戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  following prolonged tetanic stimulation (40 tetani).
2 n to study tarantula muscle before and after tetani.
3 d to 1.8 +/- 0.06 mm s(1) over the last five tetani.
4 on of either drug alone, induced LTD without tetani.
5 ency stimuli were preceded by high-frequency tetani.
6 es to intact frog muscle fibres during fused tetani.
7  the frog, was investigated during isometric tetani.
8 ) but not LTP induced by two 1-s-long 100-Hz tetani.
9 w Clostridium lineages closely related to C. tetani.
10 se caused by a toxin produced by Clostridium tetani-a Gram-positive bacillus found in high concentrat
11 due to Clostridium botulinum and Clostridium tetani among users of illegal injected drugs (IDUs) occu
12 artifacts, even while segments were in fused tetani and developing maximum tensions of more than 600
13 ed by the toxin of the bacterium Clostridium tetani and is characterised by muscle spasms and autonom
14 flashes/1000 mum(2).100 s before and after 5 tetani) and markedly decreased (to 7.7 +/- 1.6 flashes/1
15 ist blocked the LTD induction by serotonin + tetani, and co-application of a group I mGluR agonist an
16 ses, ColG from C. histolyticum, ColT from C. tetani, and ColQ1 from the Bacillus cereus strain Q1, wh
17 evelopment that exhibited summation, unfused tetani, and fused tetani in a frequency-dependent manner
18 An immunoassay for the determination of anti-tetani antibodies has been developed using a screen prin
19 ution factor should be small, while the anti-tetani antibody incubation time should be long.
20 ed during a series of thirty 0.2 s isometric tetani at L(o), the length optimal for force.
21  every 100 ms during electrically stimulated tetani at muscle lengths that varied 1.5-fold and at the
22                                       During tetani, birefringence increased with approximately the s
23 increase myoplasmic [Ca2+] during twitch and tetani, but not to reverse the shift in optimum length d
24 ism preventing hyperactivity and undesirable tetani, by preventing subsequent stimuli eliciting actio
25                                         Such tetani caused a modest AP decrease, to a similar extent
26 cted against lethal infection by Clostridium tetani cells and Bacillus anthracis spores following top
27      Phylogenetic analyses revealed known C. tetani clades as well as potentially new Clostridium lin
28 ns occur in hippocampus in vitro after brief tetani delivered to afferent pathways.
29 d identification of both C. botulinum and C. tetani demonstrates a sensitivity and specificity simila
30            We found that B. anthracis and C. tetani epitopes were the most promiscuous of the group t
31 eurotoxin (TeNT) are released by Clostridium tetani generated from spores inside a necrotic wound.
32  pathway entrance in homodimeric Clostridium tetani H(+)-PPase upon ligand binding.
33 ngens, Clostridium botulinum and Clostridium tetani, has shown that no spoVF orthologues exist in the
34 hibited summation, unfused tetani, and fused tetani in a frequency-dependent manner.
35 alysis identifies DNA from neurotoxigenic C. tetani in archaeological human samples, and a novel vari
36 eatest impact on the response, i.e. the anti-tetani incubation time and the dilution factor of the la
37                            Repetitive spaced tetani induce glutamate receptor-dependent stable restru
38 ive immune response against live Clostridium tetani infection in mice can be elicited by an adenoviru
39 n of circulating toxin and elimination of C. tetani infection, control of spasms and convulsions, mai
40  epidemiology, treatment, and prognosis of C tetani infections.
41                                  Clostridium tetani infects wounds contaminated with dirt, feces, or
42 ) after theta bursts, but not high-frequency tetani, produced a rapid, transient expression of synapt
43      The assay consisted of spiking the anti-tetani sample directly onto the toxoid modified SPE, and
44 o identify any of the 7 C. innocuum and 9 C. tetani strains tested), and 8 of 15 (53.3%) Bacteroides
45                                Evidence from tetani suggests the slow force responses result from inc
46 tect a fragment of the neurotoxin gene of C. tetani (TeNT) and was used in conjunction with previousl
47 TeNT) is an exotoxin produced by Clostridium tetani that causes paralytic death to hundreds of thousa
48 ament architecture of Alp12 from Clostridium tetani that is constructed from four protofilaments.
49 itopes of Bacillus anthracis and Clostridium tetani toxins, as these bacteria do not depend on human
50 ute stress, the LTD induction by serotonin + tetani was significantly impaired.
51                       Isometric twitches and tetani were recorded from the whole muscles and from a s
52  sequences related to modern-day Clostridium tetani, which produces the tetanus neurotoxin (TeNT) and