ライフサイエンスコーパス: thermogenesis

1 n 24 EE during COLD (i.e., less cold-induced thermogenesis).

2 their role in adipose tissue remodeling and thermogenesis.

3 in brown/beige adipocyte differentiation and thermogenesis.

4 dominated by metabolic functions related to thermogenesis.

5 of BAT in adult humans, and is indicative of thermogenesis.

6 chondrial oxidative phosphorylation to drive thermogenesis.

7 ondrial uncoupling and nonshivering adaptive thermogenesis.

8 an important fuel source fatty acid for BAT thermogenesis.

9 ondria regulating mitochondrial dynamics and thermogenesis.

10 reas CeA-DKO mice have impaired cold-induced thermogenesis.

11 anogenesis, inflammation, tissue repair, and thermogenesis.

12 nd roles in hair cycling, wound healing, and thermogenesis.

13 of lipogenesis, mitochondrial biogenesis and thermogenesis.

14 ature, created by captured sunlight or plant thermogenesis.

15 ect role in adipocyte metabolism or adaptive thermogenesis.

16 rated by the respiratory chain and increases thermogenesis.

17 es, which may serve to augment non-shivering thermogenesis.

18 ld-induced hypothermia was due to suppressed thermogenesis.

19 ficantly compromises the beige phenotype and thermogenesis.

20 pothalamus receptors to control appetite and thermogenesis.

21 with specialized roles in energy storage and thermogenesis.

22 activates brown adipose tissue and enhances thermogenesis.

23 nergy expenditure, a process called adaptive thermogenesis.

24 pated as heat in a process known as adaptive thermogenesis.

25 issue (BAT) provides a means of nonshivering thermogenesis.

26 nic program by PRDM16, a master regulator of thermogenesis.

27 -Br2 significantly inhibits brown adipocytes thermogenesis.

28 , suggesting increased reliance on BAT-based thermogenesis.

29 nal program that supports fuel oxidation and thermogenesis.

30 as a built-in rheostat negatively regulating thermogenesis.

31 eat production during cold- and diet-induced thermogenesis.

32 d mediator of brown adipose tissue-dependent thermogenesis.

33 ty acids (PUFA) promote brown adipose tissue thermogenesis.

34 icing program that suppresses adipose tissue thermogenesis.

35 ical controller of brown and beige adipocyte thermogenesis.

36 cy, which may serve to augment non-shivering thermogenesis.

37 reased energy expenditure and adipose tissue thermogenesis.

38 on and treatment of obesity by enhancing BAT thermogenesis.

39 sis, which may have contributed to increased thermogenesis.

40 oxidation is essential for optimal brown fat thermogenesis.

41 ble to achieve weight loss through increased thermogenesis.

42 sure and whether they are both necessary for thermogenesis.

43 tent with increased fatty acid oxidation and thermogenesis.

44 re in mice, highlighting a potential role in thermogenesis.

45 o prioritize translation of key proteins for thermogenesis.

46 ate and activate beige adipocytes, producing thermogenesis.

47 ocked cold-evoked or NMDA in MnPO-evoked BAT thermogenesis.

48 e electron transport chain (ETC) for fueling thermogenesis.

49 YS mice is exacerbated by brown fat adaptive thermogenesis.

50 old and other stimulators of beige adipocyte thermogenesis.

51 has also been implicated in skeletal muscle thermogenesis.

52 CA2b, resulting in SERCA2b stabilization and thermogenesis.

53 n photoreceptor that normally suppresses BAT thermogenesis.

54 reater vascularity and enhanced browning and thermogenesis.

55 ary-adipose signaling axis in the control of thermogenesis.

56 temperature in mammals through nonshivering thermogenesis.

57 Loss of FGF9 impairs BAT thermogenesis.

58 d droplets, preventing their use as fuel for thermogenesis.

59 the prominent contribution of brown AT (BAT) thermogenesis.

60 of activation of brown adipose tissue (BAT) thermogenesis.

61 in energy expenditure, most notably adaptive thermogenesis.

62 s a type of fat specialized in non-shivering thermogenesis.

63 ain high rates of lipid oxidation to support thermogenesis.

64 t includes severe obesity(1), and defects in thermogenesis(2) and lipolysis(3), both of which are adi

65 equently, IEX-1(-/-) mice exhibited enhanced thermogenesis (24 +/- 0.1 versus 22 +/- 0.1 kcal/hour/kg

66 rodent models designed to stimulate adaptive thermogenesis, a long-term increase in metabolism, prima

67 e through heat generation is termed adaptive thermogenesis, a process carried out by thermogenic adip

68 nd interscapular brown adipose tissue (iBAT) thermogenesis accompanied by reduced fat mass and improv

69 forms the adrenergic pathway responsible for thermogenesis activation into a death pathway.

70 AdKO-induced increases in thermogenesis also protected mice from cold-induced decr

71 energy balance, with particular interest in thermogenesis and browning of white adipose tissue (WAT)

72 em stimulation of brown adipose tissue (BAT) thermogenesis and browning of white adipose tissue (WAT)

73 hepatic glucose production, while enhancing thermogenesis and browning of white adipose tissue (WAT)

74 a potent driver of brown fat development and thermogenesis and cold-induced beige fat formation.

75 physiological level drives a full program of thermogenesis and converts iWAT to brown-like fat, which

76 ipose tissue (BAT) is essential for adaptive thermogenesis and dissipation of caloric excess through

77 e tissue (BAT) development and its long-term thermogenesis and energy expenditure remain unexamined.

78 a promoter, which epigenetically impairs BAT thermogenesis and energy expenditure, predisposing offsp

79 upplementation of pregnant mice on offspring thermogenesis and energy expenditure.

80 the functional relevance of PHOPSPHO1 in BAT thermogenesis and energy metabolism, we show that PHOSPH

81 ) NAD(+) metabolism in regulating whole-body thermogenesis and energy metabolism.

82 regulation of FGF21-target genes involved in thermogenesis and fatty acid oxidation in brown fat.

83 cle and neuron regeneration, enhanced muscle thermogenesis and fibrosis.

84 s the coordinated control of energy balance, thermogenesis and glucose homoeostasis.

85 t mice demonstrated increased adipose tissue thermogenesis and improved glycemia.

86 (beta-AR) potently stimulate adipose tissue thermogenesis and increase whole-body energy expenditure

87 tissue potently activates Ca(2+) cycling fat thermogenesis and increases whole-body energy expenditur

88 se mice with the IP6K inhibitor TNP enhanced thermogenesis and inhibited progression of DIO.

89 adrenergic mechanism by which Lcn2 regulates thermogenesis and lipid metabolism.

90 e energy expenditure through changes in both thermogenesis and locomotion.

91 ce involves mechanisms that affect appetite, thermogenesis and metabolism, and the outcomes of these

92 eases oxygen consumption in part by inducing thermogenesis and mitochondrial biogenesis in BAT along

93 WAT, had impaired gene programs involved in thermogenesis and mitochondrial function in BAT and a bl

94 ition in vivo represses beta-agonist-induced thermogenesis and oxygen consumption.

95 e demonstrate a novel function of Id1 in BAT thermogenesis and programming of beige adipocytes in whi

96 sion of Foxp1 in adipocytes impairs adaptive thermogenesis and promotes diet-induced obesity.

97 tively utilizes BCAA in the mitochondria for thermogenesis and promotes systemic BCAA clearance in mi

98 PGRMC2-null mice unable to activate adaptive thermogenesis and prone to greater metabolic deteriorati

99 onpathological cellular processes, including thermogenesis and protein secretion.

100 The offspring thermogenesis and related regulatory factors in adipose

101 P3 as an important mediator of physiological thermogenesis and support a renewed focus on targeting U

102 une cells and adipocytes is a determinant of thermogenesis and systemic energy balance.

103 CP1 and SLN are required to maintain optimal thermogenesis and that loss of both systems compromises

104 ve found two separate sites: one that drives thermogenesis and the other, previously unknown, that dr

105 This local thermogenesis and the resulting temperature gradient cou

106 n and beige adipocytes combust nutrients for thermogenesis and through their metabolic activity decre

107 ) has been associated with activation of BAT thermogenesis and weight loss in mice and rats.

108 as a mechanism that supports UCP1-dependent thermogenesis and whole-body energy expenditure, which o

109 dipocytes (BAs) are specialized for adaptive thermogenesis and, upon sympathetic stimulation, activat

110 s CLA's linkage with lipogenesis, lipolysis, thermogenesis, and browning of white and brown adipose t

111 hysical activity thermogenesis, diet-induced thermogenesis, and energy intake) were measured under fr

112 rticularly the linkage between inflammation, thermogenesis, and energy metabolism, is unclear.

113 plays important roles in energy expenditure, thermogenesis, and glucose homeostasis.

114 regulation of cellular stress responses and thermogenesis, and how O2 deficiency leads to metabolic

115 s surveys energy availability to engage iBAT thermogenesis, and identify AGRP neurons as a neuronal s

116 e of PHOSPHO1 as a negative regulator of BAT thermogenesis, and inhibition of PHOSPHO1 or enhancement

117 ent, causes browning of white fat, increases thermogenesis, and leads to substantial and sustained we

118 cells are fundamental for AT innervation and thermogenesis, and macrophages are required for recyclin

119 s for mitochondrial fatty acid oxidation and thermogenesis, and overall energy expenditure.

120 pid synthesis, promotes lipid catabolism and thermogenesis, and protects against diet-induced obesity

121 -specific Arg2 overexpression enhances basal thermogenesis, and protects from weight gain, insulin re

122 of brown/beige adipocyte differentiation and thermogenesis, and provide an important clue for its tar

123 nal sympathectomy compromises adipose tissue thermogenesis, and renders mice susceptible to obesity.

124 scle hypertrophy, brown adipose tissue (BAT) thermogenesis, and white adipose tissue (WAT) lipolysis

125 ; how to safely activate BAT and other organ thermogenesis; and how to sustain a negative energy bala

126 The metabolic adaptations required for thermogenesis are not fully understood.

127 ATP synthesis and thermogenesis are two critical outputs of mitochondrial

128 gnaling pathways that promote adipose tissue thermogenesis are well characterized, but the limiters o

129 ial pyruvate uptake is essential for optimal thermogenesis, as conditional deletion of Mpc1 in brown

130 Remarkably, this process supports in vivo thermogenesis, as pharmacological depletion of mitochond

131 hand, brain UGN induces brown adipose tissue thermogenesis, as well as browning and lipid mobilizatio

132 ose tissue (BAT), due to its direct roles in thermogenesis, as well as through additional mechanisms.

133 changes in muscle mitochondria contribute to thermogenesis at high altitude.

134 changes in muscle mitochondria contribute to thermogenesis at high altitudes.

135 Adaptive thermogenesis (AT) is the fat-free mass (FFM)-independen

136 el an unrecognized LepR neuron Sh2b1/SNS/BAT/thermogenesis axis that combats obesity and metabolic di

137 omponent of a SNS/brown adipose tissue (BAT)/thermogenesis axis.

138 obasal hypothalamus also impairs the SNS/BAT/thermogenesis axis; conversely, hypothalamic overexpress

139 ficantly rescue LPS, but not sympathomimetic thermogenesis blunted in UCP3KO mice.

140 Beyond thermogenesis, brown and beige fats engage other metabol

141 ompletely abrogated lipopolysaccharide (LPS) thermogenesis, but a normal response to noradrenaline.

142 mphetamine and fully inhibited noradrenaline thermogenesis, but an increased febrile response to LPS.

143 is a key site of shivering and non-shivering thermogenesis, but the importance of mitochondrial plast

144 is a key site of shivering and non-shivering thermogenesis, but the importance of mitochondrial plast

145 Mice lacking IL10 have enhanced thermogenesis, but the roles of specific cell types in t

146 thought to cause weight loss by stimulating thermogenesis, but whether FGF21 increases energy expend

147 Id1 suppresses BAT thermogenesis by binding to and suppressing PGC1alpha tr

148 tor 1alpha (PGC1alpha) controls BAT-mediated thermogenesis by regulating the expression of Ucp1 Inhib

149 on (by huddling in groups) with the costs of thermogenesis (by contributing heat).

150 Induction of nonshivering thermogenesis can be used to influence energy balance to

151 l a novel role for PLA2G2A on adipose tissue thermogenesis depending on thyroid status.

152 e (resting metabolic rate, physical activity thermogenesis, diet-induced thermogenesis, and energy in

153 consistent effect on 24-h physical activity thermogenesis (difference: 272 kcal/d; 95% CI: -254, 798

154 s were performed to investigate diet-induced-thermogenesis (DIT) and appetite sensation.

155 y, and in the bi-directional control of iBAT thermogenesis during nutrient deficiency and excess.

156 Hence, the improved thermogenesis during the development of wild boars is no

157 akfast resulted in greater physical activity thermogenesis during the morning than when fasting durin

158 unction as a developmental switch to license thermogenesis during the perinatal period.

159 e-regulated switch between ATP synthesis and thermogenesis enables cells to match outputs of mitochon

160 ects coupled (ATP production) and uncoupled (thermogenesis) energy conversion in mitochondria.

161 ose heterozygous knockouts showed defects in thermogenesis even at 30 degrees C and an inability to p

162 Thermogenesis from these adipocytes can combat obesity a

163 Cold thermogenesis had the highest representation in the dors

164 thermogenesis, its requirement for efficient thermogenesis has not been directly tested.

165 o norepinephrine (NE)-mediated regulation of thermogenesis have been a topic of debate.

166 energy-dissipating pathways that facilitate thermogenesis have been extensively described, yet littl

167 uced by Ces3 as a unique process to regulate thermogenesis in adipose tissue.

168 meostasis of retinoids and retinoid-mediated thermogenesis in adipose tissue.

169 Ces3 inhibition in vivo by showing that the thermogenesis in adipose tissues was significantly atten

170 rin's recently discovered role in regulating thermogenesis in adipose tissues.

171 itive fluorescent dye, ERthermAC, to monitor thermogenesis in BAs derived from murine brown fat precu

172 R stress-induced inhibition of lipolysis and thermogenesis in BAT.

173 -stimulated chromatin dynamics that modulate thermogenesis in BATs.

174 A2b-RyR2 pathway stimulates UCP1-independent thermogenesis in beige adipocytes.

175 ysiological and pharmaceutical activators of thermogenesis in both tissues.

176 tidylcholine (LPC), lipids shown to activate thermogenesis in brown adipocytes.

177 ty acid oxidation, oxidative metabolism, and thermogenesis in brown adipocytes.

178 Thermogenesis in brown adipose tissue (BAT) is an import

179 which POA neurons that express Opn5 regulate thermogenesis in brown adipose tissue (BAT).

180 art, to roles of Them2 in the suppression of thermogenesis in brown adipose tissue and insulin signal

181 Here we show that acutely activated thermogenesis in brown adipose tissue is defined by a su

182 program to maintain a critical capacity for thermogenesis in brown adipose tissue that can be rapidl

183 ical reactive oxygen species (ROS) initiates thermogenesis in brown and beige adipose tissues.

184 At weaning, HFD offspring had lower thermogenesis in brown and white adipose tissues compare

185 serves an indispensable role in cold-induced thermogenesis in brown fat.

186 (UCP1) plays a central role in nonshivering thermogenesis in brown fat; however, its role in beige f

187 signaling is a pathway controlling adaptive thermogenesis in brown or beige adipocytes.

188 se tissue browning and enhanced nonshivering thermogenesis in fat.

189 ctivation of brown fat underlies obesity and thermogenesis in Fgf13 heterozygous knockouts fed normal

190 f SERCA2b impairs UCP1-independent beige fat thermogenesis in humans and mice as well as in pigs, a s

191 identified as a facilitator of cold-induced thermogenesis in humans.

192 how to accurately measure individual tissue thermogenesis in humans; how to safely activate BAT and

193 irst direct evidence of UCP1-independent BAT thermogenesis in knockout mice.

194 (REE) and markers of brown and white adipose thermogenesis in lean mice.

195 cytes in white adipose tissue, and increased thermogenesis in mice, which is associated with decrease

196 ion leads to angiogenesis and UCP1-dependent thermogenesis in mouse brown and white adipose tissues.

197 inhibitors rescue mitochondrial function and thermogenesis in NCLX-null BAT, while calcium overload p

198 he metabolic O(2) demands for locomotion and thermogenesis in O(2)-thin air, but the degree to which

199 actation promoted white adipose browning and thermogenesis in offspring at weaning accompanied by per

200 lusion, maternal MFGM-PL treatment activated thermogenesis in offspring, which exerted long-term bene

201 sue upon exposure to the cold and suppresses thermogenesis in order to conserve energy reserves.

202 improved mitochondrial function, and rescued thermogenesis in Pex16-AKO mice.

203 p fever autonomically: they did not increase thermogenesis in response to a low, pyrogenic dose of LP

204 drial respiration to switch ATP synthesis to thermogenesis in response to heme.

205 Because of the dominant role of BAT-mediated thermogenesis in rodents, the role of muscle-based NST i

206 male mice in promoting adipocyte beiging and thermogenesis in SAT, in part by slanting M2 macrophage

207 This alternative pathway participates in thermogenesis in select organs of some species and is th

208 ranscriptional signature consistent with BAT thermogenesis in the context of HFD-induced obesity.

209 gy expenditure and markers of adipose tissue thermogenesis in the context of high-fat diet (HFD)-indu

210 We also built maps for cold-induced thermogenesis in unanesthetized rats and found the dorsa

211 study examined the effect of caffeine on BAT thermogenesis in vitro and in vivo.

212 se in energy intake and EE and activation of thermogenesis in WAT and brown adipose tissue were lost

213 capable of augmenting uncoupled respiration (thermogenesis) in brown adipocytes.

214 markable physiological adaptations including thermogenesis, increased intake of dietary energy, and e

215 Brown adipose tissue (BAT) thermogenesis increases energy expenditure (EE).

216 d browning expression programs, and restored thermogenesis, increasing it by a factor of 7.

217 apted to mild cold up-regulated muscle-based thermogenesis, indicated by increases in muscle succinat

218 henotypes that are consistent with defective thermogenesis; innervation can be fully rescued by resto

219 These results further confirm that SLN-based thermogenesis is a key player in muscle non-shivering th

220 Brown adipose tissue (BAT)-induced thermogenesis is a promising therapeutic target to treat

221 At the cellular level, thermogenesis is achieved through increased rates of fut

222 Adaptive thermogenesis is an energy-demanding process that is med

223 gh interscapular brown adipose tissue (iBAT) thermogenesis is an important contributor to adaptive en

224 Thermogenesis is an important homeostatic mechanism esse

225 around glucose and fatty acid metabolism and thermogenesis is found to decline with age and is implic

226 While non-shivering thermogenesis is mediated primarily by uncoupling protei

227 Adaptive thermogenesis is the cellular process transforming chemi

228 Adaptive thermogenesis is the process of heat generation in respo

229 It has been suggested that beige fat thermogenesis is tightly controlled by epigenetic regula

230 n has been implicated as being essential for thermogenesis, its requirement for efficient thermogenes

231 temic BCAA clearance, BAT fuel oxidation and thermogenesis, leading to diet-induced obesity and gluco

232 eukin-33 acts perinatally to ensure adaptive thermogenesis lifelong.

233 n response to pharmacological stimulation of thermogenesis linked to increased HDL levels in APOE*3-L

234 with key adipose tissue functions, including thermogenesis, lipid storage, and adipokine secretion.

235 nthesis is essential for regulating adaptive thermogenesis, lipolysis, and whole-body energy metaboli

236 drenergic signaling axis that acts to dampen thermogenesis, maintain tissue homeostasis, and reveal a

237 pose tissue (BAT) is an important tissue for thermogenesis, making it a potential target to decrease

238 ld exposure, the increased energy demands of thermogenesis must be counterbalanced by increased energ

239 esis is a key player in muscle non-shivering thermogenesis (NST) and can compensate for loss of BAT a

240 has been suggested as a site of nonshivering thermogenesis (NST) besides brown adipose tissue (BAT).

241 Muscle nonshivering thermogenesis (NST) was recently suggested to play an im

242 ltered insulin response/glucose handling and thermogenesis occurred prior to any functional decline i

243 Nonshivering thermogenesis occurs in brown adipose tissue to generate

244 siological activation of BAT and other organ thermogenesis occurs through beta-adrenergic receptors (

245 Recent studies suggest that thermogenesis of adipose tissues is involved in energy w

246 Adaptive thermogenesis of BAT was impaired in HFD offspring at we

247 ral raphe pallidus (rRPa) neurons influences thermogenesis of brown adipose tissue (BAT) independent

248 concept that brain alcohol sensing enhances thermogenesis of brown adipose tissue (BAT) through symp

249 of sympathetic premotor neurons controlling thermogenesis of brown adipose tissue (BAT).

250 ole of dietary macronutrient distribution on thermogenesis or energy expenditure for weight loss and

251 AKO) was not sufficient to affect adiposity, thermogenesis, or mitochondrial copy number, but knockdo

252 se tissue (BAT) is the primary non-shivering thermogenesis organ in mammals, which plays essential ro

253 uate the potential of mustard AITC to induce thermogenesis (primary outcome) and alter body temperatu

254 keletal muscle (i.e. sarcolipin (SLN)-based) thermogenesis processes play important roles in temperat

255 niques to identify a source of excitation to thermogenesis-promoting neurons in the DMH that is requi

256 c cold mimetics via activating non-canonical thermogenesis protect against obesity.

257 Our results point to a pathway for adipocyte thermogenesis regulation involving ARID5B, rs1421085, IR

258 modulates SERCA-dependent contractility and thermogenesis remain unclear.

259 Lipopolysaccharide thermogenesis requires skeletal muscle UCP3 but not UCP1

260 ate early and late phases of sympathomimetic thermogenesis, respectively.

261 Genes related to thermogenesis responded inconsistently to FGF21 treatmen

262 rons that control brown adipose tissue (BAT) thermogenesis, suggesting an additional role in energy h

263 de insight into the physiological control of thermogenesis that could inform future therapy.

264 We show that, during stimulation of BAT thermogenesis, the lipophilic gas xenon preferentially a

265 s to control UCP1-dependent and -independent thermogenesis, thereby contributing to the improvement o

266 t oxidized fatty acids to fuel Ucp1-mediated thermogenesis, thereby inhibiting lipid trafficking into

267 on its effect on brown adipose tissue (BAT) thermogenesis, though its effect on browning of white ad

268 NE enhances thermogenesis through beta3-adrenergic receptors to acti

269 DRN(Vgat) neurons are capable of regulating thermogenesis through both a "direct" descending pathway

270 vous system drives brown and beige adipocyte thermogenesis through the release of noradrenaline from

271 ate dehydrogenase activity for ATP-dependent thermogenesis through the SERCA2b pathway; beige fat the

272 to high altitude must sustain high rates of thermogenesis to cope with cold and hypoxic environments

273 to high altitude must sustain high rates of thermogenesis to cope with cold.

274 to postnatal life requires the activation of thermogenesis to maintain their core temperature.

275 ich have evolved a high capacity for aerobic thermogenesis, to determine the mechanisms of mitochondr

276 tissue (BAT), a key organ for non-shivering thermogenesis, to variations in nutritional state are no

277 g the role of mitochondrial ROS signaling in thermogenesis together with testable hypotheses for unde

278 l, our results highlight a role for AKAP1 in thermogenesis, uncoupled respiration, and regulation ene

279 but markers of mitochondrial uncoupling and thermogenesis (uncoupling protein-1, deiodinase-2, and p

280 e heat, relying on the principal effector of thermogenesis: uncoupling protein 1 (UCP1).

281 rplay between skeletal muscle- and BAT-based thermogenesis under mild versus severe cold adaptation b

282 To activate fat thermogenesis under tight spatiotemporal control without

283 al (i.e., locomotion) and physiologic (i.e., thermogenesis, vasodilatation) responses.

284 rived cytokine in the regulation of adaptive thermogenesis via a non-adrenergic pathway.

285 nesis and respiration, leading to attenuated thermogenesis via decreasing UCP1 expression.

286 1 signaling in the central amygdala controls thermogenesis via regulation of neural circuits innervat

287 Treatment of adipocytes with sLR11 inhibits thermogenesis via the bone morphogenetic protein/TGFbeta

288 he association between miR-30b/378 and brown thermogenesis was also confirmed in fish oil-fed C57/BL6

289 Significantly, the light-induced fat thermogenesis was sufficient to protect mice from diet-i

290 ncreasing visceral adiposity and in reducing thermogenesis, we assessed the existence of a possible l

291 243 treatments, oxygen consumption, and BAT thermogenesis were diminished in UCP1 KO mice, but BAT (

292 PGC1alpha, and other markers of browning and thermogenesis were elevated in IWAT and RWAT of AdKO mic

293 In contrast, changes in thermogenesis were uninvolved in the attenuation of the

294 increased body temperature, and exaggerated thermogenesis when cold-challenged.

295 ipose tissue (BAT) is an important source of thermogenesis which is nearly exclusively dependent on i

296 ed to investigate brown adipose tissue (BAT) thermogenesis, which requires mitochondrial uncoupling p

297 UCP1 Cys253 is sulfenylated during thermogenesis, while mutation of this site desensitizes

298 ase by increasing brown adipose tissue (BAT) thermogenesis, white adipose tissue (WAT) lipolysis, and

299 ed by robust expression of genes involved in thermogenesis whose transcriptome was selectively respon

300 the activation of brown adipose tissue (BAT) thermogenesis, yet the mechanisms preventing Ca(2+) dele