ライフサイエンスコーパス: thyroid peroxidase

1 ine receptor subtypes 3, 4, and 5 as well as thyroid peroxidase.

2 offspring developed Abs to thyroglobulin and thyroid-peroxidase.

3 ific targets including gastric ATPase (11%), thyroid peroxidase (14%), and anti-IgA autoantibodies ag

4 panels of single substitution analogs of the thyroid peroxidase 632-645Y epitope.

5 ion ratios and high selectivity for MPO over thyroid peroxidase and cytochrome P450 isoforms.

6 ntibodies against autoantigens, for example, thyroid peroxidase and IL-24.

7 iiodothyronine (fT3, TT3), and autoimmunity [thyroid peroxidase and thyroglobulin antibodies (TPOAb a

8 sed during its differentiation (for example, thyroid peroxidase and thyroglobulin genes).

9 ability of recombinant antigen [particularly thyroid peroxidase and thyrotropin (TSH) receptor] and o

10 , increased levels of autoantibodies against thyroid peroxidase, and also exhibited the strongest dis

11 imal overlap among anti-gastric ATPase, anti-thyroid peroxidase, and anti-transglutaminase seropositi

12 bulin, thyroid-stimulating hormone receptor, thyroid peroxidase, and sodium iodide transporter.

13 ransglutaminase antibodies (AGTAs), and anti-thyroid peroxidase (anti-TPO) antibodies were measured.

14 ontal inflamed surface area (PISA) with anti-thyroid peroxidase (anti-TPO) antibody, triiodothyronine

15 ed for anti-thyroglobulin (anti-TG) and anti-thyroid peroxidase (anti-TPO) as models.

16 ve-birth rates among euthyroid women who had thyroid peroxidase antibodies and a history of miscarria

17 in the United Kingdom underwent testing for thyroid peroxidase antibodies and thyroid function.

18 Thyroid peroxidase antibodies are associated with an inc

19 use of levothyroxine in euthyroid women with thyroid peroxidase antibodies did not result in a higher

20 Thyroid peroxidase antibodies were elevated in 24 of 62

21 unchanged when women with elevated levels of thyroid peroxidase antibodies were excluded from the ana

22 hormone levels (thyrotropin, free thyroxine, thyroid peroxidase antibodies) were measured at a mean (

23 hyroxine (T4)), thyroid-stimulating hormone, thyroid peroxidase antibodies, iodine, selenium, and mer

24 In patients who have circulating thyroid peroxidase antibodies, there is a greater risk o

25 els, thyroid-stimulating hormone levels, and thyroid peroxidase antibodies.

26 triiodothyronine (T3), free thyroxine (FT4), thyroid peroxidase antibody (Anti-TPO), and thyroid stim

27 ting hormone (TSH), free thyroxine (FT4) and thyroid peroxidase antibody (TPOAb) positivity.

28 FT4), thyroid-stimulating hormone (TSH), and thyroid peroxidase antibody (TPOAb) were obtained from m

29 combining the PRS with thyroid hormones and thyroid-peroxidase autoantibodies, and that the PRS was

30 ls of total IgE, elevated levels of IgG-anti-thyroid peroxidase, basopenia, eosinopenia, poor respons

31 2(h4) mice associated with thyroglobulin and thyroid-peroxidase, but not TSHR, Abs.

32 ation of iodide within the follicular lumen (thyroid peroxidase, DUOX2, DUOXA2), the substrate for th

33 with thyroiditis and specific for a cryptic thyroid-peroxidase epitope.

34 s), IA2 antigen (IA-2A), zinc transporter 8, thyroid peroxidase, gastric parietal cells (PCAs), tissu

35 markers such as total IgE level and IgG anti-thyroid peroxidase have been identified and, together wi

36 uggested that only approximately 2% of human thyroid peroxidase (hTPO(933)) reaches the surface of st

37 5 nM in vitro) and selectivity (>450-fold vs thyroid peroxidase in vitro), the mechanism of irreversi

38 th high selectivity for myeloperoxidase over thyroid peroxidase, limited penetration of the blood-bra

39 for TSHR Abs, with Abs to thyroglobulin and thyroid peroxidase remaining unchanged.

40 h FcepsilonRIalpha, but not thyroglobulin or thyroid peroxidase, resulted in the decreased ability to

41 likely resulted from decreased expression of thyroid peroxidase, sodium iodine symporter, and thyrogl

42 ial genetic architecture with positivity for thyroid-peroxidase-specific antibody, driven generally b

43 g expression of the sodium iodide symporter, thyroid peroxidase, TG, and thyrotropin receptor genes.

44 of mRNA transcripts for thyroglobulin (TG), thyroid peroxidase (TPO) and RET/PTC1 in the peripheral

45 serum thyroid-stimulating hormone (TSH) and thyroid peroxidase (TPO) antibody levels using regressio

46 The primary exposure was thyroid peroxidase (TPO) antibody status 7-10 years befo

47 Levels of IgG against thyroid peroxidase (TPO) are more often elevated in CSU

48 Autoantibodies to thyroid peroxidase (TPO) are the hallmark of the humoral

49 Thyroid peroxidase (TPO) autoantibody epitopes are large

50 lularly, and this might be representative of thyroid peroxidase (TPO) behavior in thyrocytes.

51 at a human TCR specific for the self-antigen thyroid peroxidase (TPO) is positively selected in the t

52 For thyroid hormone synthesis, thyroid peroxidase (TPO) molecules must be transported f

53 oantibody epitopes in amino acids 513-633 of thyroid peroxidase (TPO), a region frequently recognized

54 ession of the sodium iodide symporter (NIS), thyroid peroxidase (TPO), and TG genes.

55 tients and specific for the main autoantigen thyroid peroxidase (TPO).

56 ned, and these cells were found to reexpress thyroid peroxidase (TPO).

57 thyrotropin [TSH], free thyroxine [fT4], and thyroid peroxidase [TPO] antibodies) were assessed in 5,

58 h-affinity IgG1, kappa human autoantibody to thyroid peroxidase, was determined crystallographically

59 ccurring autoantibodies to thyroglobulin and thyroid peroxidase were unaffected.