1 and/or serum protein-bound iodine level, but thyrotoxic adverse effects were not uncommon.

2 erases to TBMEHP, which c) elicited maternal thyrotoxic and hepatotoxic effects and d) induced MNGs i

3 ression of the chemokine receptor CXCR6, and thyrotoxic capacity.

4 l helper (T(PH)) cells, as a driver of these thyrotoxic effector CD8(+) T cells.

5          Another recent study indicated that thyrotoxic hypokalaemic periodic paralysis is determined

6                                              Thyrotoxic hypokalemic periodic paralysis (TPP) is chara

7  in which mutations confer susceptibility to thyrotoxic hypokalemic periodic paralysis.

8 n these animals were rendered hypothyroid or thyrotoxic, mRNA expression of MHC isoforms remained unc

9 n non-familial hypoKPP, consisting mainly of thyrotoxic periodic paralysis (TPP) and sporadic periodi

10 thyrotoxicosis include toxic nodules and the thyrotoxic phase of thyroiditis.

11 xine and triiodothyronine causing an initial thyrotoxic phase, often followed by transient or permane