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1 he remaining 18 spectra were acquired in the thoracic spinal cord.
2 A level increases were highest in lumbar and thoracic spinal cord.
3 the lateral corticospinal tract (CST) in the thoracic spinal cord.
4 G graft in the moderately contused adult rat thoracic spinal cord.
5  period, prior to virus isolation from lower thoracic spinal cord.
6 pathetic preganglionic neurons (SPNs) in the thoracic spinal cord.
7 ation of sympathetic afferents in the caudal thoracic spinal cord.
8 d and in the intermediolateral column of the thoracic spinal cord.
9 ted peripheral nerve graft and back into the thoracic spinal cord.
10 trolateral reticular formation (rvlm) to the thoracic spinal cord.
11  investigated in the lateral horn of the rat thoracic spinal cord.
12 us, raphe magnus, and in the ventral horn of thoracic spinal cord.
13 NTF and transplanted into the contused adult thoracic spinal cord 9 d after injury.
14 ; p = 0.002) in the cervical and NT-3 in the thoracic spinal cord (98%; p = 0.009).
15  the interomedial lateral cell column of the thoracic spinal cord also contained CNTFR alpha-ir neuro
16  This included the descending projections to thoracic spinal cord and a network in brainstem, midbrai
17 ral arcuate nucleus (Arc) that innervate the thoracic spinal cord and also contain cocaine- and amphe
18 t no infectious material in the cervical and thoracic spinal cord and dorsal root ganglia (DRGs) with
19 to preganglionic, sympathetic neurons in the thoracic spinal cord and increase energy expenditure upo
20 urosemide-induced neuronal activation in the thoracic spinal cord and NTS is due to activation of sec
21 PFC that project to autonomic regions of the thoracic spinal cord and probably to other additional su
22 ferent fibers from the heart enter the upper thoracic spinal cord and synapse on cells of origin of a
23 ived identical contusion injuries to the mid-thoracic spinal cord and were allowed to recover for 6 h
24 and the intermediolateral cell column of the thoracic spinal cord, as well as the dorsomedial medulla
25 en transplanted them into the contused adult thoracic spinal cord at 9 d after injury.
26 emarkable for mild cerebellar and noticeable thoracic spinal cord atrophy.
27 xogenous L1, detectable already 1 week after thoracic spinal cord compression and immediate vector in
28 re recorded acutely 25 days following graded thoracic spinal cord compression in rats.
29 estigated whether activated microglia in the thoracic spinal cord contribute to chronic pain in a rat
30  inhibitor, would be neuroprotective after a thoracic spinal cord contusion in adult rats.
31   We used a clinically relevant rat model of thoracic spinal cord contusion injury to examine the eff
32 l medulla (RVL)) and in three regions of the thoracic spinal cord (dorsal horn, intermediate zone and
33 n, GABAergic neurons with projections to the thoracic spinal cord exist in a restricted number of med
34 bs in conjunction with the hindlimbs after a thoracic spinal cord hemisection in adult rats.
35 l administration of NEP1 40 to rats with mid-thoracic spinal cord hemisection results in significant
36 in alcohol-consuming animals in cervical and thoracic spinal cord in late-stage disease.
37 l subregions of the grey matter of the upper thoracic spinal cord in male and female lean and obese-d
38 al activity in the cuneate nucleus and upper thoracic spinal cord in rats.
39 e dorsal horn and intermediate layers of the thoracic spinal cord in the clonidine-atipamezole group
40 ial disynaptic pathways from the rvlm to the thoracic spinal cord in the rat.
41 er, FluoroGold (FG) deposited into the upper thoracic spinal cord in the same animal.
42 ta was collected for the entire cervical and thoracic spinal cord in thirty healthy adult subjects wi
43 locomotor function seen in the rat following thoracic spinal cord injuries that spare as little as 20
44  postural stability and propulsion after low thoracic spinal cord injury (SCI) in animals and humans.
45      In this study, we combined a unilateral thoracic spinal cord injury (SCI) with a cortical neurop
46 main intact following incomplete, contusive, thoracic spinal cord injury (SCI), suggesting a potentia
47                                              Thoracic spinal cord injury disrupts communication betwe
48                                              Thoracic spinal cord injury disrupts communication betwe
49  either subcortical lesion sites or sites of thoracic spinal cord injury failed to elicit corticospin
50                     Locomotor recovery after thoracic spinal cord injury is enhanced.
51                                              Thoracic spinal cord injury level was confirmed as an in
52 ide-induced activation in the NTS and caudal thoracic spinal cord is abolished by prior bilateral ren
53                                          The thoracic spinal cord is intrinsically susceptible to isc
54 emistry we find that the AT1 receptor in the thoracic spinal cord is located on neurones and ependyma
55 decreased on both sides by 50%, while in the thoracic spinal cord, it was increased by 18% on the con
56 r, BNPI immunoreactivity was absent from the thoracic spinal cord lateral horn.
57    Following spinal transection of the upper thoracic spinal cord, male Sprague-Dawley rats given leg
58 f these participants also underwent cervical-thoracic spinal cord MRI.
59 injection of Fluoro-Gold (FG) into the upper thoracic spinal cord, neurons with PPE mRNA (PPE(+) neur
60                  Complete transection of the thoracic spinal cord of adult rats evoked massive sprout
61                                          The thoracic spinal cord of adult rats was transiently compr
62 sidase (beta-gal) reporter gene into the mid-thoracic spinal cord of adult rats, transgene expression
63 4-hydroxyphenylacetic acid in the lumbar and thoracic spinal cord of ALS patients.
64 corded with spike-triggered averaging in the thoracic spinal cord of anaesthetized, paralysed cats.
65                    Here we show that, in the thoracic spinal cord of naive adult mouse, all myelinate
66 ograde fluorescence tracer injected into the thoracic spinal cord of rats.
67 ed by a fluorescent tracer injected into the thoracic spinal cord of rats.
68 of motor-evoked potentials recorded from the thoracic spinal cord of symptomatic SOD1(G93A) rats show
69                                     When the thoracic spinal cord of the North American opossum is tr
70 T1A, AT1B and AT2 receptors are expressed in thoracic spinal cord of the rat.
71  SCI, we performed contusion injuries in the thoracic spinal cord of transgenic (Tg) mice in which >9
72                       The dorsal horn of the thoracic spinal cords of rats with CP contained activate
73 FO, OVLT, SON and PVN, but not in the caudal thoracic spinal cord or NTS.
74 or cortex of rats with completely transected thoracic spinal cord produces hindlimb postural asymmetr
75 mic sympathetic preganglionic neurons in the thoracic spinal cord produces pupillary dilatation, we p
76 ks following an acute compression of the mid-thoracic spinal cord, rats displayed markedly decreased
77       Although many PPNs in the cervical and thoracic spinal cord receive the synaptic inputs from co
78 l of respiratory-related motoneurones in the thoracic spinal cord remains the subject of some debate.
79 ridging the transected dorsal columns in the thoracic spinal cord resulted in an 81% decrease in XT-1
80 us, rostral/caudal ventrolateral medulla, or thoracic spinal cord (SC).
81                         TOF-SIMS analysis of thoracic spinal cord sections was performed at 5 mum res
82 al half of the last cervical and first three thoracic spinal cord segments.
83                                              Thoracic spinal cord stimulation (SCS) is a promising th
84  in humans, stimulus parameters during upper thoracic spinal cord stimulation were adjusted to provid
85  penetrations and cell injections in the mid-thoracic spinal cord; subsequent effects on local axons
86 ing sympathetic preganglionic neurons in the thoracic spinal cord suggests that this pathway may cont
87 nsport of cholera toxin B from medullary and thoracic spinal cord sympathetic outflow areas showed la
88 infected interneurons were also found in the thoracic spinal cord (T(1)-T(13)).
89                       The terminal ileum and thoracic spinal cord (T(11)) were sampled for evaluating
90 ransported cholera toxin B injected into the thoracic spinal cord (T1) revealed that bulbospinal PPG(
91                 At the lesion epicenter (mid-thoracic spinal cord), the microenvironment created by C
92 itioned on the epidural surface of the lower thoracic spinal cord to activate the expiratory muscles.
93 itioned on the epidural surface of the upper thoracic spinal cord to activate the inspiratory interco
94                      We studied complete mid-thoracic spinal cord transected adult female rats and co
95  approximately 3 months after a complete mid-thoracic spinal cord transection at P5 in non-trained an
96 ed (step-trained 6 min/day) spinal rats (mid-thoracic spinal cord transection at post-natal day 5) at
97 MNs) and Renshaw cells (RCs) is disrupted by thoracic spinal cord transection at postnatal day 5 (P5T
98                                    Acute mid-thoracic spinal cord transection eliminates hindlimb air
99 nd standing can be reacquired after complete thoracic spinal cord transection in adult cats with appr
100 OEC transplants acutely after a complete low-thoracic spinal cord transection in adult rats.
101 nts with LAR-targeting peptides in mice with thoracic spinal cord transection injuries induce signifi
102 tion of axons across and beyond an extensive thoracic spinal cord transection lesion in adult rats.
103                               In response to thoracic spinal cord transection lesions, ephrin-B2 and
104 ding a clinical dose of lithium to rats with thoracic spinal cord transection or contusion injuries i
105 ns of ibuprofen via minipumps to rats with a thoracic spinal cord transection or contusion injury res
106 o secrete GDNF into complete and partial mid-thoracic spinal cord transection sites.
107 either partial midcervical or complete upper thoracic spinal cord transections and examined whether c
108 n 2 MSA cases and in 2 control subjects, the thoracic spinal cord was available for study.
109                         A segment of the mid-thoracic spinal cord was compressed for 60 s with a micr
110  of GABAergic neurons with projection to the thoracic spinal cord was studied by using in situ hybrid
111 al electrical stimulation delivered over the thoracic spinal cord with accelerometers that detected c

 
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