ライフサイエンスコーパス: thalami

1 l lobes, basal ganglia (BG), hippocampi, and thalami.

2 ]: 0.16, 0.49) in the basal ganglia (BG) and thalami.

3 demonstrating an enlargement in TS-affected thalami.

4 ortices of Et-treated rats, but not in their thalami.

5 eased and differ markedly between individual thalami.

6 including the right uncinate fasciculus and thalami.

7 39 vs. 1.280.5 x 10(-3) mm(2)/s, p = 0.045), thalami (1.205 vs. 1.285 x 10(-3) mm(2)/s, p = 0.031) an

8 afferents projecting to the principal optic thalami, a complex of several nuclei that resides in the

9 hibitory synapses in the nucleus reticularis thalami, a region where Nxph1 is normally expressed at h

10 was found in the mesial temporal regions and thalami, accentuating a relative signal increase in the

11 late, and insular cortices as well as in the thalami, amygdalae, and hippocampi.

12 erior divisions of nucleus paraventricularis thalami and avoided the adjoining mediodorsal thalamic n

13 hildren develop necrotizing lesions in their thalami and brainstems in the course of febrile illnesse

14 e gyrus, striatum, globus pallidus internus, thalami and cerebellum.

15 sterior cingulate white matter (WM), and the thalami and hippocampi.

16 decreased connectivity in caudate nuclei and thalami and increased connectivity in the cerebellum, po

17 Higher MTRs were seen in the thalami and pons than in the BG (P < .05), indicating ea

18 ng neural activities from motor cortices and thalami and recording hand tremor signals via accelerome

19 Focusing on the bilateral thalami and the left dorsolateral prefrontal cortex (DLP

20 mputed across the whole brain and within the thalami and white matter.

21 ementia but included the cingulate cortices, thalami, and cerebellum in patients with bvFTD, with the

22 diameter, corpus callosum, basal ganglia and thalami, and cerebellum were smaller in infants in the M

23 (DN), pons, substantia nigra (SN), pulvinar thalami, and globus pallidus (GP).

24 lobe, cerebellum, brainstem, basal ganglia, thalami, and orbitofrontal and sensorimotor cortex.

25 es, and involvement of corticospinal tracts, thalami, and spinal cord.

26 erest included putamen, caudate, hippocampi, thalami, and substantia nigra.

27 s, the executive part of both putamina, both thalami, and the cerebellum.

28 Sensory thalami are central sensory pathway stations for informa

29 k individuals with particularly small AHC or thalami are most likely to develop schizophrenia, this f

30 unctional connectivity between the DLPFC and thalami, as has been hypothesized previously.

31 n other locations such as the frontal lobes, thalami, basal ganglia and brainstem.

32 2 to 0.05], p < 0.001) and lower iron in the thalami (beta = -0.06 [-0.07 to -0.04], p < 0.001).

33 onal coupling between the left DLPFC and the thalami bilaterally.

34 of interest in the caudate nuclei, putamena, thalami, brain stem, cerebellum, and occipital cortex of

35 These changes were found in both UBOs and thalami but were only significant for NAA, NAA/creatine,

36 pm +/- 0.05 and 18.83 sec(-1) +/- 3.32), and thalami (case participants: 0 ppm +/- 0.03 and 16.49 sec

37 , patients with CIS developed atrophy of the thalami, caudate nuclei, cerebellum, and frontal, pariet

38 ADC values of thalami, CN, and CH were significantly lower in IUGR tha

39 trated bilaterally within the basal ganglia, thalami, corpus callosum, occipital, temporal, parietal

40 Diffuse lesions of the thalami, cortical neurons, or the white-matter tracts th

41 within UBOs, unaffected globus pallidus, and thalami demonstrated an age-related pattern, characteriz

42 c early involvement of the basal ganglia and thalami distinguish this illness from herpes simplex enc

43 on including nucleus dorsomedialis posterior thalami (DMP), stratum griseum et fibrosum superficiale

44 in a frontoparietal network, including both thalami, during the execution of the go/no-go task.

45 eleostean derivative of the larval eminentia thalami (EmT).

46 ally constant neurochemical signature in all thalami examined.

47 pm +/- 0.05 and 18.75 sec(-1) +/- 2.68), and thalami (first MRI: 0.01 ppm +/- 0.02 and 17.65 sec(-1)

48 f patients on MRI, and in 56 percent on CT), thalami (found in 71 percent on MRI and in 25 percent on

49 n patterns of these monoamines in postmortem thalami from 13 normal subjects (no known neurological o

50 The 2 thalami have minimal monosynaptic commissural connection

51 c tectum (TeO), LM, GLv, and n. intercalatus thalami (ICT), and a second population that only project

52 intercalatus thalami (ICT), and a second population that only project

53 se of H(2)S by examining electron microscopy thalami images of surviving mice.

54 eared as a single echogenic line between the thalami in 171 (38.9%) fetuses, as parallel echogenic li

55 th diffusion MR imaging in basal ganglia and thalami in 26 full-term neonates (age range, 1-10 days)

56 tivation was observed bilaterally within the thalami in conjunction with widespread but symmetrical c

57 dence suggests an involvement of the sensory thalami in speech recognition.

58 vation (P < or = 0.05), were observed in the thalami, insula bilaterally, anterior cingulate cortex,

59 semiovale, caudate nuclei, lentiform nuclei, thalami, internal capsules, and cerebellar hemispheres.

60 diencephalon, the insertion of the eminentia thalami into the caudal telencephalon, and the pallial-s

61 s-hemisphere coupling between left and right thalami is associated with more unstable tremor.

62 mum(2)/sec), vermis (1.26 x10(3)mum(2)/sec), thalami (left, 1.17 x10(3)mum(2)/sec; right, 1.15 x10(3)

63 3)um(2)/sec), vermis (1.26 x10(3)um(2)/sec), thalami (left, 1.17 x10(3)um(2)/sec; right, 1.15 x10(3)u

64 (P < .001) in several regions, including the thalami (left, 1.63 years +/- 0.32; right, 1.45 years +/

65 (P < .001) in several regions, including the thalami (left, 1.63 years 0.32; right, 1.45 years 0.33),

66 Nucleus reticularis thalami neurons expressed only alpha3 as the synaptic al

67 press Cav3.1, whereas in nucleus reticularis thalami neurons that express Cav3.2 and Cav3.3 channels,

68 roinjected into the nucleus parafascicularis thalami (nPf) on pain behaviors organized at different l

69 e amygdala (ACe) or nucleus parafascicularis thalami (nPf) produced dose-dependent inhibition of the

70 rotein expression in the nucleus reticularis thalami (nRt) and hippocampus, but not in the neocortex,

71 GABAergic neurons of the nucleus reticularis thalami (NRT) and thalamocortical (TC) neurons discharge

72 While the nucleus reticularis thalami (nRT) has been implicated in selective attention

73 m spikes from neurons of nucleus reticularis thalami (nRT) in brain slices from young rats and invest

74 ABAergic neurones of rat nucleus reticularis thalami (NRT) in vitro to assess pre- and postsynaptic G

75 thalamocortical (TC) and nucleus reticularis thalami (NRT) neurones, and possibly in neocortical cell

76 T-type Ca2+ channels in nucleus reticularis thalami (nRT) play a critical role in generation of low-

77 nhibitory neurons of the nucleus reticularis thalami (nRt) to fire bursts of action potentials.

78 rents) in neurons of the nucleus reticularis thalami (nRT).

79 GABAergic neurons of the nucleus reticularis thalami (NRT).

80 Right-hemisphere thalami obtained at autopsy from 14 schizophrenic and ei

81 lactate-choline ratios in basal ganglia and thalami of infants with perinatal asphyxia were predicti

82 neuron number than of neuron density in the thalami of schizophrenic patients.

83 reases in NAA ratios were most severe in the thalami of subjects with UBOs in the globus pallidus, wh

84 habenular nucleus, nucleus paraventricularis thalami, other midline-intralaminar thalamic nuclei, the

85 lues were observed in the BG relative to the thalami (P < .0001).

86 ignal in the dorsomedial aspect of bilateral thalami, periaqueductal grey matter and around the third

87 The human brain thalami play essential roles in integrating cognitive, s

88 resonance imaging (qMRI) measurements of the thalami provide important biomarkers of disease progress

89 PK binding was significantly raised in the thalami, putamen, occipital cortices, and posterior limb

90 collateralize in the nuclei posteroventralis thalami (PV), subpretectalis (SP), and interstitiopretec

91 ; P < .001) and (18)F-DPA-714 binding in the thalami (r = 0.44; 95% CI: 0.22, 0.72; P = .04) and norm

92 ontine reticular formation, and the anterior thalami remained increased during CS.

93 ur patients and indirectly, through anterior thalami (remote closed-loop), to the other four patients

94 STN DBS also increased rCBF in the bilateral thalami, right midbrain, and decreased rCBF in the right

95 tecto-fugal pathway is the nucleus rotundus thalami (Rt).

96 ateral ventricles, the lentiform nuclei, the thalami, the third ventricle and the white matter.

97 central nucleus and nucleus parafascicularis thalami) to the antinociceptive action of morphine micro

98 eep white matter, the basal ganglia, and the thalami was observed in HIV-1 infection.

99 s in the macromolecular protein pool, in the thalami when compared with the control groups.

100 neuronal loss and neuritic pathology in the thalami with the extremes of volume.

101 ignificant gender effect was observed in the thalami, with higher GABA/Water in males than in females