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1 to repeated brief optogenetic stimulation of thalamocortical afferents.
2 ynaptogenesis, elaboration and refinement of thalamocortical afferents.
3 ical changes guide subsequent alterations in thalamocortical afferents.
4 difying the source or pattern of activity in thalamocortical afferents.
5 pearance of the vibrissae-related pattern of thalamocortical afferents.
6 oximately 1 d after ingrowth of AChE-stained thalamocortical afferents.
7 i-I) was used in these same animals to label thalamocortical afferents.
8 rs IV and VI, indicating their regulation by thalamocortical afferents.
9 a pattern complementary to vibrissae-related thalamocortical afferents.
10                       In addition, embryonic thalamocortical afferents also exhibit a defasciculated
11 docarbocyanine perchlorate (DiI) labeling of thalamocortical afferents and Nissl and cytochrome oxida
12 ces using electrical stimulation of putative thalamocortical afferents, and flavoprotein autofluoresc
13 cortical activity, but not the patterning of thalamocortical afferents (application of TTX and transe
14  thalamic ventrobasal complex at P0 (just as thalamocortical afferents are innervating the cortex) an
15  that formed by the terminal distribution of thalamocortical afferents arising from the posterior nuc
16 teins are upregulated in sensory cortex when thalamocortical afferents arrive.
17 ut not for the development and plasticity of thalamocortical afferent clustering into a barrel patter
18 receive dense but transient innervation from thalamocortical afferents during the first postnatal wee
19  2) alterations in the projection pattern of thalamocortical afferents from the ventroposterior later
20 ouse barrel cortex, evoked by stimulation of thalamocortical afferents in an in vitro slice preparati
21       Specifically, in contrast to controls, thalamocortical afferents in conditional lmo4 null mice
22 ults are consistent with the conclusion that thalamocortical afferents in postnatal rats transiently
23 te body as well as the anterogradely labeled thalamocortical afferents in the auditory cortex.
24                                              Thalamocortical afferents in the visual cortex of the ad
25                                Surprisingly, thalamocortical afferents innervate the mutant cortex wi
26 lopment is thought to control the process of thalamocortical afferent innervation, segregation, and p
27 estioned by findings that the segregation of thalamocortical afferents into a somatotopic barrel patt
28 terns that undergo substantial changes after thalamocortical afferents invade the cortical plate, sug
29 o determine whether the patterning of either thalamocortical afferents or intracortical projections d
30 ted dextran amine (BDA) into the MGV labeled thalamocortical afferent patches within layer III/IV and
31 nly 20% of the mouse pups had an established thalamocortical afferent pattern in the barrel cortex at
32 tion of TTX and transection of the ION after thalamocortical afferent patterns are established), have
33                            Furthermore, many thalamocortical afferents project abnormally to widely s
34  in a decrease in the size of the patches of thalamocortical afferents representing the mystacial vib
35 c cortical topography.SIGNIFICANCE STATEMENT Thalamocortical afferents segregate in primary visual co
36 reas the MZ integrates uni- and multisensory thalamocortical afferent streams, it may ultimately infl
37  genes are established before the arrival of thalamocortical afferents, suggesting that they are inde
38 e cortex for L4 aggregation into barrels and thalamocortical afferent (TCA) segregation.
39 -specific NR1 knockout (CxNR1KO) mice, while thalamocortical afferents (TCAs) develop rudimentary whi
40 erotonin (5-HT) levels influences developing thalamocortical afferents (TCAs) in primary somatosensor
41 remarkably precise, with organized arrays of thalamocortical afferents (TCAs) that project into disti
42  transporter (5HT-T) histochemistry to label thalamocortical afferents (TCAs), we found no obvious ab
43 trinsic influences such as those mediated by thalamocortical afferents (TCAs).
44 ealed that layer I axons were collaterals of thalamocortical afferents that formed multiple divergent
45  retrograde messenger nor that they activate thalamocortical afferents, the two dominant hypotheses.
46  decrease in efficacy was more pronounced in thalamocortical afferents, thus introducing an imbalance
47                                              Thalamocortical afferents to -/- cortex form irregular p
48 tical layer IV while alternately stimulating thalamocortical afferents (via medial geniculate or down
49                     Structural plasticity in thalamocortical afferents was mostly due to elongation a
50                                 Labelling of thalamocortical afferents with HRP and staining for CO a