ライフサイエンスコーパス: thalamic nuclei

1 minative pain, such as ventral posteromedial thalamic nuclei.

2 amatergic outputs that differentially target thalamic nuclei.

3 ange feedback projections to primary sensory thalamic nuclei.

4 ular nuclei, sensory nuclei, and nonspecific thalamic nuclei.

5 he BG to directly excite neurons in specific thalamic nuclei.

6 ngling of the neurons that innervate the two thalamic nuclei.

7 rlap of cells projecting to the two anterior thalamic nuclei.

8 idus, subthalamic nucleus, and ventral motor thalamic nuclei.

9 levels of EphA7 in CT axons and ephrin-As in thalamic nuclei.

10 istributed among six cortical layers and two thalamic nuclei.

11 em form extensive projections to a number of thalamic nuclei.

12 to misrouting of interneurons into nonvisual thalamic nuclei.

13 cortices, pars opercularis and motor-related thalamic nuclei.

14 nverse correlation with frontally projecting thalamic nuclei.

15 d RGC axons in adjacent non-retino-recipient thalamic nuclei.

16 nisms influence the formation of postmitotic thalamic nuclei.

17 ative dorsal (DP) and central posterior (CP) thalamic nuclei.

18 and a PCS pattern that involved the ventral thalamic nuclei.

19 n selectively and collectively from multiple thalamic nuclei.

20 tors preferentially give rise to caudodorsal thalamic nuclei.

21 ogeneity contributes to the specification of thalamic nuclei.

22 formation, receiving input from two distinct thalamic nuclei.

23 h interval and was localized to the anterior thalamic nuclei.

24 aracentral nucleus (OPC) of the intralaminar thalamic nuclei.

25 tum, hippocampus, dentate gyrus and specific thalamic nuclei.

26 orsal, ventral anterior, and anterior medial thalamic nuclei.

27 lamic regions, as well as particular midline thalamic nuclei.

28 atosensory cortex originate in various relay thalamic nuclei.

29 rain, including the amygdala and the midline thalamic nuclei.

30 These connectivity patterns differed between thalamic nuclei.

31 ates, expression is most abundant in various thalamic nuclei.

32 MGN nucleus is very similar to that in other thalamic nuclei.

33 se reciprocal projections with corresponding thalamic nuclei.

34 antigens for calbindin and Cat-301 to reveal thalamic nuclei.

35 odality inhibitory modulation between dorsal thalamic nuclei.

36 nvolvement of visual cortex and higher order thalamic nuclei.

37 innervates cortex and possibly originates in thalamic nuclei.

38 , and specific sensory relay and association thalamic nuclei.

39 disperse activity across cortical areas and thalamic nuclei.

40 ct to the parafascicular and central lateral thalamic nuclei.

41 M), ventral lateral (VL), and posterior (Po) thalamic nuclei.

42 to regions that roughly reflected individual thalamic nuclei.

43 end on distinct inputs from sensory-specific thalamic nuclei.

44 neuropeptides, helped the identification of thalamic nuclei.

45 lls provide modulatory feedback input to all thalamic nuclei.

46 ior thalamus (pulvinar) and the medio-dorsal thalamic nuclei.

47 reuniens (Re) is the largest of the midline thalamic nuclei.

48 signals reach the cortex via sense-specific thalamic nuclei.

49 signals reach the cortex via sense-specific thalamic nuclei.

50 arietal areas are differently connected with thalamic nuclei.

51 dorsal subiculum projections to the anterior thalamic nuclei.

52 on of first (dLGN) and high-order (pulvinar) thalamic nuclei.

53 connections in the ipsilateral medial-dorsal thalamic nuclei.

54 ITCc dendrites from nociceptive intralaminar thalamic nuclei.

55 notype seen in humans with lesions of medial thalamic nuclei(1-3).

56 ecting to as few as 5, and to as many as 15, thalamic nuclei; (2) most nuclei received projections fr

57 campus make the reuniens and rhomboid (ReRh) thalamic nuclei a putatively major functional link for r

58 Identifying specific thalamic nuclei abnormalities in psychosis has implicati

59 vide evidence for differential inhibition of thalamic nuclei across brain states, where the TRN separ

60 neuronal firing via monosynaptic afferents, thalamic nuclei act as a relay station routing prefronta

61 s received bilateral lesions of anterodorsal thalamic nuclei (ADN), postsubiculum (PoS), or sham lesi

62 a PCC/RSC pattern that involved the anterior thalamic nuclei, an MPC pattern that involved the latera

63 Coupled with a lack of input from principal thalamic nuclei and a minimal layer 4, these observation

64 lso seen in various basal forebrain regions, thalamic nuclei and amygdaloid nuclei.

65 ticular nucleus in conjunction with specific thalamic nuclei and are modulated by corticothalamic and

66 axons to the designated areas within target thalamic nuclei and by progressive increase of axonal pr

67 ctivity of the suprachiasmatic and reticular thalamic nuclei and choroid epithelial cells diminished,

68 y developed, validated method for segmenting thalamic nuclei and complementary voxel-based morphometr

69 ion with the pattern of connectivity between thalamic nuclei and cortical areas or deep nuclei), whic

70 cells provide driving input to higher-order thalamic nuclei and do not innervate first-order nuclei,

71 th potential synaptic relays in the anterior thalamic nuclei and dorsolateral entorhinal cortex.

72 io between higher PDE10A expression in motor thalamic nuclei and lower PDE10A expression in striatopa

73 e show the step-wise optical fiber targeting thalamic nuclei and map the region-specific functional c

74 n the low-frequency (1-4 Hz) oscillations in thalamic nuclei and neocortical areas are essentially th

75 ow that the projections that do form between thalamic nuclei and neocortical domains have a shifted t

76 eract with primary and higher-order specific thalamic nuclei and nonspecific thalamic nuclei to carry

77 trosplenial cortex, cingulate gyrus, midline thalamic nuclei and prefrontal cortex were intensely act

78 reciprocal connections between the anterior thalamic nuclei and retrosplenial cortex, another region

79 rdependent relationship between the anterior thalamic nuclei and retrosplenial cortex, given how dysf

80 reciprocal connections between the anterior thalamic nuclei and retrosplenial/pre- and parasubicular

81 (ii) It includes multiple thalamic nuclei and six-layered cortical microcircuitry

82 nd unconditioned stimuli in the multisensory thalamic nuclei and that these BF shifts are augmented a

83 ominent connections between the intralaminar thalamic nuclei and the basal ganglia has long been esta

84 icates the anterior and mediodorsal (limbic) thalamic nuclei and the reciprocally interconnected area

85 ith potential synaptic relays in the midline thalamic nuclei and the rostral caudomedial entorhinal c

86 cells that are densest in the "nonspecific" thalamic nuclei and usually target layer 1 (L1) of multi

87 om the hippocampal formation to the anterior thalamic nuclei and vice versa impaired performance on t

88 bust IPSCs or IPSPs in other specific dorsal thalamic nuclei and vice versa.

89 nkey brain, with the greatest density in the thalamic nuclei and with moderate to low binding in the

90 e thalamus (entire thalamus and 19 bilateral thalamic nuclei) and both neocortex and brainstem ascend

91 i of the torus innervate central and lateral thalamic nuclei, and all have a weak reciprocal connecti

92 o fibers and neuropil in the analyzed dorsal thalamic nuclei, and presented no differences between ge

93 development, the cortical dependence of many thalamic nuclei, and the phenomenon of transsynaptic deg

94 ulation of cells in two or more other dorsal thalamic nuclei, and TRN-mediated inhibitory inputs can

95 (iFC) between distinct cortical networks and thalamic nuclei are among the most consistent large-scal

96 Projections to the RAIC from medial thalamic nuclei are associated with motivational/affecti

97 rly and late in the period of gestation when thalamic nuclei are becoming histologically differentiat

98 aps in the cerebral cortex and corresponding thalamic nuclei are genetically prespecified to a large

99 Our recent studies found that these thalamic nuclei are in fact derived from molecularly het

100 current study examined whether the anterior thalamic nuclei are involved in attentional processes ak

101 Midline/intralaminar/posterior dorsal thalamic nuclei are involved in forebrain circuits relat

102 shown that responses in first-order sensory thalamic nuclei are modulated by cortical areas.

103 Within this system, the anterior thalamic nuclei are prominent, both for their vital cont

104 Thalamic nuclei are thought to funnel sensory informatio

105 ns of CP and projections of all other dorsal thalamic nuclei are to the subpallium, however.

106 n, segregation, or putative absence in given thalamic nuclei are unknown.

107 ern of performance reveals that the anterior thalamic nuclei are vital for attending to those stimuli

108 ts, characteristic features of other sensory thalamic nuclei, are not encountered.

109 ipal trigeminal and ventral-posterior-medial thalamic nuclei, are substantially modulated by touch.

110 cond postnatal week, but it extends to other thalamic nuclei as well.

111 ness of CT axons to the ephrin-A gradient in thalamic nuclei, as well as by the matching levels of Ep

112 least in part, from a loss of inhibition to thalamic nuclei associated with both the sensory-discrim

113 inar (LP-pulvinar complex) are the principal thalamic nuclei associated with the elaborate developmen

114 a 3a receives the majority of its input from thalamic nuclei associated with the motor system, poster

115 SorCS3 expression marks thalamic nuclei at embryonic and early postnatal stages.

116 ent, convergent evidence places the anterior thalamic nuclei at the heart of diencephalic amnesia.

117 Selective inactivation of anterior thalamic nuclei (ATN) by microinfusion of muscimol or fl

118 ar retrosplenial cortex (RSCg), and anterior thalamic nuclei (ATN) interact to mediate diverse cognit

119 ponent of the limbic circuitry, the anterior thalamic nuclei (ATN), on the generation of new neurons

120 to the hippocampal network via the anterior thalamic nuclei (ATN).

121 gene expression were seen between different thalamic nuclei but not between diagnostic groups.

122 TAC1-lineage neurons are connected to medial thalamic nuclei by direct projections and via indirect r

123 Input to sensory thalamic nuclei can be classified as either driver or mo

124 Relay neurons in dorsal thalamic nuclei can fire high-frequency bursts of action

125 late and frontal cortices, amygdala, midline thalamic nuclei, cerebellum, and in several brainstem re

126 striatum and pallidum and increased in motor thalamic nuclei, compared to a group of matched healthy

127 iodorsal nucleus of the thalamus and midline thalamic nuclei, consistent with findings in the rhesus

128 Ventral motor thalamic nuclei contain exclusively excitatory projectio

129 Most dorsal thalamic nuclei contained subunits alpha1, alpha2, alpha

130 gests that pTH-C is the only major source of thalamic nuclei containing neurons that project to the c

131 al and parietal cortical regions and related thalamic nuclei, correlated with skill.

132 eover, TRN-mediated switching between dorsal thalamic nuclei could provide a mechanism for the select

133 t also a route through specific higher-order thalamic nuclei, creating a parallel feedforward trans-t

134 alterations are focally located in specific thalamic nuclei depending on the initial infarct locatio

135 remains one of the least explored among the thalamic nuclei despite occupying the most thalamic volu

136 Together, our findings reveal how two thalamic nuclei differentially communicate with the PFC

137 cal waves, whereas neurons from higher-order thalamic nuclei display "hub dynamics" and thus may cont

138 wever, the neuronal precursors for different thalamic nuclei display temporally distinct Gbx2 express

139 Thus, our study shows that: (1) different thalamic nuclei do not establish projections independent

140 m (NCM), the core or shell regions of dorsal thalamic nuclei, dopaminergic cell groups in the mesence

141 euronal generation was also evident in other thalamic nuclei (e.g., the lateral geniculate nucleus).

142 theless, how a distinct array of postmitotic thalamic nuclei emerge from this single developmental un

143 nteraction with ligands in the somatosensory thalamic nuclei; EphA4 affects only cortical neuronal mi

144 idespread oscillations and render subsets of thalamic nuclei especially vulnerable to pathological sy

145 In mammals, different thalamic nuclei establish highly ordered projections wit

146 that stimulation of cells in specific dorsal thalamic nuclei evokes robust IPSCs or IPSPs in other sp

147 Multiple thalamic nuclei expressed Nrg3, while detection in the s

148 suggests that innervation from PV-containing thalamic nuclei extends across superficial and middle la

149 We conclude that sensory thalamic nuclei follow the propagating cortical waves, w

150 ic correlations between the cortex and these thalamic nuclei followed the known patterns of anatomica

151 Appreciating the importance of the anterior thalamic nuclei for memory and attention provides a more

152 bodies and their projections to the anterior thalamic nuclei for spatial memory.

153 ctions to the anteromedial and anteroventral thalamic nuclei for the processing of allocentric inform

154 to test whether the hippocampus and anterior thalamic nuclei form functional components of the same s

155 vity from connected basal ganglia output and thalamic nuclei (globus pallidus-internus [GPi] and vent

156 that delta frequency bursting in particular thalamic nuclei has a causal role in producing WM defici

157 on of the specific (VB) and nonspecific (CL) thalamic nuclei has been proposed as the basis for the t

158 multiple laboratories researching different thalamic nuclei has contradicted this idea of the thalam

159 The lateral geniculate nucleus, like all thalamic nuclei, has two classically defined categories

160 Lesions involving the intralaminar thalamic nuclei have been associated with impairments in

161 Dorsal thalamic nuclei have been categorized as either "first-o

162 The lateral posterior and posterior thalamic nuclei have been implicated in aspects of visua

163 inputs bifurcates to innervate both anterior thalamic nuclei highlights the potential for parallel in

164 tor mRNA levels were found in several areas (thalamic nuclei, hippocampal CA3) with parallel increase

165 lands of Calleja, cerebral cortex, striatum, thalamic nuclei, hippocampus, amygdala, substantia nigra

166 ostral or caudal regions of the intralaminar thalamic nuclei (i.e. the central lateral or parafascicu

167 The intralaminar thalamic nuclei (IL) probably serves as a central hub in

168 ctionally-interconnecting cortical areas and thalamic nuclei, illustrating that OPCs have strikingly

169 The rostral intralaminar thalamic nuclei (ILn) are organized to activate pathways

170 sults indicate a limited role for the medial thalamic nuclei in coding for pain intensity and the aff

171 anding of the involvement of ventral midline thalamic nuclei in cognitive processes: they point to a

172 We aimed at assessing volume loss of thalamic nuclei in NMOSD.

173 ial, ventral posterior and lateral posterior thalamic nuclei in patients assessed by the Glasgow Outc

174 , EAAT2, and EAAT3 was performed in discrete thalamic nuclei in persons with schizophrenia and compar

175 ntralaminar, midline, relay, and associative thalamic nuclei in rats.

176 r from a variety of specific and nonspecific thalamic nuclei in relation to the phase of global EEG s

177 d both the dorsomedial and ventral posterior thalamic nuclei in severely disabled and vegetative head

178 tive visual cortical areas and corresponding thalamic nuclei in the embryonic rhesus monkey (Macaca m

179 -brain functional connectivity of the visual thalamic nuclei in the various populations of subjects u

180 imulation suppresses activity in both visual thalamic nuclei in vivo, moderate-frequency (10 Hz) stim

181 and mediodorsal, ventrolateral and pulvinar thalamic nuclei, in both the patients and the healthy mu

182 o area 1 were highly convergent from several thalamic nuclei including the ventral lateral nucleus (V

183 jections in area 3b were also found in other thalamic nuclei including: anterior pulvinar (Pa), ventr

184 as received connections from the same set of thalamic nuclei, including main inputs from the ventral

185 lesions of the contralateral medial auditory thalamic nuclei, including the medial division of the me

186 Thalamic nuclei, including the reticular nucleus, expres

187 al geniculate complex (MGC) and multisensory thalamic nuclei, including the suprageniculate (Sg), lim

188 the highest levels of binding were in select thalamic nuclei, including those implicated in hypoxic d

189 g stimuli that evoke strong responses in the thalamic nuclei innervating the cortex.

190 The thalamic nuclei investigated included LP, laterodorsal t

191 unction of corticothalamic pathways to relay thalamic nuclei is attention-dependent modulation of tha

192 r thalamic stroke, and the role of different thalamic nuclei is unclear.

193 rmediate nucleus (Vim), as part of the motor thalamic nuclei, is a commonly used target in functional

194 rough their innervation of a wide variety of thalamic nuclei, is effective in controlling absence sei

195 ultrastructural features of the intralaminar thalamic nuclei (ITN) projections to the globus pallidus

196 the mediodorsal nucleus, suggests that these thalamic nuclei, like RE, represent important output sta

197 gdala (BL) originates from a group of dorsal thalamic nuclei located at or near the midline, mainly f

198 Thus, the lateral posterior thalamic nuclei (LP/pulvinar) appear important for vario

199 ution supports the emerging view that limbic thalamic nuclei may contribute critically to adaptive re

200 The medial auditory thalamic nuclei may modulate activity in a short-latency

201 Thus, rostral thalamic nuclei may participate in spatial representatio

202 The anterior thalamic nuclei may represent one vital partner.

203 Future analysis of individual thalamic nuclei may reveal important, specific relations

204 ventricle or laterally adjacent mediodorsal thalamic nuclei (MD).

205 dial nucleus-or into one of the intralaminar thalamic nuclei-medial parafascicular, lateral parafasci

206 creases neuron number in three associational thalamic nuclei: mediodorsal (MD), anterior, and pulvina

207 has long been conjectured that the anterior thalamic nuclei might be key partners with the hippocamp

208 Although the midline and intralaminar thalamic nuclei (MITN) were long believed to project "no

209 ence, an increased activity of ventral motor thalamic nuclei nicely explains the refractoriness of PR

210 imulus-response properties across and within thalamic nuclei, normalize responses to diverse sensory

211 in many areas of the hypothalamus and dorsal thalamic nuclei, nucleus intercollicularis and ventricul

212 lls in midline/intralaminar/posterior dorsal thalamic nuclei of male Fmr1 knockout mice.

213 ion, retinofugal projections to midbrain and thalamic nuclei of Monodelphis domestica were investigat

214 ateral entorhinal cortex and ventral midline thalamic nuclei of neonatal rats.

215 and motor pathways within ventrolateral (VL) thalamic nuclei of the motor thalamus of macaque monkeys

216 roposterior medial, and the posterior medial thalamic nuclei of the trigeminal somatosensory pathways

217 ndin (CB) expression in cortical regions and thalamic nuclei of these pathways.

218 As is typical of primary inputs to other thalamic nuclei, parabrachiothalamic terminals are over

219 a (CTb) was injected into one of the midline thalamic nuclei-paraventricular, intermediodorsal, rhomb

220 trong mPFC projections to several additional thalamic nuclei, particularly to the mediodorsal nucleus

221 ne/intralaminar (M/IL) and ventromedial (VM) thalamic nuclei placed to spare the anterior nuclei.

222 tanding theoretical prediction that specific thalamic nuclei play a key role in controlling the spotl

223 , alongside dorsal portions of the posterior thalamic nuclei (Po), multisensory processing of informa

224 dorsal subiculum projections to the anterior thalamic nuclei produced the severest spatial working me

225 re- and postnatal development, with distinct thalamic nuclei projecting to specific cortical regions.

226 of RGS4 in the adult ventral posterolateral thalamic nuclei promotes recovery from mechanical and co

227 ior intralaminar nuclei (AILN) and posterior thalamic nuclei (PTN) to all cortical regions of the PMC

228 t both lemniscal and extralemniscal auditory thalamic nuclei receive significant corticofugal input.

229 ses a similar pathological increase in other thalamic nuclei regulated by the ZI, specifically the me

230 n pulvinar complex is a collection of dorsal thalamic nuclei related to several visual and integrativ

231 the development and organization of diverse thalamic nuclei remain largely unknown.

232 In contrast, "higher-order" thalamic nuclei respond poorly to peripheral inputs, and

233 ng evidence suggest that the ventral midline thalamic nuclei (reuniens and rhomboid) might play a sub

234 cipal extrinsic cholinergic source for these thalamic nuclei, showed a marked degree of collateraliza

235 l magnetic resonance imaging to identify the thalamic nuclei, specifically implicated in the generati

236 In addition, motor thalamic nuclei such as anterior and ventromedial, midli

237 y attributed to widespread connectivity from thalamic nuclei such as the pulvinar.

238 However, higher order thalamic nuclei, such as the pulvinar, interconnect with

239 Higher-order thalamic nuclei, such as the visual pulvinar, play essen

240 s of interconnections of cortical fields and thalamic nuclei suggest that the somatosensory system ma

241 re, the presence of input from somatosensory thalamic nuclei suggests that it plays an important role

242 in sites including the striatum, associative thalamic nuclei, superior colliculus, zona incerta, pont

243 he laterorostral part of LP (LPLR) and other thalamic nuclei surrounding LP project to dorsolateral t

244 calcium current underlies burst responses in thalamic nuclei that are important to spindle propagatio

245 y segregated pathways arising from the other thalamic nuclei that are interconnected with the frontal

246 pressed, respectively, in cortical areas and thalamic nuclei that are interconnected.

247 ults from an increase in the excitability of thalamic nuclei that have lost normal ascending inputs a

248 nformation to deeper layers of the SC and to thalamic nuclei that modulate visually guided behaviors.

249 layer 6 send a dense feedback projection to thalamic nuclei that provide input to sensory neocortex.

250 Moreover, the thalamic nuclei that provide major inputs to RWA are the

251 n those regions within the VPL and posterior thalamic nuclei that receive somatosensory information f

252 as been provided via electrodes implanted in thalamic nuclei, the cerebellum and the hippocampus usin

253 laterodorsal, anteroventral, and parateanial thalamic nuclei, the fasciculus retroflexus of Meynert,

254 , the hypothalamus, midline and intralaminar thalamic nuclei, the medial geniculate body, the periaqu

255 setup, an additional involvement of another thalamic nuclei, the parafascicular nucleus, when correc

256 ricular hypothalamic nucleus, several visual thalamic nuclei, the paranigral nucleus, several pretect

257 re relayed to the neocortex by "first-order" thalamic nuclei, the responses of which are determined b

258 We expressed ChR2 in two thalamic nuclei, the whisker motor cortex and local exci

259 l, and central medial nuclei; in the midline thalamic nuclei-the paraventricular, intermediodorsal, m

260 Three thalamic nuclei--the mediodorsal nucleus, pulvinar, and

261 ent of axonal projections from the SC to two thalamic nuclei: the dorsal lateral geniculate nucleus (

262 l inhibitory connections with several dorsal thalamic nuclei, thereby controlling attention, sensory

263 auses dramatic reorganization of postmitotic thalamic nuclei through altering the positional identity

264 der specific thalamic nuclei and nonspecific thalamic nuclei to carry out attentive visual learning a

265 trigeminovascular nociceptive, signals from thalamic nuclei to cortex and TC excitatory-inhibitory i

266 onally distinct first-order and higher-order thalamic nuclei to form molecularly defined TRN-thalamus

267 the anatomical connection from the anterior thalamic nuclei to retrosplenial cortex, and the involve

268 ay, the direct projections from the anterior thalamic nuclei to the dorsal hippocampal formation were

269 ions and specifications of connectivity with thalamic nuclei together with upcoming studies of cortic

270 Responses in ventrolateral and anterior thalamic nuclei tracked learning of the predictiveness o

271 Neurons in first-order thalamic nuclei transmit sensory information from the pe

272 waves that propagate among sensory-modality thalamic nuclei up to the cortex and that provide a mean

273 For this purpose, we measured RFC in seven thalamic nuclei using fMRI and brain glucose metabolism

274 Upon determination that thalamic nuclei VM and VL were of primary interest in th

275 Thalamic nuclei volumes were tested in a cross-sectional

276 number of c-Fos(+) neurons in ventral motor thalamic nuclei was higher in PRS rats than in unstresse

277 n V4 upon stimulus onset, variability in the thalamic nuclei was largely unaffected by visual stimula

278 the molecular basis of the specification of thalamic nuclei, we analyzed the expression patterns of

279 c feed-forward inhibition to the rest of the thalamic nuclei, we examined the effect of PCP on RtN ac

280 nization of "first-order" and "higher-order" thalamic nuclei, we followed bias-corrected sampling met

281 ents with pain, mainly lateral and posterior thalamic nuclei were affected, whereas a more anterior-m

282 tromedial, midline, reticular, and posterior thalamic nuclei were also activated.

283 Thalamic nuclei were assessed in magnetic resonance imag

284 ecting to the anteromedial and anteroventral thalamic nuclei were closely intermingled, with often on

285 he brainstem to the midline and intralaminar thalamic nuclei were examined in the rat.

286 projections to the midline and intralaminar thalamic nuclei were examined in the rat.

287 ically in first-order and higher-order relay thalamic nuclei were juxtacellularly filled with an ante

288 NMOSD) thalamic damage is controversial, but thalamic nuclei were never studied separately.

289 itive for Ca(v) 3.3, while neurons in dorsal thalamic nuclei were nonimmunoreactive.

290 Neuron numbers and volumes in these limbic thalamic nuclei were normal in the schizophrenia and bip

291 s were missing, and most of the major dorsal thalamic nuclei were not identifiable.

292 gnificant volume reductions of the following thalamic nuclei were observed in migraineurs: central nu

293 ceives input from diverse cortical areas and thalamic nuclei which are themselves interconnected.

294 tum and reticular and ventral posterolateral thalamic nuclei, which all showed synaptogyrin 1 labelin

295 The anterior thalamic nuclei, which are closely interconnected with t

296 l, depolarized alpha in posterior-projecting thalamic nuclei while (2) they engage a new, hyperpolari

297 The thalamic nuclei with abnormal volumes are densely connec

298 generation suggest possible abnormalities in thalamic nuclei with connections to other brain regions

299 a differential effect of anesthetic drugs on thalamic nuclei with disparate spatial projections, i.e.

300 striatal projections from LP and surrounding thalamic nuclei, with a focus on projections to DCS.

301 ity and function of the numerous and diverse thalamic nuclei within cortical-subcortical circuits con