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1 ) B cells produce pathogenic Abs against the thyrotropin receptor.
2 he intracellular domain of a model GPCR, the thyrotropin receptor.
3 nction and gain-of-function mutations of the thyrotropin receptor.
4 lays in the induction of autoimmunity to the thyrotropin receptor.
5 distinguish lutropin (LHR), follitropin, and thyrotropin receptors.
6                              Activating anti-thyrotropin receptor Abs are responsible for hyperthyroi
7                                          The thyrotropin receptor, also known as the thyroid-stimulat
8 of patients developed antibodies against the thyrotropin receptor and carbimazole-responsive autoimmu
9  activation of shared autoantigens including thyrotropin receptor and insulin-like growth factor-1 re
10 ies of glycoprotein hormone receptors (e.g., thyrotropin receptor) and biogenic amine receptors (e.g.
11 ss a point mutation in the gene encoding the thyrotropin receptor, and affected animals are congenita
12                                              Thyrotropin receptor antibodies (TRAb) concentration was
13 olume (1.4 vs. 0.4 cm(3), P = <0.0001), mean thyrotropin receptor antibody level titer (19.02 vs. 13.
14 on thyroid scintigraphy, remnant volume, and thyrotropin receptor antibody levels at 3 mo after ATR.
15 al presentation, thyroid function tests, and thyrotropin-receptor antibody status.
16                                              Thyrotropin receptor antigen on fibroblasts diffusely in
17 sembled models of lutropin, follitropin, and thyrotropin receptors by aligning models of their LRD, T
18 xplains how substitutions in follitropin and thyrotropin receptors distant from their apparent ligand
19 e with the extracellular domain of the human thyrotropin receptor (ETSHR).
20 responsiveness associated with inhibition of thyrotropin receptor gene expression.
21 odide symporter, thyroid peroxidase, TG, and thyrotropin receptor genes.
22 led receptors homologous to gonadotropin and thyrotropin receptors have recently been identified and
23 elix scaffold, which endowed the substituted thyrotropin receptor intracellular domain elements with
24                 GNAS mutations that impaired thyrotropin receptor signaling were associated with deve
25 ot impair key activation steps distal to the thyrotropin receptor, such as forskolin-induced adenylyl
26 xpressing either mouse (mM12 cells) or human thyrotropin receptor (TSHR) (hM12 cells).
27                                              Thyrotropin receptor (TSHR) Ab's of the stimulating vari
28        The large TSH-bound ectodomain of the thyrotropin receptor (TSHR) activates the transmembrane
29 es' disease (GD), autoantibodies bind to the thyrotropin receptor (TSHR) and cause hyperthyroidism.
30 vity of AAbeta1AR and AAM2R with stimulating thyrotropin receptor (TSHR) antibodies was evaluated bef
31                      We studied cell surface thyrotropin receptor (TSHR) by biotinylating proteins on
32 the glycoprotein hormone receptors, only the thyrotropin receptor (TSHR) cleaves (at two sites) into
33  between the cysteine-rich N terminus of the thyrotropin receptor (TSHR) ectodomain and epidermal gro
34  to generate autoantibody-reactive, secreted thyrotropin receptor (TSHR) ectodomain in mammalian cell
35                                          The thyrotropin receptor (TSHR) exists in two forms (single
36 s, the detection of circulating DTC cells by thyrotropin receptor (TSHR) mRNA measurement distinguish
37  CRE-binding protein, a Y-box protein termed thyrotropin receptor (TSHR) suppressor element protein-1
38 g mutations are, however, more common in the thyrotropin receptor (TSHR) than in its downstream trans
39 lled by thyrotropin (TSH), which acts at the thyrotropin receptor (TSHR)(2).
40 g amino acid residues 22 to 416 of the human thyrotropin receptor (TSHR), along with the baculovirus-
41  through activation of its G-protein-coupled thyrotropin receptor (TSHR), controls the synthesis of t
42                       Abs that stimulate the thyrotropin receptor (TSHR), the cause of Graves' hypert
43                                          The thyrotropin receptor (TSHR), the major autoantigen in Gr
44 y caused by autoantibodies that activate the thyrotropin receptor (TSHR).
45 ng autoantibodies (TSAb's) that activate the thyrotropin receptor (TSHR).
46 to autoantibodies that bind and activate the thyrotropin receptor (TSHR).
47 yroid-stimulating Abs (TSAbs) activating the thyrotropin receptor (TSHR).
48 ast in part, by autoantibodies targeting the thyrotropin receptor (TSHR)/insulin-like growth factor I