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1 ricular arrhythmias, 5 cardiac deaths, and 5 thromboembolic events).
2 lder age, smoking, node negativity, or prior thromboembolic event.
3 ascular event, and 14,550 had a first venous thromboembolic event.
4 ntation, new heart failure, stroke, or other thromboembolic event.
5 hemopericardium, and 5 patients (0.7%) had a thromboembolic event.
6 to death, first major haemorrhage, and first thromboembolic event.
7 One patient (1.6%) had a thromboembolic event.
8 ardial infarction, stroke, heart failure, or thromboembolic event.
9 deaths, the probable cause of death was the thromboembolic event.
10 s, non-O blood groups explain >30% of venous thromboembolic events.
11 higher incidence of both venous and arterial thromboembolic events.
12 ia who present with atypical vascular and/or thromboembolic events.
13 ted, including all-cause mortality and fatal thromboembolic events.
14 associated complications of arrhythmias and thromboembolic events.
15 bleeding risk without a favorable effect on thromboembolic events.
16 ular atrial fibrillation is a major cause of thromboembolic events.
17 ), thresholds were stable, and there were no thromboembolic events.
18 lation has been associated with some risk of thromboembolic events.
19 cal patients are at risk for fatal and major thromboembolic events.
20 long-term survival and very low incidence of thromboembolic events.
21 and fractures and increased the incidence of thromboembolic events.
22 with respect to the incidence of bleeding or thromboembolic events.
23 rophylaxis do not have an increased risk for thromboembolic events.
24 al relevance in prophylaxis and treatment of thromboembolic events.
25 f statins (or higher dose statins) on venous thromboembolic events.
26 m 0.68 to 0.72, P<0.0001, for a composite of thromboembolic events.
27 tins substantially reduce the risk of venous thromboembolic events.
28 for translation due to its association with thromboembolic events.
29 of lasofoxifene increased the risk of venous thromboembolic events.
30 , and stroke but an increased risk of venous thromboembolic events.
31 8 patients with AF, we observed 676 incident thromboembolic events.
32 proportional reporting of syncope and venous thromboembolic events.
33 dverse event of grade 3 or worse, especially thromboembolic events.
34 discussed as are recommendations to prevent thromboembolic events.
35 ficacy outcome was the composite of death or thromboembolic events.
36 age, incidence of seizures, and incidence of thromboembolic events.
37 osinophils may be required for prevention of thromboembolic events.
38 ined (HR: 0.34 vs. LT; 95% CI: 0.13 to 0.91) thromboembolic events.
39 safety parameters, including no evidence of thromboembolic events.
40 sease, especially by increasing the risk for thromboembolic events.
41 pathogenesis of atherosclerosis, leading to thromboembolic events.
42 at high risk for arrhythmic sudden death and thromboembolic events.
43 ves during pregnancy is essential to prevent thromboembolic events.
44 carotid artery stenting reduces the rate of thromboembolic events.
45 ted therapy showed significant reductions in thromboembolic events (0.27, 0.12-0.59) and death (0.37,
46 anticoagulated patients experienced nonfatal thromboembolic events (1.1%/year), whereas 13 with apica
49 ; adjusted IRR, 2.93; 95% CI, 1.54-5.55) for thromboembolic events; 11.86 (95% CI, 7.81-18.01) vs 5.3
51 d superficial surgical-site infections, 2040 thromboembolic events, 1338 myocardial infarctions, and
52 years; 30 of the patients (20%) developed 32 thromboembolic events (15 arterial and 17 venous events)
54 [2%] patients), and grade 3 or higher venous thromboembolic events (23 [8%] vs 11 [4%] patients) than
56 , were associated with an increase in venous thromboembolic events (3.8 and 2.9 cases vs. 1.4 cases p
57 idence interval [CI]: 0.5% to 1.0%) definite thromboembolic events (31 strokes) within 30 days (media
59 had little impact on the findings for venous thromboembolic events (431 [0.9%] versus 461 [1.0%], OR
60 not significantly reduce the risk of venous thromboembolic events (465 [0.9%] statin versus 521 [1.0
62 rotocol-defined withdrawal criteria (11 [4%] thromboembolic events, 5 [2%] exceeding liver enzyme thr
64 g muscles and/or joints (8/11), vascular and thromboembolic events (6/11), that is, deep vein thrombo
65 r complications (8.6% vs 0.1%; P=0.037), and thromboembolic events (8.6% vs 6.0%; P=0.037) were highe
66 requirements without increasing the risk of thromboembolic events across a wide variety of liver tra
69 dual antiplatelet treatment on bleeding and thromboembolic events after transcatheter aortic-valve i
70 factor Xa inhibitor rivaroxaban can prevent thromboembolic events after transcatheter aortic-valve r
73 site endpoint of HIT-specific complications (thromboembolic events, amputation, skin necrosis) occurr
75 ants and antiplatelet medications to prevent thromboembolic events and access thrombosis in dialysis
77 , patients with paroxysmal AF suffered fewer thromboembolic events and deaths compared with those wit
78 orted in 29 of 113 participants (25.7%) with thromboembolic events and in 11 of 34 participants (32.4
81 toring and self-adjusting therapy have fewer thromboembolic events and lower mortality than those who
82 an increased incidence of cardiovascular and thromboembolic events and more deaths during the initial
83 optical density; had a higher prevalence of thromboembolic events and reached platelet count nadir l
87 lower risk of coronary heart disease, venous thromboembolic event, and cerebrovascular disease than i
89 ents (AEs), such as intracranial hemorrhage, thromboembolic event, and progressive aortic insufficien
91 including severe heart failure, arrhythmias, thromboembolic events, and death, the majority of women
94 ile (adverse events, serious adverse events, thromboembolic events, and deaths) was similar between g
95 as not apparent for emergencies unrelated to thromboembolic events, and did not occur in a control gr
96 nd estrogen-progestin increased the risk for thromboembolic events, and etidronate increased the risk
97 Gastrointestinal side effects, hot flashes, thromboembolic events, and infections vary among drugs.
98 h) and harms, including hypertension, venous thromboembolic events, and ischemic cerebrovascular even
99 were no acute neurological complications, no thromboembolic events, and no bleeding complications.
102 rse events; raloxifene and estrogen increase thromboembolic events; and estrogen causes additional ad
103 rhythmias (aOR: 12.4; 95% CI: 11.0 to 14.0), thromboembolic events (aOR: 2.4; 95% CI: 2.0 to 2.9), pr
104 ma samples obtained from patients with prior thromboembolic events are denser and less susceptible to
106 A substantial number of patients at risk for thromboembolic events are not anticoagulated, and furthe
107 The proportion of patients with arterial thromboembolic events as defined by the Antiplatelet Tri
108 of bleeding and the composite of bleeding or thromboembolic events at 1 year were significantly less
110 ing or thromboembolism, patients with AF had thromboembolic events at higher international normalized
112 vents, but focused on the subset of arterial thromboembolic events (ATEs), comprising CV death, myoca
113 reactions; 2.6 for urticaria; 0.2 for venous thromboembolic events, autoimmune disorders, and Guillai
114 essment must address each patient's risk for thromboembolic events balanced against the risk for peri
115 reful case-by-case analysis of the risks for thromboembolic events balanced by the risks for anticoag
116 morrhage or hydrocephalus upon follow-up CT, thromboembolic events before discharge, and the 3-month
118 imator was used to compare risks of arterial thromboembolic events between cancer and noncancer group
120 im of this study was to compare the risk for thromboembolic events, bleeding, and mortality associate
121 collect prospective multicenter outcomes of thromboembolic events, bleeding, and mortality for patie
122 arboplatin, as were hypertension, infection, thromboembolic events, bleeding, and postoperative compl
123 hospitalization, but no increase in risks of thromboembolic events, bleeding-related hospitalization,
124 d thrombin activity underlies obesity-linked thromboembolic events, but the mechanistic links between
125 1.60 [CI, 1.15 to 2.23]; 2 trials) increase thromboembolic events by 4 to 7 per 1000 women per year;
126 ble electronic devices and increased risk of thromboembolic events, clinical intervention for device-
127 ll and major bleeding and at similar risk of thromboembolic events compared to nonbridged patients.
128 nd raloxifene were associated with increased thromboembolic events compared with placebo; tamoxifen w
129 se statin therapy reduced the risk of venous thromboembolic events compared with standard dose statin
130 eriprocedural risk of complications, such as thromboembolic events, compared to warfarin discontinuat
131 CTDa was associated with higher rates of thromboembolic events, constipation, infection, and neur
133 UC]); incidence of breast cancer, fractures, thromboembolic events, coronary heart disease events, st
135 nced grade 3 to 4 toxicity, and 10 (20%) had thromboembolic events (deep venous thrombosis or pulmona
136 e, myocardial infarction, and other arterial thromboembolic events defined by the Antiplatelet Triali
137 agnosis codes were used to identify arterial thromboembolic events, defined as a composite of myocard
138 utropenia grade 3 or 4: 21% vs 5%, P < .001; thromboembolic events despite aspirin prophylaxis: 23.5%
139 ic events outline a significant reduction in thromboembolic events, driven by a reduction in deep vei
140 f TTP-related death, recurrence of TTP, or a thromboembolic event during the treatment period; and a
141 f TTP-related death, recurrence of TTP, or a thromboembolic event during the trial treatment period;
142 than enoxaparin in the prevention of venous thromboembolic events during a period of immobilization
144 diagnostic strategy, defined as adjudicated thromboembolic events during the 3-month follow-up perio
145 o evaluate the previously reported excess of thromboembolic events during the 30 days after the end o
146 stroke and non-central nervous system (CNS) thromboembolic events early after discontinuation of riv
147 ade 4 adverse event in the placebo group was thromboembolic event (eight [<1%]) and the most common g
148 postmenopausal women and had lower risks of thromboembolic events, endometrial cancer, and cataracts
149 center, PREFER in AF (European Prevention of thromboembolic events-European Registry in Atrial Fibril
152 here show that systemic hypoxia accelerates thromboembolic events, functionally stimulated by the ac
153 for fractures but increased risk for stroke, thromboembolic events, gallbladder disease, and urinary
155 ll as 2-year risk of death, hospitalization, thromboembolic events, heart failure (HF), and AF progre
157 c electronic device implantation, infection, thromboembolic event, hemodialysis, and readmission).
158 bioprostheses have an elevated early risk of thromboembolic events; however, the risks and benefits o
160 nths in patients experiencing a first venous thromboembolic event in the setting of major, transient
163 d Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation (ARISTOTLE)
164 e Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation (ARISTOTLE)
165 e Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation (ARISTOTLE)
166 e Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation (ARISTOTLE)
167 e Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation (ARISTOTLE)
168 e Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation (ARISTOTLE)
169 (Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation [ARISTOTLE]
170 e Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation Trial (ARIS
171 e Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation trial who r
172 (Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation) trial, pat
173 (Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation) trial.
174 (Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation) trial.
176 (Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation; n=18 201)
177 (Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation; n=18 201),
182 b and bevacizumab may contribute to systemic thromboembolic events in patients aged 65 years or older
183 agents, addresses challenges with preventing thromboembolic events in patients at high risk and descr
185 ar-weight heparin aims to reduce the risk of thromboembolic events in patients receiving long-term vi
186 egarding the effectiveness and occurrence of thromboembolic events in patients treated with prothromb
187 e and efficacious in preventing bleeding and thromboembolic events in patients undergoing AF ablation
188 4-h infusion of tranexamic acid on death and thromboembolic events in patients with acute gastrointes
190 E constituted 11.5% of clinically recognized thromboembolic events in patients with atrial fibrillati
191 ent are largely the same as those predicting thromboembolic events in patients with atrial fibrillati
192 erapy is the standard therapy for preventing thromboembolic events in patients with atrial fibrillati
193 Therefore, carvedilol may reduce the risk of thromboembolic events in patients with heart failure, ir
194 rophylactic anticoagulation and treatment of thromboembolic events in patients with hepatic insuffici
195 of thrombus formation leading to significant thromboembolic events in patients with nonvalvular atria
200 ade 5 adverse events, including two arterial thromboembolic events, in the bevacizumab group, and 14
201 fter a median of 17 months, death or a first thromboembolic event (intention-to-treat analysis) had o
202 e modest increases in rates of hypertension, thromboembolic events, intestinal perforation, and neutr
203 levant safety end points, including arterial thromboembolic events, MI, stroke or transient ischemic
204 es, but tamoxifen increased the incidence of thromboembolic events more than raloxifene by 4 cases in
205 ositis (none vs four [10%] vs one [3%]), and thromboembolic event (none vs three [8%] vs two [5%]).
215 ring days 1-720, ten (1.2% per patient year) thromboembolic events occurred in the Fiix-PT group vers
216 e death, one nonfatal stroke, and two venous thromboembolic events occurred in the upadacitinib group
222 and nonsignificant lower risk of stroke and thromboembolic events (odds ratio =0.61, 95% confidence
224 mly assigned patients with an annual risk of thromboembolic events of 5% or more to continued warfari
225 ertension of grade 2 or higher (25% vs. 2%), thromboembolic events of grade 3 or higher (8% vs. 1%),
229 e on treatment efficacy, the impact of fatal thromboembolic events, optimal anticoagulation therapy,
231 d up to the time of intracranial hemorrhage, thromboembolic event, or progressive aortic insufficienc
232 nfections, herpes zoster, malignancy, venous thromboembolic events, or deaths were reported; one seri
234 ct of bariatric surgery on long-term risk of thromboembolic events outline a significant reduction in
236 erebrovascular disease (P<0.001), and venous thromboembolic events (P<0.001) than those reporting doi
237 ase duration; hemoglobin level; frequency of thromboembolic events, palpable splenomegaly, and splene
238 ere were 0.75 major bleeding events and 0.28 thromboembolic events per patient year of follow-up.
239 -hospital mortality or complications (major: thromboembolic event, pneumonia, sepsis, stroke, or myoc
240 (survival free of any nonsurgical bleeding, thromboembolic event, pump thrombosis, or neurological e
244 e frequent clinical complications, including thromboembolic events, seizures, fluctuations in neurolo
245 paroid (where available) reduces the risk of thromboembolic events, some of which may be life-threate
247 had lower risk-adjusted all-cause mortality, thromboembolic events, subsequent depression, alcoholism
248 ial fibrillation (AF) have increased risk of thromboembolic events such as stroke and myocardial infa
249 s finding the optimal equilibrium to prevent thromboembolic events, such as stent thrombosis and thro
252 primary breast cancer but increase risk for thromboembolic events (tamoxifen, raloxifene), endometri
255 termine the incidence of venous and arterial thromboembolic events (TEEs) in patients treated with ci
256 ied a boxed safety warning about the risk of thromboembolic events (TEEs), with TEEs reported in 0.5%
257 14 [10%]), skin rash (one [1%] vs 22 [16%]), thromboembolic events (ten [7%] vs 11 [8%]), lethargy (t
258 e predictive value of stroke risk scores for thromboembolic events (TEs) after catheter ablation of a
259 s with atrial fibrillation (AF), the risk of thromboembolic events (TEs) is variable and is influence
261 nd atrial fibrillation are at higher risk of thromboembolic events than patients with heart failure a
263 Secondary outcomes were the number of major thromboembolic events, the number of days free from coma
266 ially be used to predict the risk of diverse thromboembolic events under physiological and pathologic
267 cer patients were diagnosed with an arterial thromboembolic event vs 413 (0.11%) controls (odds ratio
268 is are associated with lower rates of venous thromboembolic events (VTE), major bleeding, and superfi
269 However, because the excess risk of venous thromboembolic events (VTEs) with cisplatin-based chemot
272 for cardiovascular safety, only one further thromboembolic event was reported (fatal pulmonary embol
273 et Trialists' Collaboration-defined arterial thromboembolic events was 1.9%, 0.9%, 1.1%, 2.1%, and 1.
276 For pulmonary embolism, history of previous thromboembolic events was identified as the only risk fa
277 proportional reporting of syncope and venous thromboembolic events was noted with data mining methods
278 ving oral anticoagulation, bleeding, but not thromboembolic events, was increased in patients with li
279 sient ischemic attack (TIA), or a peripheral thromboembolic event were randomly assigned to undergo c
280 Patient characteristics, warfarin use, and thromboembolic events were ascertained from clinical dat
281 f hospitalizations for bleeding and arterial thromboembolic events were estimated in an intent-to-tre
282 l events, cerebrovascular events, and venous thromboembolic events were estimated to be 19.6%, 21.6%,
285 nosis, the interval 30-day risks of arterial thromboembolic events were higher in cancer patients vs
287 neurysm coiling or balloon-assisted coiling, thromboembolic events were more frequent than were intra
288 events and 33 and 58 and 12 and 28 arterial thromboembolic events were observed during follow-up, re
292 nosis, the 30-day interval risks of arterial thromboembolic events were similar between cancer patien
295 diagnosis and seven of those had documented thromboembolic event while on extracorporeal membrane ox
296 hagic and ischemic stroke, or between venous thromboembolic events with or without pulmonary embolism
298 sulted in a 28% reduction in fatal and major thromboembolic events without a significant increase in
299 risk of venous and arterial fatal and major thromboembolic events without significantly increasing m
300 sociated with significantly fewer deaths and thromboembolic events, without increased risk for a seri