ライフサイエンスコーパス: theta burst stimulation

1 eled spines produced by a threshold level of theta burst stimulation.

2 tiation (LTP) was induced at both sites with theta burst stimulation.

3 s after a rTMS protocol such as intermittent theta burst stimulation.

4 ry (n = 56) olfactory bulbs before and after theta burst stimulation.

5 otocol (DePo) consisting of brief continuous theta burst stimulation.

6 ts and 12 healthy subjects, using continuous theta burst stimulation.

7 after the repeated application of continuous theta burst stimulation.

8 d SM-expressing O-LM cells to afferent fiber theta burst stimulation.

9 ency stimulation and enhanced LTP induced by theta-burst stimulation.

10 caused loss of long-term potentiation after theta-burst stimulation.

11 ral cell inhibition were also potentiated by theta-burst stimulation.

12 In addition, theta-burst stimulation, a protocol that was sufficient

13 ed from this hypothesis, induction of LTP by theta-burst stimulation activates an actin regulatory pa

14 extended course of accelerated intermittent theta burst stimulation (aiTBS), in patients with modera

15 sing an accelerated schedule of intermittent theta-burst stimulation (aiTBS), but the effectiveness o

16 Intermittent theta-burst stimulation also produced increases in outpu

17 Ca(2+) influx through L-type channels during theta burst stimulation, an action exerted via 12(S)-HPE

18 that are increasingly applied in the clinic: theta burst stimulation and accelerated scheduling.

19 mulation need to be further explored such as theta-burst stimulation and the combination of tDCS and

20 LTP in response to theta-burst stimulation and to 100-Hz tetanic stimulatio

21 stimulation, a protocol that is weaker than theta-burst stimulation and was not sufficient to induce

22 LTP was induced with theta-burst stimulation, and comparisons were made with

23 Slowly emerging membrane currents after theta burst stimulation are sensitive to the scavenger T

24 ates long-term potentiation (LTP) induced by theta burst stimulation at Schaffer collateral synapses

25 long-term potentiation (LTP) when induced by theta-burst stimulation but not high-frequency stimulati

26 transmission or on postsynaptic responses to theta burst stimulation, but nonetheless fully restored

27 ronavigated connectivity-guided intermittent theta burst stimulation (cgiTBS) at a site based on effe

28 lectrical stimulation of cortical inputs and theta burst stimulation combined with nicotine exposure

29 l processing in PMd, using either continuous theta burst stimulation (cTBS) at 80% (inhibitory cTBS)

30 we used noninvasive network-based continuous theta burst stimulation (cTBS) in human subjects (male a

31 Continuous theta burst stimulation (cTBS) is a repetitive transcran

32 hypothesis, participants received continuous theta burst stimulation (cTBS) over the left angular gyr

33 s hypothesis, we applied off-line continuous theta burst stimulation (cTBS) over the left inferior fr

34 ed in 30 healthy volunteers after continuous theta burst stimulation (cTBS) over the right cerebellar

35 spectively intermittent (iTBS) or continuous theta burst stimulation (cTBS) protocols.

36 cephalography (MEG) combined with continuous theta burst stimulation (cTBS) to (i) clarify the roles

37 We used continuous theta burst stimulation (cTBS) to condition the excitabi

38 a previous study suggesting that continuous theta burst stimulation (cTBS) to the right dlPFC can ma

39 30 patients with spatial neglect, continuous theta burst stimulation (cTBS) was applied over the left

40 Continuous theta burst stimulation (cTBS) with 600 pulses produces

41 ere, we employed a combination of continuous theta burst stimulation (cTBS), MR spectroscopy and fMRI

42 hanisms underlying the effects of continuous Theta-Burst Stimulation (cTBS) in humans are poorly unde

43 ed sham-controlled, non-invasive, continuous theta-burst stimulation (cTBS) to temporarily disrupt hu

44 shortly after the application of continuous theta-burst stimulation (cTBS) to the primary somatosens

45 For each of two TMS sessions, continuous theta-burst stimulation (cTBS) was applied to either a c

46 Theta burst stimulation delivered after infusions of Gly

47 Following theta-burst stimulation, evoked optical signals showed a

48 Theta-burst stimulation failed to induce LTP after 1 wee

49 Furthermore, MOR activation induced by theta burst stimulation in BA suppresses plastic changes

50 Long-term potentiation (LTP) induced by theta burst stimulation in the anterior piriform cortex

51 le or repeated high frequency stimulation or theta burst stimulation in the CA1 region.

52 Long-term potentiation (LTP) in response to theta burst stimulation in the hippocampus was also redu

53 Specifically, LTP induced with theta-burst stimulation in basal dendrites of hippocampa

54 of long-term potentiation (LTP) elicited by theta-burst stimulation in field CA1 of hippocampus.

55 y, we found that hippocampal LTP, induced by theta-burst stimulation in mature (>8-week-old) GluR1 kn

56 eads to deficits in associative learning and theta burst stimulation-induced LTP.

57 Glycine-induced LTP was occluded by previous theta burst stimulation-induced potentiation, indicating

58 us, GRF1 promotes LTD, whereas GRF2 promotes theta-burst stimulation-induced LTP (TBS-LTP).

59 lts provide Class I evidence that continuous theta burst stimulation is a viable add-on therapy in ne

60 show that long-term potentiation induced by theta-burst stimulation is decreased after presynaptic b

61 -term potentiation induced in area CA1 using theta-burst stimulation is particularly compromised by t

62 We conclude that theta-burst stimulation is particularly well suited to p

63 ion with high-frequency stimulation, but not theta burst stimulation, is perturbed in hippocampal CA1

64 tly altering activity in CB via intermittent theta burst stimulation (iTBS) affects PPC-M1 connectivi

65 LTP-like plasticity elicited by intermittent theta burst stimulation (iTBS) is reduced in the primary

66 or depressive disorder, and the intermittent theta burst stimulation (iTBS) protocol is replacing con

67 Accelerated intermittent theta burst stimulation (iTBS) rTMS protocols are promis

68 ing conventional 10-Hz rTMS and intermittent theta burst stimulation (iTBS) rTMS.

69 rophobia received an activating intermittent theta burst stimulation (iTBS) targeting the left poster

70 In healthy humans, we applied intermittent theta burst stimulation (iTBS) to the vermis lobule VII

71 ditions targeting the left OFC: intermittent theta burst stimulation (iTBS), expected to increase OFC

72 timulation (rMPS) and patterned intermittent theta burst stimulation (iTBS), incorporating different

73 s a novel approach to PTSD, and intermittent theta-burst stimulation (iTBS) is a new, more rapid admi

74 Intermittent theta-burst stimulation (iTBS) is a noninvasive brain st

75 Intermittent theta-burst stimulation (iTBS) is an established treatme

76 Intermittent theta-burst stimulation (iTBS) is approved by the U.S. F

77 c stimulation protocols such as intermittent theta-burst stimulation (iTBS) remain poorly understood.

78 ssociative stimulation (PAS) or intermittent theta-burst stimulation (iTBS).

79 able to respond with further potentiation to theta-burst stimulation, leading to impaired LTP.

80 ice have fewer mature dendrites and impaired theta burst stimulation long-term potentiation.

81 Successive bouts of naturalistic theta burst stimulation of field CA1 afferents markedly

82 s underwent long-term depression (LTD) after theta burst stimulation of the accessory olfactory bulb,

83 , both of which were restored by concomitant theta burst stimulation of the fimbria-fornix pathway.

84 s with drug-resistant epilepsy suggests that theta burst stimulation of the fornix may be associated

85 th a small group of cases to explore whether theta burst stimulation of the fornix might improve memo

86 Theta burst stimulation of the granule cell layer potent

87 By contrast, theta burst stimulation of the granule cell layer potent

88 Theta burst stimulation of the perforant path potentiate

89 We report here that patterned (theta burst) stimulation of the dorsal lateral geniculat

90 Theta-burst stimulation of BLA potentiated BLA-evoked re

91 Theta-burst stimulation of corticostriatal fibres produc

92 We found that theta-burst stimulation of mossy fiber input in lobule 9

93 of synaptic potentiation (E-LTP) induced by theta-burst stimulation of rat hippocampal CA1 synapses.

94 udy, we tested whether individually-tailored theta-burst stimulation of the dorsolateral prefrontal c

95 ical excitability, which is maintained after theta-burst stimulations of the cerebellum.

96 The effects of theta-burst stimulation on Kv4 channel control of the ga

97 , explored by means of transcranial magnetic theta burst stimulation over the primary motor cortex, w

98 Theta-burst stimulation over the SPC led to a more conse

99 We found that theta burst stimulation paired with postsynaptic spiking

100 ression and long-term potentiation (LTD/LTP) theta-burst stimulation paradigms, we examined whether T

101 Prolonged intermittent theta burst stimulation (piTBS) with triple doses of the

102 theta-Burst stimulation produced a strong short-term enh

103 ed for the induction of LTP in response to a theta burst stimulation protocol that depends on Ca(2+)

104 Theta-burst stimulation reliably produced postsynaptic s

105 ity in humans using network-based continuous theta burst stimulation selectively impairs behavior tha

106 rrent TMS with a modified short intermittent theta burst stimulation (sTBS) protocol.

107 Theta-burst stimulation targeting the hippocampal networ

108 Indeed, theta-burst stimulation targeting the hippocampal networ

109 male, 3 female, mean age 34 +/- 3 years) or theta burst stimulation (TBS) (n = 9, 6 male, 3 female,

110 induction of long-term potentiation (LTP) by theta burst stimulation (TBS) activates beta1 integrins,

111 Theta burst stimulation (TBS) and primed bursts (PBs) st

112 lationship between glutamate dynamics during theta burst stimulation (TBS) and the resulting magnitud

113 indings to individuals with MCI and leverage theta burst stimulation (TBS) and white matter tractogra

114 ent, long-term potentiation (LTP) induced by theta burst stimulation (TBS) at one retinal input sprea

115 LTP induced by theta burst stimulation (TBS) in hippocampal slices from

116 adult rats, coincident pre- and postsynaptic theta burst stimulation (TBS) induced LTP and we show th

117 Theta burst stimulation (TBS) is a form of repetitive tr

118 Theta burst stimulation (TBS) is a noninvasive brain sti

119 e we report that repetitive light stimuli or theta burst stimulation (TBS) of the optic nerve in the

120 In clinical practice, theta burst stimulation (TBS) presents as a more efficie

121 Theta burst stimulation (TBS) produces an extremely stab

122 Theta burst stimulation (TBS) protocols of repetitive tr

123 Activation of vChATs with theta burst stimulation (TBS) that alleviates the decay

124 Theta burst stimulation (TBS) triggered less DCV fusion

125 he memory task, brief trains of intracranial theta burst stimulation (TBS) were delivered to the baso

126 t PDE4A5 impairs long-lasting LTP induced by theta burst stimulation (TBS) while sparing long-lasting

127 Theta burst stimulation (TBS), a specific form of repeti

128 Theta burst stimulation (TBS), a specific protocol of re

129 ed in layer II/III, layer V or layer VI with theta burst stimulation (TBS), but was not observed in l

130 on the efficacy of repetitive TMS (rTMS) and theta burst stimulation (TBS), when targeting specific c

131 LTP induced by compressed theta burst stimulation (TBS), with a 10 s inter-episode

132 mpal field CA1 of both sexes but left intact theta burst stimulation (TBS)-induced actin polymerizati

133 enetic deletion of MAGL selectively enhanced theta burst stimulation (TBS)-induced long-term potentia

134 Here, we report that PKA activation restores theta burst stimulation (TBS)-induced long-term potentia

135 oA antisense oligodeoxynucleotides inhibited theta burst stimulation (TBS)-induced RhoA upregulation

136 ing high frequency stimulation (HFS), versus theta burst stimulation (TBS).

137 airs long-term potentiation (LTP) induced by theta burst stimulation (TBS-LTP).

138 ity in distal synapses upon local electrical theta-burst stimulation (TBS) and predicted proximal and

139 n acute mouse cerebellar slices, mossy fiber theta-burst stimulation (TBS) could induce either long-t

140 ory effects of 5-HT on plasticity induced by theta-burst stimulation (TBS) in two postnatal periods o

141 Theta-burst stimulation (TBS) is a form of non-invasive

142 Theta-burst stimulation (TBS) is a patterned form of rep

143 s in the synaptic response of SPNs following theta-burst stimulation (TBS) of cortical afferents.

144 Here, we determined how theta-burst stimulation (TBS) of primary afferents impac

145 In this study, we found that theta-burst stimulation (TBS) of sensory nerves induced

146 ignificant deficits in LTP induced by either theta-burst stimulation (TBS) or "pairing." Slices expos

147 Subthreshold theta-burst stimulation (TBS) produced small amplitude p

148 The theta-burst stimulation (TBS) protocol synaptic plastici

149 tag." TrkB is transiently activated by weak theta-burst stimulation (TBS) that induces only early-ph

150 of the NMDAR antagonists were observed when theta-burst stimulation (TBS) was used.

151 Theta-burst stimulation (TBS), a patterned brain stimula

152 epetitive TMS (rTMS) protocols, particularly theta-burst stimulation (TBS), and relevant rTMS-derived

153 lectroconvulsive therapy (ECT), minocycline, theta-burst stimulation (TBS), repetitive transcranial m

154 h is induced by stimulation at theta rhythm [theta-burst stimulation (TBS)-LTP], but there was no imp

155 , bursting stimulation at 25 and 100 Hz, and theta-burst stimulation (TBS).

156 inutes and 2 hours after induction of LTP by theta-burst stimulation (TBS).

157 se the LTP deficit observed in KO mice after theta-burst stimulation (TBS).

158 Theta-burst stimulation (TBS; n = 8) delivered to CA1 re

159 recovery period, either LTP (100-pulse, 5-Hz theta-burst stimulation [TBS]) or LTD (900-pulse, 1-Hz l

160 se in corticospinal excitability by applying theta burst stimulation to either the dorsolateral prefr

161 Following inhibitory theta burst stimulation to the left FEF, right FEF, or v

162 ority trial, THREE-D, comparing intermittent theta-burst stimulation to high-frequency rTMS of the le

163 and adult rat hippocampus that had undergone theta-burst stimulation to produce long-term potentiatio

164 before and immediately after TMS (continuous theta burst stimulation) to the left or right AC, and th

165 ether the repeated application of continuous theta burst stimulation trains could ameliorate spatial

166 Except for theta-burst stimulation vs sham, similar results were ob

167 -D-aspartate receptor (NMDAR) opening during theta burst stimulation was enhanced by M(1) receptor ac

168 Intermittent TMS-theta burst stimulation was used to probe long-term pote

169 explored through intermittent or continuous theta-burst stimulation was also similar in the "G" and

170 Using continuous theta burst stimulation, we demonstrated that behavioral

171 riate analysis of human fMRI, and continuous theta-burst stimulation, we identified two brain regions

172 Eight trains of continuous theta burst stimulation were applied over two consecutiv

173 ponses and long-term potentiation induced by theta-burst stimulation were decreased in nicastrin cKO

174 Theta burst stimulation, which was more effective in thi

175 ry for demanding neural computation, such as theta-burst stimulation, while glucose is sufficient for

176 ory cortex (S1) were targeted for continuous theta-burst stimulation, while stimulation over the occi

177 ered four interventions: (1) sham continuous theta burst stimulation with 150 pulses (cTBS150) follow

178 ntiation of glutamatergic transmission after theta burst stimulation with or without nicotine only oc

179 We reasoned that theta-burst stimulation would be particularly effective