ライフサイエンスコーパス: thyroid hormone

1 increases glucose utilization in response to thyroid hormone.

2 with the TR, in Pax8-KO mice, which make no thyroid hormone.

3 thyroid follicular cells and biosynthesis of thyroid hormone.

4 Iodine is an essential component of thyroid hormone.

5 the fetus is entirely dependent on maternal thyroid hormone.

6 es 3,5,3'-triiodothyronine (T3 ), the active thyroid hormone.

7 m lipid) basis, were assessed in relation to thyroid hormones.

8 understand the production and regulation of thyroid hormones.

9 omycin administration is also reduced by the thyroid hormones.

10 nd positively associated with free and total thyroid hormones.

11 d by excessive production and release of the thyroid hormones.

12 function, and factitious ingestion of excess thyroid hormones.

13 abalone, accompanied by reduced synthesis of thyroid hormones.

14 thyroid function and can alter the levels of thyroid hormones.

15 to the identification of novel biomarkers of thyroid hormone action in cardiovascular tissues; (2) st

16 e supplementation, we examined the timing of thyroid hormone action on specific brain systems.

17 tered thyroid status and mutations affecting thyroid hormone action, suggesting that these critical p

18 (D2-D3) provides cell-specific regulation of thyroid hormone activity.

19 ced by carbon tetrachloride is attenuated by thyroid hormone administration to mice, whereas aged TR

20 not demonstrate an associated disruption of thyroid hormone, although this association may have been

21 Initial screening revealed 1a (KB2115), a thyroid hormone analog, as a lead compound with low micr

22 Thyroid hormone and antibody levels were assessed.

23 e shortest larval period, highest whole-body thyroid hormone and corticosterone content, and highest

24 ([Formula: see text]), lower maternal serum thyroid hormone and lipid profiles ([Formula: see text])

25 ses during metamorphosis point to a role for thyroid hormone and retinoic acid signaling, as well as

26 om the p160 transcriptional co-activator for thyroid hormone and retinoid receptors (ACTR) and the nu

27 onitored binding experiments of activator of thyroid hormone and retinoid receptors and nuclear coact

28 ordered interaction domain from activator of thyroid hormone and retinoid receptors.

29 Serum thyroid hormones and anti-thyroid antibodies and urinary

30 in adult mammals is triggered by increasing thyroid hormones and may be a trade-off for the acquisit

31 Thyroid hormones and moderate exposure to perchlorate du

32 docrine diseases, including those related to thyroid hormones and obesity.

33 Soy supplementation has no effect on the thyroid hormones and only very modestly raises TSH level

34 Previous studies on the association between thyroid hormones and prognosis of acute ischemic stroke

35 While a number of reports concern thyroid hormones and smoltification, few and inconclusiv

36 Proton pump inhibitors, thyroid hormones, and dihydropyridine derivatives were f

37 Serum estradiol, thyroid hormones, and urinary free cortisol levels were

38 Thyroid hormones are also important regulators of fetal

39 Thyroid hormones are critical for mammalian brain develo

40 Maternal thyroid hormones are essential for fetal brain developme

41 KEY POINTS: Thyroid hormones are important regulators of growth and

42 Thyroid hormones are known regulators of adult metabolis

43 lipin expression or FAO stimulation; rather, thyroid hormones are likely to negatively regulate both

44 This may bias our view of how thyroid hormones are produced in other organisms.

45 Teleost thyroid follicles produce the same thyroid hormones as in other vertebrates: thyroxin (T4)

46 onclude, our data define a critical role for thyroid hormones as potent alphavbeta3-ligands, driving

47 ply that other potential target sites in the thyroid hormone axis should be a greater priority for bi

48 cts such as hypertriglyceridemia and altered thyroid hormone axis.

49 Thyroid hormones binding to alphavbeta3 integrin produce

50 nd plays a crucial role in processes such as thyroid hormone biosynthesis and innate host defense.

51 LIS3 as a key regulator of TSH/TSHR-mediated thyroid hormone biosynthesis and proliferation of thyroi

52 nal activation of several genes required for thyroid hormone biosynthesis, including the iodide trans

53 ) transport in the thyroid-the first step in thyroid hormone biosynthesis-with a 2 Na(+): 1 I(-) stoi

54 genes that are involved in various steps of thyroid hormone biosynthesis.

55 equired for iodide uptake, which facilitates thyroid hormone biosynthesis.

56 We found that increased intracellular thyroid hormone concentration through D3 depletion induc

57 ations between urinary perchlorate and serum thyroid hormone concentrations in models adjusted for ur

58 Thyroid hormone concentrations in the blood are stable,

59 been masked by impacts of prey abundance on thyroid hormone concentrations.

60 Thyroid hormone deficiency causes delayed craniofacial a

61 from animal and human studies indicates that thyroid hormone deficiency during early gestation alters

62 together, these data suggest that long-term thyroid hormone deficiency may drive the differentiation

63 Fetal thyroid hormone deficiency reduced oxidative phosphoryla

64 Hypothyroidism is a common condition of thyroid hormone deficiency, which is readily diagnosed a

65 -tetrabromoethylcyclohexane (beta-TBECH), on thyroid hormone deiodinase (DIO) and sulfotransferase (S

66 cts on the fetal PT to control expression of thyroid hormone deiodinases in ependymal cells (tanycyte

67 us nonthyroid tissues was-in part-induced by thyroid (hormone)-dependent signaling.

68 brain-based markers or measurable metrics of thyroid hormone-dependent perturbations in brain develop

69 Associated disruption of thyroid hormone-dependent protein synthesis in the hippo

70 etraiodothyroacetic acid (tetrac) and DAT as thyroid hormone derivatives influence gene expression af

71 rian cancer and we hypothesized that natural thyroid hormone derivatives may antagonize these actions

72 To conclude, the cytotoxic potential of thyroid hormone derivatives, tetrac, triac and T1AM, in

73 ecules besides trace amines (TAs), including thyroid hormone-derivatives like 3-iodothyronamine (T1AM

74 Thyroid hormones did not differ among the four groups, w

75 higher in anadromous salmon, whereas plasma thyroid hormones did not differ.

76 rapid ossification and hypertrophy; second, thyroid hormone directly affects hypochord formation and

77 One possible molecular target of thyroid hormone disrupting chemicals (THDCs) is transthy

78 and the hazard and risk assessment of these thyroid hormone disrupting chemicals.

79 esses in adult and developing organisms, and thyroid hormone disruption is of high concern for neurod

80 a tiered screening and testing approach for thyroid hormone disruption, using the levels of assessme

81 and the potential clinical impact regarding thyroid hormones disruptions in early pregnancy is neede

82 d the potential windows of susceptibility to thyroid hormone disturbances related to study visit of s

83 ice that were developmentally overexposed to thyroid hormone due to a Dio3 mutation.

84 t of infant visual attention is sensitive to thyroid hormone during the early prenatal period, when t

85 gs of this study highlight the importance of thyroid hormones during pregnancy for normal development

86 Small changes in thyroid hormones during pregnancy, including changes wit

87 Thyroid hormone excess, by contrast, accelerates develop

88 yroid gland provides insufficient amounts of thyroid hormone for the needs of peripheral tissues.

89 Water consumption, thyroid hormone function, behavioral responses, and skul

90 RATIONALE: Thyroid hormones have been linked with various proathero

91 Thyroid hormones have long been known to have a range of

92 thetic mammalian gene circuit that maintains thyroid hormone homeostasis by monitoring thyroid hormon

93 The physiological control of thyroid hormone homeostasis by the feedback loops involv

94 transport and intracellular pH regulation to thyroid hormone homeostasis.

95 ence that administration of supraphysiologic thyroid hormone improves depressive symptoms in patients

96 one receptors are required for the action of thyroid hormone in fetal cardiomyocytes.

97 d perchlorate exposure and the importance of thyroid hormone in fetal neurodevelopment.

98 n of type 2 deiodinase (D2), which activates thyroid hormone in skeletal muscle is upregulated by acu

99 ion is also required for the biosynthesis of thyroid hormone in vertebrates, and there is evidence fo

100 a-analysis to assess the prognostic value of thyroid hormones in AIS.

101 late exposure to alter circulating levels of thyroid hormones in pregnant women.

102 ve been suggested to interfere with maternal thyroid hormones in the second or third trimesters, but

103 he perinatal period and examined the role of thyroid hormones in these processes.

104 metric pigmentation and, via cross-talk with thyroid hormones, in modulating eye migration.

105 The thyroid hormone-inactivating (TH-inactivating) enzyme ty

106 ed the hippocampal expression changes in the thyroid hormone-inactivating enzyme, deiodinase-III (Dio

107 A-seq analysis of gene expression suggests a thyroid hormone-independent endocrine signaling pathway

108 nscriptomic responses included alteration of thyroid hormone induced bZip protein (thibz), deiodinase

109 Thus, developmental levels of thyroid hormone influence the epigenetic information of

110 Conversely, exogenous thyroid hormones inhibit zebrafish heart regeneration.

111 solated fetal sheep islets studied in vitro, thyroid hormones inhibited beta cell proliferation in a

112 ncreatic beta cell is therefore sensitive to thyroid hormone, insulin and leptin before birth, with p

113 kers confirmed the requirement for an intact thyroid hormone-integrin interaction in ERK activation.

114 the clinical syndrome of excess circulating thyroid hormones, irrespective of the source.

115 Thyroid hormone is a pleiotropic factor that controls ma

116 Thyroid hormone is critical for normal brain development

117 integrin, a plasma membrane receptor, binds thyroid hormones (L-thyroxine, T4; 3,5,3'-triiodo-L-thyr

118 ve data collection, and measurement of serum thyroid hormone levels (which were not possible in this

119 ns thyroid hormone homeostasis by monitoring thyroid hormone levels and coordinating the expression o

120 , this synthetic circuit sensed pathological thyroid hormone levels and restored the thyrotrophic fee

121 exposure has been associated with decreased thyroid hormone levels in animals, but human studies are

122 PFHxS lowered thyroid hormone levels in both dams and offspring in a d

123 ed the degree to which phthalates may affect thyroid hormone levels in particularly susceptible popul

124 atistical evidence supporting the utility of thyroid hormone levels in prognosis of acute stroke.

125 of urinary phthalate metabolites and plasma thyroid hormone levels in samples collected at up to fou

126 ssociation between maternal TCS exposure and thyroid hormone levels of mothers and newborns.

127 , internal chemical dosimetry, and placental thyroid hormone levels were determined.

128 ome or standardized mean difference (SMD) of thyroid hormone levels with 95% confidence intervals (95

129 neral term for excess circulating and tissue thyroid hormone levels, whereas hyperthyroidism specific

130 ns of PBBs and PCBs with thyroid disease and thyroid hormone levels.

131 elationship between maternal urinary TCS and thyroid hormone levels.

132 anges in food intake, physical activity, and thyroid hormone levels.

133 and thyroid function via the measurement of thyroid hormone levels.

134 generation inhibitors and have no detectable thyroid hormone-like activity.

135 In skeletal muscle, physical exercise and thyroid hormone mediate the peroxisome proliferator-acti

136 No evidence of thyroid hormone-mediated neurobehavioral disruption in o

137 d biosynthesis, cholesterol biosynthesis and thyroid hormone metabolic processes.

138 Therefore, we investigated the role of thyroid hormone metabolism and signaling in colorectal C

139 or roles in cellular antioxidant defense and thyroid hormone metabolism.

140 Thyroid hormones modulate not only multiple functions in

141 suggesting comparable epigenetic effects of thyroid hormone on both the male and female ancestral ge

142 in view of the confounding effect of excess thyroid hormone on immune responses, spontaneously arisi

143 rdiovascular outcomes through treatment with thyroid hormone or thyromimetic drugs.

144 f maternal urinary phthalate metabolites and thyroid hormone parameters during pregnancy.

145 potential effects in estrogen, androgen, and thyroid hormone pathways, and it is one of the only regu

146 l hydrocarbon receptor, stress-response, and thyroid hormone pathways.

147 Thyroid hormones play a key role in modulating myocardia

148 ite their obvious biological importance, the thyroid hormone precursor protein, thyroglobulin (Tg), h

149 vironmental perchlorate exposures may affect thyroid hormone production during pregnancy.

150 iodide uptake into the thyroid and decrease thyroid hormone production.

151 the present study tested the hypotheses that thyroid hormones promote beta cell proliferation in the

152 Thyroid hormone promotes expression of peroxisome prolif

153 ning silencing mediator of retinoic acid and thyroid hormone receptor (SMRT) and nuclear receptor cor

154 and silencing mediator of retinoic acid and thyroid hormone receptor (SMRT) corepressors and is larg

155 restingly, EBI was found to be a very potent thyroid hormone receptor (THR) agonist, while NH-3 is an

156 rofile in maternal serum for activity at the thyroid hormone receptor (THR) and ryanodine receptor (R

157 t mediates ligand-independent actions of the thyroid hormone receptor (TR) during development and in

158 o describe transcriptional regulation by the thyroid hormone receptor (TR).

159 Loss of thyroid hormone receptor (TR)alpha1 abolishes T(3) signa

160 We found that the thyroid hormone receptor (TRalpha 3) has a differential

161 ses of pomc promoter sequences revealed that thyroid hormone receptor 1beta-binding motif insertions

162 ASO-T3 (NAT3) and ApoB-ASO-T3 (AAT3) enhance thyroid hormone receptor activity.

163 yroid hormone triiodothyronine and synthetic thyroid hormone receptor agonists, such as sobetirome (G

164 Thyroid hormone receptor alpha (THRA) gene mutations, vi

165 Mutations in the thyroid hormone receptor alpha 1 gene (THRA) have recent

166 These results reveal that thyroid hormone receptor alpha1 is required for normal n

167 In the retina, thyroid hormone receptor beta (thrb) is required for exp

168 a (PPARG), glucocorticoid receptor (GR), and thyroid hormone receptor beta (THRB), when exposed to 14

169 ing in KO mice, likely a result of decreased thyroid hormone receptor beta expression without Mdr2.

170 (RTH) disorders, due to mutations in either thyroid hormone receptor beta or alpha (beta: female n =

171 ated that PI3K/Akt signaling is important in thyroid hormone receptor beta(PV/PV) knock-in (PV) mice

172 X6 (Sex Determining Region Y-Box 6) and cTR (Thyroid hormone receptor beta).

173 binding of the transcription factor THRbeta (thyroid hormone receptor beta).

174 is suboptimal because of lower expression of thyroid hormone receptor beta.

175 Down-regulation of the thyroid hormone receptor beta1 (TRbeta) appears to be as

176 mponent of the cone precursor circuitry, the thyroid hormone receptor beta2 (TRbeta2), enables the ab

177 methyltransferase Dot1L is a coactivator for thyroid hormone receptor during Xenopus development.

178 CIP4 (Cdc42-interacting protein 4)/TRIP10 (thyroid hormone receptor interactor 10) was identified a

179 We conclude that TRbeta2-46 is an oncogenic thyroid hormone receptor isoform that promotes SKP2 expr

180 Inhibition of thyroid hormone receptor locally in the retina is a ther

181 ia SMRT (silencing mediator for retinoid and thyroid hormone receptor).

182 rotein, vitellogenin, estrogen receptor, and thyroid hormone receptor, demonstrated that blood is a u

183 We show that the RNA-binding protein THRAP3 (thyroid hormone receptor-associated protein 3) regulates

184 s a liver-directed, orally active, selective thyroid hormone receptor-beta agonist designed to improv

185 Thyroid hormone receptors (TRs) are critical endocrine r

186 these T(3) -induced changes are mediated via thyroid hormone receptors (TRs) or by non-genomic mechan

187 processes through modulation of the nuclear thyroid hormone receptors and several other proteins.

188 findings define an important function of the thyroid hormone receptors and suggest TR ligands could h

189 Thyroid hormone receptors are required for the action of

190 otein and silencing mediator of retinoid and thyroid hormone receptors to a newly identified putative

191 effect is mediated via the genomic action of thyroid hormone receptors, with little evidence for non-

192 CoR1) and silencing mediator for retinoid or thyroid-hormone receptors (SMRT) are the best characteri

193 1 (NCoR1)/silencing mediator for retinoid or thyroid-hormone receptors (SMRT) corepressors in skin ke

194 1 (NCoR1)/silencing mediator for retinoid or thyroid-hormone receptors (SMRT) corepressors or histone

195 Thyroid hormones regulate essential biological functions

196 ptor (TSHR) on the thyroid gland, triggering thyroid hormone release.

197 reated, resulting in a dramatic reduction in thyroid hormone replacement dosage, and 2) the identific

198 Thyroid hormone replacement has been used for more than

199 The standard treatment is thyroid hormone replacement therapy with levothyroxine.

200 a status that is readily treatable with oral thyroid hormone replacement therapy.

201 s applied to participants with resistance to thyroid hormone (RTH) disorders, due to mutations in eit

202 R) that act as agonists and induce excessive thyroid hormone secretion, releasing the thyroid gland f

203 ell decisions in the retinal tissue based on thyroid hormone signaling activity.

204 Thus, chemicals that can affect thyroid hormone signaling during pregnancy are of great

205 Specifically, blockade of thyroid hormone signaling may potentiate heart regenerat

206 Thyroid hormone signaling plays a critical role in activ

207 Inactivation of thyroid hormone signaling reduces mouse cardiomyocyte po

208 metabolic mechanism by which CR-CSCs exploit thyroid hormone signaling to facilitate their self-renew

209 autoregulatory feedback loop that modulates thyroid hormone signaling.

210 key elements of the response to photoperiod, thyroid hormone signalling components were assessed in t

211 deiodinase DIO2, a critical modulator of the thyroid hormone signalling pathway.

212 d as a component of an enzyme that activates thyroid hormone; since this discovery, the relevance of

213 Deregulation of deiodinase function and thyroid hormone status has been implicated in tumorigene

214 luate the potential contribution of aberrant thyroid hormone status to the epigenetic inheritance of

215 yperthyroidism is characterised by increased thyroid hormone synthesis and secretion from the thyroid

216 Because thyroid hormone synthesis is affected by iodine deficien

217 Thyroid hormone synthesis is stimulated by TSH activatin

218 an thyroglobulin) that can lead to defective thyroid hormone synthesis, resulting in congenital hypot

219 pollutants are known to adversely affect the thyroid hormone system, and major gaps have been identif

220 In humans, the thyroid hormones T(3) and T(4) are synthesized in the th

221 ice over a 10 wk period or treated them with thyroid hormone (T(3)) for 5 wk.

222 fepristone, the HPT axis-based treatments of thyroid hormones (T(3) and T(4)), and the HPG axis-based

223 esis of antisense-oligonucleotides (ASO) and thyroid hormone T3 conjugates for obesity treatment.

224 tive peptide that combines glucagon with the thyroid hormone T3 lowers lipid levels, improves glucose

225 However, the thyroid hormone T3 up-regulated mitoIK, ATP, conferring

226 Here we show that exogenous thyroid hormone (T3) administration increases cardiomyoc

227 Thyroid hormone (T3) affects development and metabolism

228 Glucagon and thyroid hormone (T3) exhibit therapeutic potential for m

229 with both processes taking place when plasma thyroid hormone (T3) levels are high.

230 We have previously shown that exogenous thyroid hormone (T3) stimulates cardiomyocyte proliferat

231 We have been studying thyroid hormone (T3)-dependent amphibian metamorphosis i

232 Here we report that a thyroid hormone (T3)-free window, with or without a demy

233 and rT3 separately) and measuring changes in thyroid hormone (T4, T3, rT3, and 3,3'-T2) concentration

234 ctive human liver subcellular fractions with thyroid hormones (T4 and rT3 separately) and measuring c

235 ermore, we demonstrate that BMP4 is a direct thyroid hormone target and is involved in a positive aut

236 sm, a metabolic disease characterized by low thyroid hormone (TH) and high thyroid-stimulating hormon

237 Thyroid hormone (TH) and retinoic acid (RA) within the t

238 Thyroid hormone (TH) and TH receptors (TRs) alpha and be

239 mediated I(-) uptake plays a pivotal role in thyroid hormone (TH) biosynthesis.

240 cumulation in the thyroid, the first step in thyroid hormone (TH) biosynthesis.

241 e AOPs help to establish links between these thyroid hormone (TH) disrupting molecular events and adv

242 of a heterotopia in a rat model, induced by thyroid hormone (TH) disruption during gestation.

243 ltiple animal studies have shown PBDEs to be thyroid hormone (TH) disruptors.

244 3) is considered to be the primary bioactive thyroid hormone (TH) due to its high affinity for TH nuc

245 Interestingly, both FOXO1 and thyroid hormone (TH) have similar effects on carbohydrat

246 Forkhead box O (FoxO) proteins and thyroid hormone (TH) have well established roles in card

247 Thyroid hormone (TH) is a critical regulator of perinata

248 Thyroid hormone (TH) is critical for the maintenance of

249 An effect of thyroid hormone (TH) on erythropoiesis has been known fo

250 Thyroid hormone (TH) regulates diverse developmental eve

251 Thyroid hormone (TH) regulates many cellular events unde

252 effects can occur in tissues that depend on thyroid hormone (TH) regulation for normal physiologic f

253 ing postnatal day 7-10, when serum levels of thyroid hormone (TH) rise.

254 Their DNT effects are suspected to involve thyroid hormone (TH) signaling disruption.

255 Recent studies have implicated thyroid hormone (TH) signaling in cone photoreceptor via

256 Thyroid hormone (TH) signaling plays an important role i

257 Thyroid hormone (TH) signaling promotes tissue maturatio

258 Thyroid hormone (TH) signaling regulates cell proliferat

259 nts the potential for chemical disruption of thyroid hormone (TH) signaling through multiple molecula

260 ganic contaminants that can compete with the thyroid hormone (TH) thyroxine (T4) for binding to trans

261 Inactivating mutations in the thyroid hormone (TH) transporter Monocarboxylate transpo

262 that control the concentration of the active thyroid hormone (TH) triiodothyronine through regioselec

263 al week, a period physiologically similar to thyroid hormone (TH)-regulated metamorphosis in anuran a

264 ogical treatments to highlight the role that thyroid hormones (TH) play in sensory development and de

265 gh the production and subsequent function of thyroid hormones (TH).

266 Here, we investigate whether thyroid-hormones (TH) and their receptors (TR) coordinat

267 Calcitonin is a 32-amino acid thyroid hormone that can form amyloid fibrils.

268 ate (SMR), and elevate whole-body content of thyroid hormone (the primary morphogen controlling metam

269 Thyroid hormones (THs) are essential for establishing la

270 ley syndrome patients.SIGNIFICANCE STATEMENT Thyroid hormones (THs) are essential to establish the st

271 efore studied whether maternally transferred thyroid hormones (THs) exert context-dependent effects o

272 Thyroid hormones (THs) operate numerous physiological pr

273 Since it is unknown whether thyroid hormones (THs) regulate mitochondrial function i

274 r, the association of low-exposure POPs with thyroid hormones (THs) remains unclear.

275 of the pituitary hormone thyrotropin and the thyroid hormones thyroxine and triiodothyronine) are som

276 of thyroid function, including those for the thyroid hormones thyroxine and triiodothyronine, are amo

277 a stress hormone acting synergistically with thyroid hormone to accelerate metamorphosis).

278 rocarbon receptor activity or binding to the thyroid hormone transport protein was found.

279 chemicals (THDCs) is transthyretin (TTR), a thyroid hormone transporter in vertebrates.

280 Deficiency of the thyroid hormone transporter monocarboxylate transporter

281 Since then, a subgroup of thyroid hormone-treated patients with residual symptoms

282 iagnosed before delivery who did not receive thyroid hormone treatment during pregnancy (IRR=1.37, 95

283 evaluated whether the risk was moderated by thyroid hormone treatment during pregnancy.

284 Thyroid hormone treatment in juveniles enhanced NR1 gene

285 Plasma thyroid hormone (tri-iodo-l-thyronine; T(3) ) concentrat

286 Here, we show that the thyroid hormone triiodothyronine (T3), through binding t

287 Thyroid hormone triiodothyronine and synthetic thyroid h

288 ns blood plasma levels of estradiol (E2) and thyroid hormones (TSH, T3t, T4t) were also determined.

289 Levothyroxine (LT4) is a form of thyroid hormone used to treat hypothyroidism.

290 ions in concentrations of corticosterone and thyroid hormones was observed.

291 Thyroid hormones were measured in a subset without thyro

292 yroglobulin (TG) is the protein precursor of thyroid hormones, which are essential for growth, develo

293 ver the perinatal period and is dependent on thyroid hormones, with potential consequences for neonat

294 ds birth and is dependent on the presence of thyroid hormones, with potential implications for the he

295 ablation with rhTSH and those prepared with thyroid hormone withdrawal (3 years, 1.5% vs 2.1%; 5 yea

296 terval between surgery and RITh was 18 d for thyroid hormone withdrawal and 25 d for rhTSH (P < 0.01)

297 A higher radiation exposure using thyroid hormone withdrawal for several weeks compared wi

298 d thyroid carcinoma is performed either with thyroid hormone withdrawal or with administration of rec

299 Subsequently, after 4-6 wk of thyroid hormone withdrawal patients were treated with 5.

300 human thyroid-stimulating hormone (rhTSH) or thyroid hormone withdrawal.