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1 for visualization of the thoracic duct ( TD thoracic duct ).
2 f trunk lymphatic vessels and did not form a thoracic duct.
3 l circuits connecting to lymph nodes and the thoracic duct.
4 mph nodes and traffic into the blood via the thoracic duct.
5 from lymphoid tissues into blood, the human thoracic duct.
6 vessels and large lymphatic trunks like the thoracic duct.
7 formation of the PAC and, subsequently, the thoracic duct.
8 bsequently heal, permitting DCs to enter the thoracic duct.
9 ump in both mesenteric lymphatics and in the thoracic duct.
10 al effusions, ascites, and dilatation of the thoracic duct.
11 pumping capacity and morphologically changed thoracic duct.
12 muscle myosin heavy chain (SM-MHC), whereas thoracic duct and arterioles expressed both SMA and SMB
13 le activities and MLC(20) phosphorylation of thoracic duct and cervical lymphatics were determined in
14 e collecting lymphatic vessels including the thoracic duct and cisterna chyli, and presenting as chyl
15 atic contractions in isolated human vessels (thoracic duct and mesenteric lymphatics) maintained unde
17 pment of the lymphatic system, including the thoracic duct, and that alpha9 deficiency could be one c
19 ults show lymphatic endothelial cells of the thoracic duct arise from primitive veins through a novel
20 n is transiently expressed in the developing thoracic duct at embryonic day 14, but expression is rap
23 ting intestinal lymph DCs were collected via thoracic duct cannulation from B27-transgenic and contro
27 ing of PKG-Ialpha protein in the wall of rat thoracic duct confirmed its localization inside TD muscl
29 all models used in vivo (3 murine models and thoracic duct development in zebrafish) and in vitro (ly
30 t both Cx37 and Cx43 are required for normal thoracic duct development, including valve formation.
31 igher frequency of LALM (29% vs 9%, P<.001), thoracic duct dilatation (4% vs 0, P=.3), pleural effusi
32 mmon in TSC/LAM, while lymphatic involvement-thoracic duct dilatation, chylous pleural effusion, asci
33 lcified pulmonary nodules, pleural effusion, thoracic duct dilatation, hepatic and renal angiomyolipo
35 re, we report on our initial experience with thoracic duct drainage and autotransfusion in patients w
36 rds of 8 patients who underwent percutaneous thoracic duct drainage with autotransfusion as part of t
40 gulatory mechanism that maintains pumping in thoracic duct in an energy-saving/efficient mode: it imp
41 ed ascites in eight (10%), dilatation of the thoracic duct in seven (9%), and hepatic AML in three (4
43 sults from retrograde flow of chyle from the thoracic duct into lymphatic tributaries with defective
44 Although retrograde flow of chyle from the thoracic duct is considered a potential mechanism underl
45 phatic network in the zebrafish, whereas the thoracic duct is initially dispensable for lymphatic fun
46 Less-invasive surgical procedures such as thoracic duct ligation by video-assisted thoracoscopy ar
50 se agonists produce lymphopenia in blood and thoracic duct lymph by sequestration of lymphocytes in l
51 ed in the intestinal (afferent) and efferent thoracic duct lymph of rats during the course (0 to 289
56 y analyzed the TCR Vbeta repertoires of CD4+ thoracic duct lymphocytes (TDL) collected during the ini
57 positively selected miHA-specific donor CD8+ thoracic duct lymphocytes (TDL) collected from irradiate
58 y almost 100-fold as measured by enumerating thoracic duct lymphocytes (TDL) obtained early post-tran
62 perimental data on migration of 51Cr-labeled thoracic duct lymphocytes (TDLs) via major lymphoid and
63 d functional analysis of positively selected thoracic duct lymphocytes 4 days after transplant and by
67 luorescent tracer revealed that lymph in the thoracic duct of these mice could enter the thoracic cav
68 ntestinal lymph was collected by cannulating thoracic ducts of mesenteric lymphadenectomized animals.
69 agent through the lymphatic system to the TD thoracic duct outlet was 244 seconds (range, 201-387 sec
71 nsible for the self-regulatory adjustment of thoracic duct pumping to changes in lymph flow pattern.
72 ACNA1C were consistently detected from human thoracic duct samples examined and the CaV1.2 protein wa
73 either lymphatic embolization procedures or thoracic duct stenting with covered stents to exclude re
75 : 2 patients (8%) with traumatic leak from a thoracic duct (TD) branch, 14 patients (56%) with pulmon
76 of the lymphatic network with blood from the thoracic duct (TD) in both neonatal and mature mice.
80 acic duct decompression (TDD), rerouting the thoracic duct to the pulmonary venous atrium to treat mu
87 roperitoneal lymphatics, cisterna chyli, and thoracic duct were viewed with an accuracy of 23 of 25 (
89 both the active peak and plateau tensions of thoracic duct, whereas only the active peak tension of c
90 phatic contraction, we hypothesized that the thoracic duct would be more sensitive to the modulation