ライフサイエンスコーパス: thiobarbituric acid

1 and TMA), oxidative (peroxides value, K230, thiobarbituric acid and K270) and sensory analyses were

2 We used in situ chemical trapping with 2-thiobarbituric acid and mass spectrometry with a deutera

3 ptible to lipid peroxidation (as measured by thiobarbituric acid) and to cell kllling within a 90-min

4 al production as determined with a cell-free thiobarbituric acid assay.

5 We identified a thiobarbituric acid derivative, 5376753, that allosteric

6 Merbarone (5-[N-phenyl carboxamido]-2-thiobarbituric acid) is an anticancer drug that inhibits

7 Reactive oxygen species (mumole/min) and thiobarbituric acid (mM/mg/protein) levels increased whi

8 (2)-isoprostanes, malondialdehyde (MDA), and thiobarbituric acid reacting substances (TBARS), in the

9 ilting, loss of chlorophyll, and increase in thiobarbituric acid reacting substances.

10 amounts of glucose, urea, triglycerides, and thiobarbituric acid-reacting substances in diabetic rats

11 y weight ratio, urinary albumin, and urinary thiobarbituric acid-reacting substances.

12 thin-layer chromatography and the periodate-thiobarbituric acid reaction, we found that the hepatopa

13 Thiobarbituric acid reactive material (TBARM) in tissue

14 roxidation in retinal homogenates was by the thiobarbituric acid reactive species (TBARS) formation

15 mulsion was tested by the hydroperoxides and thiobarbituric acid reactive species (TBARS) measurement

16 in liver homogenates from Wistar rats by the thiobarbituric acid reactive species test.

17 ioxidant activity, as assessed indirectly by thiobarbituric acid reactive substance (TBARS) formation

18 rase (AST), lactate dehydrogenase (LDH), and thiobarbituric acid reactive substance (TBARS), and sali

19 Spectrofluorometry was used to analyze thiobarbituric acid reactive substance (TBARS); ferric-r

20 Heat caused increased thiobarbituric acid reactive substance levels (an indica

21 MDA was measured as thiobarbituric acid reactive substance.

22 ia and the high K+-mediated increase in lung thiobarbituric acid reactive substance.

23 cellular adhesion molecule-1 (p = 0.001) and thiobarbituric acid reactive substances (p = 0.001) as w

24 We measured diaphragmatic concentrations of thiobarbituric acid reactive substances (TBAR), a marker

25 creased the hexanal content (0.21mug/ml) and thiobarbituric acid reactive substances (TBARS) (2.56mug

26 es, and capacity to inhibit the formation of thiobarbituric acid reactive substances (TBARS) (IC(50)

27 ent and composition, free fatty acids (FFA), thiobarbituric acid reactive substances (TBARS) and fluo

28 from W-MR-Al had lower peroxide value (PV), thiobarbituric acid reactive substances (TBARS) and non-

29 tent, total volatile basic nitrogen (TVB-N), thiobarbituric acid reactive substances (TBARS) and pero

30 Results from thiobarbituric acid reactive substances (TBARS) and sens

31 The thiobarbituric acid reactive substances (TBARS) assay is

32 on via oxidative stress was also detected by thiobarbituric acid reactive substances (TBARS) assay.

33 of fish oil oxidation was studied using the thiobarbituric acid reactive substances (TBARS) assay.

34 owth and lipid/protein spoilage, maintaining Thiobarbituric Acid Reactive Substances (TBARS) at 1.7 m

35 glycated albumin content, by measurement of thiobarbituric acid reactive substances (TBARs) for lipi

36 Peroxide value (PV) and thiobarbituric acid reactive substances (TBARS) had incr

37 oxy-9,11-octadecadienoic acid (13-HODE), and thiobarbituric acid reactive substances (TBARS) in 252 w

38 Serum MDA levels were measured as thiobarbituric acid reactive substances (TBARS) in 634 p

39 However, thiobarbituric acid reactive substances (TBARS) increase

40 extract inhibited lipid peroxidation in the thiobarbituric acid reactive substances (TBARS) method (

41 Oxidative stability was evaluated by thiobarbituric acid reactive substances (TBARS) on day (

42 xidation was monitored in parallel using the thiobarbituric acid reactive substances (TBARS) test.

43 Lipid hydroperoxides and thiobarbituric acid reactive substances (TBARS) were ana

44 Lower total volatile base (TVB) and thiobarbituric acid reactive substances (TBARS) were det

45 ity (ORAC) and lipid peroxidation assayed as thiobarbituric acid reactive substances (TBARS) were mea

46 9-hydroxyoctadecadieneoic acid (9-HODE), and thiobarbituric acid reactive substances (TBARS) were mea

47 Thiobarbituric acid reactive substances (TBARS) were red

48 Serum ALT value, and hepatic thiobarbituric acid reactive substances (TBARS), and hep

49 AP), 2, 2'-diphenyl-1-picrylhydrazyl (DPPH), thiobarbituric acid reactive substances (TBARs), and Thi

50 for acidity index (IA), peroxide index (IP), thiobarbituric acid reactive substances (TBARS), conjuga

51 profiles, formation of hydroperoxides (PV), thiobarbituric acid reactive substances (TBARS), fluores

52 2-thiobarbituric acid reactive substances (TBARS), hexanal

53 Hydroperoxide, thiobarbituric acid reactive substances (TBARS), malondi

54 The increases in thiobarbituric acid reactive substances (TBARS), p-anisi

55 lates) with glutathione peroxidase (GPx) and thiobarbituric acid reactive substances (TBARS), while a

56 y acids (FFA), lipid hydroperoxides (PV) and thiobarbituric acid reactive substances (TBARS).

57 nally analyzed by two Raman spectroscopy and thiobarbituric acid reactive substances (TBARS).

58 OHdG), superoxide dismutase (Cu-Zn SOD), and thiobarbituric acid reactive substances (TBARS).

59 (PFP, 1.18-1.32 mmol peroxides/kg mince) and thiobarbituric acid reactive substances (TBARS, 0.30-0.3

60 ct C1 inhibited efficiently the formation of thiobarbituric acid reactive substances (TBARS; IC(50) =

61 as determined by conjugated dienes (CDs) and thiobarbituric acid reactive substances (TBARSs) product

62 cells were treated with ox-LDL (50 microg/mL thiobarbituric acid reactive substances 12 to 16 nmol/mg

63 ed by measuring lipid peroxides (measured as thiobarbituric acid reactive substances [TBARS]) in reti

64 Thiobarbituric acid reactive substances activity indicat

65 yeast molds and restricted the formation of thiobarbituric acid reactive substances and biogenic ami

66 was assessed by histology and measurement of thiobarbituric acid reactive substances and NOX-related

67 Peroxide value, concentration of thiobarbituric acid reactive substances and oxygen uptak

68 There were no differences in thiobarbituric acid reactive substances and reduced glut

69 ough determination of the peroxide value and thiobarbituric acid reactive substances as well as analy

70 eptibility to lipid peroxidation by both the thiobarbituric acid reactive substances assay and the fl

71 oxidants in preventing lipoperoxidation in a thiobarbituric acid reactive substances assay.

72 y measuring malondialdehyde levels using the thiobarbituric acid reactive substances assay.

73 g, pH variation) using Conjugated Dienes and Thiobarbituric acid reactive substances assays.

74 al volatile base content, peroxide value and thiobarbituric acid reactive substances but high sensory

75 of skeletal muscle carbonylated proteins and thiobarbituric acid reactive substances during hyperammo

76 evealed the greatest capacity to inhibit the thiobarbituric acid reactive substances formation (IC(50

77 d value, peroxide value, anisidine value and thiobarbituric acid reactive substances value) during st

78 edium and high oxidative groups according to thiobarbituric acid reactive substances values after 9da

79 Thiobarbituric acid reactive substances values and lipox

80 The increased trypan blue uptake and thiobarbituric acid reactive substances were inhibited b

81 hydroxy--deoxyguanosine, and H2O2 and plasma thiobarbituric acid reactive substances were significant

82 ease in mean F2-isoprostane and no effect on thiobarbituric acid reactive substances when compared to

83 TBARS (thiobarbituric acid reactive substances) test was used t

84 cts (4-hydroxy-2-nonenal, hexanal, propanal, thiobarbituric acid reactive substances), protein oxidat

85 es of oxidative stress, urinary excretion of thiobarbituric acid reactive substances, 8-hydroxy--deox

86 lactate dehydrogenase, laccase) and damage (thiobarbituric acid reactive substances, acetylcholinest

87 tile base nitrogen, trimethylamine nitrogen, thiobarbituric acid reactive substances, ATP catabolism

88 Quantitative measurements of thiobarbituric acid reactive substances, conjugated dien

89 , evidenced by an 80% reduction (P<0.001) in thiobarbituric acid reactive substances, effective inhib

90 and alterations in the heat shock response, thiobarbituric acid reactive substances, heat shock prot

91 ed bacterial cells show an enhanced level of thiobarbituric acid reactive substances, indicating the

92 n (4-hydroxy-2-nonenal, 4-HNE; hexanal, HEX; thiobarbituric acid reactive substances, TBARS), and pro

93 Lipid oxidation, assessed as thiobarbituric acid reactive substances, was significant

94 ed with decreased urinary levels of H2O2 and thiobarbituric acid reactive substances.

95 by 2-hydroxyethidine, and lipid peroxides by thiobarbituric acid reactive substances.

96 The minimum 2-thiobarbituric acid reactive values were observed for th

97 ght ratio (W/D), tissue albumin index (TAI), thiobarbituric acid-reactive material content (TBARM), a

98 rotein and lactate dehydrogenase (LDH), lung thiobarbituric acid-reactive species, and lung histology

99 ues, was subject to analysis by conventional thiobarbituric acid-reactive substance (TBARS) and ferro

100 e, anserine, homocarnosine, pentosidine, and thiobarbituric acid-reactive substance (TBARS) contents.

101 The peroxide value (PV) and thiobarbituric acid-reactive substance (TBARS) value of

102 Thiobarbituric acid-reactive substance and oxidized and

103 s compared with H441 showed less increase in thiobarbituric acid-reactive substance and phosphatidylc

104 of low density lipoprotein was measured by a thiobarbituric acid-reactive substance, which was confir

105 throcytes glutathione peroxidase (FB400) and thiobarbituric acid-reactive substances (FB100, FA400, F

106 lement increased the plasma concentration of thiobarbituric acid-reactive substances (TBARS) (P: = 0.

107 esulted in a fivefold increased formation of thiobarbituric acid-reactive substances (TBARS) and acti

108 Lipid peroxidation was measured as thiobarbituric acid-reactive substances (TBARS) and alph

109 n forms and quercetin, and reduced levels of thiobarbituric acid-reactive substances (TBARS) and carb

110 after the heat treatment, there were higher thiobarbituric acid-reactive substances (TBARs) and chol

111 de value (PV) was found and large amount of, thiobarbituric acid-reactive substances (TBARS) and hexa

112 The increases in thiobarbituric acid-reactive substances (TBARS) and p-an

113 significantly reduced (p<0.05) the levels of thiobarbituric acid-reactive substances (TBARS) and prot

114 synergism in inhibition of the formation of thiobarbituric acid-reactive substances (TBARS) and prov

115 d symptoms, exercise, ejection fraction, and thiobarbituric acid-reactive substances (TBARS) as an in

116 n a lipid system was determined by using the thiobarbituric acid-reactive substances (TBARS) assay.

117 Glucose, insulin, and thiobarbituric acid-reactive substances (TBARS), a marke

118 Furthermore, the content of thiobarbituric acid-reactive substances (TBARS), and cat

119 els of lipid peroxidation products including thiobarbituric acid-reactive substances (TBARS), glutath

120 e authors analyzed the association of plasma thiobarbituric acid-reactive substances (TBARS), glutath

121 dation, breath ethane (an in vivo assay) and thiobarbituric acid-reactive substances (TBARS, an in vi

122 a lipid antioxidant, suppressed increases in thiobarbituric acid-reactive substances (TBARSs; a measu

123 ompanied by decreases in the amount of total thiobarbituric acid-reactive substances -TBARS and gluta

124 olvents exhibited the lowest peroxide value, thiobarbituric acid-reactive substances and beany odour

125 Both proteins block accumulation of thiobarbituric acid-reactive substances and conjugated d

126 n of 8-epi-PGF2 alpha coincided with that of thiobarbituric acid-reactive substances and lipid hydrop

127 O-deethylase activity), or oxidative stress (thiobarbituric acid-reactive substances and ratios of re

128 develop normally, and their plasma levels of thiobarbituric acid-reactive substances do not differ fr

129 volume in 1 second (FEV1) with 1) levels of thiobarbituric acid-reactive substances in plasma (p-TBA

130 d deletions in kidney mitochondrial DNA, and thiobarbituric acid-reactive substances in plasma, toget

131 d prevented the diabetes-induced increase in thiobarbituric acid-reactive substances in serum and sig

132 low density lipoprotein (measured as either thiobarbituric acid-reactive substances or the oxidant s

133 Thiobarbituric acid-reactive substances were higher prec

134 se, hair copper, urinary copper, and urinary thiobarbituric acid-reactive substances were measured du

135 one-binding globulin, F(2)-isoprostanes, and thiobarbituric acid-reactive substances were measured up

136 ion, hair copper concentrations, and urinary thiobarbituric acid-reactive substances were significant

137 incubated with 10 microgram/ml oxLDL (10-15 thiobarbituric acid-reactive substances) and blocking an

138 Lipid peroxidation (thiobarbituric acid-reactive substances) increased after

139 otype in which oxidative damage (measured as thiobarbituric acid-reactive substances) was significant

140 of lipid peroxidation (conjugated dienes and thiobarbituric acid-reactive substances) were also great

141 xposure to IL-1alpha, TNF-alpha, and ox-LDL (thiobarbituric acid-reactive substances, 13.4 nmol/mg LD

142 Thiobarbituric acid-reactive substances, a less specific

143 37, suppressed formation of 8-epi-PGF2alpha, thiobarbituric acid-reactive substances, and lipid hydro

144 f several markers of oxidative status (i.e., thiobarbituric acid-reactive substances, erythrocyte glu

145 stress, as exemplified by the generation of thiobarbituric acid-reactive substances, expression of h

146 Thiobarbituric acid-reactive substances, markers of oxid

147 the diabetes-associated increases in plasma thiobarbituric acid-reactive substances, mitochondrial D

148 s of headspace aldehydes, malonaldehyde, and thiobarbituric acid-reactive substances.

149 ipid peroxidation was determined by assaying thiobarbituric acid-reactive substances.

150 The commonly used thiobarbituric-acid-reactive-substances (TBARS) assay to

151 by free radical-mediated lipid peroxidation (thiobarbituric acid reactivity), which could be suppress

152 s of lipid peroxidation (lipid peroxides and thiobarbituric acid-reactivity), plasma chelatable iron,

153 90, 180 days) at -18 C on pH, total acidity, thiobarbituric acid, residual nitrite-nitrate, color and

154 re widely followed, including measurement of thiobarbituric acid substances and the sole use of fluor

155 trichloroacetic acid (TCA), reaction with 2-thiobarbituric acid (TBA) and quantification with ultrap

156 , pH, total volatile basic-nitrogen (TVB-N), thiobarbituric acid (TBA) as well as free amino acid (FA

157 etermined by assay of snap frozen tissue for thiobarbituric acid (TBA) concentrations (nmol/g tissue

158 PLs showed a proportionate relationship with thiobarbituric acid (TBA) number, an indicator of lipid

159 ts, total volatile basic nitrogen (TVBN) and thiobarbituric acid (TBA) of the dried milkfish samples

160 de value, iodine value specification number, thiobarbituric acid (TBA) value and colour of pistachio

161 ificantly reduced peroxidase value (POV) and thiobarbituric acid (TBA) values during storage period.

162 cing systems, according to peroxide (PV) and thiobarbituric acid (TBA) values.

163 3) products of reactive oxygen species (ROS; thiobarbituric acid [TBA]-reacting material, and dichlor

164 lipid peroxidation further confirmed by the thiobarbituric acid (TBAR) assay.

165 Thiobarbituric acid (TBARS) test demonstrated that compa

166 n in mitochondrial membrane fragments by the thiobarbituric acid test and by measurement of nonrespir

167 Results of thiobarbituric acid tests showed that in bulk fish oil,

168 rowth (total count: over 1.6 log CFU/g), pH, thiobarbituric acid value (TBA), and total volatile nitr

169 measuring Free Fatty Acids, peroxide value, Thiobarbituric Acid value and color value during 12 mont

170 rtified muffins exhibited lower peroxide and thiobarbituric acid values during a 14 day storage than

171 Anisidine and thiobarbituric acid values indicated the formation of se