戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 modifying disease-associated morbidities of -thalassemia intermedia.
2  condition compared with other forms of beta thalassemia intermedia.
3 xhibited increased embryonic death and alpha-thalassemia intermedia.
4 equently evaluated in a murine model of beta-thalassemia intermedia.
5 l hemoglobinuria, beta-thalassemia major, or thalassemia intermedia.
6 lable state reported to exist in severe beta-thalassemia intermedia.
7 two individuals of Greek Cypriot origin with thalassemia intermedia.
8 mouse hereditary anemia model, Hbbth3/+ beta-thalassemia intermedia.
9 eostasis in the Hbbth3/+ mouse model of beta-thalassemia intermedia.
10 of Hbb(Th3/+) mice (Th3/+), a mouse model of thalassemia intermedia.
11 mited iron overload in a mouse model of beta-thalassemia intermedia.
12  of hepatocyte Tfrc in a mouse model of beta-thalassemia intermedia ameliorated hepcidin deficiency a
13 s for this mutation, father and son, who had thalassemia intermedia and an apparent dominant mode of
14 ad in untransfused patients affected by beta-thalassemia intermedia and Hamp modulation provides impr
15                        Individuals with beta-thalassemia intermedia and hemoglobinopathies of equival
16 ot cover other hemoglobinopathies, including thalassemia intermedia and sickle cell anemia, in which
17  hydrops fetalis, hemoglobin H disease, beta-thalassemia intermedia, beta-thalassemia major/Cooley's
18 a(A)(2)) could be achieved in mice with beta-thalassemia intermedia following engraftment with bone m
19          Here we used a murine model of beta-thalassemia intermedia (Hbb(th1/th1) mice) to investigat
20 atment of mouse models of HH (Hfe(-/-)) and -thalassemia intermedia (Hbb(th3/+)) with Tmprss6 siRNA f
21  the bone marrow of animals affected by beta-thalassemia intermedia (Hbbth3/+).
22  exacerbates the phenotypic severity of beta-thalassemia intermedia in mice.
23 lso activated to reduce the severity of beta-thalassemia intermedia in the Hbbth1/th1 murine model.
24                                         beta-Thalassemia intermedia is a disorder characterized by in
25 rload in hereditary hemochromatosis and beta-thalassemia intermedia is caused by hepcidin deficiency.
26  liver, spleen, heart, and kidney tissues of thalassemia intermedia mice (Hbb(th3/+)).
27                  In contrast, in anemic beta-thalassemia intermedia mice, there is altered progressio
28 nt but incomplete phenotypic correction in a thalassemia intermedia mouse model.
29 se studies have shown correction of the beta-thalassemia intermedia phenotype and a partial, variable
30 verload and anemia consistent with both beta-thalassemia intermedia (th3/+) and major (th3/th3).
31 pothesis, we exploited a mouse model of beta-thalassemia intermedia, Th3/(+) We observed that HO inhi
32 CD46-transgenic mice and in a mouse model of thalassemia intermedia that our in vivo approach resulte
33 ts with beta-thalassemia major (TM) and beta-thalassemia intermedia (TI) were consecutively recruited
34 emoglobin (HbF) levels and morbidity in beta-thalassemia intermedia (TI), we analyzed data from 63 un
35  of beta-thalassemia, a model for human beta-thalassemia intermedia, we previously demonstrated that
36 is greatly increased in Hbb(th3/+) mice with thalassemia intermedia, where it contributes to the supp
37                                      In beta-thalassemia intermedia, which does not require blood tra