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1  thyroid damage and select the predictors of thyroid dysfunction.
2 nd identifying patterns of severity-specific thyroid dysfunction.
3 ossible association with renal, muscular and thyroid dysfunction.
4 nganese detoxification and manganese-induced thyroid dysfunction.
5 compared to children born to mothers with no thyroid dysfunction.
6 atment of screen-detected, undiagnosed overt thyroid dysfunction.
7  with systemic reactions, renal disease, and thyroid dysfunction.
8  relationships between BDE-209 exposures and thyroid dysfunction.
9 ical entities that encompass mild degrees of thyroid dysfunction.
10 vide a candidate target for the treatment of thyroid dysfunction.
11 ce, and rhythm disturbances that result from thyroid dysfunction.
12 s determined the proportion of patients with thyroid dysfunction.
13 d to have laboratory evidence of subclinical thyroid dysfunction.
14 r than 60 years, and others at high risk for thyroid dysfunction.
15 ease and have few or no signs or symptoms of thyroid dysfunction.
16  22% black), 17% reported medication use for thyroid dysfunction.
17    Vigilance is required to detect and treat thyroid dysfunction.
18  patient's thyroid cells as the cause of the thyroid dysfunction.
19                                 Of interest, thyroid dysfunction (10 cases of hypothyroidism and 1 of
20 95 patients met the criteria for subclinical thyroid dysfunction (127 were hyperthyroid, and 168 were
21 years; range, 18 to 60 years), 36% developed thyroid dysfunction: 19 became hyperthyroid and 14 hypot
22                                              Thyroid dysfunction after exposure to low or moderate do
23 udies that define subgroups of patients with thyroid dysfunction amenable to specific preventive stra
24  examine the association between subclinical thyroid dysfunction and depressive symptoms in all prosp
25 cting on the association between subclinical thyroid dysfunction and depressive symptoms.
26  Data on the association between subclinical thyroid dysfunction and fractures conflict.
27 exist on the association between subclinical thyroid dysfunction and heart failure events.
28 re at increased risk of atezolizumab-induced thyroid dysfunction and lower risk of death in triple ne
29 l disease, suggesting a relationship between thyroid dysfunction and periodontal health.
30 ine Society in late 2007, and a symposium on thyroid dysfunction and pregnancy was hosted by the Amer
31 f necessary, to avoid increasing the risk of thyroid dysfunction and related adverse health effects i
32 onal analysis to determine the prevalence of thyroid dysfunction and risk factors for abnormal thyroi
33                      Additional attention to thyroid dysfunction and screening in this age group is r
34  which unleash immune responses and can have thyroid dysfunction and vitiligo as adverse events.
35  should be aimed at detection of subclinical thyroid dysfunction and whether persons with mildly abno
36 uctive thyroiditis (e.g., amiodarone-induced thyroid dysfunction) and factitious hyperthyroidism.
37 ncluding type 2 diabetes mellitus [T2DM] and thyroid dysfunction), and osteoporosis.
38 st media include hypersensitivity reactions, thyroid dysfunction, and contrast-induced nephropathy.
39 rction, obesity, smoking, diabetes mellitus, thyroid dysfunction, and endurance exercise training all
40 thyroiditis, iodine-induced and drug-induced thyroid dysfunction, and factitious ingestion of excess
41 terial hypertension, osteoarthritis, asthma, thyroid dysfunction, and large organ fibrosis.
42 e of shared genetic architecture for MCV and thyroid dysfunction, and mineral metabolism disorders.
43 with the traditional notion that subclinical thyroid dysfunction, and subclinical hypothyroidism in p
44 rmal thyroid physiology, the consequences of thyroid dysfunction, and the effects of therapy in 3 car
45  of cancer and its treatment such as anemia, thyroid dysfunction, and the neurotoxicity of cancer che
46        Inexpensive blood tests for diabetes, thyroid dysfunction, and vitamin B12 deficiency allowed
47            Although assigning a diagnosis of thyroid dysfunction appears quite simple, this is often
48                   Although clinical signs of thyroid dysfunction are usually absent in affected males
49 s that secrete thyrotropin, and drug-induced thyroid dysfunction, are also important causes.
50  lines seemed to successfully compensate for thyroid dysfunction, as serum thyroxine/triiodothyronine
51 d must be differentiated from other forms of thyroid dysfunction, as treatment differs.
52 lly relevant association between subclinical thyroid dysfunction at baseline and depressive symptoms
53 nvestigating exacerbating conditions such as thyroid dysfunction, but without specifying the impact o
54 has a critical role in fetal development and thyroid dysfunction can adversely affect obstetric outco
55                                              Thyroid dysfunction can adversely affect somatic growth
56                                              Thyroid dysfunction commonly occurs with amiodarone ther
57                                              Thyroid dysfunction continued for 28 years after transpl
58 and uterine epithelial morphogenesis and how thyroid dysfunction could lead to female infertility.
59                                  Subclinical thyroid dysfunction, defined as thyroid-stimulating horm
60 , including weight gain, insulin resistance, thyroid dysfunction, endothelial dysfunction, and oxidat
61 e (TSH) has increased identification of mild thyroid dysfunction, especially in elderly patients.
62 endelian randomization to assess the role of thyroid dysfunction for cardiovascular disease (CVD).
63                               After dividing thyroid dysfunction groups into five subgroups, interest
64 notyped in patients with GDM and gestational thyroid dysfunction (GTD), a pathology commonly found in
65                                     Although thyroid dysfunction has been linked to heart failure, it
66 ted in opioid users, but clinically relevant thyroid dysfunction has not been identified.
67 raumatic brain injury, rheumatoid arthritis, thyroid dysfunction, hearing impairment, and handgrip st
68       Finally, data clarifies the effects of thyroid dysfunction, hyperparathyroidism, acromegaly and
69 amine the frequency of amiodarone-associated thyroid dysfunction in adults with congenital heart dise
70     Our goal was to assess the prevalence of thyroid dysfunction in association with 131I exposure du
71 lance of benefits and harms of screening for thyroid dysfunction in nonpregnant, asymptomatic adults.
72                            The prevalence of thyroid dysfunction in older adults is nearly 25%.
73 ation between clinicopathologic features and thyroid dysfunction in patients with NS.
74 ts the importance of determining the role of thyroid dysfunction in the onset and progression of mang
75 nditions including diabetes, kidney disease, thyroid dysfunction, iron deficiency and myeloproliferat
76                                              Thyroid dysfunction is a global health concern, causing
77                                              Thyroid dysfunction is a known complication after hemato
78                                  Subclinical thyroid dysfunction is a risk factor for developing symp
79            We show that atezolizumab-induced thyroid dysfunction is associated with longer survival.
80                                              Thyroid dysfunction is associated with poor prognosis in
81 tion of multiple physiological functions and thyroid dysfunction is associated with substantial morbi
82                        Amiodarone-associated thyroid dysfunction is common in adults with congenital
83        Even though treatment for subclinical thyroid dysfunction is controversial, office-based scree
84 ients found by screening to have subclinical thyroid dysfunction is inconclusive.
85 of the efficacy of treatment for subclinical thyroid dysfunction is inconclusive.
86                                              Thyroid dysfunction is more common in adult patients wit
87                                              Thyroid dysfunction is suggested as a potential determin
88 and evidence informing treatment targets for thyroid dysfunction largely come from studies of middle-
89 sial, office-based screening to detect overt thyroid dysfunction may be indicated in women older than
90  P2 =.0049; vitiligo, P2 < 10(-6)), although thyroid dysfunction may have been related more to the le
91 , and amiodarone >200 mg/d a risk factor for thyroid dysfunction (odds ratio, 4.0; P=0.60).
92 were significant risk factors for developing thyroid dysfunction (odds ratios, 3.0 and 7.00; P=0.04 a
93 fect of treating screen-detected subclinical thyroid dysfunction on health outcomes.
94 ases such as diabetes nephrotic syndrome and thyroid dysfunction on the metabolism of apoB-containing
95 -infectious pneumonitis, had no uncontrolled thyroid dysfunction or diabetes, had no active brain met
96                                              Thyroid dysfunction, osteoarthritis, diabetes, irritable
97 iated with opium consumption (P = 0.046) and thyroid dysfunction (P = 0.037).
98                                              Thyroid dysfunction rates are increased in patients with
99 nadequate to determine whether screening for thyroid dysfunction reduced cardiovascular disease or re
100                         The NS patients with thyroid dysfunction showed higher urine protein, creatin
101 was different among NS patients in different thyroid dysfunction subgroups.
102 s indicate a direct role of IFN-gamma in the thyroid dysfunction that occurs in autoimmune thyroiditi
103  conditions represent the earliest stages of thyroid dysfunction, the benefits of detecting and treat
104      The prevalence of overt and subclinical thyroid dysfunction, the evidence for the efficacy of tr
105  more common in younger patients, those with thyroid dysfunction, those not seen in person, and in th
106 gation into the fundamental biology relating thyroid dysfunction to the development of cardiovascular
107 ties for future scientific research relating thyroid dysfunction to the progression of cardiovascular
108 us children born to mothers with no recorded thyroid dysfunction using Poisson regression models.
109 apsulated hESC-derived progenitor cells, and thyroid dysfunction was maintained for the duration of t
110            Multivariate analysis showed that thyroid dysfunction was more likely if patients were les
111  medication were controlled for, subclinical thyroid dysfunction was not associated with depression,
112 he local production of IFN-gamma can lead to thyroid dysfunction, we have generated transgenic mice t
113 progression, and consequences of subclinical thyroid dysfunction were also reviewed.
114 ets were independent risk factors predicting thyroid dysfunction, while higher albumin and hemoglobin
115 rms of screening for subclinical and "overt" thyroid dysfunction without clinically obvious symptoms,

 
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