased fertility, are common i
6 E2F-1 are viable and fertile, yet experience testicular atrophy and exocrine gland dysplasia.
7 e demonstrate that Kdm2a deficiency leads to testicular atrophy and male infertility.
8 mbryonic Sertoli cells (SCs) leads to severe testicular atrophy and male sterility owing to rapid dep
9 sks of infertility, testicular hypofunction, testicular atrophy, and a composite of low sperm concent
10 o the developmental arrest of spermatocytes, testicular atrophy, and infertility.
11                       Males exhibited marked testicular atrophy associated with increased apoptosis o
12 in the absence of liver dysfunction produces testicular atrophy by reduction in mitosis, maturation a
13 encephalopathy Leigh syndrome, develop early testicular atrophy caused by a complete arrest of sperma
14 e a knock-in mouse model that reproduces the testicular atrophy, diminished fertility, and systemic s
15 der increased gonadotrophin drive because of testicular atrophy inducing events.
16 ing cell-type-specific markers, we find that testicular atrophy is due to severe loss of germ cells,
17 pecia, skin atrophy, kyphosis, osteoporosis, testicular atrophy, lipofuscin accumulation in renal pro
18 ad unilateral obstruction with contralateral testicular atrophy or epididymal obstruction.
19  However, PRL1(-/-)/PRL2(+/-) male mice show testicular atrophy phenotype similar to PRL2(-/-) mice.
20  display signs of premature aging, including testicular atrophy, reduced LC and Sertoli cell (SC) num
21 ands attributed to androgen effects, such as testicular atrophy, seminiferous tubule diameter reducti
22  At post-natal day 200, random appearance of testicular atrophy was noted in exposed offspring, and l