ライフサイエンスコーパス: tick bite

1 eri B31 via the natural transmission mode of tick bite.

2 susceptible to B. burgdorferi infection via tick bite.

3 ated with the AGS sensitization event of the tick bite.

4 d States, nearly all AGS patients reported a tick bite.

5 ansmitted within just 15 minutes following a tick bite.

6 uitment of leukocytes to the location of the tick bite.

7 oinfections that most commonly occur after a tick bite.

8 haped and unable to infect mice by needle or tick bite.

9 C3H/HeN mice via either needle injection or tick bite.

10 nd imply host exposure to alpha-Gal during a tick bite.

11 ere non-infectious in mice when delivered by tick bite.

12 nvasion and erythema migrans formation after tick bite.

13 g with the host inflammatory response to the tick bite.

14 Many patients do not recall a tick bite.

15 ially by syringe inoculation or naturally by tick bite.

16 h N40 by syringe than in those inoculated by tick bite.

17 tral North Carolina with a recent history of tick bite.

18 an infectious dose of B. burgdorferi B31 by tick bite.

19 ht additional evidence for the connection to tick bites.

20 B) are acquired after Ixodes ricinus-complex tick bites.

21 as alpha-gal syndrome also requires avoiding tick bites.

22 sually transmitted to humans via mosquito or tick bites.

23 ht additional evidence for the connection to tick bites.

24 ia burgdorferi, which is transmitted through tick bites.

25 ave shown to develop immunity after repeated tick bites.

26 at can allow IgE to decrease is avoidance of tick bites.

27 interfere with alpha-gal allergy elicited by tick bites.

28 E) virus (TBEV) is transmitted to humans via tick bites.

29 associated with total IgE levels and recent tick bites.

30 he Flaviviridae family and is transmitted by tick bites.

31 e virus is transmitted to humans by infected tick bites.

32 rom homologous substances transferred during tick bites.

33 ed associations between IgE to alpha-Gal and tick bites.

34 k bites has been shown effective in reducing tick bites.

35 Most cases (69%) were attributed to tick bites.

36 led to establish a new infection in mice via tick bites.

37 d in ng/ micro l, was used as a biomarker of tick bites.

38 in wooded environments that are conducive to tick bites.

39 o of the four infected persons were aware of tick bites.

40 se tick bites, as measured by a biomarker of tick bites.

41 and their persistence in the blood prior to tick bites.

42 gdorferi, is introduced into human hosts via tick bites.

43 ick typhus even in the absence of recognised tick-bites.

44 ified 142 patients (61.2%) with diagnoses of tick bites, 40 patients (17.2%) with LD, and 50 patients

45 32% of direct charges were for patients with tick bites, 48% were for patients with LD, and 20% were

46 He reported a tick-bite 6 weeks before without erythema migrans follow

47 Of the 142 patients seen for tick bites, 95 (67%) underwent serologic testing for LD.

48 with Ixodes scapularis acquire resistance to tick bites, a phenomenon, known as tick immunity, that i

49 sympatric isolates of A. phagocytophilum via tick bite and challenged 16 weeks later by Ixodes scapul

50 ice were infected with E. phagocytophila via tick bite and challenged either 12 or 16 weeks later by

51 e assessed human skin and blood samples upon tick bite and developed a human skin explant model mimic

52 eins (19ISP) to enhance the recognition of a tick bite and diminish I. scapularis engorgement on a ho

53 transmitted within as little as 15 min of a tick bite and enter the central nervous system (CNS) to

54 catae (a new world RF spirochete species) by tick bite and monitored the immune responses generated i

55 The connection between tick bites and a-Gal sensitization was further supported

56 The connection between tick bites and alpha-Gal sensitization was further suppo

57 ruses (TBFVs) infect mammalian hosts through tick bites and can cause various serious illnesses, such

58 ly acquired in adulthood as a consequence of tick bites and has a regional distribution that largely

59 ly acquired in adulthood as a consequence of tick bites and has a regional distribution that largely

60 s and mediators are mobilized to the site of tick bites and play a role in resistance against ticks a

61 The pathogenic relationship between tick bites and sensitization to red meat, alpha-Gal, and

62 esting this period may carry higher risk for tick bites and subsequent contraction of tick-borne dise

63 icomponent vaccine that will protect against tick bites and the pathogens they transmit.

64 estimate the frequency of Ixodes scapularis tick bites and the resulting incidence of Lyme disease i

65 to increase or decrease risk of black-legged tick bites and tick-borne disease.

66 History of tick bites and tick-related febrile illness were assesse

67 essment of symptomatic cases associated with tick bites and vector surveillance efforts.

68 ported White race, 86% reported a history of tick bite, and 75% met the criteria for anaphylaxis base

69 collected from all animals for 14 days after tick bite, and spirochete densities were assessed by qua

70 th strain N40 by syringe, with strain N40 by tick bite, and with strain Pbi by syringe).

71 the 301 patients, 275 (91%) were exposed to tick bites, and 165 (54%) were bitten by a tick.

72 thorough exposure history, particularly for tick bites, and awareness of clinical features may promp

73 d LD, presumptive cases of LD, patients with tick bites, and diagnostic tests ordered for LD.

74 esumptive LD, seen and given prophylaxis for tick bites, and having diagnostic tests indicate that re

75 This type of allergy is induced by tick bites, and therapeutic options for this skin-driven

76 es and those of others strongly suggest that tick bites are a cause, if not the only significant caus

77 This provides further evidence that tick bites are associated with IgE responses to alpha-Ga

78 In the United States, lone star tick bites are associated with the development of AGS.

79 Tick bites are considered an important route of sensitiz

80 drate contained in red meat) allergy, pigeon tick bite (Argax reflexus), wheat-dependent exercise-ind

81 ck bite prevention and consequently decrease tick bites, as measured by a biomarker of tick bites.

82 Tick bites, associated with the secretion of tick saliva

83 istatin is a RAGE antagonist that suppresses tick bite-associated inflammation, allowing successful b

84 otic therapy for prophylaxis of LD following tick bites, but the extent to which physicians in endemi

85 hermore, we explored the natural response to tick bites by profiling antigens recognized by IgG isola

86 given within 72 hours after an I. scapularis tick bite can prevent the development of Lyme disease.

87 nterleukin (IL)-12 and IL-1beta, following a tick bite, compared with wild-type animals.

88 Tick bites constitute the primary sensitization source,

89 wherein non-natural host species exposed to tick bites develop an immune response that thwarts subse

90 No patients with tick bite developed clinical LD.

91 of the human population exposed to lone star tick bites develops AGS.

92 product was incapable of infecting mice via tick bite even though the mutant was (i) infectious in m

93 nt failed to establish infection in mice via tick bite, exogenous supplementation of GlcNAc into unfe

94 tive to doxycycline for treatment of African tick bite fever in some selective situations.

95 African tick bite fever is the most commonly encountered travel-

96 tization event has been linked to an initial tick bite, followed by consumption of red meat containin

97 as well as from others with Lyme disease and tick-bitten guinea pigs and mice, to identify 199 recogn

98 n-impregnated clothing for the prevention of tick bites has been shown effective in reducing tick bit

99 E to alpha-Gal and also the association with tick bites have been increasing worldwide.

100 Tick bites have been shown to transmit a novel form of s

101 ment of sensitization has been attributed to tick bites; however, the possible role of other parasite

102 Body regions where tick bites human vary depending on the tick species and

103 rythema migrans developed at the site of the tick bite in a significantly smaller proportion of the s

104 s with thrombocytopenia and leukopenia after tick bite in Hokkaido, Japan, in 2019 and 2020, respecti

105 R, 3.55; 95% CI, 1.38-9.18), and an attached tick bite in the past 5 years (OR, 3.99; 95% CI, 2.22-7.

106 pite only 84 individuals (17.2%) recalling a tick bite in the past 5 years, 178 (36.5%) had evidence

107 lood samples from patients with a history of tick bite in the preceding 2 months at Mudanjiang Forest

108 index patient acquired the disease through a tick bite in the province of Avila - 300 km away from th

109 Of note, the duration of the tick bites in both cases was very short.

110 informed decisions concerning management of tick bites in HGE-endemic areas.

111 ammalian meat allergy and has been linked to tick bites in North America, Australia, and Eurasia.

112 y regional, reflecting the important role of tick bites in sensitization, and is more common in demog

113 We review the role of tick bites in the development of alpha-gal-specific IgE

114 The immune response to tick bites includes T cells specific for tick protein, w

115 o food and medication in patients at risk of tick bites including travellers.

116 owever, despite the increasing prevalence of tick bite-induced allergies, the mechanisms by which cut

117 nse in Lyme disease patients, and in mice by tick bite inoculation, provides evidence that the Rev pr

118 Alpha-gal syndrome, which is associated with tick bites, is a rising cause of IgE-mediated food anaph

119 ination of B. burgdorferi from the site of a tick bite likely occurs, in part, via attachment to host

120 The unusual link with tick bites may be explained by the fact that allergic ce

121 ch are likely to be present at the site of a tick bite, may play a role in preparing the organism for

122 lmost exclusively in the limb closest to the tick bite (median incubation period, 66 days).

123 were made to infect immunodeficient mice by tick bite or inoculation of tick contents.

124 were completely protected against a natural tick bite or subcutaneous (s.c.) inoculation of 297.

125 . burgdorferi sensu strictu by syringe or by tick bite or with strain Pbi of B. burgdorferi genospeci

126 effectiveness of these pesticides to prevent tick bites or human tick-borne diseases is unknown.

127 ted dogs, and four had a reported history of tick bite preceding the illness.

128 5-year-old female with a history of multiple tick bites presented with fever and pancytopenia.

129 attitudes, and reported behaviors related to tick bite prevention and consequently decrease tick bite

130 ients should be counseled about mosquito and tick bite prevention when receiving rituximab and other

131 Mice infected with B31 by tick bite produced antibodies that recognized each of th

132 Lyme disease begins at the site of a tick bite, producing a primary infection with spread of

133 nty-eight patients (55%) with a diagnosis of tick bite received antibiotic therapy.

134 ing, most patients consulting physicians for tick bites received prophylactic antibiotic therapy of u

135 Most interestingly, multiple tick bites reduced the likelihood of contracting Lyme bo

136 develop an influenzalike illness following a tick bite should include HGE.

137 f severe nocturnal anaphylaxis due to pigeon tick bite showing the diagnostic value of the extract an

138 on of topical antibiotics to the skin at the tick bite site could eradicate Borrelia burgdorferi infe

139 is article, we describe NET formation at the tick bite site in vivo in mice.

140 ctive in eliminating B. burgdorferi from the tick bite site or in preventing dissemination to other t

141 eliosis, disseminate hematogenously from the tick bite site to the joints, the heart, and the central

142 igrans (EM) skin lesion that develops at the tick bite site typically between 7 and 14 days after inf

143 cond that causes only local infection at the tick bite site, and a third that causes systemic disease

144 DIA3 modulates inflammatory responses at the tick bite site, potentially facilitating spirochete surv

145 ors influence B. burgdorferi survival at the tick bite site.

146 manifestation called erythema migrans at the tick bite site.

147 Tick saliva deposited into the tick-bite site is composed of an array of immunomodulato

148 s within neutrophil granulocytes that invade tick bite sites are likely ingested by the vector, to be

149 h a transient expression in the host only at tick-bite sites.

150 ssues of the somatic quadrant closest to the tick bite than in tissues further from the site of infec

151 es could be used to study the factors within tick bites that cause allergies and to investigate how s

152 With repeated tick bites, the T cells develop a strong Th2 signal with

153 Rash, history of tick bite, thrombocytopenia, and hyponatremia were often

154 sease, spirochetes spread from the site of a tick bite to colonize multiple tissue sites, causing mul

155 After tick-bite transmission back to mice, the process was rev

156 Importantly, tick bite upregulated RAGE ligands in skin, and endogeno

157 m is often abundant and aggressive, and many tick bite victims receive multiple bites.

158 Nymphs remain attached to adult tick-bite victims longer than they remain attached to ch

159 albeta1-(3)4GlcNAc-R (alpha-gal) following a tick bite was associated with allergies to red meat, cet

160 , disease caused by Borrelia turicatae after tick bite was compared in 2 rhesus macaques in which rad

161 B. miyamotoi by subcutaneous inoculation or tick bite were collected for immunoblotting against B. m

162 nted with a definite Ixodes scapularis (Say) tick bite were measured to determine the utility of the

163 clinical symptoms, antibiotic treatments, or tick bites were not more frequent in E. equi- or B. burg

164 Persons with tick bites were prospectively studied to determine if th

165 cut with encephalitis, who had a recent deer tick bite, were evaluated by the relevant serologic test

166 n, but may also be relevant to itching after tick bites which can last for weeks.

167 They appear to be stimulated only by tick bites which induce production of alpha-gal specific

168 crobial treatment after an Ixodes scapularis tick bite will prevent Lyme disease.

169 observed with serum from a mouse infected by tick bite, with the recognition of two additional bands

170 CCHFV) is a priority pathogen transmitted by tick bites, with no vaccines or specific therapeutics ap

171 us (TBEV), which is usually transmitted by a tick-bite, with increasing incidence in northeastern Eur

172 olina presenting with fever and a history of tick bite within the preceding 14 days.

173 n order to determine whether transmission by tick bite would alter the dissemination dynamics and dis

174 immunodominant antigens in humans exposed to tick bites, yet vaccination with the three selected TSGP