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1 atory synaptic circuits, corticostriatal and thalamostriatal.
2 vely modulates the reinforcing properties of thalamostriatal activity.
3         We conclude that the main excitatory thalamostriatal afferents differ in many of their charac
4 n microscopic analysis of the synaptology of thalamostriatal afferents to the matrix compartments fro
5 nd dopamine release induced by activation of thalamostriatal afferents.
6 lar glutamate transporter type 2 (VGluT2) by thalamostriatal afferents.
7 ow from the patch and matrix compartments by thalamostriatal afferents.
8 imary brain developmental disorder affecting thalamostriatal and callosal pathways, also present in t
9 time window for synaptic integration between thalamostriatal and corticostriatal inputs, which might
10 oach in awake mice of both sexes to identify thalamostriatal and corticostriatal neurons during extra
11 characterizing the sound-evoked responses of thalamostriatal and corticostriatal neurons, our work de
12 cate that DCS is a region of convergence for thalamostriatal and corticostriatal projections from reg
13 ostriatal) and vGluT2-positive (i.e., mostly thalamostriatal) axo-spinous glutamatergic synapses usin
14                We have previously found that thalamostriatal axodendritic terminals are reduced as ea
15 rging with glutamatergic corticostriatal and thalamostriatal axon terminals at dendritic spines of me
16 onnectivity, it was found that activation of thalamostriatal axons in a way that mimicked the respons
17 h the DYT1 dystonia mutation, stimulation of thalamostriatal axons, mimicking a response to salient e
18 resynaptic mGluR1a labeling of glutamatergic thalamostriatal boutons and, less frequently, dopaminerg
19                              The dynamics of thalamostriatal, but not corticostriatal, synapses were
20 r SAPAP family member, SAPAP4, is present at thalamostriatal, but not corticostriatal, synapses.
21 gether, our findings describe a hypothalamic-thalamostriatal circuit that suppresses reward-seeking b
22 ptic transmission in the corticostriatal and thalamostriatal circuits of Sapap3 KO mice and littermat
23 ices over striatal activity through distinct thalamostriatal circuits.
24 lice preparation that preserved cortico- and thalamostriatal connectivity, it was found that activati
25 is rewiring involves corticostriatal but not thalamostriatal contacts onto MSNs.
26 cumbal opioid injections rapidly dysregulate thalamostriatal ensemble dynamics, weaken thalamostriata
27 indings demonstrate that corticostriatal and thalamostriatal glutamatergic axo-spinous synapses displ
28 erential pattern of synaptic organization of thalamostriatal glutamatergic inputs to the patch and ma
29                                              Thalamostriatal input, dopaminergic input, as well as in
30  indicate that A1R-SD at corticostriatal and thalamostriatal inputs to DLS can be additive and that A
31  is reproduced by activating ChR2-expressing thalamostriatal inputs, which synchronize cholinergic in
32 e and that A1R-SD in DMS occurs primarily at thalamostriatal inputs.
33 imaging in vivo, we identify paraventricular thalamostriatal neuronal ensembles that are inhibited up
34 i/SNr to determine the relationships between thalamostriatal neurons and basal ganglia afferents.
35 odulation rate in their overall firing rate, thalamostriatal neurons convey information about the pre
36 ic, and chemogenetic experiments reveal that thalamostriatal neurons innervate accumbal parvalbumin i
37 stantia nigra pars reticulata (SNr), and the thalamostriatal parafascicular nucleus (PF)] and with pr
38 avoidance learning, directly implicating the thalamostriatal pathway in reward-based learning.
39 excitatory synapses from corticostriatal and thalamostriatal pathways and their postsynaptic targets
40 racterize the role(s) of corticostriatal and thalamostriatal pathways in regulating basal ganglia act
41 epileptogenesis and disconnection of cortico-thalamostriatal pathways through hemispherotomy or neuro
42          These data suggest that the loss of thalamostriatal PF neurons in Parkinson's Disease is a p
43                                       As the thalamostriatal projection is heterogeneous, we set out
44 a3e (encoding Sema3E) is highly expressed in thalamostriatal projection neurons, whereas in the stria
45  plasticity suggest that corticostriatal and thalamostriatal projection systems code information in t
46  and functional specificity of basal ganglia-thalamostriatal projections and discusses various aspect
47 atal afferents but strikingly different from thalamostriatal projections arising from the parafascicu
48                                   Additional thalamostriatal projections arose from VA, VL pars cauda
49    We used anterograde axonal tracing to map thalamostriatal projections from LP and surrounding thal
50          It also discusses the importance of thalamostriatal projections from the caudal intralaminar
51                                 In primates, thalamostriatal projections from the centromedian (CM) a
52      This study examines the organization of thalamostriatal projections from ventral tier nuclei tha
53                    Although the existence of thalamostriatal projections has long been known, the rol
54                      These results implicate thalamostriatal projections in the pathophysiology of PD
55  cortex and raise the possibility that VA/VL thalamostriatal projections neurons have divergent conne
56                                        VA/VL thalamostriatal projections terminate in broad, rostroca
57                          Corticostriatal and thalamostriatal projections utilize glutamate as their n
58 nd vGluT2, as markers of corticostriatal and thalamostriatal projections, respectively, we demonstrat
59  reinforce a self-initiated action, and that thalamostriatal reinforcement is constrained by mGlu(2)
60          Thus, we examined the regulation of thalamostriatal self-stimulation by mGlu(2).
61 rsely, blockade of these receptors increased thalamostriatal self-stimulation, suggesting that endoge
62                            Although previous thalamostriatal studies emphasize projections from the i
63                                We found that thalamostriatal synapses differ significantly in their p
64 ittle is known about how corticostriatal and thalamostriatal synapses differ.
65 n functional and anatomical rearrangement of thalamostriatal synapses specifically in direct-pathway
66        SAPAP isoforms at corticostriatal and thalamostriatal synapses were detected using immunostain
67                              In contrast, at thalamostriatal synapses, a single afferent volley decre
68 atum (DLS) and on synaptic plasticity in the thalamostriatal synapses.
69     In contrast to corticostriatal synapses, thalamostriatal synaptic activity is unaffected by Sapap
70 te thalamostriatal ensemble dynamics, weaken thalamostriatal synaptic innervation of downstream neuro
71 ess the possibility that degeneration of the thalamostriatal system could underlie some of the defici
72 siological data that support the role of the thalamostriatal system in action selection, attentional
73  elicits cognitive deficits and disrupts the thalamostriatal system in mice.
74 anding of the neural mechanisms by which the thalamostriatal system integrates and regulates the basa
75 high degree of functional specificity of the thalamostriatal system through which CM/Pf may provide a
76 e the pathophysiology of corticostriatal and thalamostriatal systems in PD.
77 cleus indicate that both corticostriatal and thalamostriatal terminals express presynaptic GluR6/7 an
78 o chemogenetic and optogenetic inhibition of thalamostriatal terminals reversed motor deficits in dop
79 ce also acquired operant self-stimulation of thalamostriatal terminals when ChR2 expression was viral
80 ing properties of optogenetic stimulation of thalamostriatal terminals, which are associated with ves
81               Overall, our findings reveal a thalamostriatal to parvalbumin interneuron circuit that
82 netic induction of long-term potentiation of thalamostriatal transmission in DMS CINs rescued alcohol
83 ated A1R-mediated inhibition of cortico- and thalamostriatal transmission using in vitro whole-cell,