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1 nt of which may be whole genome duplication (tetraploidy).
2 y accelerates the spontaneous development of tetraploidy.
3 evere damage and the eventual development of tetraploidy.
4 o genes expected from the ancestral salmonid tetraploidy.
5  in the first S phase following induction of tetraploidy.
6 omosomes, multipolar spindles, and increased tetraploidy.
7 opy-neutral loss-of-heterozygosity (LOH) and tetraploidy.
8  and aneuploidy but not with binucleation or tetraploidy.
9  resulting in whole-genome reduplication and tetraploidy.
10 tin bridges, cleavage furrow regression, and tetraploidy.
11 DNA damage responses, p53 stabilization, and tetraploidy.
12 rkers identified the genetic requirements of tetraploidy.
13 Gp1balpha concurrent with their promotion of tetraploidy.
14       Most duplicate genes are removed after tetraploidy.
15 hich results in near-diploid aneuploidy, not tetraploidy.
16 3 RNAi permits both arrest insensitivity and tetraploidy.
17 ch correlated with an increased incidence of tetraploidy.
18 l expression of Plk1 protein correlates with tetraploidy.
19 aberrant mitoses with extra centrosomes, and tetraploidy.
20 effects of PARP inhibition on development of tetraploidy.
21 A synthesis, resulting in the acquisition of tetraploidy.
22       In maize, a species that experienced a tetraploidy 5-12 million years ago, we show that in addi
23 y (16 of 18) and 90% of tested patients with tetraploidy (9 of 10) had an IgH translocation.
24                                              Tetraploidy, a common feature in cancer, results in the
25 hat favors F-actin polymerization and limits tetraploidy, a starting point for tumorigenesis.
26 on, doublets were diploidized with ancestral tetraploidy already blurred.
27                                              Tetraploidies and balanced segments inject bias for thos
28  chromosome segregation errors, resulting in tetraploidy and aneuploidy.
29                      An equivalent period of tetraploidy and body plan evolution may have ended for a
30 the organism because it immediately produces tetraploidy and centrosome amplification, which is thoug
31 plicable to the recently observed pattern of tetraploidy and gene conversion in asexual, bdelloid rot
32 in and its downstream target, MTMC1, promote tetraploidy and other forms of GI.
33 s expected from this combination of defects, tetraploidy and polyploidy are consequences of APC inhib
34 y increased centrosome aberration frequency, tetraploidy, and aneuploidy in nonirradiated HMEC.
35 able as evidenced by spontaneous DNA damage, tetraploidy, and aneuploidy.
36 ome bridges and mis-segregation in anaphase, tetraploidy, and faster mitotic slippage in the presence
37 n intestinal commensal generated aneuploidy, tetraploidy, and gammaH2AX foci in HCT116, RKO, and YAMC
38 d microtubule dynamics, prometaphase arrest, tetraploidy, and mitotic cell death.
39 totic defects associated with high levels of tetraploidy/aneuploidy.
40                                      Ancient tetraploidies are found throughout the eukaryotes.
41 megakaryocytic endomitosis and c-Myc-induced tetraploidy are mechanistically linked by their reliance
42  caused by HPV-16 where bypass of arrest and tetraploidy are separable consequences of p53 loss with
43                                              Tetraploidy, arising from a cytokinesis defect, is known
44  that induction of senescence is critical to tetraploidy arrest.
45 s, overexpression of Plk1 in cells generates tetraploidy but does not confer resistance to arrest.
46 NA-mediated inhibition of Gp1balpha prevents tetraploidy by both c-Myc and MTMC1, whereas Gp1balpha o
47                                              Tetraploidy can arise from various mitotic or cleavage d
48                                   Given that tetraploidy can have deleterious consequences, such as g
49 -proficient cells to spontaneously evade the tetraploidy checkpoint degenerates to uncontrolled polyp
50  provide a functional demonstration that the tetraploidy checkpoint does not exist in normal mammalia
51 proposal that normal mammalian cells have a "tetraploidy checkpoint" that arrests binucleate cells in
52  as yet ill-defined mechanistic basis of the tetraploidy checkpoint, the involvement of a tumor-suppr
53 vented through activation of a p53-dependent tetraploidy checkpoint.
54                                              Tetraploidy constitutes a genomically metastable state t
55   Anticancer agents often cause drug-induced tetraploidy (DIT) in cancer cells.
56 but a general mechanism for the induction of tetraploidy during tumorigenesis is lacking.
57 nforced expression of cyclin B1 also induces tetraploidy, either after mitotic spindle inhibition or
58                                        Thus, tetraploidy enhances the frequency of chromosomal altera
59 c allopolyploidy and result from 28 distinct tetraploidy events plus an additional six hexaploidy eve
60 delling and counter-selection, we found that tetraploidy evolved because it confers immediate fitness
61                            Once established, tetraploidy facilitated novel genetic routes for adaptat
62    Antimyeloma effects included induction of tetraploidy followed by apoptosis.
63                                 However, the tetraploidy genome and long generation-time of the commo
64    We propose that, similar to aneuploidy or tetraploidy, haploidy triggers a p53-dependent response
65                                              Tetraploidy has long been of interest to both cell and c
66 the detection of moderate gains (such as tri-tetraploidy) has been a challenge in cancer research.
67              The E6 mutant fails to generate tetraploidy; however, the presence of E7 is sufficient t
68  oesophageal adenocarcinoma, aneuploidy, and tetraploidy in a cohort of 350 people with Barrett's oes
69            Thus, the announcement in 1999 of tetraploidy in a mammal, the South American red vizcacha
70 spindle poisons accelerate the appearance of tetraploidy in cells either lacking functional p53 or ov
71 NI of TMPRSS2-ERG had generalized aneuploidy/tetraploidy in contrast to tumors without TMPRSS2-ERG CN
72 overexpression alone is sufficient to induce tetraploidy in established and primary cells.
73 uclei and the progressive accumulation of G1 tetraploidy in human diploid fibroblasts.
74   To explore the origins and consequences of tetraploidy in the African clawed frog, we sequenced the
75                        After the most recent tetraploidy in the Arabidopsis lineage, most gene pairs
76 4-8); however, the immediate consequences of tetraploidy in the first interphase are not known.
77 ion of p75(NTR) in the induction of neuronal tetraploidy in the mouse neocortex.
78  Nbs1 fragment induced DNA rereplication and tetraploidy, in NBS-deficient but not NBS-proficient cel
79                 Our results demonstrate that tetraploidy increases exponentially over the life span o
80 o pathway kinase LATS2 is a key mechanism of tetraploidy-induced cell-cycle arrest.
81 amage marker remains at control levels after tetraploidy induction.
82                                              Tetraploidy is accompanied by significantly higher level
83                                              Tetraploidy is an aneuploidy-permissive condition that c
84 on of nonfractionated cells, suggesting that tetraploidy is an important mediator of Aurora-B-induced
85         This potentially oncogenic effect of tetraploidy is countered by a p53-dependent barrier to p
86 ting mitosis without chromosome segregation, tetraploidy is frequent in late-stage erythroblasts, the
87                              In the striatum tetraploidy is mainly associated with long-range project
88                                            A tetraploidy left Arabidopsis thaliana with 6358 pairs of
89                             We conclude that tetraploidy mainly affects long-range projection neurons
90  cyclin D2, elucidating a possible route for tetraploidy-mediated genomic instability in carcinogenes
91 neuploidy (n=35 cases; 0.25 [0.12-0.54]) and tetraploidy (n=45 cases; 0.44 [0.22-0.87]).
92                                 We show that tetraploidy occurs in 15% (n = 29) of the Inga species w
93 s as well as partial triploidies and partial tetraploidies of portions of chromosome 22q were mapped
94                                 Although the tetraploidy of maize is ancient, biased gene loss and ex
95 wo sets of four ribosomal RNA genes confirms tetraploidy of this clone.
96 m human tumors and mouse models suggest that tetraploidy, one example of polyploidy, can promote tumo
97   The same selective benefit also maintained tetraploidy over long evolutionary timescales, inhibitin
98 e a high degree of structural and functional tetraploidy over more than 180 million years, the reduct
99 0, 16, and 18 was associated with aneuploidy/tetraploidy (P = 0.037, 0.013, and 0.054, respectively).
100                               Once acquired, tetraploidy persists in most cases examined.
101 es the rate of cell proliferation results in tetraploidy, premature appearance of irreversible hyperp
102 o GPI 6150 did not induce the development of tetraploidy, suggesting that, aside from its catalytic f
103 ent of their three independent, fractionated tetraploidies sum to a powerful comparative genomic syst
104 eals phenotypic consequences of whole-animal tetraploidy that make C. elegans an excellent model for
105                              For all studied tetraploidies, the loss of duplicated genes, known as ho
106                       Following most ancient tetraploidies, the two subgenomes are distinguishable be
107 nterest, even though the definitive proof of tetraploidy, the presence of four copies of each chromos
108 Plk1, suggesting that loss of p53 results in tetraploidy through upregulation of Plk1.
109  De Lange make important connections between tetraploidy, tumorigenesis, and telomere crisis-a common
110   To explore the origins and consequences of tetraploidy, we sequenced the allotetraploid A. hypogaea
111 to the resistance of arrest and induction of tetraploidy, we used an E6 mutant unable to degrade p53
112 mpanying the DNA damage response, LT induces tetraploidy, which is also dependent on Bub1 binding.
113  the metals caused a persistent induction of tetraploidy, which was not noted in hTERT- cells.
114 us, sustained expression of Aurora-B induces tetraploidy, which, in turn, facilitates genomic instabi
115 i-quadruplets still reflecting the ancestral tetraploidy with clear signs of advanced rediploidizatio
116    The pattern is consistent with degenerate tetraploidy with numerous segmental deletions, some in o
117 ation) and ploidy abnormalities (aneuploidy, tetraploidy) within each Barrett's esophagus segment of

 
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