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1 nical diagnosis, magnitude of early and late thyroidal 131I uptake values, and the 4- to 24-hr 131I u
2 plications, such as aberrations of tracheal, thyroidal, and vascular structures, were determined with
3                 Interestingly, the subset of thyroidal CD3(+)CD4(+)Tbet(+)RORgammaT(-) effector Th1 c
4 IL-6 during EAGD induction in the setting of thyroidal CD40 overexpression and showed decreased level
5 g a murine model of GD, we demonstrated that thyroidal CD40 overexpression augmented the production o
6 al autoimmune GD (EAGD), whereas deletion of thyroidal CD40 suppressed disease.
7 the effective half-lives of 131I in both the thyroidal component and the extrathyroidal component dur
8  is unknown and the presence of an intrinsic thyroidal defect has not been conclusively demonstrated.
9 of symptoms before diagnosis and presence of thyroidal disease, non-coeliac food intolerance or gastr
10 ases (8 % vs 40 % and 25 %, P = 0.028), more thyroidal diseases (18 % vs 16 % and 13 %, P = 0.043) an
11 terized by elements of both hypothalamic and thyroidal dysfunction.
12 g export, affected individuals suffer from a thyroidal ER storage disease characterized by a distende
13 at iodine and IFN-gamma cooperate to promote thyroidal expression of ICAM-1 in this mouse model of th
14 ender, tumor size, histologic variant, extra-thyroidal extension (ETE), and central and lateral nodal
15  averaged 2.2 +/- 0.2 cm and 11.9% had extra-thyroidal extension.
16 isruptions in kallikrein-3 levels, steroidal-thyroidal hormone balance, and antioxidant defense trigg
17 d-stimulating hormone deficiency (9.2%), and thyroidal hypothyroidism (5.8%).
18 -) would thus directly inhibit the newborns' thyroidal I(-) uptake.
19                                          Non-thyroidal illness syndrome (NTIS), characterized by low
20 ltered thyroid function (TF), especially non-thyroidal illness syndrome (NTIS), is common in hospital
21 mble of changes is collectively known as non-thyroidal illness syndrome (NTIS).
22 roid function test abnormalities seen in non-thyroidal illness usually resolve spontaneously, treatme
23 l illness." In psychiatric patients with non-thyroidal illness, patterns of thyroid function test abn
24 non is now more commonly referred to as "non-thyroidal illness." In psychiatric patients with non-thy
25  during sepsis often occur in the absence of thyroidal illness; however, the mechanisms involved in t
26 he initial ICAM-1 kinetics was paralleled by thyroidal infiltration of CD45(+) hemopoietic cells, whi
27                       The partially impaired thyroidal iodide organification in Pendred's syndrome su
28 1I uptake ratio was evaluated as an index of thyroidal iodide retention in hyperthyroid patients.
29 ated a recombinant MV-Edm encoding the human thyroidal iodide symporter (NIS).
30      Adding an allowance for accumulation of thyroidal iodine stores would produce an EAR of 72 mug a
31                                        Rapid thyroidal iodine turnover may contribute to 131I therapy
32  located in the lower portion of both of the thyroidal lobes in 556 children, the echographic shape w
33  ophthalmopathy(GO) is the most common extra-thyroidal manifestation of GD.
34                        We show that, whereas thyroidal Na(+)/I(-) symporter expression is thyroid-sti
35 d-eye disease (TED) is the most common extra-thyroidal presentation of graves' disease.
36                                              Thyroidal production of triiodothyronine (T3) is absent
37 due to incomplete thyrotropin suppression, a thyroidal radioiodide uptake of < 1%, and a low saliva t
38 , resulting in increased serum thyroxine and thyroidal radioiodide uptake.
39 itive transthyroidal gradient indicating the thyroidal release of DIT in each patient.
40 sue did not significantly change with age or thyroidal state.
41 r suppressor activity of THRB, the effect of thyroidal status on longevity, and the occurrence of tis
42 te significantly to the relative increase in thyroidal T3 production in patients with Graves' disease
43 TG) at residues Tyr(5) and Tyr(130), whereas thyroidal T3 production may originate in several differe
44                                              Thyroidal T4 production results from iodination of thyro
45                    In addition, depletion of thyroidal TH content was slower during iodine deficiency
46 ma is rare and results from abnormal ectopic thyroidal tissue iodine metabolism.
47 nduction of gross chromosomal aberrations in thyroidal TPC-1 cells following overexpression of PBF an
48    Of the 71 masses, 41 were thymic, 16 were thyroidal, two were esophageal cancers, six were trachea
49 so in many other normal tissues; conversely, thyroidal uptake of [131I]FIBG was 2-3-fold lower than t
50                         The 4- to 24-hr 131I thyroidal uptake ratio is a practical substitute for exa