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1 r decreasing gradient along the cervical and thoracic cord.
2 toneurons, with no change in the brachial or thoracic cord.
3 ctive fibers at T1, T4, T8, and T10 of human thoracic cord.
4  that extend past cervical segments into the thoracic cord.
5 ners is not well known, particularly for the thoracic cord.
6 ammatory disease with a predilection for the thoracic cord.
7 or imaging and magnetization transfer ratio; thoracic cord: 3D T2), together with disability, pain an
8  6-7 days after transplantation into the low thoracic cord and by 10 days had spread rostrally to the
9 e intermediolateral (IML) cell column of the thoracic cord and the sacral parasympathetic nucleus (SP
10 ere located within the cervical cord, 18% in thoracic cord, and 19% in the lumbosacral spinal cord.
11       The corresponding relationships in the thoracic cord are unknown owing to technical difficultie
12 n the generation of locomotor outputs in the thoracic cord by acting at both the premotoneuronal and
13 vical cord cross-sectional area (P = 0.038), thoracic cord cross-sectional area (P = 0.043), cervical
14               Dorsal hemisections of the mid thoracic cord did not alter the immunohistochemical sign
15 oss-sectional area of the cervical and upper thoracic cord (down to T3 level) was calculated with the
16 ed Disability Status Scale score followed by thoracic cord GM area and brain GM volume.
17 Patients with relapsing MS (RMS) had smaller thoracic cord GM areas than did age- and sex-matched con
18                                              Thoracic cord GM areas were correlated with lower limb f
19                                              Thoracic cord GM atrophy can be detected in vivo in the
20  through the lesion after transection of the thoracic cord in developing opossums and that the critic
21 pinal cord injury patients (8-8 cervical and thoracic cord injuries) within 1.5 years after injury an
22            Persistent, caudally located (ie, thoracic) cord lesions in AQP4-Ab-positive patients asso
23                       Patients with isolated thoracic cord myelitis at the time of pain onset were si
24 excitability of propriospinal neurons in the thoracic cord of the adult mice with a complete thoracic
25 l root recordings, we showed that, while the thoracic cord possesses an intrinsic rhythmogenic capaci
26 odels of SCI in rodents induce damage in the thoracic cord, resulting primarily in white matter disru
27  analysis of the gene expression profiles of thoracic cord samples from rats carrying the G93A SOD1 g
28 pport the idea that the rvlm projects to the thoracic cord via disynaptic, intrareticular pathways pa
29                                     When the thoracic cord was transected at postnatal day (PD) 5 and